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Lɛ wi lan mɔ bɔt blɔd tɛst fɔ no if pɔsin gɛt korona at sik.

Lɛ wi lan mɔ bɔt blɔd tɛst fɔ no if pɔsin gɛt korona at sik.

Yu dɔn ɛva wɔnda aw yu at gɛt wɛlbɔdi? Sɔntɛnde, wi kin gɛt sik dɛn we gɛt fɔ du wit at ɛn wi nɔ kin ivin no. Bɔt gud tin na dat, blɔd tɛst dɛn de we kin ɛp fɔ no dɛn tin ya we kin mek pɔsin gɛt prɔblɛm kwik kwik wan. So tide, lɛ wi tɔk bɔt wetin na dɛn blɔd tɛst ya ɛn aw dɛn go ɛp yu fɔ no if yu gɛt at sik, mɔ di korona at sik (CAD). Lɛ wi tray fɔ ɔndastand dis simpul wan, lɛk se yu de tɔk to yu padi.

Wetin mek yu nid fɔ du dɛn blɔd tɛst ya?

Naw yu go mɔs de wɔnda se, "Wetin mek a nid dɛn blɔd tɛst ya?" Nɔto ɔlman nid dɛn wantɛm wantɛm. Bɔt sɔm tin dɛn de we dɔktɔ kin tɛl yu fɔ du dɛn tɛst ya. Si if dɛn rizin ya apin to yu:

  • Di we aw yu de it: Bɔku tɛm yu kin it tin dɛn we nɔ fayn ɛn we gɛt bɔku ɔyl, shuga, ɛn flawa.
  • Famili istri: If ɛnibɔdi na yu famili, mɔ yu mama, papa, ɔ brɔda ɛn sista dɛn, dɔn gɛt korona at sik (CAD) bifo i ol 50 ia .
  • Fat ɔ we yu de gɛt bɔku bɔku bɔdi: If yu bɔdi wet pas aw i fɔ bi.
  • Nɔ ɛksesaiz: If di bɔdi nɔ de du ɛksɛsayz inof insay di de.
  • Smok: If yu na pɔsin we de smok ɔ yu de yuz ɔda kayn tabak (e.g., betel nat ɔ betel lif).
  • Ɔda mɛrɛsin: If yu dɔn ɔlrɛdi gɛt sik dɛn lɛk Dayabitis, Ay Blɔd Prɛshɔn, ɔ if yu bin dɔn gɛt at atak ɔ strok bifo.

If yu gɛt wan ɔ mɔ pan dɛn tin ya, i go fayn fɔ mek yu tɔk to dɔktɔ ɛn du blɔd tɛst lɛk dis.

Udat dɛn kin du dɛn blɔd tɛst ya?

Bɔku tɛm, na pɔsin we dɛn tren fɔ du dis kin tek blɔd sɛmpul frɔm yu. Wi kin kɔl dɛm ``Phlebotomist'' ɔ ``Phlebotomy technician''. Dɔn dɛn kin sɛn dis blɔd sɛmpul to wan mɛdikal lɛbɔretri. Wan ``Mɛdikal laboratori tɛknishian'' de de. I kin yuz spɛshal mashin dɛn fɔ mɛzhɔ di difrɛn tin dɛn we de na yu blɔd ɛn mek wan ripɔt.

Wetin dɛn blɔd tɛst ya kin mɛzhɔ fɔ no if pɔsin gɛt at sik?

Okay, naw lɛ wi si wetin rili dɛn blɔd tɛst ya de luk fɔ. Dis na wae dɔktɔ dɛn kin yuse fɔ gɛt aidia bɔt yu risk fɔ gɛt at sik.

Kɔlɔstrel ɛn tin dɛn we gɛt fɔ du wit am

  • Tכtal Kכlestכl (TC): Dis na di tכtal kכlestכl we de na yu bכdi. I gɛt fɔ du wit di risk we yu gɛt fɔ gɛt at sik.
  • Triglycerides (TG): Dis na wan kayn fat bak. Dɛn tin ya gɛt fɔ du wit at sik bak. di simpul shuga dεm, simpul kabohaydret dεm (lεk bred, rays), fεt it dεm, εn alkol we wi de it kin mek dεn `(TG)` lεvεl dεm ya go כp. Dis kin ridyus if yu du gud ɛksesaiz.
  • High-Density Lipoprotein (HDL): Dis na wetin wi kin kɔl "gud kɔlɔstrel." Di mɔ we i de na di blɔd, na di mɔ di risk fɔ gɛt at sik nɔ go bɔku.
  • Low-Density Lipoprotein (LDL): Dis na wetin wi kin kɔl "bad kɔlɔstrel." Di mɔ we i de na di blɔd, na di mɔ yu go gɛt at sik.
  • Lipoprotein (a) (Lp(a)): dis kin fכm we wan protin we dεn kכl apo(a) de atak LDL kכlestכl. If in lɛvɛl ay na di blɔd, i kin mek i gɛt at atak, strok, blɔd we kin klɔt, fat kin de na di at dɛn afta dɛn dɔn ɔpreshɔn di korona at baypas, ɛn di korona at kin smɔl bak afta dɛn dɔn du angioplasty. Dis kin rɔn bak na famili. So, if pɔsin na di famili dɔn gɛt at sik we i yɔŋ, di dɔktɔ go tɛl yu fɔ du dis tɛst.
  • Apolipoprotein B (ApoB): Dis na wan men protin we de insay di kɔlɔstrel. Sɔm stɔdi dɛn sho se di ApoB lɛvɛl na bɛtɛ indikɛtɔ fɔ risk pas di LDL lɛvɛl nɔmɔ. di hכy ApoB lεvεl dεm de sho se di nכmba fכ sכm sכm, dεns LDL patikyula dεm de inkrεs.

