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Yu tink se rɛd yist rays we de mek yu kɔlɔstrel nɔ bɔku, rili fayn fɔ yu?

Yu tink se rɛd yist rays we de mek yu kɔlɔstrel nɔ bɔku, rili fayn fɔ yu?

Yu na pɔsin bak we de wɔri bɔt ay kɔlɔstrel lɛvɛl ɛn de luk fɔ sɔlv fɔ am? Na dɛn kayn tɛm ya, sɔm pipul dɛn kin luk fɔ tin dɛn we "natural" pas di mɛrɛsin dɛn we dɔktɔ gi dɛn. We yu de luk lɛk dat, yu go dɔn yɛri di nem "rɛd yist rays". Bɔku tɛm dɛn kin sɛl dɛn tin ya as natura l sɔpɔt we de mek di kɔlɔstrel nɔ bɔku. Bɔt yu tink se i rili simpul so? Lɛ wi tɔk klia wan bɔt dis tide.

Wetin na Rɛd Yist Rays?

Fɔ tɔk am simpul wan, rɛd yist rays na prɔdak we dɛn mek bay we dɛn de fɛmɛnt rays we dɛn de yuz wan spɛshal kayn yist we dɛn kɔl *Monascus purpureus*. Dɛn dɔn de yuz am as tradishɔnal mɛrɛsin na Eshia kɔntri dɛn lɛk Chaina fɔ lɔng lɔng tɛm. Dɛn kin yuz am bak fɔ mek it kɔlɔ ɛn fɔ mek tin nɔ pwɛl.

Dis rɛd yist rays kin gɛt bɔku tin dɛn we dɛn se kin ɛp fɔ kɔntrol kɔlɔstrel.

  • Monacolins: di mכst imכtant pan dεm na monacolin K. Dis na di men aktif ingridiεnt fכ dis.
  • Ɔda tin dɛn we dɛn kin yuz fɔ mek am: Apat frɔm dat, i gɛt bak sterol, isoflavones, ɛn "healthy fats."

Di wɔd "natural" kin mek yu fil fayn. Bɔt wi nid fɔ rili tek tɛm. Bikɔs dɛn nɔ dɔn du ɛni lɔng tɛm, bɔku risach bɔt dis yet. So i nɔr klia wetin na di rayt doz, ɔr if i sef fɔ tek lɔng tɛm. I kin ivin denja fɔ sɔm pipul dɛn.

Di impɔtant tin na dat di tin dɛn we dɛn kin yuz fɔ mek rɛd yist rays kin difrɛn bad bad wan frɔm wan brand to ɔda brand, so wetin dɛn kin tɔk bɔt wan brand nɔ kin apin to ɔda wan.

Dis na rili ‘supliment’? Ɔ na mɛrɛsin?

Dis na di kwɛstyɔn we kin mek bɔku pipul dɛn kɔnfyus. I sɔprayz fɔ no se di ansa na dat, i kin bi ɔl tu .

Memba aw a bin se di most impotant ingredient fo dis na monacolin K? Dis kemikal we dɛn kɔl monacolin K rili fiba di aktif kemikal we de insay di drɔg we dɛn kɔl lovastatin , we dɔktɔ dɛn kin se fɔ mek di kɔlɔstrel nɔ bɔku. Dat min se pan kemikal, dɛn na di sem.

Dis na di rizin we mek big prɔblɛm kin kam. na wan say, dεn de sכl am as "natural supliment". Bɔt institiushɔn dɛn lɛk FDA (Food and Drug Administration) na Amɛrika se bikɔs i gɛt wan tin we de insay wan mɛrɛsin, dɛn nɔ go ebul fɔ sɛl am if dɛn nɔ gɛt lɔ. Bikɔs dɛn kin sho se if dɛn tek dɛn tin ya we dɛn nɔ kɔntrol di rayt we, dɛn kin mek di mɔsul dɛn pwɛl bad bad wan ɛn ivin gɛt sik na di kidni.

