Wetin na di A1C tɛst we de tɛl yu bɔt yu blɔd shuga lɛvɛl? Lɛ wi tɔk bɔt am!

Yu go mɔs dɔn yɛri dɔktɔ dɛn se, "Yu nid fɔ du A1C tɛst." Ɔ if pɔsin na yu famili gɛt dayabitis, yu go dɔn yɛri bɔt dis A1C tɛst. Wetin na dis A1C tɛst rili? Wetin wi kin lan frɔm am? Tide, wi go tɔk bɔt dis A1C tɛst insay wan simpul we we yu go ɔndastand.

Wetin na di A1C tɛst?

Fɔ tɔk am simpul wan, di A1C tɛst na tɛst we de mɛzhɔ yu blɔd shuga, ɔ ``Glucose'', lɛvɛl fɔ di pas tri mɔnt. Dɛn gi di rizɔlt dɛn as pasɛnt. Di mɔ di pasɛnt we yu gɛt, na di mɔ yu blɔd shuga go bɔku.

Dɔktɔ dɛn kin yuz di A1C tɛst fɔ sɔm men rizin dɛn:

• Identify di stej bifo dayabitis (`(Prediabetes)`). Dat min se, wan stej we de nia fɔ gɛt dayabitis.
• Fɔ no if yu gɛt tayp 2 dayabitis.
• If yu gɛt tayp 2 dayabitis ɔ tayp 1 dayabitis, i impɔtant fɔ ɔndastand aw yu tritmɛnt plan de wok fayn fayn wan. Yu A1C lɛvɛl kin ɛp yu ɛn yu dɔktɔ fɔ disayd if yu nid fɔ chenj yu tritmɛnt plan.

Aw dɛn kin du di A1C tɛst?

Dɔktɔ dɛn kin tɛst A1C insay tu we dɛn:

• Bɔku tɛm, dɛn kin pul smɔl blɔd frɔm wan vein ɛn sɛn am na lɛb fɔ mek dɛn tɛst am. Dis na di we aw dɛn kin du am pas ɔl. dis dεn kכl am ``Phlebotomist'' we de pul bכdi.
• Di ɔda we na fɔ tek wan drop blɔd na yu finga ɛn tɛst am. Dis kin gi yu rizulyt insay sɔm minit. Bɔt, dɛn kin jɔs yuz dis we fɔ si aw yu tritmɛnt de go, ɛn dɛn nɔ kin yuz am fɔ no if yu gɛt nyu sik.

Test we yu de yuz vein kin kɔrɛkt pas finga prik tɛst.

Wetin na ɔda nem fɔ di A1C tɛst?

Dɛn kin kɔl dis A1C tɛst wit sɔm ɔda nem dɛn. Yu kin yɛri dɛn nem dɛn ya bak:

• Ɛmoglobin A1C `(Hɛmoglobin A1C)`
• HbA1c `(HbA1c)`
• Ɛmoglobin we gɛt glayket
• Glycohemoglobin test `(Glycohemoglobin test)`

Wetin na di blɔd shuga (Glucose)?

wi bכdi de gεt dis `(Glucose)` כ shuga mεntal wan frכm di kabohaydret dεm we de insay di it dεm we wi de it. Dis na di men tin we de gi wi bɔdi in sɛl dɛn ɛnaji. wi bכdi de kכri dis `(Glucose)` to di sεl dεm we de כlsay na di bכdi.

Bɔku tin dɛn we kin apin na wi bɔdi kin ɛp fɔ mek di shuga we de na wi blɔd nɔ rich wan patikyula say. di mכst imכtant rol fכ dis na di כmon ``Insulin`` we wi pankrias de mek.

If yu blɔd shuga lɛvɛl de ɔp ɔltɛm (haypa glycemia), we min se yu A1C sɛf de ay, bɔku tɛm i kin min se yu gɛt dayabitis. Dayabitis kin apin we di pankrias nɔ de mek insulin, ɔ we di insulin we i de mek nɔ de du, ɔ we di bɔdi nɔ ebul fɔ yuz insulin fayn fayn wan.

Ustɛm a nid fɔ du A1C tɛst?