Ɔda impɔtant tin dɛn we de na di blɔd ɛn tin dɛn we de sho se pɔsin de du am

  • Complete Blood Count with Differential (CBC): Dis tɛst de sho di nɔmba fɔ di rɛd blɔd sɛl dɛn, wayt blɔd sɛl dɛn, ɛn di pletlɛt dɛn we de na yu blɔd. I kin no bɔt tin dɛn lɛk we pɔsin nɔ gɛt bɛtɛ blɔd, we na we pɔsin nɔ gɛt rɛd blɔd sɛl.
  • Homocysteine ​​(Hcy): Dis na amino asid. If dis kin bɔku na di blɔd, dat kin mek pɔsin gɛt at sik.
  • Ɛmoglobin A1c (HbA1c): Dis blɔd tɛst de ɛp fɔ no if pɔsin gɛt dayabitis. Dayabitis na wan sik wae de mek pɔrsin gɛt korona at sik (CAD). Dis tɛst de sho di avɛrej blɔd shuga lɛvɛl fɔ di pas tu to tri mɔnt.
  • Fasting Blood Glucose / Fasting Blood Sugar: Dis de mek yu no yu blɔd shuga lɛvɛl na mɔnin afta yu dɔn fast fɔ wan nɛt. I kin no bifo yu gɛt shuga ɛn dayabitis, ɛn dɛn ɔl tu kin mek yu gɛt at sik.
  • Creatine Kinase (CK): dis na wan εnzym we de insay di mכsul dεm. di lεvεl dεm we i de gi kin inkrεs if yu de tek mεdikeshכn dεm we de lכs kכlestכl (e.g., statin).
  • Alanine Aminotransferase (ALT) ɔ SGPT: Dis na ɛnzaym we de na di liva. I kin go ɔp we yu tek mɛrɛsin dɛn we de mek yu nɔ gɛt bɔku kɔlɔstrel.
  • Aspartate Transaminase (AST) ɔ SGOT: Dis na ɔda ɛnzaym na di liva. I kin go ɔp bak we yu de tek mɛrɛsin dɛn we de mek yu nɔ gɛt bɔku kɔlɔstrel.
  • Fibrinogen: .Dis na prɔtin we de insay yu blɔd. I de ɛp fɔ mek yu blɔd klɔt. Bɔt if i tu ay, i kin mek yu gɛt at atak mɔ ɛn mɔ.
  • Thyroid-Stimulating Hormone (TSH): Dis de sho aw yu tayroyd gland de wok fayn fayn wan. Dɛn dɔn tɔk se we tayroyd nɔ de wok fayn, dat kin mek i gɛt at sik ɛn at nɔ kin bit fayn.
  • Ultra-sensitive C-reactive Protein (us-CRP) כ High-sensitivity CRP: Dis de sho se inflameshn de na di bכdi vεsul dεm. Di mɔ in lɛvɛl go ɔp, na di mɔ di risk fɔ gɛt at ɛn blɔd vesel sik.
  • di urine Albumin-Creatinine Ratio (uACR): dis de mכsu di amoun fכ di protin we de insay di urine. Ivin smɔl prɔtin we de na di urine kin mek pɔsin gɛt at sik. Dis tɛst impɔtant mɔ fɔ pipul dɛn we gɛt dayabitis.
  • MPO (Myeloperoxidase): Dis na tin bak we de sho se yu gɛt inflamɛns. Di ay levul dɛm fɔ dis kin gɛt fɔ du wit di risk fɔ gɛt at atak, strok, ɛn nid fɔ gɛt korona at baypas ɔpreshɔn. If dis gɛt ay levul kin sho bak se di sik we de na di at ɛn di blɔd de wɔs.
  • Vitamin D: Dis na vaytamɛn we kin sɔlv di fat. i de rigul di kalsiכm εn fכsfεt lεvεl na di bכdi. Dɛn tin ya kin ɛp fɔ mek di bon dɛn gɛt wɛlbɔdi. If yu nɔ gɛt bɛtɛ vitamin D, dat kin mek yu gɛt difrɛn prɔblɛm dɛn we kin mek yu gɛt wɛlbɔdi. Dɔn bak, if i nɔ izi fɔ yu fɔ tek di statin (kɔlestrol mɛrɛsin), dis vaytamɛn D we yu nɔ gɛt kin bi wan rizin.
  • Trimethylamine N-oxide (TMAO): dis na di baktri dεm we de na yu gכt de mek am. I de bɔku bɔku wan na mit, eg, ɛn tin dɛn we dɛn mek wit milk. supliment dεm lεk choline, lecithin, εn L-carnitine kin inkrεs di TMAO lεvεl dεm bak. If di TMAO bɔku na di blɔd, i kin mek pɔsin gɛt at sik, strok, ɛn atrɔsklerɔsis.
  • Amino-terminal, pro-Brain Natriuretic Peptide (NT-proBNP): dis protin de mek na yu at. I kin kɔmɔt na di blɔd we yu at gɛt ɛkstra strɛs. di ay lεvεl fכ dis protin de sho se i gεt hכy risk fכ at sik εn i kin sho bak se di at de wok dכn. Dɛn kin du ɔda tɛst dɛn fɔ no mɔ.
  • Serum Creatinine (CR): Dis kin gɛt fɔ du wit di wok we di kidni dɛn de du. If di kidni dɛn nɔ de wok fayn, i kin put strɛs pan di at. Di kidni we nɔ de wok fayn na wan sik we kin mek pɔsin gɛt at sik mɔ ɛn mɔ. Ɛn di at kin afɛkt di kidni dɛn bak.
  • Atherosclerotic Cardiovascular Disease (ASCVD) Risk Score: Dis skכl de prεdikt yu risk fכ gεt ``atherosclerotic'' ivent (lεk hat atak כ strok) insay di nεks 10 ia.

