Yu gɛt bɔku bɔku eosinophils, we na wan kayn wayt blɔd sɛl, na yu bɔdi? Lɛ wi tɔk bɔt Hypereosinophilic Syndrome!

Sɔntɛnde, yu kin fil lɛk se sɔntin nɔ fayn na yu bɔdi, se yu de gɛt prɔblɛm ɔltɛm, bɔt yu nɔ ebul fɔ no ustɛm i bi? Sɔm sik dɛn de we nɔ kin bɔku ɛn we kin kɔmplikt smɔl. Tide wi go tɔk bɔt wan pan dɛn kayn sik dɛn de. insay dis, di nכmba fכ di eosinophils, we na wan spεshal kayn wayt blכd sεl na wi bכdi, de inkrεs pasmak.

Wetin na di Hypereosinophilic Syndrome?

Fɔ tɔk am simpul wan, Hypereosinophilic Syndrome (HES) na wan sik we nɔ kin apin so ɔltɛm we di nɔmba fɔ di eosinophils , we na wan kayn wayt blɔd sɛl na wi bɔdi, bɔku pasmak. Naw yu kin de wɔnda wetin na dɛn eosinophils ya.

Tink bɔt am lɛk fɔt. Difrɛn kayn sojaman dɛn de fɔ protɛkt dis fɔt. Semweso, difrɛn kayn wayt blɔd sɛl dɛn de na wi blɔd we de fɛt jem ɛn protɛkt wi frɔm sik dɛn. Eosinophils na wan pan dɛn spɛshal sojaman dɛn de. Dɛn men wok na fɔ fɛt di parasayt dɛn we de kam insay wi bɔdi ɛn fɔ ansa we alɛji apin.

nכmal wan, di eosinofil dεm de mek 5% to 7% pan di tכtal wayt bכdi sεl dεm na di hεlty pכsin in bכdi. Dat min se bitwin 100 ɛn 500 eosinofil dɛn de pan wan maykrolit blɔd. Bɔt we yu gɛt sɔntin lɛk alɛji, dɛn eosinofil ya kin bɔku smɔl. Wi kin kɔl da kɔndishɔn de eosinophilia .

כltu, insay hypereosinophilic syndrome (HES), dis kכndyushכn we dεn kכl eosinophilia kin bi mכr siriכs, εn eosinophil prodakshכn kin apin pan hכy rεt, we kin mek dεn sεl dεm ya kin gɛda pasmak na di bכdi εn difrεn tisu dεm na di bכdi. we di eosinofil dεm de inkrεs dis we, dεn sεl dεm ya kin bigin fכ pwεl imכtant כgan dεm lεk wi at, lכng, skin, εn nεv sεstem. If dɛn nɔ trit dis sik fayn, i kin mek pɔsin in layf de pan denja. Bɔt di gud nyus na dat if dɛn no di sik kwik kwik wan ɛn bigin fɔ trit am fayn, pas 80% pan di pipul dɛn we gɛt HES de liv fayn ivin fayv ia afta dɛn dɔn no se i gɛt am.

Udat kin gɛt di sik we dɛn kɔl hypereosinophilic syndrome (HES)?

Dis kɔndishɔn we dɛn kɔl HES na rili rare . So i nɔ kin izi fɔ mek dɔktɔ dɛn tɔk ustɛm bɔku pipul dɛn gɛt dis sik. Pan ɔl we ɛnibɔdi kin gɛt dis sik, i kin kam pan pipul dɛn we ol bitwin 20 ɛn 50 ia .

Wetin kin mek pɔsin gɛt di sik we dɛn kɔl hypereosinophilic syndrome (HES)?

infakt, insay plεnti kes dεm fכ HES, risechכr dεm stil nכ fכgεst εksεkεt wetin de mek dis sכdεn inkrεs pan di eosinophil kכnt. Bɔt dɛn dɔn no sɔm tin dɛn ɔ sɔm tin dɛn we kin mek dis apin:

• Mayloproliferative diseases: Dis na wan grup of sik wae de sho se di blɔd sɛl dɛn we de na wi bon mɛrɔ de bɔku pasmak. Fɔ tɔk am simpul wan, i tan lɛk se di bon mɛro de wok tumɔs.
• di εlevεt lεvεl dεm fכ Interleukin-5: Dis na protin we wi wayt bכdi sεl dεm de mek. we dis de inkrεs, di prodakshכn fכ di eosinofil dεm de stimulate.
• wan jεnεtik abnכmaliti we de mek di sεl dεm gro kwik kwik wan: sכmtεm, dεn sεl dεm ya kin gro we dεn nכ ebul fכ kכntrכl bikoz fכ di chenj dεm na di jin dεm.