If yu gɛt dayabitis, yu fɔ gɛt A1C tɛst tu tɛm insay di ia fɔ si aw yu tritmɛnt plan de wok fayn. Yu dɔktɔ go tɛl yu ustɛm dɛn fɔ du dis.

If dɛn nɔr dɔn no se yu gɛt dayabitis, bɔt yu gɛt dɛn sik ya, dɔktɔ kin ɔda fɔ mek dɛn du A1C tɛst:

• Fɔ tɔsti pasmak ɔltɛm (`(Polydipsia)`)
• Fɔ pis pasmak
• Di vishɔn we nɔ klia
• Fɔ fil taya ɔltɛm (`(Fatigue)`)

Yu kin gɛt A1C tɛst bak fɔ si if yu de pan denja fɔ gɛt tayp 2 dayabitis. Di tin dɛn we kin mek pɔsin gɛt dis sik na:

• Fɔ gɛt pɔsin na yu famili we gɛt tayp 2 dayabitis.
• If yu bin dɔn gɛt gestational dayabitis bifo.
• Fat.
• Nɔ de du bɔku tin dɛn ɔ fɔ du ɛksɛsayz.
• Bikɔs i dɔn pas 35 ia.

Aw dɛn kin kɔl A1C?

Di A1C tɛst na sɔntin we dɛn kɔl ɛmoglobin . Ɛmoglobin na di pat pan wi rɛd blɔd sɛl dɛn we de kɛr ɔksijɛn go ɔlsay na di bɔdi. We glukɔs de na yu blɔd, i kin stik to dis ɛmoglobin. Dɛn kɔl dis glycation . Di mɔ glukɔs de na yu blɔd, na di mɔ dis stik de go ɔp. εn, dis stik kin las fכ lεk tri mכnt – we na bכt di avεrej layfspan fכ wan rεd bכdi sεl.

di A1C tεst de mכsu di avrej glukכs we de atak εmoglobin ova tεm. Bikɔs i de mɛzhɔ di blɔd shuga lɛvɛl as tɛm de go, di A1C tɛst kin tɛl wi mɔ bɔt yu wɛlbɔdi pas wan blɔd shuga tɛst.

A nid fɔ fast bifo di A1C tɛst?

Nɔ, yu nɔ nid fɔ fast bifo yu tek A1C tɛst.

Wetin kin apin we yu du A1C tɛst?

Na dis na wetin yu kin ɛkspɛkt frɔm A1C tɛst, we involv fɔ tek blɔd frɔm wan vein:

• We yu sidɔm na chia, phlebotomist (pɔsin we de pul blɔd) go luk fɔ wan vein na yu an we izi fɔ pul blɔd frɔm. Bɔku tɛm, dɛn kin pul di blɔd frɔm insay yu an, jɔs biɛn yu ɛlb.
• We dɛn dɔn fɛn wan vein, dɛn kin klin di say ɛn disinfɛkt am.
• Dɔn dɛn kin put smɔl nidul insay di vein ɛn tek blɔd sɛmpul. Dis kin fil lɛk se yu prɛk am smɔl.
• Afta dɛn dɔn put di nidul, dɛn kin gɛda smɔl blɔd insay wan tɛst tyub.
• We dɛn dɔn gɛda inof blɔd fɔ di tɛst, dɛn kin pul di nidul ɛn stɔp di blɔd bay we dɛn put prɛshɔn pan di say wit kɔtɔn ɔ goz.
• Fɔ dɔn, put plasta pan di eria ɛn yu dɔn.

We dɛn de du A1C tɛst, we dɛn kin du bay we dɛn tek blɔd na wan finga, dɛn tin ya kin apin:

• Wan dɔktɔ aks yu us finga fɔ tek blɔd.
• yu finga dεm go wayp wit alkol εn dεn go mek sכm pankshכn wit wan sכmכl nidul we dεn kכl ``Lancet'' (dis kin bi insay sכmכl plastic divays).
• Dɛn go swɛt yu finga, pul wan drop blɔd, ɛn gɛda am insay wan smɔl plastic kɔntena.
• Afta dɛn dɔn tek inof blɔd fɔ di tɛst, dɛn go gi yu wan kɔtɔn ɔ goz fɔ ol yu finga fɔ stɔp di blɔd.
• Bɔku tɛm, dɛn kin gɛt di rizɔlt insay sɔm minit.