Tink bɔt am dis we, ɛni wan pan dɛn tɛst ya tan lɛk ditektiv. I tek ɛni clue fɔ pies togɛda di wan ol pikchɔ. Semweso, dɔktɔ dɛn kin luk dɛn tɛst ripɔt ya ɛn kam fɔ no bɔt yu at in wɛlbɔdi.

Aw yu kin pripia bifo yu du blɔd tɛst?

Bifo dɛn pul yu blɔd, i impɔtant fɔ aks yu dɔktɔ if yu nid fɔ stɔp fɔ tek ɛni mɛrɛsin, vaytamɛn, ɔ sɔpɔtmɛnt we yu de tek. Sɔm mɛrɛsin dɛn kin afɛkt di tin dɛn we kin kɔmɔt frɔm di blɔd tɛst. Bɔt nɔ ɛva stɔp fɔ tek ɛni mɛrɛsin if yu dɔktɔ nɔ advays yu.

Sɔm blɔd tɛst kin mek yu fast fɔ sɔm awa (bɔku tɛm 8-12 awa) bifo di tɛst. So aks yu dɔktɔ bɔt dis bifo yu du di tɛst.

Wetin kin apin we dɛn de du di tɛst?

Phlebotomist kin put nidul insay wan vein na yu ɔp an ɔ insay yu ɛlbo. Yu kin fil lɛk se yu de swɛt we dɛn de put di nidul insay di vein. Di blɔd we dɛn kin pul kin tek lɛk wan minit so.

Wetin kin apin afta dɛn dɔn pul blɔd?

Afta dɛn dɔn tek di blɔd sɛmpul, di pɔsin we de mɛn di blɔd kin pul di nidul, ol di say we di blɔd sɛmpul de, put plasta ɔ kɔtɔn, ɛn put tin we de tay.

Wetin na di rizulyt dɛn we a kin gɛt? Wetin dɛn de tɛl mi?

Di tin dɛn we go apin go difrɛn difrɛn wan bay di difrɛn tin dɛn we de na yu blɔd. Yu dɔktɔ go tɔk to yu bɔt yu rizɔlt ɛn tɛl yu wetin fɔ du nɛks.

Na sɔm tipik valyu dɛm we dɛn kin ɛkspɛkt fɔ sɔm impɔtant tɛst dɛm we gɛt fɔ du wit at sik risk:

  • Tɔtɔl Kɔlestɔl (TC): .
  • Ej 20 ɔ yɔŋ: Bitwin 75 - 169 mg/dL.
  • Ej 21 ɔ pas dat: Bitwin 100 - 199 mg/dL.
  • Triglisɛrɛyd (TG): If yu nɔ rich 150 mg/dL i fayn. If i pas 500 - 1000 mg/dL, risk de fɔ gɛt pankrias. Dis lɛvɛl kin pasmak pan pipul dɛm wae fat ɛn pipul dɛm wae gɛt shuga.
  • di hכy dεnsiti lipoprotein (HDL - "gud kכlestכl"): di hכy dis, di bεtε.
  • Man: i pas 45 mg/dL.
  • Fem: i pas 55 mg/dL.
  • di lכw dεnsiti lipoprotein (LDL - "bad kכlestכl"): di lכw di bεtε. Di lɛvɛl we dɛn de ɛkspɛkt go difrɛn difrɛn wan dipen pan yu wɛlbɔdi kɔndishɔn:
  • If yu gɛt at sik, blɔd vesel sik, dayabitis, ɔ yu de pan rili ay risk fɔ gɛt at sik: I nɔ rich 70 mg/dL.
  • If yu gɛt `(Mɛtabolik Sindrɔm)` ɔ pas wan tin we kin mek yu gɛt at sik: I nɔ rich 100 mg/dL.
  • If yu gɛt smɔl risk fɔ gɛt korona at sik: I nɔ rich 130 mg/dL.
  • Kɔmplit blɔd kɔnt (CBC): .
  • di wayt bכdi sεl kכnt: 5,000 - 10,000 (fכ wan maykrolit).
  • εmatokrit (di כmכnt fכ bכdi we mek wit rεd bכdi sεl dεm): Man dεm 40% - 55%; Uman dɛn 36% - 48%.
  • εmoglobin (di pat pan di rεd bכdi sεl dεm we de kכri כksijεn): Man dεm 14 - 18 gm/dL; Di uman dɛn 12 - 16 gm/dL.
  • Lipoprotein (A) (Lp(a)): less dan 30 mg/dL i gud.
  • Apolipoprotein B (ApoB): I nɔ rich 100 mg/dL i fayn.
  • Homocysteine ​​(Hcy): less dan 15 mcmol/L i gud.
  • Ɛmoglobin A1c (HbA1c): .
  • Hεlth lεvεl: 6% כ less.
  • Pridiabetes: 5.7% - 6.4%.
  • Dayabitis: 6.5% ɔ pas dat.
  • Target fɔ pɔrsin wae gɛt dayabitis: 6.5% - less dan 7%.
  • Fast Blɔd Glukɔs: If yu nɔ rich 100 mg/dL i fayn.
  • Prɛdiabɛtis: 100 - 125 mg/dL.
  • Dayabitis: 126 mg/dL ɔ pas dat pan tu difrɛn tɛm dɛn.
  • Kriatin kinaz (CK): Nɔmal rɛnj 30 - 220 U/L.
  • Alanin aminotransfεrayz (ALT/SGPT): Nכmal rεnj 5 - 45 U/L.
  • Aspartat transaminase (AST/SGOT): Nɔmal rɛnj 7 - 40 U/L.
  • Fibrinogen: Nɔmal rɛnj 200 - 400 mg/dL.
  • Tayroyd Stimulεt Hכmon (TSH): di כptimal rεnj na 0.4 - 5.5 mIU/mL. (Dis kin difrɛn if yu bin dɔn gɛt tayroyd tritmɛnt bifo tɛm).
  • Ultra-sεnsitiv C-rεaktiv protin (us-CRP):
  • Lɔw risk: less dan 2.0 mg/L.
  • Modaret risk: 2.0 - 3.0 mg/L.
  • High risk: i pas 3.0 mg/L.
  • Yurin albumin-kriatinin rεshכ (uACR): lεs dan 30 mg/g i gud.
  • MPO: Les dan 420 pmol/L na gud tin.
  • Vitamin D: Di rεnj כptimal na 31 - 80 ng/ml.
  • Trimethylamin N-ɔksayd (TMAO): .
  • Lɔw risk: less dan 6.2 uM.
  • Modaret risk: 6.2 - 9.9 uM.
  • High risk: 10 uM ɔ mɔ.

