Wi rili ɔndastand di A1C tɛst fɔ dayabitis? (HbA1c) (HbA1c) ɛn .

Yu de tek mɛrɛsin fɔ dayabitis? Ɔ ɛnibɔdi na yu famili gɛt dayabitis? Dɔn yu go dɔn yɛri bɔt di tɛst we dɛn kɔl ‘A1C’. Dɔktɔ dɛn kin aks yu fɔ du dis tɛst ɔltɛm, nɔto so? Sɔntɛnde, we yu si di valyu dɛn we de na dis ripɔt, yu kin fred smɔl. So, tide wi go tɔk rili simpul ɛn padi biznɛs bɔt wetin dis A1C bi, wetin i rili de mɛzhɔ, ɛn aw wi fɔ ɔndastand in rizɔlt.

Fɔ tɔk am simpul wan, wetin na A1C tɛst?

Di A1C tɛst na simpul blɔd tɛst we de mɛzhɔ yu avɛrej blɔd glukɔs lɛvɛl fɔ di pas tri mɔnt. Dɛn gi di rizɔlt dɛn as pasɛnt. Di mɔ di pasɛnt we yu gɛt, na di mɔ yu blɔd shuga go bɔku.

Tink bɔt am dis we. We yu chɛk yu blɔd shuga wit glukomita na os, yu kin jɔs gɛt wan snɛpsho fɔ yu blɔd shuga lɛvɛl da tɛm de. Na laik di nomba of runs we dem skor fo wan single bol fo kriket mats. Bɔt di A1C tɛst nɔ tan lɛk dat. I tan lɛk di batting avrej we batsman gɛt ova di kɔs fɔ wan ɔl tɛnament. Dat min se wi kin si di big pikchɔ, nɔto jɔs fɔ wan de, bɔt fɔ tri mɔnt .

Dɛn kin kɔl dis tɛst wit sɔm ɔda nem dɛn. Yu ripɔt kin rayt am bak lɛk:

• Ɛmoglobin we dɛn kɔl A1C
• HbA1c
• Ɛmoglobin we gɛt glayket
• Glycohemoglobin tɛst we dɛn kin du

Wetin mek wi de du dis A1C tɛst?

Dɔktɔ dɛn kin yuz dis tɛst fɔ sɔm men rizin dɛn.

• Fɔ no bifo yu gɛt dayabitis: Dis kin ɛp yu fɔ no bifo tɛm if yu gɛt ay risk fɔ gɛt dayabitis.
• Diagnose Type 2 Diabetes: Dis na impɔtant tɛst fɔ no if pɔrsin wae gɛt dis sik gɛt dayabitis.
• Si aw dɛn de kɔntrol yu shuga: If yu dɔn ɔlrɛdi gɛt tritmɛnt fɔ dayabitis (Tayp 1 ɔ Tayp 2), dis kin ɛp yu fɔ no aw yu tritmɛnt plan (mɛrɛsin, it, ɛksɛsayz) de wok fayn fayn wan. Bay yu ɛn yu dɔktɔ kin disayd if yu nid fɔ mek ɛni chenj na yu tritmɛnt plan.

Aw dɛn kin kɔl A1C? Lɛ wi luk am smɔl wit sayɛns.

Fɔ ɔndastand dis, wi fɔs nid fɔ no smɔl bɔt di rɛd blɔd sɛl dɛn we de na wi blɔd.

Insay wi rɛd blɔd sɛl dɛn, wan prɔtin de we dɛn kɔl ɛmoglobin . Tink bɔt am lɛk bɔs we de kɛr ɔksijɛn ɔlsay na wi bɔdi. Naw, glukɔs (shuga) we de kɔmɔt na di it we wi de it kin ad to wi blɔd. Dis glukɔs we de na di blɔd kin atak di ɛmoglobin bɔs. Wi kin kɔl dis prɔses glycation.Dat.

As yu blɔd shuga de go ɔp, di shuga we de tay pan ɛmoglobin de go ɔp bak. di imכtant tin na dat, dis shuga we dεn bכnd de de fכ di layf we di rεd bכdi sεl de liv (we na 90 dez, כ lεk 3 mכnt).

So wetin di A1C tεst de du na fכ mכsu us pasεnshכn pan di tכtal εmoglobin na da shuga-bכnd hεmoglobin protin de. Na dat mek wi kin gɛt 3 mɔnt avrej frɔm dis.

Ɛnitin de we yu nid fɔ pripia bifo yu tek dis tɛst?

Nɔ, dis na di big tin we izi fɔ du fɔ dis tɛst. Yu nɔ nid fɔ fast fɔ awa bifo yu tek di A1C tɛst. Yu kin tek dis tɛst ɛni tɛm pan di de.

Tu we dɛn fɔ du di tɛst

Tu men we de fɔ du di A1C tɛst. Yu dɔktɔ go disayd di we we go fayn fɔ yu bay wetin yu nid.