Ɛni risk de fɔ di A1C tɛst?

Blɔd tɛst na tin we rili kɔmɔn ɛn we nid fɔ du fɔ mek dɛn du mɛdikal tɛst. Di risk dɛm fɔ ɔl tu di kayn A1C tɛst dɛm rili smɔl. Yu kin gɛt sɔm brus ɔ pen usay dɛn pul di blɔd ɔ usay dɛn chuk yu finga, bɔt dis go wɛl kwik kwik wan.

Wetin mi A1C valyu min?

Dɛn gi di rizɔlt fɔ wan A1C tɛst as pasɛnt. dis nכmba de tεl yu aw bכku pan di εmoglobin protin de glycated, we min se i de atak glukכs. Di mɔ di pasɛnt, na di mɔ yu blɔd shuga dɔn de ɔp fɔ sɔm mɔnt dɛn we dɔn pas.

di A1C lεvεl dεm fכ no di sik na dεn wan ya:

• If i nɔ rich 5.7%, dat min se yu nɔ gɛt dayabitis. (Hɛlth lɛvɛl) .
• If i de bitwin 5.7% ɛn 6.4%, dɛn kin tek am se na prɛdiabɛtis. Dis min se dayabitis dɔn nia fɔ bigin.
• If yu gɛt 6.5% ɔ pas dat , i kin sho se yu gɛt tayp 2 dayabitis (`(Type 2 Diabetes)`) ɔ tayp 1 dayabitis (`(Type 1 Diabetes)`).

If yu dɔn gɛt dayabitis, yu A1C rizɔlt de sho aw yu tritmɛnt plan dɔn wok fayn fɔ di pas tri mɔnt. Dis tritmɛnt kin inklud pils, insulin injɛkshɔn, fɔ tɛst yu blɔd shuga ɔltɛm, ɛn fɔ chenj yu layf (lɛk aw yu de it ɛn ɛksesaiz). Bay yu A1C valyu, yu ɛn yu dɔktɔ kin disayd if ɛni chenj na yu tritmɛnt plan nid fɔ de.

Di impɔtant tin fɔ mɛmba na dat yu A1C (if yu gɛt dayabitis) na jɔs wan avɛrej pan yu blɔd shuga lɛvɛl fɔ sɔm mɔnt. I nɔr min sayn fɔ yu wɛl bɔdi ɔr di las tin fɔ disayd if yu de liv wɛl bɔdi layf. Mɛmba se yu A1C kin chenj ɔlsay na yu layf, ɛn tin dɛn de we yu kin du fɔ kɔntrol yu dayabitis ɛn fɔ mek yu A1C lɛvɛl bɛtɛ, if nid de.

Wetin na A1C ɛn ɛstimat nɔmal glukɔs (eAG)?

Sɔm lab dɛn kin gi A1C rizɔlt as pasɛnt, ɛn di kɔrɛspɔndɛnsdi εstimat avεrej glukכs (eAG) valyu dεn gi bak.

di `(eAG)` kכlkyushכn de kכnvכlt di A1C pasεnshכn to di sem yunit dεm (miligram pan wan dεsilita - mg/dL כ milimol pan wan lita - mmol/L) we yu de yuz wit yu os glukכs mita. Jɔs lɛk aw yu A1C na avɛrej, di `(eAG)` na wan valyu we de sho yu avɛrej blɔd shuga lɛvɛl fɔ di pas tri mɔnt.

fכ egzampl, wan A1C we na 7% ikwal to `(eAG)` 154 mg/dL (8.6 mmol/L). Wan A1C we na 9% ikwal to `(eAG)` 212 mg/dL (11.8 mmol/L).

Wetin na nɔmal A1C valyu?

Fɔ pɔsin we nɔ gɛt dayabitis, di nɔmal A1C valyu nɔ rich 5.7%.

