We yu luk pan blɔd tɛst, yu go mɔs pe atɛnshɔn tumɔs pan kɔlɔstrel, mɔ di bad kɔlɔstrel, LDL. Bikɔs wi ɔl no se we di LDL lɛvɛl go ɔp, di chans fɔ gɛt sik dɛn lɛk at atak ɛn strok kin bɔku. Bɔt di stori nɔ de dɔn de. Stɔdi dɔn sho se 50% pan di pipul dɛn we dɔn gɛt at atak nɔ bin gɛt ay LDL lɛvɛl. So, dɔktɔ dɛn kin yuz ɔda tɛst fɔ no di prɔblɛm kɔrɛkt wan. Na dat wi kin kɔl di C-Reactive Protein, ɔ CRP tɛst.
Wetin na CRP rili?
Fɔ tɔk am simpul wan, C-Reactive Protein (CRP) na wan protin we wi liva de mek. We inflamɛns de , dat na, swel we sɔm damej de mek, sɔmsay na yu bɔdi, dis CRP lɛvɛl de go ɔp na di blɔd.
Tink bɔt am dis we. Bad kɔlɔstrel (LDL) nɔ jɔs de stik na di wɔl dɛn na wi blɔd vesel, i de pwɛl dɛn bak. Dis damej na wetin wi kɔl inflammation. I tan lɛk smɔl wund. we di bכdi de tray fכ hεl dis damej, i de mek spεshal protin dεm we de wok lεk "εp tim." CRP na wan pan dɛn kayn prɔtin dɛn de. dat min se if inflameshn de sכmsay na di bכdi, wi kin si am frכm di CRP lεvεl. I tan lɛk se we wi si smok, wi kin tek am se faya de sɔmsay.
Wetin mek dɔktɔ go tɛl yu fɔ du CRP tɛst?
di CRP tεst de mכsu di כmכnt כf C-Reactive Protein na yu bכdi. Dis kin gi yu dɔktɔ gud aidia bɔt di lɛvɛl we di inflamɛns de na yu bɔdi.
Sɔm stɔdi dɛn dɔn sho se CRP na bɛtɛ prɛdiktɔ fɔ di risk fɔ di sik we dɛn kɔl kadiovaskular sik (CVD) pas LDL kɔlɔstrel. Bɔt, i nɔto patikyula tɛst fɔ gɛt at sik. I jɔs de sho if inflamɛns de na di bɔdi. Di inflamɛns kin bi bikɔs ɔf at sik ɔ ɔda tin.
Ɔda kes dɛn de we dɛn kin yuz dis tɛst:
- Autoimmune Diseases: Dɛn kin du dis tɛst bak fɔ pipul dɛm wae gɛt sik lɛk Lupus ɔr Rheumatoid Arthritis. Bikɔs dɛn sik ya kin mek bak di bɔdi inflamɛns. Dis tɛst kin ɛp bak fɔ si if di mɛrɛsin we dɛn gi kin ridyus da inflamɛns de. Bɔt dis tɛst nɔ kin no usay di inflamɛns de na di bɔdi.
- Infεkshכn: dεn kin du dis tεst if dεn saspek fכ wan sistεm infεkshכn lεk sepsis, fכngal infεkshכn, כ bon infεkshכn.
- Sɛkɔn at atak risk: Dɛn kin yuse am bak fɔ si wetin de risk fɔ gɛt ɔda at atak fɔ pɔrsin wae dɔn ɔlrɛdi gɛt hat atak.
- Ɔda sik dɛn:Dis kin ɛp bak fɔ no bɔt sik dɛn lɛk inflammatory bowel disease ɛn lung disease.
Aw fɔ ɔndastand di valyu dɛn we de na di CRP ripɔt?
Yu dɔktɔ kin ɔda dis CRP tɛst if yu gɛt sayn dɛn fɔ infekshɔn, lɛk fiva, kol, at bit kwik kwik wan, yu de blo mɔ ɛn mɔ, yu nɔs, ɛn yu de vɔmit.
Dis na wan rili simpul blɔd tɛst. Bɔku tɛm, dɛn kin pul smɔl blɔd frɔm wan vein we de na yu an. I nɔ nid ɛni spɛshal pripiamɛnt (lɛk fɔ fast). Nɔ pen nɔ de pas smɔl say we de at usay dɛn put di nidul. Bɔt sɔm mɛrɛsin dɛn we yu de tek kin afɛkt di rizɔlt fɔ dis tɛst. So mek shɔ se yu tɛl yu dɔktɔ bɔt ɔl di mɛrɛsin dɛn we yu de tek.