(If di TMAO lɛvɛl bɔku, yu dɔktɔ kin se yu fɔ lɛf LDL ɛn kɔntrol ɔda tin dɛn we kin mek yu gɛt prɔblɛm. Dɛn kin tɛl yu bak se yu fɔ tek aspirin we nɔ bɔku tu tɛm insay di de ɛn fala di it we dɛn kin it na Mɛditarenian.)

  • NT-proBNP: Lεs dan 125 pg/mL na gud tin.
  • Sεrum Kriatinin (CR): .
  • Fem: 0,58 - 0,96 mg/dL.
  • Man dɛn: 0,73 - 1,22 mg/dL.
  • ASCVD risk skɔ: .
  • Lɔw risk: <5% .
  • Bɔdalayn risk: 5-7.4%
  • Mɔdarɛt risk: 7.5-<20%
  • Di ay risk: >20%

Impɔtant: Dɛn tin ya na jɔs avrej valyu dɛn. Yu fɔ kɔmpia di tin dɛn we yu dɔn fɛn to bɔku ɔda tin dɛn lɛk yu ej, yu man ɔ uman, ɛn ɔda sik dɛn we yu gɛt. So , i impɔtant fɔ tɔk bɔt dɛn rizɔlt ya wit yu dɔktɔ.

Aw lɔng i kin tek fɔ no di tin dɛn we kin apin?

Bɔku tɛm, di blɔd tɛst rizɔlt kin tek sɔm dez ɔ mɔ fɔ kam bak. Aks yu dɔktɔ aw lɔng i go tek fɔ gɛt yu rizɔlt.

A fɔ go to di dɔktɔ bak?

If yu nɔ gɛt yu tɛst rizɔlt afta lɛk tu wiks, kɔl yu dɔktɔ. We yu dɔn gɛt di rizɔlt, yu dɔktɔ go tɔk bɔt am wit yu ɛn tɛl yu wetin fɔ du nɛks.

Mɛmba se pan ɔl we blɔd tɛst kin gi yu gud aidia bɔt yu risk fɔ gɛt at sik, dɛn wan ya nɔ go ebul fɔ tɛl yu fɔ tru se yu gɛt di sik. If yu blɔd tɛst sho se yu gɛt bɔku prɔblɛm, yu dɔktɔ kin tɛl yu fɔ du ɔda tɛst. Nɔr frayd fɔ tɔk to yu dɔktɔ bɔt ɛni kwɛstyɔn ɔr kɔnsyusɔn wae yu gɛt ɔl dis prɔses.

Mɛsej we dɛn kin kɛr go na os

Fɔ tɔk am simpul wan, dɛn blɔd tɛst ya na fayn tin fɔ no if pɔsin gɛt at sik kwik kwik wan. Bay di infɔmeshɔn wae dɛn gi yu, yu kin chenj yu layf ɛn if nid de, yu kin bigin fɔ trit yu. Dɔn, yu gɛt mɔ chans fɔ protɛkt yusɛf frɔm siriɔs at sik.

Mɛmba:

  • If yu gɛt tin dɛn we kin mek yu gɛt at sik (di wan dɛn we wi bin dɔn tɔk bɔt fɔs), mek dɛn du dɛn tɛst ya pan dɔktɔ advays.
  • Tɔk opin wan wit yu dɔktɔ bɔt di rizɔlt.
  • Nɔ wɔri if di rizɔlt sho se di risk bɔku. I jɔs min se yu nid fɔ tek tɛm kia fɔ yu wɛlbɔdi smɔl.
  • Yu kin mek yu at gɛt wɛlbɔdi bay we yu de it tin dɛn we fayn, du ɛksɛsayz ɔltɛm , ɛn avɔyd fɔ smok .

Jɔs lɛk aw yu at valyu to yu, na so i rili valyu bak fɔ no bɔt yu at wɛlbɔdi!


` At sik, blɔd tɛst, kɔlɔstrel, korona at sik, at wɛlbɔdi, CAD, Blɔd Tɛst, Kɔlestɔl, Koronari At Sik, At Wɛlbɔdi, Prɛventiv Ɛlth

⚠️ Important: The medical articles and information on Nirogi Lanka are for general awareness only, and are by no means a substitute for professional medical advice, diagnosis, or treatment. For any medical problem you have, consult a qualified physician immediately.