Wetin yu A1C ripɔt min?

Okay, naw wi kam to di pat we impɔtant pas ɔl. Wetin di pasɛnt we de na yu ripɔt min?

We dɛn de no if pɔsin gɛt sik, dɛn kin put di A1C valyu dɛn lɛk dis:

Wetin na di gol fɔ pɔsin we gɛt dayabitis?

If yu dɔn ɔlrɛdi gɛt dayabitis, yu A1C target go dipen pan yu ej, wɛlbɔdi, ɛn ɔda tin dɛn. Yu nɔ go ebul fɔ sɛt da gol de fɔ yusɛf. Yu fɔ tɔk to yu dɔktɔ fɔ no di rayt target fɔ yu.

Bɔt in jɔnal, di advays we dɛn kin gi bɔku pan di big pipul dɛn we gɛt dayabitis na fɔ kip dɛn A1C dɔŋ 7% . If yu kɔntinyu fɔ gɛt dis lɛvɛl, i kin mek yu nɔ gɛt bɔku pan di prɔblɛm dɛn we kin apin bikɔs ɔf dayabitis.

Wetin na di risk dɛm fɔ gɛt ɛlevɛt A1C lɛvɛl?

if yu gεt hכy A1C lεvεl fכ lכng tεm i kin pwεl difrεn כgan dεm na di bכdi. Dis tan lɛk we yu de rɔsti wan pat pan ayɛn insay sɔlvushɔn we gɛt bɔku shuga. Di men tin dɛn we kin apin kin bi:

• Ay (Retinopathy): I kin apin we pɔsin nɔ de si ɔ i kin blaynd.
• Kidni (Nephropathy): I kin mek di kidni nɔ wok fayn.
• Nyuropathy: Kכndishכn dεm lεk fכ sכmtεm sכmtεm, pen, εn lכs fכ fil na di limb dεm kin apin.
• At ɛn blɔd vesel dɛm: Di risk fɔ gɛt at atak ɛn strok kin bɔku.
• Gastroparesis: di bεlε kin wok fayn.

Ɔda tin dɛn de we kin afɛkt di rizɔlt fɔ di A1C tɛst?

Yɛs. Sɔntɛnde yu A1C rizɔlt kin lay ɔ i kin pas yu rial blɔd shuga lɛvɛl. Bɔku rizin dɛn de we mek dis kin bi.

apat frכm dat, di jεnεtik kכndishכn dεm we dεn k כl εmoglobin vεry כnt kin mek bak di A1C risכlt dεm difrεn. If yu gɛt wan sik lɛk anemia ɔ yu nɔ shɔ bɔt yu A1C rizɔlt, mek shɔ se yu tɛl yu dɔktɔ. I go tek dɛn tin dɛn de na kɔna fɔ mek yu ebul fɔ chɛk yu rizɔlt kɔrɛkt wan.

Di A1C valyu nɔmɔ na infεkt fɔ mεzhɔ kכntrכl pan dayabεtis?

A1C na wan rili impɔtant ɛn yusful mɛzhɔmɛnt we wi dɔn de yuz fɔ bɔku bɔku ia. Bɔt tide, we di tin dɛn we dɛn de yuz fɔ mek tɛknɔlɔji dɔn go bifo, sɔm we dɛn de we de mek wi ebul fɔ si mɔ tin dɛn.

Imajin dis: Sɔmbɔdi in A1C na 7%. Tu we dɛn de we kin apin.

1. Di pɔsin in blɔd shuga lɛvɛl kin stebul ɔltɛm arawnd 154 mg/dL.

2. Ɔ, di pɔsin in blɔd shuga lɛvɛl kin chenj bad bad wan ɔl di de (e.g., i go go ɔp to 300 ɛn afta dat i go bak dɔŋ to 50). di avrej fכ dεn big fכlt dεm ya kin hכy lεk 7%.

Yu si, nɔto so? Ivin if di A1C valyu na di sem, di difrɛns pan dɛn blɔd shuga kɔntrol de big. Di sɛkɔn pɔsin in bɔdi kin pwɛl mɔ.

Dis na di say we di Continuous Glucose Monitoring (CGM) divays dεm εn wan mεzhɔmεnt we dεn kכ l Time in Range (TIR) ​​de kam in. CGM kin trak aw yu bכdi shuga lεvεl de chenj כl di de. TIR de mכsu aw mכch tεm insay di de yu bכdi shuga lεvεl de insay wan sכm rεnj (e.g., 70-180 mg/dL).

Pan ɔl we dɛn we ya na mɔ advans we dɛn pas A1C, di A1C tɛst na di wan we kɔmɔn ɛn izi fɔ du naw, ɛn di tɛst we impɔtant pas ɔl na Sri Lanka.