Yu gɛt dayabitis? Ɔ yu dɔktɔ dɔn tɛl yu fɔ tek wan tɛst we dɛn kɔl C-Peptide? We yu yɛri dis nem, yu go fred smɔl ɛn yu go want fɔ no mɔ. "Wetin bi dis nyu test? Wetin i de luk fo?" Sɔntɛm yu de tink. Bɔt nɔ wɔri atɔl. Dis na wan rili simpul tɛst wae de gi wi bɔrku impɔtant infɔmeshɔn fɔ mɛn yu shuga. Tide, wi go tɔk bɔt dis C-Peptide tɛst ditayli, insay wan we we yu go ɔndastand rili simpul wan.
Fɔ tɔk am simpul wan, wetin na C-peptide?
Fɔ ɔndastand dis, lɛ wi tink bɔt am dis we. Di pankrias we de na wi bɔdi tan lɛk smɔl faktri. Di men tin we dis faktri de mek na wan ɔmon we dɛn kɔl insulin . Insulin na wan rili impɔtant ɔmon we de kɔntrol di shuga (glucose) we de na wi blɔd ɛn ɛp wi bɔdi in sɛl dɛn fɔ yuz da shuga de as ɛnaji.
Naw, we di pankrias de mek dis insulin, i de pul wan smɔl pat bak we tay pan am. Na da pat de wi kɔl C-peptide . fכ bi prεsis, wan insulin mכlikul de fכm frכm wan big mכlikul we dεn kכl ‘proinsulin’. we di insulin pat separet frכm dis εn kכmכt insay di bכdi, di rεst C-pεptida pat de kכmכt bak insay di bכdi.
di imכtant tin na dat εvri tεm we wan insulin mכlikul kכmכt frכm di pankrias, wan ikwal amoun fכ C-pεptida de kכmכt bak. Dat min se if wi mɛzhɔ di C-peptide we de na pɔsin in blɔd, wi go ebul fɔ no ɔmɔs insulin dɛn yon bɔdi, we na dɛn yon pankrias, de mek. Dis na di valyu we pas ɔl fɔ dis tɛst.
Impɔtant: Wan C-peptide tɛst nɔ kin ebul fɔ no if pɔsin gɛt dayabitis. Dɛn kin no if yu gɛt dayabitis bay we yu du blɔd glukɔs tɛst ɔ HbA1c tɛst. Dɛn kin yuz C-peptide fɔ gi mɔ ditayli infɔmeshɔn bɔt di kɔndishɔn fɔ pɔsin we dɔn ɔlrɛdi gɛt dayabitis.
Wetin mek dɔktɔ go tɛl yu fɔ du dis tɛst?
Bɔku rizin dɛn de we mek yu dɔktɔ go tɛl yu fɔ du dis tɛst. I tan lɛk impɔtant pruf fɔ ditektiv we de chɛk wan sik.
- Fɔ no di kayn shuga we yu gɛt: Tu men kayn dayabitis de, Tayp 1 dayabitis ɛn Tayp 2 dayabitis . Di tu kayn dɛn kin wok difrɛn we na di bɔdi.
- Insay Tayp 1 dayabitis , di bɔdi in imyun sistɛm kin pwɛl di sɛl dɛn we de mek insulin na di pankrias. Dis kin mek di bɔdi nɔ kin mek insulin. So, if yu luk di C-peptide lεvεl fכ dis kayn pכsin, i go rili lכw.Ɔ i kin bi ziro.
- Insay Tayp 2 dayabitis , pan ɔl we di pankrias kin mek insulin fayn fayn wan fɔs, di bɔdi in sɛl dɛn nɔ kin ansa fayn to da insulin de (insulin resistance). Dɔn, di pankrias de mek mɔ insulin fɔ kɔmpɛns di tin we nɔ de. So, we pɔsin we gɛt Tayp 2 dayabitis bigin, di C-peptide lɛvɛl kin nɔmal ɔ i kin ay .
- Mekɔp aw di tritmɛnt go wok fayn: If yu de tek mɛrɛsin fɔ dayabitis, dis tɛst kin chɛk if dɛn mɛrɛsin dɛn de de mek yu pankrias wok ɛn mek insulin fayn fayn wan.
