Udat na lipidɔlɔjis - spɛshal pɔsin pan kɔlɔstrel? Lɛ wi fɛn ɔndastand!

Yu dɔktɔ dɔn tɛl yu se yu kɔlɔstrel bɔku smɔl? Ɔ yu de wɔri smɔl bikɔs sɔmbɔdi na yu famili gɛt ay kɔlɔstrel ? Dɔn dis atikul go rili impɔtant to yu. Tide wi go tɔk bɔt wan ‘Lipidologist’, dɔktɔ we spɛshal pan fat lɛk kɔlɔstrel.

Udat rili na Lipidɔlɔjis?

Fɔ tɔk am simpul wan, Lipidologist na dɔktɔ we spɛshal pan di kayn fat dɛn we de na wi bɔdi, mɔ di kɔlɔstrel . Sɔntɛnde, dɛn fat ya we dɛn kɔl lipid kin gɛda na wi blɔd vesel dɛn ɛn stɔp dɛn. Yu kin dɔn yɛri bɔt ‘bad kɔlɔstrel’. Di men wok we dɛn dɔktɔ ya de du na fɔ ɛp pipul dɛn we gɛt lipid disɔda.

Imajin, dɛn dɔktɔ ya de ɛp wi bay we dɛn nɔ jɔs du wan simpul blɔd tɛst ɛn yuz dɛn sɛns. Dɛn ɔndastand gud gud wan bɔt dɛn lipid abnɔmal tin ya, ɛn bak di tin dɛn we kin mek pɔsin gɛt at sik ɛn sik dɛn we de mek pɔsin gɛt at ɛn blɔd . rεsכch dεn sho se we wi bכdi in lεvεl dεm fכ ‘bad kכlestכl’ (LDL - Low-Density Lipoprotein) inkrεs, fεt dεposit (atheroma) kin bכku insay di bכdi vεsul dεm. dis bildup fכ fεt dεposit dεm na wetin wi kכl ‘atherosclerosis’.

Naw, luk, we dis `(LDL)` kכlestכl, dat bad kכlestכl, bכku na wi bכdi vεsul dεm, dεn kin bigin fכ kכl dεm. Na dat mek di lipidɔlɔjis dɛn kin wɔri mɔ bɔt di kayn `(LDL)` we de na yu blɔd. If yu `(LDL)` lɛvɛl ay, yu de pan ay risk fɔ gɛt at atak ɔ strok . So, dɛn dɔktɔ ya kin tray fɔ ɛp yu fɔ kɔntrol yu ay kɔlɔstrel lɛvɛl ɛn ridyus yu risk fɔ gɛt at sik ɛn blɔd vesel sik.

At sik ɛn vaskul sik na di men tin dɛn we kin mek pɔsin day ɛn disabled ɔlsay na di wɔl. Lipidɔlɔjis dɛm kin yuz dɛn ɛkspɛriɛns fɔ ɛp pipul dɛm wae de pan risk fɔ gɛt dɛn kayn sik ya bikɔs dɛn gɛt ay kɔlɔstrel.

Yu tink se spɛshal sɛtifiket de fɔ bi lipodɔlɔjis?

Yɛs, i rili de. Wan ɔganayzeshɔn de na Amɛrika we dɛn kɔl ``American Board of Clinical Lipidology``, ɛn dɛn kin gɛt dis spɛshal sɛtifiket afta dɛn dɔn pas ɛgzam we dɛn kin du. Fɔ mek dɔktɔ apia fɔ dis ɛgzam, i fɔ gɛt valid mɛdikal laysens we i gɛt naw. Dɔn bak, dɛn fɔ dɔn dɔn tu ia klinik ɛkspiriɛns, ɔ dɛn fɔ dɔn gɛt spɛshal sɛtifiket frɔm ɔda mɛdikal bɔd. Afta dɛn dɔn gɛt dis sɛtifiket, dɛn dɔktɔ ya gɛt fɔ ridyus am ɛvri 10 ia. Dis min se dɛn de lan nyu tin dɛn ɔltɛm.

Wetin rili lipodɔlɔjis kin du?

Lipidɔlɔjis kin trit pipul dɛn mɔ we gɛt lipid disɔda. Na sɔm ɛgzampul dɛn ya:

• Hyperlipidemia: Dis na we di fat (especially cholesterol) na di bכdi de go כp pas di nכmal lεvεl.
• Hypercholesterolemia: Dis na we di lɛvɛl fɔ di bad kɔlɔstrel we wi bin tɔk bɔt, dat na, LDL, kin go ɔp bad bad wan.
• Hypolipidemia: Dis na di ɔda say. I min se di kɔlɔstrel lɛvɛl na di blɔd smɔl pas aw i fɔ bi. Dis kin bi prɔblɛm bak sɔntɛnde.
• Famili haypa kɔlɔstrelemia: Dis na sɔntin we spɛshal smɔl. na di jin dεm de kכz am, we min se di lεvεl fכ LDL kכlestכl rili hכy, we de pas dכn tru jεnereshכn.
• Hypertriglyceridemia: Dis na we ɔda kayn fat we dɛn kɔl triglycerides de na di blɔd tumɔs. Dis nɔ fayn bak fɔ di at.

