Yu blɔd shuga smɔl? (Hypoglycemia) Lɛ wi tɔk bɔt dis ditayli!

Tide wi go tɔk bɔt wan sik we kin afɛkt bɔku pipul dɛn, mɔ di wan dɛn we gɛt shuga, ɛn sɔntɛnde ivin di wan dɛn we nɔ gɛt shuga. Dat na di blɔd shuga we nɔ de bɔku, ɔ we wi de tɔk bɔt mɛrɛsin, di blɔd shuga we nɔ gɛt bɛtɛ blɔd. I impɔtant fɔ no bɔt dis bikɔs i kin rili siriɔs.

Wetin na haypoglycemia? Lɛ wi ɔndastand am simpul wan.

Fɔ tɔk am simpul wan, haypoglycemia kin apin we yu blɔd shuga, ɔ glukɔs, go dɔŋ pas di lɛvɛl we yu gɛt wɛlbɔdi. Wi kin kɔl dis bak ‘lɔ blɔd shuga.’ Dis na fayn tin wae kin apun pan pipul dɛm wae gɛt shuga, mɔr di wan dɛm wae gɛt tayp 1 dayabitis.

In jɛnɛral, fɔ pɔsin we gɛt dayabitis, dɛn kin tek am se di blɔd shuga lɛvɛl go dɔŋ pas 70 mg/dL (miligram pan wan desilita) ɔ 3.9 mmol/L (milimol pan wan lita). Fɔ pɔsin we nɔ gɛt dayabitis, haypoglycemia na we di lɛvɛl go dɔŋ pas 55 mg/dL (3.1 mmol/L). Dɛn kayn tɛm dɛn de, yu nid fɔ it ɔ drink sɔntin we gɛt shuga ɔ kabɔhaydrɛt kwik kwik wan. Bɔt if dis sik kam bad bad wan (siv hapoglycemia), i kin mek pɔsin in layf de pan denja. If na so i bi, yu nid fɔ gɛt tritmɛnt fɔ glukagon we yu nid ɔ fɔ ɛp yu kwik kwik wan.

Wetin mek ‘blɔd shuga’ impɔtant to wi bɔdi?

Wi bɔdi kin gɛt glukɔs ɔ shuga frɔm di it dɛn we wi de it, mɔ frɔm it dɛn we gɛt kabɔhaydrɛt. Dis glukɔs na di men tin we wi bɔdi de gi wi ɛnaji. I tan lɛk petrol fɔ motoka. Dis glukɔs de kɛr wi blɔd go na ɛvri sɛl na di bɔdi fɔ mek i gɛt ɛnaji. Dis glukɔs impɔtant mɔ fɔ wi bren. Fɔ mek di bren wok fayn, i nid fɔ gɛt glukɔs ɔltɛm.

If yu nɔ gɛt dayabitis, yu bɔdi kin ɛp fɔ kip yu blɔd shuga lɛvɛl insay wan wɛlbɔdi rɛnj tru bɔku prɔses. di כmon insulin – we wi pankrias de mek – na di big wan we de kכntribyut to dis. Glucagon na ɔda impɔtant ɔmon we de insay dis prɔses.

Bɔt pipul dɛn we gɛt dayabitis nid fɔ tek mɛrɛsin fɔ dayabitis we dɛn de it ɛn insulin we dɛn mek fɔ kɔntrol di shuga we dɛn gɛt na dɛn blɔd. Dɛn nid bak fɔ chenj di we aw dɛn de liv dɛn layf. Bɔku tɛm, di blɔd shuga we smɔl kin kam bikɔs ɔf tumɔs insulin na di bɔdi – ɔ bikɔs i kin mek am na di wɔl ɔ bikɔs yu de tek bɔku insulin. Di blɔd shuga we smɔl kin kam bak bikɔs ɔf ɔda prɔblɛm dɛn we gɛt fɔ du wit di ɔmon ɛn mɛtabolik.

If yu blɔd shuga lɛvɛl go dɔŋ pas di wɛlbɔdi lɛvɛl, yu kin bigin fɔ gɛt sɔm sayn dɛm lɛk fɔ shek shek ɛn at bit fast. If i drɔp tumɔs, i kin mek pɔsin in layf de pan denja. Bikɔs, lɛk aw a bin dɔn tɔk, di bren nid fɔ gɛt glukɔs ɔltɛm fɔ mek i ebul fɔ wok fayn fayn wan. If glukɔs nɔ de, di bren nɔ go ebul fɔ wok.