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Lɛ wi lan mɔ bɔt blɔd tɛst fɔ no if pɔsin gɛt korona at sik.
Prɛventiv ƐlthJuly 5, 2026

Lɛ wi lan mɔ bɔt blɔd tɛst fɔ no if pɔsin gɛt korona at sik.

Yu dɔn ɛva wɔnda aw yu at gɛt wɛlbɔdi? Sɔntɛnde, wi kin gɛt sik dɛn we gɛt fɔ du wit at ɛn wi nɔ kin ivin no. Bɔt gud tin na dat, blɔd tɛst dɛn de we kin ɛp fɔ no dɛn tin ya we kin mek pɔsin gɛt prɔblɛm kwik kwik wan. So tide, lɛ wi tɔk bɔt wetin na dɛn blɔd tɛst ya ɛn aw dɛn go ɛp yu fɔ no if yu gɛt at sik, mɔ di korona at sik (CAD). Lɛ wi tray fɔ ɔndastand dis simpul wan, lɛk se yu de tɔk to yu padi.

Wetin mek yu nid fɔ du dɛn blɔd tɛst ya?

Naw yu go mɔs de wɔnda se, "Wetin mek a nid dɛn blɔd tɛst ya?" Nɔto ɔlman nid dɛn wantɛm wantɛm. Bɔt sɔm tin dɛn de we dɔktɔ kin tɛl yu fɔ du dɛn tɛst ya. Si if dɛn rizin ya apin to yu:

  • Di we aw yu de it: Bɔku tɛm yu kin it tin dɛn we nɔ fayn ɛn we gɛt bɔku ɔyl, shuga, ɛn flawa.
  • Famili istri: If ɛnibɔdi na yu famili, mɔ yu mama, papa, ɔ brɔda ɛn sista dɛn, dɔn gɛt korona at sik (CAD) bifo i ol 50 ia .
  • Fat ɔ we yu de gɛt bɔku bɔku bɔdi: If yu bɔdi wet pas aw i fɔ bi.
  • Nɔ ɛksesaiz: If di bɔdi nɔ de du ɛksɛsayz inof insay di de.
  • Smok: If yu na pɔsin we de smok ɔ yu de yuz ɔda kayn tabak (e.g., betel nat ɔ betel lif).
  • Ɔda mɛrɛsin: If yu dɔn ɔlrɛdi gɛt sik dɛn lɛk Dayabitis, Ay Blɔd Prɛshɔn, ɔ if yu bin dɔn gɛt at atak ɔ strok bifo.

If yu gɛt wan ɔ mɔ pan dɛn tin ya, i go fayn fɔ mek yu tɔk to dɔktɔ ɛn du blɔd tɛst lɛk dis.

Udat dɛn kin du dɛn blɔd tɛst ya?

Bɔku tɛm, na pɔsin we dɛn tren fɔ du dis kin tek blɔd sɛmpul frɔm yu. Wi kin kɔl dɛm ``Phlebotomist'' ɔ ``Phlebotomy technician''. Dɔn dɛn kin sɛn dis blɔd sɛmpul to wan mɛdikal lɛbɔretri. Wan ``Mɛdikal laboratori tɛknishian'' de de. I kin yuz spɛshal mashin dɛn fɔ mɛzhɔ di difrɛn tin dɛn we de na yu blɔd ɛn mek wan ripɔt.

Wetin dɛn blɔd tɛst ya kin mɛzhɔ fɔ no if pɔsin gɛt at sik?

Okay, naw lɛ wi si wetin rili dɛn blɔd tɛst ya de luk fɔ. Dis na wae dɔktɔ dɛn kin yuse fɔ gɛt aidia bɔt yu risk fɔ gɛt at sik.

Kɔlɔstrel ɛn tin dɛn we gɛt fɔ du wit am

  • Tכtal Kכlestכl (TC): Dis na di tכtal kכlestכl we de na yu bכdi. I gɛt fɔ du wit di risk we yu gɛt fɔ gɛt at sik.
  • Triglycerides (TG): Dis na wan kayn fat bak. Dɛn tin ya gɛt fɔ du wit at sik bak. di simpul shuga dεm, simpul kabohaydret dεm (lεk bred, rays), fεt it dεm, εn alkol we wi de it kin mek dεn `(TG)` lεvεl dεm ya go כp. Dis kin ridyus if yu du gud ɛksesaiz.
  • High-Density Lipoprotein (HDL): Dis na wetin wi kin kɔl "gud kɔlɔstrel." Di mɔ we i de na di blɔd, na di mɔ di risk fɔ gɛt at sik nɔ go bɔku.
  • Low-Density Lipoprotein (LDL): Dis na wetin wi kin kɔl "bad kɔlɔstrel." Di mɔ we i de na di blɔd, na di mɔ yu go gɛt at sik.
  • Lipoprotein (a) (Lp(a)): dis kin fכm we wan protin we dεn kכl apo(a) de atak LDL kכlestכl. If in lɛvɛl ay na di blɔd, i kin mek i gɛt at atak, strok, blɔd we kin klɔt, fat kin de na di at dɛn afta dɛn dɔn ɔpreshɔn di korona at baypas, ɛn di korona at kin smɔl bak afta dɛn dɔn du angioplasty. Dis kin rɔn bak na famili. So, if pɔsin na di famili dɔn gɛt at sik we i yɔŋ, di dɔktɔ go tɛl yu fɔ du dis tɛst.
  • Apolipoprotein B (ApoB): Dis na wan men protin we de insay di kɔlɔstrel. Sɔm stɔdi dɛn sho se di ApoB lɛvɛl na bɛtɛ indikɛtɔ fɔ risk pas di LDL lɛvɛl nɔmɔ. di hכy ApoB lεvεl dεm de sho se di nכmba fכ sכm sכm, dεns LDL patikyula dεm de inkrεs.