So, we dɛn de trit dɛn sik ya, pɔsin we de stɔdi bɔt lipid kin gi yu wan ɔ mɔ mɛrɛsin . Dɛn kin advays yu bak fɔ chenj di we aw yu de liv yu layf . Fɔ ɛgzampul, aw fɔ kɔntrol yu it ɛn ɛksesaiz. insay sכm spεshal kes dεm, dεn kin yuz tritmεnt bak we dεn kכl `(Lipoprotein apheresis)`. Dis involv fɔ tek sɔm pan yu blɔd tru spɛshal mashin, pul di bad `(LDL)` kɔlɔstrel frɔm am, dɔn yu go bak di blɔd we yu dɔn klin to yu bɔdi. Dis nɔto sɔntin we dɛn kin du fɔ ɔlman, bɔt i kin rili bɛnifit di wan dɛn we nid am.

Udat na di fɔs dɔktɔ we wi fɔ si if wi gɛt bɔku kɔlɔstrel?

As yu kam fɔ no se yu kɔlɔstrel lɛvɛl dɔn ay smɔl, yu kin si yu praymari kia (PCP) . Sɔntɛnde wi kin kɔl am bak yu famili dɔktɔ. I kin ɛp yu fɔ kɔntrol yu kɔlɔstrel na di bɛsis lɛvɛl.

Bɔt if dɛn tritmɛnt ya nɔ de wok fɔ yu, ɔ if yu gɛt prɔblɛm fɔ kɔntrol yu kɔlɔstrel lɛvɛl, yu kin tink bɔt fɔ go to dɔktɔ we de mɛn yu bɔdi. Dɛn ɔndastand dip kɔlestɔl ɛn di difrɛn tritmɛnt dɛn we de. Dɛn men gol na fɔ ɛp yu fɔ ridyus di risk fɔ gɛt at sik ɔr blɔd vesel sik.

Sɔm pipul dɛn kin tink se ɔl di wan dɛn we de stɔdi bɔt di lipid na di wan dɛn we de mɛn dɛn at. Bɔt nɔto so i bi. Bɔku pan di lipidɔlɔjis dɛn gɛt spɛshal no bɔt intanɛnt mɛrɛsin ɔ famili mɛrɛsin . Infakt, wan sɔv we dɛn bin du insay 2010 sho se 50% pan di mɛmba dɛn na di Nashɔnal Lipid Asɛmbli bin gɛt dis kayn spɛshal wok. So, yu kin aks yu rɛgyula dɔktɔ if i gɛt ɛni spɛshal no bɔt lipidɔlɔji.

Lɛ wi lan smɔl mɔ bɔt di fild fɔ lipidɔlɔji.

Lipidɔlɔji na jɔs wan branch pan mɛrɛsin we de stɔdi, no, ɛn trit lipid disɔda.

Wan lipɔlɔjis sabi dɛn tin ya gud gud wan:

• Aw di lipoprotein dεm de wok: dεn tin ya lεk motoka dεm we de transpכt fεt (lεk kכlestכl εn triglisεrayd) rawnd wi bכdi.
• Aw Atherosclerosis De Divεlכp: Dat fεt we de bכku insay di bכdi we wi bin dכn tכk bכt.
• Aw di it ɛn ɛksesaiz kin afɛkt di kɔlɔstrel lɛvɛl: Aw di it we wi de it ɛn di ɛksɛsayz we wi de du kin afɛkt wi kɔlɔstrel.
• Difrɛn tritmɛnt dɛn de fɔ mek pɔsin gɛt ay kɔlɔstrel: mɛrɛsin, chenj di we aw pɔsin de liv in layf, ɛn ɔda tin dɛn.
• Aw fɔ trit pipul dɛm wae gɛt ɛritayt lipid disɔda: Fɔ ɛgzampul, kɔndishɔn dɛm lɛk ``Familial hypercholesterolemia``.
• Aw fɔ asɛs yu risk fɔ gɛt hat sik ɛn blɔd vesel sik yuse risk factors: Fɔ no yu risk bay tin dɛm lɛk yu ej, famili histri, ɛn if yu de smok ɔr nɔ smok.