Aw kɔmɔn tin na di haypoglycemia?

Hypoglycemia na tin we kin apin to pipul dɛn we gɛt dayabitis, mɔ di wan dɛn we de yuz insulin. Wan stɔdi sho se 4 pan ɔl 5 pipul dɛn we gɛt tayp 1 dayabitis, ɛn lɛk af pan pipul dɛn we gɛt tayp 2 dayabitis we de tek insulin, bin ripɔt se dɛn gɛt smɔl shuga na dɛn blɔd wan tɛm insay 4 wik.

Pipul wae de tek meglitinides ɔ sulfonylureas fɔ tayp 2 dayabitis kin gɛt bɔrku risk bak fɔ gɛt low blɔd shuga. Yu kin gɛt haypoglycemia ivin if yu nɔ gɛt dayabitis, bɔt i nɔ kin apin so ɔltɛm.

Wetin na di sayn dɛm wae de sho se yu gɛt haypoglycemia (lɔ blɔd shuga)?

Di sayn dɛm fɔ hapo glycemia kin bigin kwik kwik wan. Dɛn kin difrɛn bak frɔm wan pɔsin to ɔda pɔsin. Ivin pan di sem pɔsin, di sayn dɛm kin difrɛn frɔm wan tɛm to ɔda wan.

Dɛn sayn ya kin trik smɔl. Bɔt dɛn kin gi yu gud wɔnin fɔ tek akshɔn bifo yu blɔd shuga go dɔŋ ivin. Na sɔm pan di sayn dɛm wae de sho se yu gɛt dis sik:

• Wan nature we de shek shek ɔ we de shek.
• Bɔdi we nɔ gɛt layf.
• Swet ɛn fil kol.
• Big big angri (polyphagia).
• Di at rit de go ɔp.
• Diziz ɔ filin fɔ spin.
• I nɔ kin izi fɔ mek yu pe atɛnshɔn ɔ kɔnfyus.
• Fɔ wɔri ɔltɛm ɔ fɔ vɛks ɔltɛm.
• Di kɔlɔ we di skin kin chenj (pallor).
• Fɔ fil lɛk se yu lip, yu tɔŋ, ɔ yu chɛst dɔn swɛ.

Di sayn dɛm wae de sho se yu gɛt siriɔs haypoglycemia na:

• Blɔr vishɔn ɔ dabl vishɔn.
• Tɔk we nɔ de tɔk fayn.
• Nɔ balans ɔ i nɔ kin izi fɔ du wok.
• Fɔ mek pɔsin nɔ ebul fɔ no wetin fɔ du.
• Di sik dɛn we kin mek pɔsin sik.
• Nɔbɔdi nɔ kin no natin.

Siriɔs haypoglycemia kin mek pɔsin in layf de pan denja! I nid fɔ go to dɔktɔ wantɛm wantɛm. Pan ɔl we i nɔ kin apin so ɔltɛm, if dɛn nɔ trit am, i kin mek pɔsin kɔma ɛn ivin day.

Sɔntɛnde, yu blɔd shuga kin go dɔŋ we yu de slip. Wi kin kɔl dis na nɛt haypoglycemia. Di sayn dɛm kin bi:

• Nɔ slip ɔ slip we nɔ de rɛst.
• Klos ɔ bed we de wet bikɔs yu de swet.
• Fɔ ala na yu slip.
• Bad drim dɛn.
• Fɔ fil taya pasmak, kɔnfyus, ɔ fɔ fil bad we yu wek.

Ustɛm di sayn dɛn we de sho se pɔsin gɛt aypoglycemia kin apia?

Di glukɔs lɛvɛl wae de sik kin bigin fɔ pipul dɛm wae gɛt shuga kin difrɛn frɔm pɔrsin to ɔda pɔrsin. Tipikli, pipul dɛm wae gɛt shuga kin bigin fɔ gɛt sɔm kayn sik wae dɛn blɔd shuga drɔp to 70 mg/dL ɔr smɔl pas dat. Bɔt if yu blɔd shuga go dɔŋ kwik kwik wan, yu kin gɛt sɔm sayn dɛn ivin bifo tɛm.