- Fɔ no if yu nid fɔ tek insulin injɛkshɔn: Ɛspɛshali if yu gɛt Tayp 2 dayabitis fɔ lɔng tɛm, yu pankrias kin wik smɔl smɔl ɛn stɔp fɔ mek insulin. da tεm de, yu C-pεptida lεvεl dεm go dכn sכmtεm. Na da tɛm de yu dɔktɔ go disayd se na tɛm fɔ mek yu bigin fɔ tek insulin injɛkshɔn frɔm ɔdasay.
- Difrɛns bitwin insulin we di bɔdi de mek ɛn insulin we dɛn injɛkt: Dis na impɔtant pɔynt. Imajin se yu na pɔsin we de yuz insulin injɛkshɔn. If wi luk di insulin lɛvɛl na yu blɔd, i de sho ɔl tu di insulin we yu bɔdi de mek ɛn di insulin we yu injɛkt. Bɔt C-pɛptida nɔ tan lɛk dat. εksojen insulin nכ gεt C-pεptida. So, we yu luk di C-peptide lεvεl, yu kin no kכrekt כl di insulin we yu bכdi stil de prodyuz, ivin we yu de tek injεkshכn.
- Fɛn di tin we kin mek yu blɔd shuga nɔ bɔku (haypoglycemia): Sɔm pipul dɛn kin gɛt di blɔd shuga we kin denja. Dis tɛst kin ɛp fɔ no if di tin we kin mek pɔsin gɛt insulin na di bɔdi we pasmak ɔ ɔda tin we kin mek i apin.
Aw fɔ tek di tɛst ɛn pripia
Yu nɔ nid fɔ fred fɔ tek dis tɛst. Na wan rili simpul prɔses.
Aw yu kin du di tɛst?
Bɔku tɛm, dis kin min fɔ tek blɔd sɛmpul . Jɔs lɛk aw yu kin du blɔd tɛst ɔltɛm, nɔs ɔ lab tɛknishian go tek smɔl blɔd frɔm wan vein na yu an. I go fil lɛk smɔl sting, lɛk se ant bit am, na dat nɔmɔ.
Na smɔl tɛm nɔmɔ dɛn kin du dis tɛst wit urine sample . If dɛn kayn tin ya apin, bɔku tɛm dɛn kin nid fɔ tek 24 awa urine sample. Yu dɔktɔ ɔ di lɛbɔretri go gi yu instrɔkshɔn ɛn kɔntena fɔ dis.
Yu nid fɔ rɛdi bifo di tɛst?
Yɛs, sɔntɛnde, dɛn kin nid fɔ pripia smɔl.
- Fast: Yu dɔktɔ kin aks yu fɔ lɛf fɔ it ɔ drink fɔ 8 to 12 awa bifo yu du di tɛst. Bɔt bɔku tɛm, dɛn nɔ kin ban fɔ drink wata.
- Pɔst-stimuleshɔn tɛst: .Ɔda tɛm, yu dɔktɔ kin want fɔ mek yu it sɔntin (sɔntin we gɛt shuga) ɛn chɛk yu C-peptide lɛvɛl afta yu pankrias dɔn stimulate. So ɔltɛm fala yu dɔktɔ in instrɔkshɔn dɛn fayn fayn wan.
- Mɛrɛsin: If yu de tek ɛni mɛrɛsin fɔ ɔda sik, mek shɔ se yu tɛl yu dɔktɔ bɔt am. Sɔm mɛrɛsin kin afɛkt di rizɔlt fɔ dis tɛst, so dɛn kin aks yu fɔ stɔp fɔ tek dɛn fɔ sɔm tɛm wan ɔ tu de bifo di tɛst.
No big risk nɔ de fɔ dis tɛst. Yu kin gɛt sɔm brus we dɛn pul di blɔd, bɔt dis go dɔn afta sɔm tɛm.
Wetin di tɛst rizɔlt dɛn se?
