Yu gɛt ay blɔd shuga na mɔnin, ilɛksɛf yu gɛt dayabitis? Mek wi lan bɔt dis ‘Somogyi ifɛkt’?

Yu na pɔsin we de tek insulin fɔ Dayabitis Mɛlitus? If na so, yu dɔn notis se yu blɔd shuga dɔn go ɔp wantɛm wantɛm we yu wek na mɔnin? Ivin if yu tek yu mɛrɛsin na nɛt ɛn tek tɛm, i kin sɔprayz fɔ no se yu stil gɛt am na mɔnin? Wan rizin fɔ dat kin bi di ‘Somogyi ifɛkt’. Lɛ wi tɔk bɔt dis smɔl mɔ .

Wetin na di Somogyi ifɛkt?

Fɔ tɔk am simpul wan, di Somogyi ifɛkt na we pɔsin we gɛt dayabitis we de tek insulin gɛt di blɔd shuga lɛvɛl we de go dɔŋ wantɛm wantɛm (haypoglycemia) na nɛt, dɔn di blɔd shuga lɛvɛl go ɔp wantɛm wantɛm (haypa glycemia) na mɔnin. Dis kin apin bikɔs sɔm ɔmon dɛn we de na wi bɔdi de wok. Imajin, na midul nɛt, yu blɔd shuga kin drɔp wantɛm wantɛm, ɛn yu bɔdi kin tink se, "O, dis man go gɛt prɔblɛm," ɛn i kin pul ɔmon fɔ sev am. Na dat mek yu blɔd shuga de go ɔp na mɔnin.

Dɛn gi am in nem to Dɔktɔ Maykɛl Somogyi, we na dɔktɔ we bin fɔs fɛn am insay di ia 1930. Sɔntɛnde dɔktɔ dɛn kin kɔl am ‘ribound hyperglycemia’.

Bɔt na sɔntin we yu fɔ no. Di Somogyi ifekt stil na ‘tiori’ bitwin di wan dɛm we de du risach, we min se na jɔs wan opinion. Dat min se i nɔ pruv 100%, bɔt sayɛns pruf de fɔ sɔpɔt am. Trade, dɔktɔ dɛn bin de tink se na dis na di men rizin we mek di shuga de bɔku na mɔnin. Bɔt nyu risach, mɔ di risach we dɛn de du we dɛn de yuz kɔntinyu glukɔs monitarin (CGM), dɔn mek pipul dɛn aks kwɛstyɔn bɔt dis aidia.

Bɔku ɔda rizin dɛn de we mek di blɔd shuga lɛvɛl kin go ɔp na mɔnin, nɔto jɔs di Somogyi ifɛkt:

• Di kayn mɛrɛsin we yu de tek nɔ go du, bikɔs i de kɔmɔt na yu bɔdi na nɛt.
• Wan tin we dɛn kɔl di Dawn phenomenon. (Wi go tok abaut dis tu)
• Di kayn mɛrɛsin we dɛn bin de tek we dɛn de it dina nɔ bin mach di mɔni we dɛn bin de it.
• Fɔ gɛt insulin rɛsistɛns.

Wetin na di difrɛns bitwin di Somogyi ifɛkt ɛn di Dawn fenomen?

Okay, naw yu go de wɔnda, ‘Wetin na dis dawn fenomen?’ Dɛn tu tin ya na tin dɛn we kin mek di blɔd shuga go ɔp na mɔnin pan pipul dɛn we gɛt dayabitis.

di dכn fεnomen na di nכ mal inkris na di bכdi shuga lεvεl we de kכz fכ di rilis fכ sεvεra כmon dεm (lεk kכtisol εn growth כmon) na di εli mכnin. Dis na nɔmal tin.

Bɔt insay di Somogyi ifɛkt, di ɔmon dɛnsɛf de wok, bɔt i kin apin bikɔs di shuga kin drɔp wantɛm wantɛm (haypoglycemia) na nɛt. Insay di dawn fenomen, shuga nɔ de go ɔp bikɔs ɔf dis kayn drɔp na di shuga.

Wan ɔda impɔtant tin na dat di dawn fenomen, pan di tin dɛn we kin mek di blɔd shuga go ɔp na mɔnin , kin bɔku pas di Somogyi ifɛkt.