Fɔ tru, lipidɔlɔji na nyu spɛshal wok pan mɛrɛsin. I bin jɔs bigin fɔ divɛlɔp insay di fɔs 2000 dɛm. Bɔt i dɔn bi spɛshal tin we rili impɔtant bikɔs di prɔblɛm we de wit ay kɔlɔstrel na prɔblɛm fɔ bɔku pipul dɛn tide . Jɔs tink bɔt, na Amɛrika nɔmɔ, dɛn se i pas 80 milyɔn pipul dɛn we dɔn pas 20 ia we gɛt bɔku kɔlɔstrel. I tan lɛk se dis tin de bɔku na Sri Lanka bak.

Wetin kin apin we yu go si lipɔlɔjis?

We yu go to lipidɔlɔjis, di dɔktɔ go tek blɔd sɛmpul fɔs frɔm yu. Na dat.Dɛn kin du wan lipid panɛl tɛst. Dis kin mek yu no di difrɛn kayn kɔlɔstrel (lɛk LDL ɛn HDL) ɛn triglisɛrɛyd dɛn na yu blɔd kɔrɛkt wan.

Dɔn, di dɔktɔ go du wan ɛgzam fɔ di bɔdi . I go tɔk to yu bak bɔt yu mɛdikal istri . E fayn fɔ tɛl yu famili histri, bikɔs sɔmtɛm dɛn kayn sik ya kin de na famili. Di dɔktɔ go aks yu bak bɔt di tin dɛn we yu de du ɛvride . Fɔ ɛgzampul, yu kin yuz tabak ɔ yu kin yuz ɛksɛsayz? Ɔl dis go ɛp yu fɔ mek wan tritmɛnt plan we go fayn fɔ yu.

We yu bigin fɔ tek mɛrɛsin we de mek yu nɔ gɛt bɔku bɔku kɔlɔstrel ɛn chenj yu layf, i go mɔs bi se yu dɔktɔ we de mɛn yu lipidɔlɔjis go nid fɔ si yu bak. Dis na bikɔs yu nid fɔ de chɛk yu blɔd kɔlɔstrel ɔltɛm fɔ mek shɔ se yu tritmɛnt de wok. Yu dɔktɔ kin nid fɔ tray ɔda tritmɛnt if wan tritmɛnt nɔ wok.

If yu de kɔntinyu fɔ gɛt prɔblɛm fɔ kɔntrol yu kɔlɔstrel, nɔ fred fɔ aks yu dɔktɔ fɔ mek dɛn sɛn yu to dɔktɔ we de mɛn yu bɔdi . Dɛn kin yuz dɛn ɛkspɛriɛns fɔ mek wan tritmɛnt plan we go fayn fɔ yu. Sɔm pipul dɛn, mɔ di wan dɛn we gɛt bɔku bɔku kɔlɔstrel na dɛn famili, kin nid fɔ gɛt mɔ advans tritmɛnt ɔ fɔ gɛt kɔmbayn tritmɛnt. So, nɔ shem fɔ aks fɔ ɛp.

So, wetin na di impɔtant tin dɛn we wi fɔ mɛmba frɔm dis stori? (Mɛsej we dɛn kin tek go na os)

Okay, so wi don tok plenti boht wan lipodologist tide. Fɔ tɔk smɔl, na sɔm tin dɛn we yu fɔ mɛmba:

• Lipidɔlɔjis na dɔktɔ we spɛshal pan lipid, lɛk kɔlɔstrel. Dɛn kin ɛp yu fɔ ridyus di risk fɔ gɛt at sik.
• If i nɔ izi fɔ yu fɔ kɔntrol yu kɔlɔstrel wit di tritmɛnt we yu de gi yu ɔltɛm , i go fayn fɔ mek yu go to dɔktɔ we de mɛn yu bɔdi.
• Lipidɔlɔjis dɛn go luk ɔltin frɔm yu famili mɛdikal istri to yu layf ɛn mek wan tritmɛnt plan we go fayn fɔ yu .
• Ay kɔlɔstrel kin bi ɛnimi we nɔ de tɔk natin. So, i rili impɔtant fɔ mek dɛn tɛst yu blɔd ɛn fala wetin yu dɔktɔ tɛl yu fɔ du.
• If yu gɛt ɛni kwɛstyɔn, nɔ fred fɔ aks dɔktɔ. Yu na di wan we fɔ bisin mɔ bɔt yu wɛlbɔdi biznɛs.

So, nɔ fred di kɔlɔstrel, tray fɔ manej am fayn . Lipidɔlɔjis kin bi di bɛst pɔsin fɔ ɛp yu pan da joyn de.

` Kɔlɔstrel, Lipidɔlɔjis, Fat, At sik, LDL Kɔlestɔl, Lipidɔlɔjis, Kɔlestɔl, Lipid dizayd