Di wod "cholesterol" de mek plenti of wi fil likl frayd, no so? Bikɔs wi ɔl no se if kɔlɔstrel bɔku, di chans fɔ gɛt siriɔs sik lɛk at sik ɛn strok kin bɔku. Bɔt di gud nyus na dat, wi kin kɔntrol dis sik if wi it gud it ɛn liv fayn layf. Ɛn if nid de, mɛrɛsin dɛn de we di dɔktɔ kin gi am. Wit ɔl dis, dɔktɔ ɛn sayɛnsman dɛn stil de du risach ɛn fɛn nyu tin dɛn bɔt kɔlɔstrel. So lɛ wi si wetin dɛn dɔn fɛn.
Nɔto ɔlman na di sem, naw di tritmɛnt spɛshal fɔ yu!
Trade, dɔktɔ dɛn bin de tink se ɔlman in kɔlɔstrel fɔ bi di sem. Bɔt naw da we de fɔ si tin dɔn chenj. Naw, we yu dɔktɔ luk yu kɔlɔstrel ripɔt, i nɔ jɔs de disayd bay dɛn valyu dɛn de, bɔt i de disayd bak bay ɔda tin dɛn we de mek yu gɛt at sik.
Imajin, yu yon:
- Blɔd prɛshɔn
- Di blɔd shuga lɛvɛl
- Ej
- We
I kin bisin bak bɔt tin dɛn lɛk dis. Fɔ tɔk am simpul wan, if yu gɛt ay risk fɔ gɛt at sik, yu dɔktɔ go advays yu fɔ kip yu kɔlɔstrel lɛvɛl smɔl as yu ebul. Dis min se yu tritmɛnt kin difrɛn frɔm di tritmɛnt we dɛn gi yu padi.
Ɛksesaiz na di fɔs mɛrɛsin bifo mɛrɛsin!
Lɛ wi se yu gɛt smɔl ay kɔlɔstrel ɛn smɔl ay blɔd prɛshɔn, bɔt yu ɔl risk fɔ gɛt at sik nɔ bɔku. If na so i bi, yu dɔktɔ nɔ go gi yu mɛrɛsin wantɛm wantɛm. Frɔm di las gaydlain frɔm di American Heart Association, di fɔs layn fɔ tritmɛnt na fɔ sidɔm smɔl ɛn muv mɔ. Dat min se yu fɔ du ɛksɛsayz.
fyzikal aktiviti kin ridyus di "bad" kכlestכl, כ LDL (Low-Density Lipoprotein) , na yu bכdi bay 3 to 6 miligram (mg/dL). Nɔto dat nɔmɔ, i kin mek di blɔd prɛshɔn go dɔŋ bak.
Di bɛst we fɔ mek yu swet na fɔ du lɛk 150 minit ɛksesaiz we nɔ gɛt bɛtɛ trɛnk insay di wik. Bɔt nɔ tink se i at fɔ fɛn bɔku tɛm. Ivin jɔs 5 ɔ 10 minit we yu de du ɛksɛsayz ɛvride, dat kin mek big difrɛns pan yu kɔlɔstrel lɛvɛl. So mek am yu abit fɔ du sɔntin ɛvride, lɛk fɔ waka ɔ fɔ rayd baysikul.
Nɔto jɔs statin, ɔda nyu tritmɛnt dɛn de!
Bɔku tɛm, dɔktɔ dɛn kin gi wan klas ɔf mɛrɛsin we dɛn kɔl `Statins` as tritmɛnt fɔ kɔlɔstrel. Bɔt dɛn drɔgs ya nɔ de wok fɔ ɔlman. Sɔm pipul dɛn nɔ kin gɛt di tin dɛn we dɛn bin de op fɔ gɛt frɔm dɛn mɛrɛsin ya, ɛn ɔda pipul dɛn kin gɛt sɔm bad bad tin dɛn. If yusɛf gɛt di sem prɔblɛm, nɔ wɔri. Naw, sɔm ɔda tin dɛn de we yu kin du fɔ dat.