Wetin na di sayn dɛm fɔ di Somogyi ifɛkt?

Di men sayn fɔ di Somogyi ifɛkt na dat yu blɔd shuga lɛvɛl de go ɔp we yu wek na mɔnin. If yu luk yu glukomita ɔ if yu yuz CGM (Continuous Glucose Monitoring) divays, i go sho se yu shuga lɛvɛl bɔku na mɔnin.

Dipen pan aw yu blɔd shuga ay, yu kin gɛt sɔm kayn sik dɛn lɛk dis we yu wek na mɔnin:

• Fɔ tɔsti ɔltɛm (polydipsia).
• Fɔ fil angri pas aw i kin fil.
• Fɔ pis bɔku tɛm .
• Ed de at.
• Fɔ fil bad ɛn vɛks (irritability).

Wetin mek di Somogyi ifɛkt kin apin?

akɔdin to di tiori bɔt di Somogyi ifɛkt, if yu blɔd shuga lɛvɛl drɔp tumɔs (haypoglycemia) na di midul nɛt we yu de slip bikɔs ɔf di insulin we yu de tek, yu bɔdi go tray fɔ ‘sev’ yu frɔm da lɔw shuga stet de.

di bכdi de rilis sεvεra כmon dεm fכ dis ‘rεskyu mishכn’. Di men wan dɛn na:

• Adrenaline we gɛt di sik
• Kɔtikosterɔyd dɛn
• Ɔmon we de mek pɔsin gro
• Glukagɔn we dɛn kɔl Glucagon

Dɛn ɔmon dɛn ya kin mek yu liva pul mɔ glukɔs ɔ shuga pas aw i kin kɔmɔt na yu blɔd. Dis we aw di glukɔs de kɔmɔt de mek di shuga na yu blɔd go ɔp. If yu gɛt dayabitis, yu pankrias nɔ de mek insulin fɔ kɔntrol dis shuga lɛvɛl we de go ɔp, ɔ i nɔ de mek ɛni wan atɔl. Dis na di rizin we mek yu blɔd shuga de kɔntinyu fɔ bɔku.

Insay pɔsin we nɔ gɛt dayabitis, di bɔdi kin kɔntrol di blɔd shuga ɛn i kin mek i nɔ gɛt wan patikyula say. Na dat mek di Somogyi ifɛkt nɔ de afɛkt pipul dɛn we nɔ gɛt dayabitis.

Aw yu go fɛn dɔktɔ lɛk dis?

Fɔ tru, di Somogyi ifɛkt nɔto rili kɔmɔn tin we kin mek di blɔd shuga go ɔp na mɔnin, so i kin at fɔ no smɔl. Ɛn, lɛk aw a bin dɔn tɔk, di las risach we dɛn dɔn du sho se sɔntɛm nɔto dat kin mek di blɔd shuga go ɔp na mɔnin. So i kin tranga, if nɔto i nɔ pɔsibul, fɔ mek dɔktɔ no di sik kɔrɛkt wan bay we i se, ‘Okay, na dis.’

If bɔku tɛm yu gɛt ay blɔd shuga na mɔnin, i go fayn fɔ tɔk to yu dɔktɔ we de ɛp yu fɔ kɔntrol yu dayabitis. I go luk aw yu blɔd shuga de chenj ɛn tray fɔ fɛn di tin we de mek yu gɛt am. Di we we go wok pas ɔl na fɔ yuz wan CGM (Continuous Glucose Monitoring) divays.

If yu nɔ de yuz CGM, yu dɔktɔ go tɛl yu fɔ chɛk yu blɔd shuga bɔku tɛm insay di de. Fɔ ɛgzampul:

• Tu awa afta dɛn dɔn it ivintɛm it.
• Bifo yu go slip.
• Na midul nɛt (fɔ ɛgzampul, arawnd 2 ɔ 3 oklɔk mɔnin).
• We yu wek na mɔnin.

Dɔn bak, dɛn kin tɛl yu fɔ rayt tin dɛn lɛk us it dɛn yu de it na nɛt, ɔmɔs yu de it, ɛn if yu de du ɛksɛsayz na nɛt, us kayn ɛksesaiz yu de du ɛn fɔ aw lɔng yu de du.