Wetin dɛn kin tek as "nɔmal" ɛn wɛl bɔdi fɔ pipul dɛm wae gɛt shuga kin dipen pan yu gol dɛm ɛn yu akses to dayabitis mɛnejɛmɛnt mɛrɛsin ɛn ikwipmɛnt. Yu ɛn yu dɔktɔ go wok togɛda fɔ no yu target A1C rɛnj. Dis kin chenj ɔlsay na yu layf.

Jɛnɛral wan, di American Diabetes Association se bɔku pan di big pipul dɛn we gɛt dayabitis fɔ tray fɔ gɛt A1C we na 7% ɔ smɔl pas dat .

Yu A1C target kin pas 7% pan dɛn sityueshɔn ya:

• If di layf we pɔsin kin liv nɔ bɔku.
• If yu gɛt bɔku tɛm we yu gɛt siriɔs lɔw blɔd shuga (`(Hypoglycemia)`) ɔ yu nɔ no bɔt dɛn kayn episɔd ya (`(Hypoglycemia unawareness)`).
• If yu gɛt siriɔs prɔblɛm dɛn frɔm dayabitis (lɛk sik na yu kidni we nɔ de dɔn, yu nerve damej, ɔ yu at ɛn blɔd sik).

na di כda say, fכ uman dεm we gεt tayp 1 dayabεtis we dεn bεlε, dכkta dεn kin jεnarali rεkכmand fכ kip di A1C lεvεl na 6.5% כ lכw כl di bεlε. dis dεn de du fכ ridyus di hεlth risk to di pikin εn fכ mek di pikin nכ gro pasmak (fetal macrosomia).

Wetin na wan denja lɛvɛl fɔ A1C?

As yu A1C lɛvɛl de go ɔp, mɔ if i de ay fɔ bɔku ia, yu kin gɛt prɔblɛm dɛn lɛk:

• Di prɔblɛm dɛn we kin apin na di yay (`(Retinopathy)`) .
• Kidni prɔblɛm (`(Nɛfropati)`) .
• Nεv prכblεm (`(Nyuropati)`) .
• Bɛlɛ we nɔ de wok fayn (Gastroparesis) .
• At sik
• Strok

Risach dɔn sho se pipul dɛm wae gɛt shuga kin ridyus dɛn risk fɔ gɛt dayabitis kɔmplikeshɔn bay wae dɛn de kip A1C lɛvɛl dɔŋ 7% .

Bɔt mɛmba se ɔda tin dɛn lɛk di tin dɛn we yu gɛt frɔm yu jɛnɛtiks ɛn aw lɔng yu dɔn gɛt dayabitis, kin afɛkt bak aw yu gɛt prɔblɛm wit dayabitis.

Yu tink se di A1C tɛst rili kɔrɛkt?

Sɔm tin dɛn kin afɛkt di kɔrɛkt we aw dɛn de du A1C tɛst, lɛk:

• jεnεtik fכktכr, fכ egzampl, εmoglobin vεryכnt dεm
• Ɔda tin dɛn we kin apin to pɔsin we sik
• Sɔm mɛrɛsin dɛn ɛn tin dɛn we dɛn kin yuz fɔ it
• Mistek we dɛn kin mek we dɛn de gɛda, transpɔt, ɔ prosɛs di sɛmpul dɛn fɔ tɛst

Dɛn tin ya kin mek yu rizɔlt bi `fals low` ɔ `falsely high`. Bɔku pan dɛn tin ya kin apin bikɔs yu rɛd blɔd sɛl dɛn kin liv ɛn di wɛlbɔdi we kin chenj.

Hemoglobin Variant ɛn A1C Rizult

di hεmoglobin vεryכnt dεm kin afekt di risכlt fכ sכm A1C tεst dεm.

Di kayn ɛmoglobin we de na yu blɔd na wetin yu gɛt frɔm yu jin dɛn. Difrɛn kayn dɛn de. Di wan we kin kɔmɔn pas ɔl na ɛmoglobin A. Ɔda kayn ɛmoglobin we nɔ kin bɔku, dɛn kin kɔl dɛn ɛmoglobin we dɛn kin kɔl ɛmoglobin we dɛn kin yuz.