Lɛ wi luk dɛn nyu tritmɛnt ya na tebul. Da we de i kin izi fɔ ɔndastand.
| Di we aw dɛn kin trit am | Aw i de wok | Speshal pɔynt dɛn |
|---|---|---|
| PCSK9 inhibitɔ dɛn | wan protin we dεn kכl PCSK9, we di liva de mek, de ambɔg di we aw di bad kכlestכl (LDL) de kכmכt na di bכdi. dis dכg de blכk da PCSK9 protin de. | Wan vaysin, we dɛn kin gi ɛvri 2 wik, kin wok fayn pas statin fɔ pipul dɛn we gɛt famili aypa kɔlɔstrelemia , we na wan sik we dɛn kin gɛt frɔm dɛn mama ɛn papa. |
| siRNA tɛrapi (e.g. inklisiran) | Dis na nyu teknɔlɔji. Na we fɔ trit sik bay we wi de chenj di we aw wi jin dɛn de wok. dis dכg de lכs di LDL lεvεl bay we i de afekt di jin we de mek di PCSK9 protin. | Wan vaysin we dɛn kin gi ɛvri sɔm mɔnt. I fayn fɔ pipul dɛn we gɛt sik lɛk atherosclerotic cardiovascular disease (ASCVD) . |
| Bempedoik asid we dɛn kɔl | Lɛk statin, dɛn mɛrɛsin ya kin wok bak bay we dɛn kin mek i nɔ izi fɔ mek di bɔdi gɛt kɔlɔstrel. | Dɛn kin tek dis as pil. I kin ridyus di LDL lɛvɛl bay lɛk 15%. Naw dɛn kin gi am to pipul dɛn we gɛt famili histri bɔt kɔlɔstrel ɔ ASCVD pasɛnt. |
Di tin we impɔtant pas ɔl:Yu dɔktɔ fɔ disayd if dɛn mɛrɛsin ya fayn fɔ yu. Nɔ ɛva bigin ɔ stɔp fɔ tek mɛrɛsin fɔ yusɛf.
Nanoteknɔlɔji de ‘it’ di fat we de na di blɔd vesel dɛn
We di kɔlɔstrel lɛvɛl go ɔp, di fat (plek) kin bigin fɔ gɛda insay wi blɔd vesel, ɔ di at. dis na mεdikal dεm we dεn kכl `atherosclerosis.` As tεm de go, dεn dεposit dεm ya de mek di bכdi fכ fכlכ εn i de mek di risk fכ at sik εn strכk bכku.
Bɔt sayɛnsman dɛn dɔn mek wan smɔl tin naw we kin "it" dɛn ɔyl layers ya. Dɛn kɔl am `nanoparticle`. Dat min se, sɔntin we smɔl sote pɔsin nɔ go si am wit in yay. pan tap we dis de na di εkspiriεns stej naw, insay di fכs tεm dεn kin yuz wan dכg we gεt dis `nanoparticle` fכ trit `atherosclerosis`.
Aw di gut wɛlbɔdi kin afɛkt di kɔlɔstrel
Fɔ lɔng tɛm naw, pipul dɛn we de stɔdi bɔt dis dɔn tink se di wɛlbɔdi na wi intestinal kin afɛkt di kɔlɔstrel lɛvɛl bak, bɔt i nɔ bin klia klia wan aw dat apin.
Bɔt naw dɛn dɔn kam fɔ no se probayotiks (gud baktri dɛm) ɛn prɛbayɔtiks (fud fɔ dɛn gud baktri dɛm ya) kin ridyus bad kɔlɔstrel (LDL) ɛn triglisɛrɛyd (ɔda kayn fat na di blɔd). pan tap dat, dεn gud baktri dεm ya na di gכt kin inkrεs di lεvεl dεm fכ "gud" kכlestכl, כ HDL (High-Density Lipoprotein) .
If yu want fɔ tek probayotik ɔ prɛbayɔtik sɔpɔtmɛnt, tɔk to yu dɔktɔ fɔs. Dɛn stil de du risach bɔt di doz we dɛn nid fɔ gɛt fɔ gɛt rizɔlt, ɛn if pɔsin tek am tumɔs, i kin mek di bɛlɛ at.
Mɛsej we dɛn kin kɛr go na os
- Kɔlɔstrel kɔntrol na in yon fɔ ɛnibɔdi. Dɛn kin disayd fɔ trit yu nɔr jɔs de pan yu kɔlɔstrel lɛvɛl, bɔt ɔda tin dɛm wae kin mek yu gɛt dis sik. So tɔk to yu dɔktɔ ɔltɛm.
- Di fɔs ɛn impɔtant tin we yu fɔ du fɔ ɛni tritmɛnt na fɔ chenj yu layf, mɔ fɔ du ɛksɛsayz ɔltɛm .
- If di statin dɛm wae dɛn kin yuse nɔr fayn fɔ yu, nɔr wɔri. Naw, ɔda tritmɛnt dɛn de we go wok fayn.
- Wi kin op fɔ gɛt mɔ advans tritmɛnt fɔ kɔlestɔl mɛnejɛmɛnt tumara bambay.
- Nɔ bigin ɛni mɛrɛsin ɔ sɔpɔtmɛnt we yu nɔ go to dɔktɔ.
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