Na we di dɔktɔ gɛda ɔl dis infɔmeshɔn nɔmɔ, i go ebul fɔ no wetin mek di blɔd shuga bɔku na mɔnin.

Aw dɛn kin trit di Somogyi ifɛkt?

Afta yu ɛn yu dɔktɔ dɔn wok togɛda fɔ no aw yu blɔd shuga lɛvɛl de biev na nɛt, dɛn go advays yu bɔt chenj dɛn we yu kin mek fɔ ɛp fɔ mek yu blɔd shuga nɔ go ɔp na mɔnin. Mɛmba se ɔlman in bɔdi difrɛn, ɛn bɔku tin dɛn de we kin afɛkt di blɔd shuga. So, wetin yu kin du fɔ mek yu nɔ gɛt mɔnin blɔd shuga kin difrɛn frɔm wan pɔsin to ɔda pɔsin.

Yu dɔktɔ kin tɛl yu tin dɛn lɛk:

• Fɔ chenj di doz fɔ yu dayabitis mɛrɛsin ɔ insulin.
• Fɔ chenj di kayn it dɛn we yu de it fɔ dina. Fɔ ɛgzampul, yu go nid fɔ it tin dɛn we gɛt bɔku prɔtin, kabɔhaydrɛt, ɔ fat, ɔ yu go nid fɔ it smɔl pan dɛn.
• If i pɔsibul, if yu chenj to insulin pɔmp, dat go mek yu ebul fɔ kɔntrol di insulin we yu de tek mɔ kɔrɛkt wan.
• Fɔ chenj di tɛm fɔ ɛksesaiz na nɛt.

Us prɔblɛm dɛn dis kin mek?

Ilɛksɛf di Somogyi ifɛkt na in rili de mek di blɔd shuga go ɔp na mɔnin ɔ nɔto so, i de sho tu impɔtant tin dɛn. Pipul wae gɛt dayabitis fɔ tray fɔ avɔyd dɛn tu prɔblɛm ya:

• Kɔntinyu fɔ mek di blɔd shuga go ɔp.
• Di blɔd shuga lɛvɛl we smɔl (haypoglycemia) kin apin na nɛt.

Kɔntinyu fɔ inkrisayz di blɔd shuga lɛvɛl

As yu blɔd shuga lɛvɛl de kɔntinyu fɔ bɔku, yu HbA1c (A1C) lɛvɛl go go ɔp, ɛn yu Time in Range (TIR) ​​go go dɔŋ. Di HbA1c tɛst de mɛzhɔ yu avɛrej blɔd shuga lɛvɛl fɔ di pas tri mɔnt. TIR na di avrej pasɛnt de we yu blɔd shuga lɛvɛl de insay di target rɛnj.

Di mɔ yu A1c ɛn TIR lɛvɛl de wɔs, mɔ if dɛn dɔn de de fɔ lɔng tɛm, na di mɔ yu risk fɔ gɛt kɔmplikeshɔn frɔm dayabitis. Sɔm ɛgzampul dɛn bɔt dɛn prɔblɛm ya na:

• Di prɔblɛm dɛn we kin apin to pɔsin in yay (Retinopathy) .
• Prɔblɛm dɛn na di kidni (Nephropathy) .
• Nyuropati we pɔsin kin gɛt
• At sik

Risach dɔn sho se pipul dɛm wae gɛt shuga kin ridyus dɛn risk fɔ gɛt dɛn prɔblɛm ya if dɛn kip dɛn A1c lɛvɛl dɔŋ 7%.

Di shuga we yu gɛt na yu blɔd na mɔnin kin bi prɔblɛm we nɔ de dɔn, ɛn if yu nɔ trit yu, yu blɔd shuga kin kɔntinyu fɔ bɔku fɔ sɔm awa ɛvride. Dis kin mek yu gɛt mɔ prɔblɛm wit dayabitis as tɛm de go.

Wan nɛt we di blɔd shuga kin shɔt

Fɔ bɔku pipul dɛn we gɛt dayabitis, di blɔd shuga we nɔ bɔku, ɔ we di blɔd shuga go dɔŋ pas 70 mg/dL (miligram pan wan dɛsilita).