If yu gɛt wan kayn ɛmoglobin we nɔ de mek yu gɛt dayabitis, bɔt i kin afɛkt di A1C rizɔlt. Lab dεm gεt difrεn we fכ tεst A1C pan blכd wit wan hεmoglobin vεryכnt.

Sɔm pan di kayn dɛn we dɛn kin si mɔ na:

• Ɛmoglobin C trayt : Dis kin apin mɔ pan blak pipul dɛn, pipul dɛn we kɔmɔt na Wɛst Afrika, ɛn pipul dɛn we kɔmɔt na Sawt ɛn Sɛntral Amɛrika, di Karibian, ɛn Yurop.
• Hemoglobin D trait : Dis kin apin to pipul dɛm wae de liv na Chaena, India, Turkey, Brazil, ɛn sɔm pat dɛm na Yurop.
• Hemoglobin E trait : Dis kin apin mɔ pan pipul dɛn we kɔmɔt Eshia na Amɛrika, mɔ di wan dɛn we kɔmɔt na Sawt Is Eshia.
• Hemoglobin S trait : Dis kin apin mɔ pan blak pipul ɛn Hispanik Amɛrikin pipul dɛn.

We dɛn du blɔd tɛst, dɛn kin no di kayn ɛmoglobin we difrɛn. If yu tink se yu kin gɛt wan kayn ɛmoglobin we kin afɛkt yu A1C rizɔlt, tɔk to yu dɔktɔ.

Rizin dɛm wae mek yu A1C kin smɔl pas aw i rili bi

Dɛn kɔndishɔn ɛn sityueshɔn ya kin mek yu A1C rizɔlt smɔl pas yu rial lɛvɛl:

• Yuz rɔm pasmak (`(Alcohol use disorder)`)
• Blɔd transfyushɔn
• Krכnik kidni fεil (`(Krכnik kidni fεil)`)
• Erythropoietin-stimulating ejen dεm (ESA dεm) .
• Blɔd we de kɔmɔt pasmak (`(Ɛmoraji)`) .
• Fɔ liv na ay ay ples
• Fɔ tek tin dɛn we dɛn kin yuz fɔ mek ayɛn
• Sirosis na di liva (`(Sirosis na di liva)`)
• We uman gɛt bɛlɛ
• Sikl sel anemia (`(Sikl sel anemia)`)
• Sfɛrosaytɔsis ɛn ɛmolaytik anemia (`(Sfɛrosaytɔsis ɛn ɛmolaytik anemia)`)

Rizin dεm we mek A1C valyu dεm kin hכy pas aw dεn rili bi

Dɛn kɔndishɔn ɛn sityueshɔn ya kin mek yu A1C rizɔlt ay pas aw i rili bi:

• Anemia, fɔ ɛgzampul, anemia we nɔ gɛt ayɔn, we pɔsin nɔ gɛt bɛtɛ blɔd we i gɛt infekshɔn, ɔ we i nɔ gɛt bɛtɛ blɔd we i gɛt kansa.
• Sɔm mɛrɛsin dɛn, lɛk mɛrɛsin dɛn we de mek pɔsin nɔ ebul fɔ fɛt di sik ɛn di wan dɛn we de mek pɔsin nɔ ebul fɔ fɛt di sik we dɛn kɔl protease inhibitor.
• di triglisεrayd dεm we de inkrεs na di bכdi (`(Hypertriglyceridemia)`)
• Fɔ transplant ɔgan dɛn
• Talasɛmia (`(Talasɛmia)`) .
• Vitamin B12 we nɔ de (`(Vitamin B12 we nɔ de)`)

Yu tink se dɛn kin mɛn dayabitis ivin if di A1C lɛvɛl bɔku?

Yɛs, yu kin ebul. Jɔs bikɔs yu gɛt ay A1C (above 6.5%) nɔ min se yu gɛt dayabitis. Sɔm mɛrɛsin dɛn lɛk stɛroyd, ɔ ɔda tin dɛn lɛk sik kin mek yu blɔd shuga go ɔp fɔ sɔm tɛm. Ɔda tin dɛm lɛk anemia kin mek yu A1C rizɔlt tan lɛk se i ay pas aw i rili bi. Chans de bak fɔ mek di tɛst nɔ kɔrɛkt.