Wetin na di sayn dɛm fɔ HES?

Di sayn we kin sho se yu gɛt HES na we yu gɛt skin rɔsh ɔ ayv. כda simptom dεm de dipכnt pan usay na di bכdi di eosinophil lεvεl dεm de εlevεt.

Tink bɔt am dis we:

• If di at afɛkt: Yu kin gɛt sɔm sayn dɛm lɛk Kɔnjɛstiv Hat Failure (wan tin we fiba di at we nɔ de wok fayn), Cardiomyopathy (we di at mɔsul wik), Myocarditis (di at mɔsul we de inflamɛns), ɔ Pericardial Effusion (we wata kin gɛda rawnd di at).
• If di lכng dεm afekt: Yu kin gεt infεkshכn dεm na di כp respiratory infεkshכn כltεm, lεk kol εn flu, kכf, εn i kin at fכ brith.

Apat frɔm dat, dɛn kin si bak di kɔmɔn sayn dɛm lɛk fɔ taya, fiva, bɔdi pen, ɛn fɔ lɛf fɔ it bɔku bɔku it .

Aw dɔktɔ dɛn kin no se pɔsin gɛt di sik we dɛn kɔl hypereosinophilic syndrome (HES)?

Fɔ no HES kin bi kɔmplikɛt prɔses. Dɔktɔ dɛn kin yuz "process of elimination," we min se dɛn fɔ mek shɔ se nɔ ɔda tin nɔ de we kin mek i apin.

Fɔ ɛgzampul, if yu gɛt tin dɛn we de mek yu bɔdi it ɔltɛm, yu dɔktɔ kin chɛk fɔs fɔ sɔm tin dɛn we de apin na yu skin ɛn tɛl yu fɔ gi yu tritmɛnt fɔ dɛn.

If yu sik stil de afta dɛn tritmɛnt ya, yu dɔktɔ kin du blɔd tɛst fɔ chɛk fɔ si if di eosinophil lɛvɛl dɔn go ɔp na yu blɔd. If i tan lɛk se yu sik gɛt fɔ du wit yu liva prɔblɛm, ɛn afta yu dɔn pul ɔda tin dɛn we kin mek yu gɛt dis sik, yu dɔktɔ kin du blɔd tɛst bak we yu liva de wok . Sɔntɛnde, dɛn kin nid fɔ tek bayɔpsi (we dɛn tek wan pat pan di tisu) fɔ kɔnfɔm se dɛn dɔn no se i gɛt HES.

Bikɔs dis sik kin tek sɔm tɛm fɔ no bɔt dis sik, yu kin sik fɔ sɔm mɔnt, yu kin taya, ɛn yu nɔ kin ebul fɔ no di rayt tin we de apin. Bɔt i impɔtant fɔ peshɛnt.

Aw dɔktɔ dɛn kin trit di sik we dɛn kɔl hypereosinophilic syndrome (HES)?

Di men gol fɔ dɔktɔ dɛn na fɔ ridyus di ay levul fɔ di eosinophils na yu blɔd ɛn tisu dɛn. Di tritmɛnt dɛm wae dɛn kin yuse fɔ gɛt dis na:

• Kɔtikosterɔyd: Dɛn wan ya na pawaful mɛrɛsin dɛn we de mek pɔsin nɔ fil bad.
• Kimotɛrapi drɔgs: Drɔg dɛn we dɛn kin gi fɔ kil kansa sɛl dɛn. dεn kin yuz dεn tin ya bak insay lכw dכz fכ kכntrכl di eosinophil sεl dεm.

Apat frɔm dat, sɔm tɛm dɛn kin yuz di tritmɛnt dɛn we dɛn kin tɔk bɔt lɛk Imatinib ɛn monoklɔnal antibɔdi tritmɛnt dɛn.

Yu tink se dɛn kin mek dɛn nɔ gɛt di sik we dɛn kɔl hypereosinophilic syndrome (HES)?

I sɔri fɔ no se, bikɔs wi nɔ no di rayt tin we kin mek bɔku pan di HES kes dɛn, i nɔ kin izi fɔ tɔk ɛni patikyula tin bɔt aw fɔ mek dɛn nɔ gɛt am.