We di shuga na di blɔd go dɔŋ, bɔku pipul dɛn kin gɛt sayn dɛn lɛk dis:

• Fɔ fil lɛk se yu bɔdi de shek.
• Fɔ fil se yu rili angri (polyphagia).
• Swet ɛn fil kol.
• Diziz, filin fɔ layt ed.
• Di at rit de go ɔp.

Bɔt if yu blɔd shuga go dɔŋ we yu de slip, i kin tek sɔm tɛm fɔ mek yu wek ɛn notis di sayn dɛn. Dis kin denja, ɛn if yu gɛt siriɔs ay blɔd blɔd, dat kin mek yu layf de pan denja. Di sayn dɛm we de sho se yu blɔd shuga nɔ bɔku na nɛt kin difrɛn smɔl, lɛk:

• Inshɔmnia, fɔ fil bad fɔ slip.
• Fɔ fil taya ɛn nɔ de pe atɛnshɔn we yu wek na mɔnin.
• Fɔ gɛt bad drim dɛn.
• Di klos ɔ bed we yu de wɛr kin wet bikɔs yu de swet.

I nɔ mata us sik yu gɛt we de mek yu tink se yu gɛt smɔl shuga na yu blɔd, i impɔtant fɔ chɛk yu blɔd shuga lɛvɛl fɔ si aw i smɔl. Dis go ɛp yu fɔ disayd aw fɔ trit am. Fɔ trit haypoglycemia, yu fɔ it fast-akting carbohydrates, we na shuga (lɛk banana ɔ apul jus).

If bɔku tɛm yu blɔd shuga nɔ bɔku na nɛt, mek shɔ se yu tɔk to yu dɔktɔ bɔt dat. Tugeda, una kin ajɔst yu dayabitis mɛnejɛmɛnt plan fɔ mek i nɔ apin igen.

Ustɛm a fɔ go to dɔktɔ?

If yu de gɛt ay shuga na yu blɔd ɔltɛm na mɔnin, i impɔtant fɔ tɔk to yu dɔktɔ we de ɛp yu fɔ kɔntrol yu dayabitis. I kin tɛst yu blɔd shuga lɛvɛl, no if na di Somogyi ifɛkt na di kɔz, ɔ if ɔda tin de apin, ɛn rikomɛnd tritmɛnt plan we rayt fɔ yu.

If yu gɛt bɔku blɔd shuga na mɔnin, dat kin rili mek yu vɛks ɛn yu nɔ kin fil fayn. No wan-sayz-fit-ɔl sɔlv nɔ de fɔ dis. So i kin tek sɔm tɛm fɔ fɛn di rayt sɔlv fɔ yu. Yu dɔktɔ kin ɛp yu fɔ disayd aw fɔ du di bɛst tin. Nɔ fred fɔ aks fɔ dɛn ɛp. If yu tek akshɔn naw, dat kin ɛp yu fɔ avɔyd prɔblɛm dɛn we kin apin fɔ shɔt tɛm ɛn fɔ lɔng tɛm.

Fɔ dɔn, di tin we impɔtant pas ɔl (Take-Home Message) .

Okay, so naw yu don geht beta andastan di Somogyi effekt we wi bin tok boht. Mɛmba se nɔto dis nɔmɔ de mek di shuga bɔku na mɔnin. Bɔt, i impɔtant fɔ no bɔt am.

• Di men tin na dat, if yu de gɛt ay blɔd shuga ɔltɛm na mɔnin, nɔ ignore am. Mek shɔ se yu tɔk to yu dɔktɔ.
• If yu de yuz CGM (Continuous Glucose Monitoring), aks yu dɔktɔ fɔ ɛp yu fɔ analayz di data fayn fayn wan.
• Lɔw blɔd shuga (haypoglycemia) na nɛt kin rili denja. If yu gɛt ɛni wan pan dɛn sik ya, tɛl yu dɔktɔ bɔt dɛn bak.
• Tɔk bɔt aw yu de it, ɛksesaiz, ɛn aw yu fɔ tek mɛrɛsin wit yu dɔktɔ ɛn ajɔst dɛn to wetin go wok fayn fɔ yu.

Fɔ liv wit dayabitis na prɔblɛm, bɔt if yu de mɛn yu fayn fayn wan, yu kin liv fayn layf. No fred, okay?

` Somogyi ifekt, dayabitis, mɔnin shuga spayk, haypoglycemia, haypa glycemia, insulin