Sɔm pipul dɛn we gɛt dayabitis kin gɛt sayn dɛn we de sho se dɛn gɛt ay blɔd ivin we dɛn gɛt bɔku bɔku glukɔs. Dis na bikɔs we di blɔd shuga lɛvɛl go ɔp fɔ lɔng tɛm (chronic hyperglycemia), di bɔdi kin yus to wetin dɛn kin tek as ‘nɔmal’ lɛvɛl. di lεvεl we dεn kin si di sכmtεm dεm fכ lכw bכdi shuga de chenj.

Sɔm pipul dɛn kin gɛt smɔl shuga na dɛn blɔd bɔt dɛn nɔ kin gɛt sɔm kayn sik, ɔr dɛn nɔ kin notis am. Dɔktɔ dɛn kɔl dis haypoglycemia unawareness . Dɛn pipul ya kin gɛt siriɔs haypoglycemia ɛn dɛn kin nid fɔ go to dɔktɔ.

Nɔr no bɔt haypoglycemia kin apin pan pipul dɛm wae gɛt dayabitis wae dɔn gɛt low blɔd shuga lɛvɛl fɔ lɔng tɛm. Dɛn bɔdi kin stɔp fɔ sho di sayn dɛm we dɛn blɔd shuga lɛvɛl go dɔŋ.

If yu gɛt haypoglycemia unawareness, i impɔtant fɔ tɛl yu padi ɛn fambul dɛm bɔt am so dat dɛn go ɛp yu if yu blɔd shuga go dɔŋ tumɔs. Yu go si se dɛn tin ya go ɛp yu bak:

• Yuz wan Continuous Glucose Monitoring (CGM) divays we de wɔn yu we yu blɔd shuga nɔ bɔku.
• Chek yu blɔd shuga lɛvɛl wit yu an ɔltɛm.
• Dayabitis alert dog – Dɛn dɔn tren dɛn dɔg ya spɛshal wan fɔ alert yu we yu blɔd shuga smɔl.

Us prɔblɛm dɛn kin apin bikɔs di blɔd shuga nɔ bɔku?

Siriɔs haypoglycemia we de te fɔ lɔng tɛm kin mek pɔsin in layf de pan denja. I kin mek bak yu gɛt prɔblɛm dɛn lɛk:

• Bɔku ɔgan dɛn we nɔ de wok fayn.
• Di at we nɔ de wok fayn.
• Di at we de mek di at nɔ de wok.
• Di bren we de pwɛl ɔltɛm.
• Kɔma stet.
• Day.

Dɔn bak, fɔ yuz ebi ebi mashin dɛn, lɛk fɔ drayv, we yu blɔd shuga nɔ bɔku, dat kin rili denja. Yu go mɔs gɛt aksidɛnt. If yu gɛt dayabitis, chɛk yu blɔd shuga ɔltɛm bifo yu yuz ebi ebi mashin dɛn.

Wetin kin mek pipul dɛn we gɛt shuga gɛt blɔd shuga nɔ bɔku?

Hypoglycemia kin apin we yu blɔd shuga lɛvɛl go dɔŋ pas di wɛlbɔdi lɛvɛl. Dis kin apun fɔ bɔrku rizin fɔ pipul dɛm wae gɛt shuga. Hypoglycemia kin apin if yu it, ɛksesaiz, ɛn di mɛrɛsin dɛn we yu de tek fɔ gɛt dayabitis nɔ balans.

Dis na de tin wae kin apin wae pipul dɛm wae gɛt shuga kin gɛt haypoglycemia:

• Fɔ tek insulin tumɔs, fɔ tek di rɔng kayn insulin, ɔ fɔ injekt insulin insay di mɔsul instead fɔ tek di fat tisu.
• Nɔr fɔ di tɛm fɔ it insulin ɛn kabɔhaydrɛt fayn fayn wan (fɔ ɛgzampul, fɔ wet tumɔs fɔ it afta yu dɔn tek insulin fɔ it).
• Fɔ tek tumɔs ɔ pas di doz we dɛn tɛl yu fɔ tek di mɛrɛsin fɔ dayabitis.
• Fɔ bi mɔ aktif (ɛksɛsayz) pas aw i kin bi.
• Fɔ drink rɔm we yu nɔ it.
• Fɔ it let ɔ fɔ skip fɔ it.
• Di it dɛn nɔ gɛt bɛtɛ fat, prɔtin, ɛn fayv.

If yu gɛt bɛlɛ ɛn yu gɛt tayp 1 dayabitis, yu go gɛt smɔl blɔd shuga insay di fɔs tri mɔnt bikɔs ɔf di chenj dɛn we di ɔmon dɛn de chenj.