Ɔda impɔtant tin dɛn we de na di blɔd ɛn tin dɛn we de sho se pɔsin de du am

  • Complete Blood Count with Differential (CBC): Dis tɛst de sho di nɔmba fɔ di rɛd blɔd sɛl dɛn, wayt blɔd sɛl dɛn, ɛn di pletlɛt dɛn we de na yu blɔd. I kin no bɔt tin dɛn lɛk we pɔsin nɔ gɛt bɛtɛ blɔd, we na we pɔsin nɔ gɛt rɛd blɔd sɛl.
  • Homocysteine ​​(Hcy): Dis na amino asid. If dis kin bɔku na di blɔd, dat kin mek pɔsin gɛt at sik.
  • Ɛmoglobin A1c (HbA1c): Dis blɔd tɛst de ɛp fɔ no if pɔsin gɛt dayabitis. Dayabitis na wan sik wae de mek pɔrsin gɛt korona at sik (CAD). Dis tɛst de sho di avɛrej blɔd shuga lɛvɛl fɔ di pas tu to tri mɔnt.
  • Fasting Blood Glucose / Fasting Blood Sugar: Dis de mek yu no yu blɔd shuga lɛvɛl na mɔnin afta yu dɔn fast fɔ wan nɛt. I kin no bifo yu gɛt shuga ɛn dayabitis, ɛn dɛn ɔl tu kin mek yu gɛt at sik.
  • Creatine Kinase (CK): dis na wan εnzym we de insay di mכsul dεm. di lεvεl dεm we i de gi kin inkrεs if yu de tek mεdikeshכn dεm we de lכs kכlestכl (e.g., statin).
  • Alanine Aminotransferase (ALT) ɔ SGPT: Dis na ɛnzaym we de na di liva. I kin go ɔp we yu tek mɛrɛsin dɛn we de mek yu nɔ gɛt bɔku kɔlɔstrel.
  • Aspartate Transaminase (AST) ɔ SGOT: Dis na ɔda ɛnzaym na di liva. I kin go ɔp bak we yu de tek mɛrɛsin dɛn we de mek yu nɔ gɛt bɔku kɔlɔstrel.
  • Fibrinogen: .Dis na prɔtin we de insay yu blɔd. I de ɛp fɔ mek yu blɔd klɔt. Bɔt if i tu ay, i kin mek yu gɛt at atak mɔ ɛn mɔ.
  • Thyroid-Stimulating Hormone (TSH): Dis de sho aw yu tayroyd gland de wok fayn fayn wan. Dɛn dɔn tɔk se we tayroyd nɔ de wok fayn, dat kin mek i gɛt at sik ɛn at nɔ kin bit fayn.
  • Ultra-sensitive C-reactive Protein (us-CRP) כ High-sensitivity CRP: Dis de sho se inflameshn de na di bכdi vεsul dεm. Di mɔ in lɛvɛl go ɔp, na di mɔ di risk fɔ gɛt at ɛn blɔd vesel sik.
  • di urine Albumin-Creatinine Ratio (uACR): dis de mכsu di amoun fכ di protin we de insay di urine. Ivin smɔl prɔtin we de na di urine kin mek pɔsin gɛt at sik. Dis tɛst impɔtant mɔ fɔ pipul dɛn we gɛt dayabitis.
  • MPO (Myeloperoxidase): Dis na tin bak we de sho se yu gɛt inflamɛns. Di ay levul dɛm fɔ dis kin gɛt fɔ du wit di risk fɔ gɛt at atak, strok, ɛn nid fɔ gɛt korona at baypas ɔpreshɔn. If dis gɛt ay levul kin sho bak se di sik we de na di at ɛn di blɔd de wɔs.
  • Vitamin D: Dis na vaytamɛn we kin sɔlv di fat. i de rigul di kalsiכm εn fכsfεt lεvεl na di bכdi. Dɛn tin ya kin ɛp fɔ mek di bon dɛn gɛt wɛlbɔdi. If yu nɔ gɛt bɛtɛ vitamin D, dat kin mek yu gɛt difrɛn prɔblɛm dɛn we kin mek yu gɛt wɛlbɔdi. Dɔn bak, if i nɔ izi fɔ yu fɔ tek di statin (kɔlestrol mɛrɛsin), dis vaytamɛn D we yu nɔ gɛt kin bi wan rizin.
  • Trimethylamine N-oxide (TMAO): dis na di baktri dεm we de na yu gכt de mek am. I de bɔku bɔku wan na mit, eg, ɛn tin dɛn we dɛn mek wit milk. supliment dεm lεk choline, lecithin, εn L-carnitine kin inkrεs di TMAO lεvεl dεm bak. If di TMAO bɔku na di blɔd, i kin mek pɔsin gɛt at sik, strok, ɛn atrɔsklerɔsis.
  • Amino-terminal, pro-Brain Natriuretic Peptide (NT-proBNP): dis protin de mek na yu at. I kin kɔmɔt na di blɔd we yu at gɛt ɛkstra strɛs. di ay lεvεl fכ dis protin de sho se i gεt hכy risk fכ at sik εn i kin sho bak se di at de wok dכn. Dɛn kin du ɔda tɛst dɛn fɔ no mɔ.
  • Serum Creatinine (CR): Dis kin gɛt fɔ du wit di wok we di kidni dɛn de du. If di kidni dɛn nɔ de wok fayn, i kin put strɛs pan di at. Di kidni we nɔ de wok fayn na wan sik we kin mek pɔsin gɛt at sik mɔ ɛn mɔ. Ɛn di at kin afɛkt di kidni dɛn bak.
  • Atherosclerotic Cardiovascular Disease (ASCVD) Risk Score: Dis skכl de prεdikt yu risk fכ gεt ``atherosclerotic'' ivent (lεk hat atak כ strok) insay di nεks 10 ia.