We yu dɔn gɛt yu rizɔlt, di tebul we de dɔŋ ya go ɛp yu fɔ ɔndastand di valyu dɛn. Bɔt mɛmba se dɛn valyu ya kin difrɛn smɔl frɔm wan lab to ɔda lab. So mek shɔ se yu sho yu ripɔt to yu dɔktɔ ɛn gɛt advays.
| Inflameshɔn Lɛvɛl | Di valyu fɔ CRP (mg/dL) . | Posisibul minin/rizin dɛn |
|---|---|---|
| Mɔdaret Ɛleveshɔn | 1,0 - 10,0 mg/dL | Ɔtoimyun sik dɛm lɛk rεumatoid at, lupus, hat atak, pankrias, brɔnkayt. |
| Mak Ɛlevɛshɔn | 10,0 - 50,0 mg/dL | Akyu baktri ɔ vayral infɛkshɔn, inflamɛns na di blɔd vesel (Systemic Vasculitis), siriɔs aksidɛnt ɔ injuri. |
| Sivɛri Ɛleveshɔn | I pas 50.0 mg/dL | Bɔrku tɛm (90%) i kin kam wit wan akyu baktriyal infεkshɔn. Kɔndishɔn lɛk dengi, malaria, ɛn ɛpatitis C kin mek am bak. |
wan CRP lεvεl we pas 10 mg/dL dεn kin jεnarali kכnsidr am as sayn fכ sכm inflameshn. Bɔt dis tɛst nɔmɔ nɔ go ebul fɔ no wetin de mek i apin. Yu dɔktɔ go nid fɔ du mɔ tɛst fɔ no wetin mek i apin.
Wan spɛshal CRP tɛst (hs-CRP) we de luk pan di risk fɔ gɛt at sik
Wan tɛst de we gɛt mɔ sɛnsitiv pas di standad CRP tɛst, we dɛn kin yuz fɔ asɛs di risk fɔ gɛt di sik we dɛn kɔl kadiovaskular sik (CVD). Dɛn kɔl am Ay-Sɛnsitiviti CRP (hs-CRP) .
Dis tɛst kin yus mɔ fɔ pipul dɛm wae gɛt 10%-20% risk fɔ gɛt hat atak insay di nɛks 10 ia. di CRP lεvεl, lεk di kכlestכl lεvεl, kin εp fכ no di risk fכ at sik. Wan stɔdi dɔn sho se we pɔsin gɛt ay CRP lɛvɛl, dat kin mek i gɛt at atak tri tɛm.
Na dis na aw fɔ intaprit hs-CRP rizɔlt:
| hs-CRP valyu (mg/L) 1.1. | Risk fɔ gɛt sik we dɛn kɔl kadiovaskular sik (CVD). |
|---|---|
| I nɔ rich 1.0 mg/L | Low risk |
| 1.0 mg/L to 3.0 mg/L | Modaret risk |
| Mɔ pas 3.0 mg/L | High risk |
Bikɔs di CRP lɛvɛl kin difrɛn smɔl frɔm wan de to di ɔda, i bɛtɛ fɔ mek dɛn du dis tɛst tu tɛm, tu wiks apat, fɔ no kɔrɛkt wan fɔ no if yu gɛt at sik. Dɔn bak, fɔ gɛt ay hs-CRP lɛvɛl nɔ min se yu gɛt at sik. So i impɔtant fɔ kɔmpia am wit yu LDL kɔlɔstrel lɛvɛl fɔ gɛt di ful pikchɔ.
Wetin yu fɔ du if yu CRP lɛvɛl ay?
If yu CRP lɛvɛl ay, di tritmɛnt go dipen pan di kɔz. If yu CRP lɛvɛl go ɔp bikɔs ɔf wan risk factor fɔ gɛt at sik, sɔm tin dɛn de we yu kin du fɔ ridyus da risk de:
- Yu fɔ it tin dɛn we go ɛp yu at: It tin dɛn we nɔ gɛt bɔku ɔyl, sɔl, ɛn shuga, ɛn we gɛt bɔku frut ɛn vɛjitebul.
- Kɔntrol di kɔlɔstrel.
- Mek yu bɔdi gɛt di rayt wet.
- Ɛksesaiz ɔltɛm.
- If yu gɛt dayabitis ɔ yu gɛt ay blɔd prɛshɔn, kɔntrol dɛn fayn fayn wan.
- If yu de smok, stɔp wantɛm wantɛm.
- Limit fɔ drink rɔm.
Statin, we na wan kayn mɛrɛsin we de mek pɔsin nɔ gɛt bɔku bɔku kɔlɔstrel, kin ɛp fɔ ridyus di risk fɔ gɛt at sik pan pipul dɛn we gɛt ay CRP. Bɔt if yu kɔlɔstrel lɛvɛl nɔmal, statin nɔ kin bi di rayt ansa fɔ ay CRP lɛvɛl. I bɛtɛ fɔ tɔk to yu dɔktɔ bɔt us tritmɛnt we go fayn fɔ yu.
Mɛsej we dɛn kin kɛr go na os
- Di CRP tɛst de mɛzhɔ inflamɛns na di bɔdi, nɔto wan patikyula sik.
- Dis kin rili yusful fɔ asɛs di kɔndishɔn dɛm lɛk di risk fɔ gɛt at sik, infɛkshɔn, ɛn ɔtoimyun sik dɛm.
- If yu CRP lɛvɛl ay, yu dɔktɔ kin nid fɔ du mɔ tɛst fɔ no di kɔz.
- wan mכr sεnsitiv tεst we dεn kכl hs-CRP dεn de yuz fכ spεshal wan fכ ases di risk fכ at sik.
- Di chenj na yu layf impɔtant fɔ manej di ay CRP lɛvɛl, bɔt mek shɔ se yu go to yu dɔktɔ fɔ ɛni tritmɛnt.
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