Wetin kin mek pipul dɛn we nɔ gɛt dayabitis gɛt ay blɔd blɔd?

Tu men kayn haypoglycemia de pan pipul dɛm wae nɔr gɛt dayabitis: riaktiv haypoglycemia ɛn fasting hypoglycemia.

Riaktiv Haypoglycemia

Riaktiv haypoglycemia na we di blɔd shuga lɛvɛl smɔl afta yu dɔn it. Bɔku tɛm, i kin apin insay tu to 4 awa afta yu dɔn it.

Risach pipul dɛn nɔ no gud gud wan wetin mek dis kin apin, bɔt dɛn tink se na bikɔs we pɔsin it sɔm it dɛn, mɔ di simpul tin dɛn we gɛt kabɔhaydrɛt lɛk wayt rays, pɔt, bred, kek, ɛn swit, dat kin mek di blɔd shuga go ɔp wantɛm wantɛm, dɔn i kin go dɔŋ wantɛm wantɛm.

Dɔn bak, sɔm ɔpreshɔn dɛn we dɛn kin du fɔ lɛ pɔsin nɔ gɛt bɔku bɔku bɔdi (bariatric ɔpreshɔn) kin mek pɔsin gɛt riaktiv haypoglycemia. Afta ɔpreshɔn lɛk gastric bypass ɔpreshɔn, di bɔdi kin tek di shuga kwik kwik wan. Dis kin mek dɛn mek mɔ insulin. Dis kin mek yu gɛt ay blɔd blɔd.

Bɔku tɛm, di bɔdi kin kɔrɛkt dis sik fɔ insɛf, bɔt if yu it sɔm tin dɛn we gɛt kabɔhaydrɛt, dat kin mek yu nɔ gɛt di sik kwik kwik wan.

Fasting Hypoglycemia we yu de fast

Bɔrku pipul dɛm wae nɔr gɛt shuga nɔr kin gɛt haypoglycemia ivin afta dɛn dɔn fast fɔ lɔng tɛm. Dis na bikɔs di ɔmon dɛn na di bɔdi ɛn di glukɔs we dɛn dɔn kip de kɔntrol di shuga we de na di blɔd.

Bɔt sɔm sik ɛn kɔndishɔn kin mek pipul dɛn we nɔ gɛt dayabitis gɛt fasting hypoglycemia. Dɛn tin ya na:

• Fɔ drink rɔm pasmak:alkol de mek di bכdi nכ ebul fכ mek nyu glukכs sεl dεm (gluconeogenesis). If yu nɔ it fayn fɔ sɔm dez ɛn drink bɔku, yu bɔdi kin dɔn di glukɔs we i dɔn kip (glycogen). Dis kin mek yu gɛt haypoglycemia, we na we yu bɔdi nɔ ebul fɔ rigul di shuga na yu blɔd fayn fayn wan.
• Bɔrku sik: Hypoglycemia kin apin pan siriɔs sik, lɛk ɛnd stej liva sik, sepsis, fast, ɔr kidni fail. dis na biכs yu bכdi de yuz di glukכs we de naw fכ enεji fast pas aw i kin mek nyu glukכs frכm di it we yu de it.
• di adrenal insufficiency: Insay dis kכndishכn, di kכtisol lεvεl dεm de lכs pas nכmal. Bikɔs kɔtisol de ɛp fɔ kɔntrol di blɔd shuga bay we i de mek i bɔku, if di kɔtisol nɔ bɔku, dat kin mek i gɛt ay blɔd shuga.
• Nɔn-islet sɛl tumor haypoglycemia (NICTH): Dis na wan sik we nɔ kin apin so ɔltɛm bɔt we kin rili siriɔs. insay dis kכndyushכn, wan tכmכro de rilis tu mכch כmon we dεn kכl Insulin-like Growth Factor 2 (IGF-2). Dis ɔmon de wok lɛk insulin. we IGF-2 εlevεt, haypoglycemia de apin. NICTH kin kam bikɔs ɔf difrɛn kayn benign (nɔ gɛt kansa) ɛn malignant (kansa) tumor.
• Insulinoma: Dis na kansa we nɔ kin apin so ɔltɛm we kin kam na di pankrias. I de mek tumɔs insulin, we de mek yu gɛt haypoglycemia – bɔku tɛm na ali mɔnin.