Tink bɔt am dis we, ɛni wan pan dɛn tɛst ya tan lɛk ditektiv. I tek ɛni clue fɔ pies togɛda di wan ol pikchɔ. Semweso, dɔktɔ dɛn kin luk dɛn tɛst ripɔt ya ɛn kam fɔ no bɔt yu at in wɛlbɔdi.

Aw yu kin pripia bifo yu du blɔd tɛst?

Bifo dɛn pul yu blɔd, i impɔtant fɔ aks yu dɔktɔ if yu nid fɔ stɔp fɔ tek ɛni mɛrɛsin, vaytamɛn, ɔ sɔpɔtmɛnt we yu de tek. Sɔm mɛrɛsin dɛn kin afɛkt di tin dɛn we kin kɔmɔt frɔm di blɔd tɛst. Bɔt nɔ ɛva stɔp fɔ tek ɛni mɛrɛsin if yu dɔktɔ nɔ advays yu.

Sɔm blɔd tɛst kin mek yu fast fɔ sɔm awa (bɔku tɛm 8-12 awa) bifo di tɛst. So aks yu dɔktɔ bɔt dis bifo yu du di tɛst.

Wetin kin apin we dɛn de du di tɛst?

Phlebotomist kin put nidul insay wan vein na yu ɔp an ɔ insay yu ɛlbo. Yu kin fil lɛk se yu de swɛt we dɛn de put di nidul insay di vein. Di blɔd we dɛn kin pul kin tek lɛk wan minit so.

Wetin kin apin afta dɛn dɔn pul blɔd?

Afta dɛn dɔn tek di blɔd sɛmpul, di pɔsin we de mɛn di blɔd kin pul di nidul, ol di say we di blɔd sɛmpul de, put plasta ɔ kɔtɔn, ɛn put tin we de tay.

Wetin na di rizulyt dɛn we a kin gɛt? Wetin dɛn de tɛl mi?

Di tin dɛn we go apin go difrɛn difrɛn wan bay di difrɛn tin dɛn we de na yu blɔd. Yu dɔktɔ go tɔk to yu bɔt yu rizɔlt ɛn tɛl yu wetin fɔ du nɛks.

Na sɔm tipik valyu dɛm we dɛn kin ɛkspɛkt fɔ sɔm impɔtant tɛst dɛm we gɛt fɔ du wit at sik risk:

  • Tɔtɔl Kɔlestɔl (TC): .
  • Ej 20 ɔ yɔŋ: Bitwin 75 - 169 mg/dL.
  • Ej 21 ɔ pas dat: Bitwin 100 - 199 mg/dL.
  • Triglisɛrɛyd (TG): If yu nɔ rich 150 mg/dL i fayn. If i pas 500 - 1000 mg/dL, risk de fɔ gɛt pankrias. Dis lɛvɛl kin pasmak pan pipul dɛm wae fat ɛn pipul dɛm wae gɛt shuga.
  • di hכy dεnsiti lipoprotein (HDL - "gud kכlestכl"): di hכy dis, di bεtε.
  • Man: i pas 45 mg/dL.
  • Fem: i pas 55 mg/dL.
  • di lכw dεnsiti lipoprotein (LDL - "bad kכlestכl"): di lכw di bεtε. Di lɛvɛl we dɛn de ɛkspɛkt go difrɛn difrɛn wan dipen pan yu wɛlbɔdi kɔndishɔn:
  • If yu gɛt at sik, blɔd vesel sik, dayabitis, ɔ yu de pan rili ay risk fɔ gɛt at sik: I nɔ rich 70 mg/dL.
  • If yu gɛt `(Mɛtabolik Sindrɔm)` ɔ pas wan tin we kin mek yu gɛt at sik: I nɔ rich 100 mg/dL.
  • If yu gɛt smɔl risk fɔ gɛt korona at sik: I nɔ rich 130 mg/dL.
  • Kɔmplit blɔd kɔnt (CBC): .
  • di wayt bכdi sεl kכnt: 5,000 - 10,000 (fכ wan maykrolit).
  • εmatokrit (di כmכnt fכ bכdi we mek wit rεd bכdi sεl dεm): Man dεm 40% - 55%; Uman dɛn 36% - 48%.
  • εmoglobin (di pat pan di rεd bכdi sεl dεm we de kכri כksijεn): Man dεm 14 - 18 gm/dL; Di uman dɛn 12 - 16 gm/dL.
  • Lipoprotein (A) (Lp(a)): less dan 30 mg/dL i gud.
  • Apolipoprotein B (ApoB): I nɔ rich 100 mg/dL i fayn.
  • Homocysteine ​​(Hcy): less dan 15 mcmol/L i gud.
  • Ɛmoglobin A1c (HbA1c): .
  • Hεlth lεvεl: 6% כ less.
  • Pridiabetes: 5.7% - 6.4%.
  • Dayabitis: 6.5% ɔ pas dat.
  • Target fɔ pɔrsin wae gɛt dayabitis: 6.5% - less dan 7%.
  • Fast Blɔd Glukɔs: If yu nɔ rich 100 mg/dL i fayn.
  • Prɛdiabɛtis: 100 - 125 mg/dL.
  • Dayabitis: 126 mg/dL ɔ pas dat pan tu difrɛn tɛm dɛn.
  • Kriatin kinaz (CK): Nɔmal rɛnj 30 - 220 U/L.
  • Alanin aminotransfεrayz (ALT/SGPT): Nכmal rεnj 5 - 45 U/L.
  • Aspartat transaminase (AST/SGOT): Nɔmal rɛnj 7 - 40 U/L.
  • Fibrinogen: Nɔmal rɛnj 200 - 400 mg/dL.
  • Tayroyd Stimulεt Hכmon (TSH): di כptimal rεnj na 0.4 - 5.5 mIU/mL. (Dis kin difrɛn if yu bin dɔn gɛt tayroyd tritmɛnt bifo tɛm).
  • Ultra-sεnsitiv C-rεaktiv protin (us-CRP):
  • Lɔw risk: less dan 2.0 mg/L.
  • Modaret risk: 2.0 - 3.0 mg/L.
  • High risk: i pas 3.0 mg/L.
  • Yurin albumin-kriatinin rεshכ (uACR): lεs dan 30 mg/g i gud.
  • MPO: Les dan 420 pmol/L na gud tin.
  • Vitamin D: Di rεnj כptimal na 31 - 80 ng/ml.
  • Trimethylamin N-ɔksayd (TMAO): .
  • Lɔw risk: less dan 6.2 uM.
  • Modaret risk: 6.2 - 9.9 uM.
  • High risk: 10 uM ɔ mɔ.