Pan ɔl dɛn lɔ ya, bɔku pipul dɛn kin bay dɛn kayn tin dɛn de na di Intanɛt. BɔtDi men prɔblɛm na dat yu nɔ gɛt we fɔ no ɔmɔs aktif ingridiɛnt (monacolin K) de na di bɔtul we yu bay.

Di bɛnifit dɛn we rɛd yist rays gɛt

Bɔku impɔtant bɛnifit dɛn de we pipul dɛn we de tek rɛd yist rays kin ɛkspɛkt. Lɛ wi luk dɛn na wan tebul.

Yuzful tin Aw i kin apin (insay simpul wɔd dɛn) .
Fɔ ridyus "bad" kɔlɔstrel (LDL) . di kεmikכl we de insay, we dεn kכl monokolin K, de ridyus di kכlestכl we di liva de mek. Dis na di rayt we aw di statin mɛrɛsin dɛn we dɔktɔ dɛn kin gi kin wok.
Fɔ ridyus di risk fɔ gɛt strok We di kɔlɔstrel nɔ bɔku, di risk fɔ mek di blɔd klɔt na di blɔd vesel dɛn kin go dɔŋ. Bɔku tɛm, dɛn denja blɔd ya kin mek pɔsin gɛt strok ɛn at atak.
Fɔ mek di blɔd go fayn fayn wan we di kכlestכl dεposit (plak) dεm na di bכdi vεsul dεm dכn dכn, di bכdi fכ fכlכ kin izi, we min se di bכdi in כgan dεm εn mכsul dεm de gεt bεtε כksijεn.

Aw dis rili wok fɔ mek di kɔlɔstrel nɔ bɔku?

sכm risεch dεn sho se rεd yist rays prodakt dεm we gεt monacolin K kin ridyus di tכtal kכlestכl lεvεl bכku bכku wan, spεshal wan "bad" kכlestכl (LDL). Wan stכdi sho se if yu tek 2.4 gram pan de, i de ridyus di LDL lεvεl bay 22% εn di tכtal kכlestכl bay 16% ova 12 wik.

Bɔt di big prɔblɛm we de ya na...

dis rizulεt dεm de dipכnt pan aw mכch monacolin K de insay di prכdak we yu bay. Di amount kin difrɛn bad bad wan frɔm wan prɔdak to ɔda prɔdak. Insay wan stɔdi, we dɛn bin de tɛst difrɛn brand dɛn na di makit, sɔm nɔ bin gɛt monacolin K atɔl (0%), we ɔda wan dɛn bin gɛt lɛvɛl we ay lɛk 0.58%.

Dat min se di bɔtul we yu pe fɔ nɔ gɛt ɛnitin fɔ mek yu nɔ gɛt bɔku kɔlɔstrel atɔl, ɔ i kin gɛt di sem tin we di pil gɛt. I tan lɛk se na lɔtri.

Sayd ɛfɛkt ɛn risk dɛn we yu fɔ no bɔt!

Nɔ tek am se bikɔs na "natural" nɔr sayd ɛfɛkt nɔ de. Dat na big mistek. Rɛd yist rays kin gɛt di sem risk ɛn sayd ɛfɛkt lɛk di statin mɛrɛsin dɛn we dɔktɔ gi.

Smɔl smɔl sayd ɛfɛkt dɛn we dɛn kin ripɔt:

  • Ed de at
  • At bɔn
  • Bɛlɛ nɔ kin fil fayn

Bɔt wi nid fɔ no bak bɔt mɔ siriɔs risk dɛn . Dɛn tin ya na di sem tin wit di sayd ɛfɛkt dɛn we di mɛrɛsin we dɛn kɔl lovastatin kin gɛt.

  • Prɔblɛm dɛn na di mɔsul dɛn: Di mɔsul dɛn kin pen ɛn wikɛd.
  • Prɔblɛm dɛn na di liva: Di ɛnzaym dɛn na di liva kin bɔku.

So, di pipul dɛn we de kam biɛn ya ɛn di wan dɛn we de tek dɛn mɛrɛsin ya nɔ fɔ yuz rɛd yist rays atɔl .