Pan ɔl we i nɔ kin apin so ɔltɛm, i kin kam bak wit di mɛrɛsin dɛn we nɔ gɛt natin fɔ du wit dayabitis, lɛk beta-blɔka ɛn sɔm antibayɔtiks.

If yu gɛt haypoglycemia we yu nɔ gɛt dayabitis, i impɔtant fɔ go to dɔktɔ.

Aw pipul dɛm wae gɛt shuga kin no se pɔrsin wae gɛt blɔd pasmak?

Di wangren we fɔ no fɔ tru if yu gɛt haypoglycemia na fɔ tɛst yu blɔd shuga lɛvɛl wit glukomita. Bɔku pan dɛn tin ya kin yuz wan smɔl nidul we dɛn kɔl lancet fɔ tek wan drop blɔd na di tip na yu finga.

Wan kɔntinyu glukɔs monitarin (CGM) divays na fayn tin fɔ ɛp yu fɔ no ɛn mek yu nɔ gɛt bɔku blɔd shuga. Bikɔs yu kin sɛt am fɔ wɔn yu we yu blɔd shuga smɔl. Dɛn CGM alɛt ya kin fayn mɔ pan tin dɛn we di blɔd shuga we nɔ bɔku kin denja, lɛk we yu de slip ɔ we yu de drayv.

If yu gɛt sayn dɛn fɔ hapo glycemia, bɔt no we nɔ de fɔ chɛk yu blɔd shuga, trit di hypoglycemia.

Aw pipul dɛm wae nɔr gɛt shuga kin no se pɔrsin wae gɛt blɔd pasmak?

If yu dɔktɔ tink se yu gɛt aypoglycemia, i kin chɛk yu blɔd shuga ɛvri sɔm awa we yu de fast fɔ sɔm dez. Dɛn kin du ɔda tɛst fɔ no di ɔndalayn kɔz fɔ di haypoglycemia. Fɔ ɛgzampul, dɛn kin du imej tɛst fɔ si if na kansa de mek di blɔd shuga nɔ bɔku.

If yu gɛt riaktiv haypoglycemia, yu dɔktɔ kin tɛl yu fɔ du tɛst we dɛn kɔl Mixed-Meal Tolerance Test (MMTT). Insay dis MMTT, yu fɔs drink spɛshal drink we gɛt prɔtin, fat, ɛn shuga. Dis drink de mek yu blɔd glukɔs lɛvɛl go ɔp, ɛn dis kin mek yu bɔdi mek mɔ insulin. Dɔn, dɔktɔ kin chɛk yu blɔd glukɔs bɔku tɛm insay di nɛks fayv awa.

Aw dɛn kin trit haypoglycemia (we blɔd shuga nɔ bɔku)?

Dɛn kin trit mild ɛn mɔdaret haypoglycemia bay we yu it ɔ drink sɔntin we gɛt shuga (kabɔhaydrɛt).

Di American Diabetes Association rεkomεnd di "15-15 rul" fכ trit wan mild כ mכdarεt haypoglycemia episod:

• It ɔ drink 15 gram kabɔhaydrɛt we de wok kwik kwik wan fɔ mek yu blɔd shuga go ɔp.
• Afta 15 minit, chɛk yu blɔd shuga lɛvɛl.
• If i stil de dכn 70 mg/dL, tek כda 15 gram fast-akt kכbaidret.
• Ripit dis te yu blɔd shuga lɛvɛl at le 70 mg/dL.

Fɔ no ɔmɔs kabɔhaydrɛt de insay di it dɛn we yu de it, rid di tin dɛn we de sho di it dɛn we yu de it. Na sɔm it dɛn we gɛt lɛk 15 gram kabɔhaydrɛt we de wok kwik kwik wan:

• Wan smɔl frut, fɔ ɛgzampul, af banana.
• 4 ɔns (af kɔp) frut jus ɔ rɛgyula sɔft drink (nɔto it).
• Wan tebul spɔnj shuga, ɔni ɔ srɔp.
• Wan tiub we gɛt instant glukɔs jel (luk di instrɔkshɔn dɛn).
• 3 ɔ 4 glukɔs tablɛt dɛn (luk di instrɔkshɔn dɛn).

If yu gɛt sayn dɛm fɔ hapo glycemia, bɔt no we nɔ de fɔ chɛk yu blɔd shuga, yuz di 15-15 lɔ te yu fil fayn.