(If di TMAO lɛvɛl bɔku, yu dɔktɔ kin se yu fɔ lɛf LDL ɛn kɔntrol ɔda tin dɛn we kin mek yu gɛt prɔblɛm. Dɛn kin tɛl yu bak se yu fɔ tek aspirin we nɔ bɔku tu tɛm insay di de ɛn fala di it we dɛn kin it na Mɛditarenian.)

  • NT-proBNP: Lεs dan 125 pg/mL na gud tin.
  • Sεrum Kriatinin (CR): .
  • Fem: 0,58 - 0,96 mg/dL.
  • Man dɛn: 0,73 - 1,22 mg/dL.
  • ASCVD risk skɔ: .
  • Lɔw risk: <5% .
  • Bɔdalayn risk: 5-7.4%
  • Mɔdarɛt risk: 7.5-<20%
  • Di ay risk: >20%

Impɔtant: Dɛn tin ya na jɔs avrej valyu dɛn. Yu fɔ kɔmpia di tin dɛn we yu dɔn fɛn to bɔku ɔda tin dɛn lɛk yu ej, yu man ɔ uman, ɛn ɔda sik dɛn we yu gɛt. So , i impɔtant fɔ tɔk bɔt dɛn rizɔlt ya wit yu dɔktɔ.

Aw lɔng i kin tek fɔ no di tin dɛn we kin apin?

Bɔku tɛm, di blɔd tɛst rizɔlt kin tek sɔm dez ɔ mɔ fɔ kam bak. Aks yu dɔktɔ aw lɔng i go tek fɔ gɛt yu rizɔlt.

A fɔ go to di dɔktɔ bak?

If yu nɔ gɛt yu tɛst rizɔlt afta lɛk tu wiks, kɔl yu dɔktɔ. We yu dɔn gɛt di rizɔlt, yu dɔktɔ go tɔk bɔt am wit yu ɛn tɛl yu wetin fɔ du nɛks.

Mɛmba se pan ɔl we blɔd tɛst kin gi yu gud aidia bɔt yu risk fɔ gɛt at sik, dɛn wan ya nɔ go ebul fɔ tɛl yu fɔ tru se yu gɛt di sik. If yu blɔd tɛst sho se yu gɛt bɔku prɔblɛm, yu dɔktɔ kin tɛl yu fɔ du ɔda tɛst. Nɔr frayd fɔ tɔk to yu dɔktɔ bɔt ɛni kwɛstyɔn ɔr kɔnsyusɔn wae yu gɛt ɔl dis prɔses.

Mɛsej we dɛn kin kɛr go na os

Fɔ tɔk am simpul wan, dɛn blɔd tɛst ya na fayn tin fɔ no if pɔsin gɛt at sik kwik kwik wan. Bay di infɔmeshɔn wae dɛn gi yu, yu kin chenj yu layf ɛn if nid de, yu kin bigin fɔ trit yu. Dɔn, yu gɛt mɔ chans fɔ protɛkt yusɛf frɔm siriɔs at sik.

Mɛmba:

  • If yu gɛt tin dɛn we kin mek yu gɛt at sik (di wan dɛn we wi bin dɔn tɔk bɔt fɔs), mek dɛn du dɛn tɛst ya pan dɔktɔ advays.
  • Tɔk opin wan wit yu dɔktɔ bɔt di rizɔlt.
  • Nɔ wɔri if di rizɔlt sho se di risk bɔku. I jɔs min se yu nid fɔ tek tɛm kia fɔ yu wɛlbɔdi smɔl.
  • Yu kin mek yu at gɛt wɛlbɔdi bay we yu de it tin dɛn we fayn, du ɛksɛsayz ɔltɛm , ɛn avɔyd fɔ smok .

Jɔs lɛk aw yu at valyu to yu, na so i rili valyu bak fɔ no bɔt yu at wɛlbɔdi!


` At sik, blɔd tɛst, kɔlɔstrel, korona at sik, at wɛlbɔdi, CAD, Blɔd Tɛst, Kɔlestɔl, Koronari At Sik, At Wɛlbɔdi, Prɛventiv Ɛlth

⚠️ Important: The medical articles and information on Nirogi Lanka are for general awareness only, and are by no means a substitute for professional medical advice, diagnosis, or treatment. For any medical problem you have, consult a qualified physician immediately.

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