If a gɛt hypereosinophilic syndrome (HES), wetin a fɔ ɛkspɛkt? (Di we aw di sik de go bifo)

De fiuja kɔs fɔ yu sik, ɔr "prognosis," de dipen pan yu patikyula kɔndishɔn. Fɔ ɛgzampul, if yu HES na sɔm patikyula tin, lɛk bon mɛro sik, yu dɔktɔ go mɔs gi yu tritmɛnt we dɛn go yuz fɔ da sik de.

Bɔt if yu dɔktɔ nɔ ebul fɔ no wetin mek yu gɛt HES, di tritmɛnt go pe atɛnshɔn fɔ kɔntrol yu sik dɛn. Fɔ ɛgzampul, if HES afɛkt yu lɔng ɛn yu kin gɛt infɛkshɔn ɔltɛm, di tritmɛnt go tray fɔ kɔntrol dɛn infɛkshɔn dɛn de.

Jɛnɛral wan, di kwik we dɛn no bɔt HES ɛn trit am, na di mɔ i nɔ go gɛt prɔblɛm fɔ lɔng tɛm.

Aw a go kia fɔ mi wɛlbɔdi biznɛs?

As wi bin dɔn tɔk, dɔktɔ dɛn kin trit HES wit kɔtikosterɔyd ɛn kemotɛrapi. Dɛn tu tritmɛnt ya kin gɛt sayd ɛfɛkt . So, tɔk to yu dɔktɔ bɔt dɛn sayd ɛfɛkt ya ɛn wetin yu kin du fɔ mek dɛn nɔ ambɔg yu.

• Tek yu mɛrɛsin dɛn di rayt tɛm.
• Du wetin di dɔktɔ tɛl yu fɔ du.
• It di it we balans.
• Tray fɔ rilaks as yu ebul.
• Stay strɔng na yu maynd.

Ustɛm a fɔ go to mi dɔktɔ?

If dɛn dɔn no se yu gɛt HES, ɛn yu sik dɛn de kɔntinyu ɔ de wɔs, go to yu dɔktɔ wantɛm wantɛm. Dɔn bak, mek shɔ se yu atɛnd di klinik apɔntinmɛnt dɛn we yu dɔn plan fɔ du.

Ustɛm a fɔ go na di Imejɛnsi Rum?

Wi no se wan sik we dɛn kɔl hypereosinophilic syndrome (HES) kin afɛkt difrɛn ɔgan dɛn, ivin di at. So, if HES afɛkt yu at, ɛn yu gɛt sɔm sayn dɛn lɛk we yu de fil pen wantɛm wantɛm na yu chɛst, yu chɛst de tayt, yu nɔs, ɛn yu de swet , yu fɔ go na di imejensi rum wantɛm wantɛm. Dɛn tin ya kin bi sayn fɔ sho se yu gɛt at atɔk.

Us kwɛstyɔn dɛn a fɔ aks mi dɔktɔ?

Bikɔs HES na wan sik wae nɔr kin bɔrku, yu kin gɛt bɔrku kwɛstyɔn bɔt am. Na sɔm kwɛstyɔn dɛn we yu kin aks fɔ bigin:

• Dɔktɔ, wetin rili na dis Hypereosinophilic Syndrome?
• Wetin mek dis bin apin to mi?
• Yu tink se dɛn kin trit dis?
• Wetin na di tritmɛnt dɛn we dɛn kin gɛt?
• Wetin na di sayd ɛfɛkt dɛm we di tritmɛnt kin gɛt?
• Dis HES sityueshɔn kin kam bak?
• A go ebul fɔ wɛl ɔltogɛda?

Fɔ dɔn, tin dɛn fɔ mɛmba (Take-Home Message) .

Hypereosinophilic syndrome (HES) na wan sik wae nɔr kin bɔrku ɛn sɔmtɛm kin at fɔ no. If yu gɛt HES, yu kin dɔn sik fɔ sɔm mɔnt, yu kin sɔfa wit difrɛn sik dɛn, ɛn yu nɔ kin fil fayn.

Bɔt di gud nyus na dat , wae dɛn dɔn no se yu gɛt HES, fayn fayn tritmɛnt dɛn de. I kin tek sɔm mɔnt fɔ mek yu tritmɛnt sho di rizɔlt, so tray fɔ peshɛnt. Dɔn bak, if yu gɛt prɔblɛm fɔ bia wit de prɔblɛm wae de kam wit pɔrsin wae gɛt dis sik fɔ lɔng tɛm, tɔk to yu dɔktɔ. Dɛn kin tɛl yu bɔt program ɛn tin dɛn we go ɛp yu. Mɛmba se nɔto yu wan de!

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