Nɔ fred di nɔmba dɛn we de na yu tɛst ripɔt. Na yu dɔktɔ nɔmɔ go ebul fɔ ɛksplen di rizɔlt kɔrɛkt wan, bikɔs dɛn nid fɔ kɔmpia am wit ɔda tin dɛn lɛk di shuga we yu gɛt na yu blɔd ɛn di mɛrɛsin dɛn we yu de tek.
dis na sכm tכpik C-pεptida lεvεl dεm. Bɔt mɛmba se dɛn valyu ya kin difrɛn smɔl frɔm wan lab to ɔda lab.
| Rizult lɛvɛl | Nɔmal rɛnj |
|---|---|
| Avrej valyu | 0.5 ng/mL to 2.0 ng/mL (nanogram/mililita) . |
If di C-pεptida lεvεl lכw (Lכw C-pεptida) .
if yu C-peptide lεvεl de dכn di nכmal rεnj, i min se yu pankrias de prodyuz bכku insulin כ nכ de prodyuz insulin.
Imajin se dɛn dɔn du tɛst we yu blɔd shuga lɛvɛl rili ay ɛn yu C-pɛptida lɛvɛl rili smɔl. Dat na strɔng sayn fɔ Tayp 1 dayabitis . Bikɔs we yu blɔd shuga bɔku, pankrias we gɛt wɛlbɔdi fɔ mek bɔku insulin. Bɔt if i nɔ du dat, i min se di pankrias nɔ de wok.
pan tap dat, כda kכndyushכn dεm we kin mek di C-pεptida lεvεl dεm lכw inklud:
- Tayp 2 dayabitis we dɔn de fɔ lɔng tɛm: We yu dɔn gɛt Tayp 2 dayabitis fɔ lɔng tɛm, di pankrias kin taya ɛn di insulin we dɛn kin mek nɔ kin bɔku.
- Addison’s disease: Na wan sik wae de mek di adrenal gland dɛm nɔr de wok fayn.
- Liva sik.
- Fɔ tek insulin injɛkshɔn: If yu tek insulin na do, dat kin mek di bɔdi nɔ gɛt insulin.
If di C-pεptida lεvεl hכy (High C-peptide) .
if yu C-peptide lεvεl hכy pas di nכmal rεnj, i min se yu pankrias de wok pasmak εn de prodyuz tu mכch insulin.
Di men rizin fɔ dis na di fɔs tɛm we pɔsin kin gɛt Tayp 2 dayabitis . Dis na we di bɔdi in sɛl dɛn nɔ de ansa to insulin (insulin resistance), ɛn di pankrias gɛt fɔ wok tu tɛm pas aw i fɔ mek insulin fɔ kɔmpɛns di tin we nɔ de. di sem tεm, di C-pεptida lεvεl dεm de inkrεs bak.
pan tap dat, כda kכndyushכn dεm we kin mek di C-pεptida lεvεl dεm hכy inklud:
- Insulin resistance: Dɛn kin si am bak pan prɛ-dayabɛt.
- Cushing syndrome: Na wan sik we di bɔdi de mek tumɔs pan di ɔmon we dɛn kɔl kɔtisol.
- di kidni dεm we de wok fayn: We di kidni dεm nכ ebul fכ pul di C-pεptida fayn fayn wan, in lεvεl dεm na di bכdi de go כp.
- Insulinoma: Na wan kayn tumbu we nɔ kin bɔku ɛn we kin mek insulin we pasmak.
- di potashכm lεvεl dεm na di bכdi de dכn (Hypokalemia).
Ilɛk wetin go apin, na yu dɔktɔ go gi yu di kɔrɛkt we fɔ ɛksplen am ɛn di nɛks tin dɛn we yu fɔ du. So mek shɔ se yu gɛt di ripɔt ɛn si am.
Mɛsej we dɛn kin kɛr go na os
- C-Peptide na wan tɛst we rili valyu we de mɛzhɔ ɔmɔs insulin yu bɔdi, yu yon pankrias, de mek.
- Dis tɛst kin rili ɛp dɔktɔ dɛn fɔ no kɔrɛkt wan if yu gɛt Tayp 1 ɔ Tayp 2 dayabitis.
- Dis infɔmeshɔn impɔtant fɔ no if yu dayabitis tritmɛnt de wok ɔ if na tɛm fɔ mek yu bigin fɔ tek insulin injɛkshɔn.
- Ivin if yu yuz insulin injɛkshɔn, dis tɛst kin no ɔmɔs insulin yu bɔdi de mek insay.
- Nɔ jomp to kɔnklushɔn bay di tɛst rizɔlt. Ɔltɛm tɔk to yu dɔktɔ bɔt dis ɛn fala in instrɔkshɔn dɛn di rayt we.
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