Dɔktɔ dɛn kin yuz pas wan tɛst fɔ no if pɔsin gɛt dayabitis. Fɔ ɛgzampul, dɛn kin aks yu fɔ du fast blɔd glukɔs tɛst ɔ A1C tɛst. Bɔt yu dɔktɔ go tek tɛm luk yu rizɔlt ɛn tɔk bɔt am wit yu.

A1C de tɛl yu kɔrɛkt wan bɔt aw fɔ mɛn sik dɛn we gɛt dayabitis?

Fɔ lɔng tɛm naw, dɔktɔ ɛn pipul dɛn we gɛt dayabitis dɔn de yuz di A1C tɛst mɔ fɔ no aw dɛn tritmɛnt de wok fayn.

Bɔt mɔ fɔ pipul dɛn we gɛt tayp 1 dayabitis, di blɔd shuga kin chenj bad bad wan fɔ sɔm dez, wik, ɛn mɔnt. So, A1C de bays pan wan min ɛn nɔto ɔltɛm i de gi kɔrɛkt pikchɔ bɔt di manɛjmɛnt.

Fɔ ɛgzampul, tink bɔt dis: Sɔmbɔdi in blɔd shuga kin chenj ɔltɛm, bɔt in A1C kin bi 7%. כda pכsin in bכdi shuga lεvεl de kכnsistεnt arawnd 154 mg/dL, εn dεn A1C kin bi 7% bak.

Dis biɛn tɛm, wit di introdɔkshɔn fɔ kɔntinyu glukɔs monitarin (CGM) divays dɛm , dɔktɔ dɛm ɛn pipul dɛm wae gɛt dayabitis dɔn si se Time in Range (TIR) ​​de gi yu wan mɔr yusful ɛn kɔrɛkt pikchɔ bɔt dayabitis mɛnejɛmɛnt.

di TIR na di tεm we yu bכdi shuga lεvεl de insay di tεm we dεn rεkomεnd. Dɛn kin mɛzhɔ di TIR as pasɛnt. di blכd shuga tכgεt rεnj kin difrεn frכm pכsin to pכsin, bכt wan tכpik tכgεt rεnj de bitwin 70 εn 180 mg/dL. Fɔ bɔku big pipul dɛn we gɛt tayp 1 ɔ tayp 2 dayabitis, dɔktɔ dɛn kin se dɛn fɔ tray fɔ kip di TIR pas 70% (lɛk 17 awa pan 24 awa de).

Wit `(CGM)` ɛn `(TIR)`, dɔktɔ ɛn pipul dɛm wae gɛt shuga kin si aw ɔltɛm dɛn blɔd shuga lɛvɛl kin ay ɔr smɔl. Dis kin ɛp dɛn fɔ mek dɛn tritmɛnt rijim dɛn kɔrɛkt wan.

Fɔ dɔn, yu fɔ se...

I kin mek yu at pwɛl we yu A1C rizɔlt nɔr de lɛk aw yu bin de ɛkspɛkt ɔr pas aw yu bin de ɛkspɛkt. Bɔt mɛmba se, if yu gɛt bɔku A1C, dat nɔ min se yu gɛt dayabitis. Bɔku tin dɛn de we kin mek di rizɔlt nɔ kɔrɛkt. Yu dɔktɔ go tɛl yu if yu nid fɔ du mɔ tɛst. Nɔ fred fɔ aks yu dɔktɔ kwɛstyɔn dɛn. Dɛn de de fɔ ɛp yu.

If yu gɛt dayabitis, i impɔtant fɔ mɛmba se yu A1C na jɔs wan tɛmporari avɛrej pan yu blɔd shuga lɛvɛl. Tin dɛn de we yu kin du fɔ mek yu A1C lɛvɛl bɛtɛ. If yu de fil bad fɔ kɔntrol yu dayabitis, tɔk to yu dɔktɔ. Tugeda, una kin mek wan plan fɔ manej yu dayabitis ɛn rich yu A1C gol dɛm.

` A1C, HbA1c, dayabitis, blɔd shuga, glukɔs, ɛmoglobin, dayabitis tɛst