Yɔŋ pikin dɛn kin nid smɔl smɔl kabɔhaydrɛt fɔ trit di haypoglycemia. Aks yu pikin in dɔktɔ bɔt dis.

Aw dɛn kin trit siriɔs haypoglycemia?

Sivεr haypoglycemia nid difrεnt tritmεnt pas mild כ mכdarεt haypoglycemia. If pɔsin we yu sabi gɛt aypoglycemia ɛn i nɔ de tɔk fayn, i de kɔnfyus, ɔ i nɔ de no natin, .Nɔ gi dɛn tin fɔ it ɔ wata. Dɛn bin ebul fɔ chok.

If yu gɛt siriɔs haypoglycemia, yu fɔ yuz imejensi glukagon. Dis na wan kayn sintetik glukagon. dipכnt pan di kayn, dεn kin gi am as injεkshכn כ as nos pauda (dray nos spεray). sεntetik glukכgn de gi signal fכ yu liva fכ rilis di glukכs we dεn stכr. Dis kin mek yu blɔd shuga lɛvɛl go ɔp.

If di glukagon na injεkshכn, injεkt am na in bכtכk, an, כ thigh akɔdin to di instrכkshכn dεm we de na di kit. If di glukagon na nos paoda, put am insay wan nos akɔdin to di instrɔkshɔn dɛn we de na di paket.

Wans i dɔn gɛt kɔnshɛns bak (bɔku tɛm insay 5 to 15 minit), i kin fil lɛk se i de nɔs ɛn vɔmit. If i de ledɔm, tɔn am na in sayd fɔ mek i nɔ chok if i vɔmit.

Nɔ shek fɔ kɔl 911 fɔ ɛp. If pɔsin nɔ no natin, i nɔ gɛt glukagon, ɔ yu nɔ no aw fɔ yuz am, kɔl 911 wantɛm wantɛm.

Yu tink se dɛn kin mek di blɔd shuga nɔ bɔku?

Bɔt i sɔri fɔ no se i nɔ kin izi fɔ mek yu nɔ gɛt bɔku blɔd shuga we yu gɛt dayabitis. Bɔt yu kin tray fɔ chenj yu dayabitis mɛnejɛmɛnt plan fɔ ridyus di nɔmba fɔ di hapoglycemic episɔd dɛm wae yu kin gɛt.

Dis kin izi fɔ du wans yu ɛn yu dɔktɔ dɔn ɔndastand wetin de mek yu blɔd shuga nɔ bɔku. Gi yu dɔktɔ bɔku tin bɔt di tɛm we yu blɔd shuga nɔ bɔku. Sɔntin we simpul lɛk fɔ chenj di tɛm we yu de tek yu mɛrɛsin, it, ɛn ɛksesaiz kin ebul fɔ sɔlv di prɔblɛm.

Dɛn step ya kin ɛp fɔ mek yu nɔ gɛt bɔku blɔd shuga:

• Tek ɔl yu mɛrɛsin dɛn jɔs lɛk aw dɛn tɛl yu fɔ tek.
• Fɔ fala yu dɔktɔ in advays bɔt aw fɔ it ɛn ɛksesaiz.
• Yuz wan kɔntinyu glukɔs monitarin divays (CGM), ɔ chɛk yu blɔd shuga lɛvɛl ɔltɛm – bifo ɛn afta yu it, bifo ɛn afta ɛksɛsayz, ɛn we yu de slip.
• Afta yu dɔn trit wan episod we gɛt lɔw blɔd shuga, rayt di ditel dɛm fɔ di episɔd. Put di ditel dɛm lɛk di tɛm, wetin yu it i nɔ tu te yet, if yu bin de du ɛksɛsayz, wetin yu bin de si, ɛn wetin na yu glukɔs lɛvɛl. Dis go ɛp yu ɛn yu dɔktɔ fɔ mek wan plan fɔ mek yu nɔ gɛt bɔku blɔd shuga tumara bambay.

Wetin kin apin if a gɛt haypoglycemia?

De luk fɔ hapoglycemia pan pipul dɛm wae nɔr gɛt dayabitis de dipen pan di ɔndalayn kɔz. If yu gɛt dayabitis, if yu gɛt ay blɔd blɔd ɔltɛm, dat kin mek yu denja. I kin mek pipul dɛn nɔ no bɔt di haypoglycemia. Dis min se yu nɔ go notis di wɔnin sayn dɛm we de sho se yu blɔd shuga nɔ bɔku.If yu gɛt smɔl blɔd shuga ɔltɛm, i rili impɔtant fɔ fɛn dɔktɔ we go ɛp yu fɔ kɔntrol yu dayabitis.

Aw a kin kia fɔ misɛf?

If yu gɛt dayabitis ɔ ɔda sik we de mek yu gɛt ay blɔd blɔd, wɛr mɛdikal alert nɛks ɔ breslɛt, ɔ kɛr mɛdikal ID kad wit yu so dat ɔda pipul dɛn go no aw fɔ ɛp yu we ɛnitin apin.

Dis na step dɛm wae yu kin tek fɔ mek yu nɔr gɛt siriɔs haypoglycemia:

• Ɔltɛm kip snek wit yu na os ɛn we yu de go na do – tin dɛn lɛk frut snek, glukɔs tab, ɔ jus bɔks.
• Ɛksplen to di wan dɛn we yu kin spɛn tɛm wit bɔt di haypoglycemia ɛn siriɔs hapoglycemia, ɛn aw dɛn kin ɛp yu if yu gɛt ɛnitin we apin.
• Ɔltɛm, yu fɔ kɛr imejensi glukagon. Tich di wan dɛn we yu lɛk aw fɔ yuz am ɛn ustɛm fɔ yuz am.

If yu gɛt dayabitis, nɔ tray fɔ trit yu blɔd shuga pasmak bay we yu it bɔku tin dɛn we gɛt kabɔhaydrɛt. Dis kin mek yu blɔd shuga kam bak ɔp. I kin tranga fɔ stɔp fɔ it pasmak bikɔs yu bɔdi de "fayt ɔ flay" rispɔns. Afta yu dɔn trit yusɛf wit 15 gram kabɔhaydrɛt, sɛt tayma fɔ 15 minit bifo yu it ɛni mɔ shuga.

Ustɛm a fɔ go to mi dɔktɔ bɔt haypoglycemia?

If yu gɛt dayabitis ɛn yu de gɛt lɔw blɔd shuga ɔltɛm, tɔk to dɔktɔ we de ɛp yu fɔ kɔntrol yu dayabitis. I kin ɛp yu fɔ mek wan plan fɔ manej. Dis kin inklud fɔ chenj di mɛrɛsin we yu de tek, di tin dɛn we yu fɔ it, ɔ di we aw yu de du ɛksɛsayz.

If yu gɛt smɔl blɔd shuga we yu nɔ gɛt dayabitis, tɔk to yu dɔktɔ. I go nid fɔ rɔn tɛst fɔ no di men tin we mek i apin. Sɔntɛnde, de sik kin siriɔs. So, i rili impɔtant fɔ go to yu dɔktɔ.

Wetin na di difrɛns bitwin Hypoglycemia ɛn Hyperglycemia?

Fɔ tɔk am simpul wan, haypoglycemia na we di blɔd shuga nɔ bɔku. ‘Hypo-’ min ‘lɔ’. Hyperglycemia na di blɔd shuga we bɔku. ‘Hyper-’ min ‘ay’ ɔ ‘ɛksɛs’.

Fɔ dɔn, tin dɛn fɔ mɛmba (Take-Home Message) .

Hypoglycemia na wan kɔmɔn sik wae kin afɛkt pipul dɛm wae gɛt shuga. If dɛn nɔ trit am kwik, i kin mek pɔsin in layf de pan denja. I impɔtant fɔ lan yu yon wɔnin sayn dɛm ɛn fɔ tɛst yu blɔd shuga lɛvɛl ɔltɛm fɔ mek yu nɔ gɛt siriɔs aypa blɔd.If yu gɛt smɔl blɔd shuga ɔltɛm, wok wit yu dɔktɔ fɔ mek wan plan fɔ mɛn yu shuga. Di mɔ yu no bɔt wetin kin mek yu episɔd, na di mɔ i go izi fɔ mek yu plan.

If yu gɛt smɔl blɔd shuga we yu nɔ gɛt dayabitis, yu fɔ rili go to dɔktɔ. Pan ɔl we i nɔ kin izi fɔ mek di rizin we mek i siriɔs, i kin apin. So, i bɛtɛ fɔ mek dɛn tɛst yu kwik kwik wan pas fɔ du am leta. Tek kia ɔf yu wɛlbɔdi!

` Hypoglycemia, low blood sugar, dayabitis, low blood glucose, di simptom dɛm, tritmɛnt, glucagon