Wonin: Nɔ tek rɛd yist rays pan dɛn kayn tin ya
If yu gɛt dɛn wɛlbɔdi prɔblɛm ya: If yu de tek dɛn mɛrɛsin ya:

  • Kidni sik
  • Liva sik
  • If yu gɛt bɛlɛ
  • If yu na mama we de gi pikin in bɛlɛ
  • If yu gɛt alɛji fɔ fɛns ɔ yist

  • Ɔda kɔlɔstrel mɛrɛsin dɛn (e.g., statin dɛn lɛk lovastatin, atorvastatin, simvastatin) .
  • Ɔda kɔlɔstrel mɛrɛsin dɛn lɛk Gemfibrozil, Fenofibrate
  • Drug dεm we de sכpres di imyun (e.g. cyclosporine) .
  • Antifungal mεdikeshכn dεm (e.g. kεtokonazכl, flukonazכl) .
  • Sɔm antibayɔtiks, lɛk ɛrythromycin
  • Protease inhibitors fɔ HIV

Dɛn kin intarakt bak wit mɛrɛsin fɔ blɔd prɛshɔn ɛn tayroyd disɔda. So, ilɛksɛf yu gɛt wɛlbɔdi, mek shɔ se yu tɔk to yu dɔktɔ bifo yu yuz rɛd yist rays ɔ ɛni ɔda supamakit .**

Mɛsej we dɛn kin kɛr go na os

  • Rɛd yist rays nɔto jɔs "nɛchral." Di kɔmpawnd we de insay, we na monokolin K, gɛt kemikal dɛn we fiba di mɛrɛsin we dɛn kɔl lovastatin, we dɔktɔ dɛn kin gi am.
  • Di amount of dis aktif ingridiɛnt na prodak dɛm na di makit nɔ de rigul, so yu nɔ gɛt ɛni we fɔ no ɔmɔs, ɔ if ɛni wan de, de insay di bɔtul we yu bay.
  • Risk de fɔ gɛt sayd ɛfɛkt dɛn we fiba di statin drɔgs (espɛshali to di liva ɛn mɔsul dɛm).
  • If yu gɛt ay kɔlɔstrel, instead fɔ disayd fɔ yusɛf ɛn yuz supamakit lɛk dis, di bɛst ɛn sef tin fɔ du na fɔ go to dɔktɔ we sabi du di wok ɛn gɛt di rayt advays ɛn tritmɛnt.

Rɛd Yist Rays, kɔlɔstrel, kɔlɔstrel, lovastatin, statin, at wɛlbɔdi, sayd ɛfɛkt dɛn

Frequently Asked Questions (FAQ)

Aw dis rili wok fɔ mek di kɔlɔstrel nɔ bɔku?

sכm risεch dεn sho se rεd yist rays prodakt dεm we gεt monacolin K kin ridyus di tכtal kכlestכl lεvεl bכku bכku wan, spεshal wan "bad" kכlestכl (LDL). Wan stכdi sho se if yu tek 2.4 gram pan de, i de ridyus di LDL lεvεl bay 22% εn di tכtal kכlestכl bay 16% ova 12 wik.

⚠️ Important: The medical articles and information on Nirogi Lanka are for general awareness only, and are by no means a substitute for professional medical advice, diagnosis, or treatment. For any medical problem you have, consult a qualified physician immediately.

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Yu tink se rɛd yist rays we de mek yu kɔlɔstrel nɔ bɔku, rili fayn fɔ yu?

Yu tink se rɛd yist rays we de mek yu kɔlɔstrel nɔ bɔku, rili fayn fɔ yu?

Yu na pɔsin bak we de wɔri bɔt ay kɔlɔstrel lɛvɛl ɛn de luk fɔ sɔlv fɔ am? Na dɛn kayn tɛm ya, sɔm pipul dɛn kin luk fɔ tin dɛn we "natural" pas di mɛrɛsin dɛn we dɔktɔ gi dɛn. We yu de luk lɛk dat, yu go dɔn yɛri di nem "rɛd yist rays". Bɔku tɛm dɛn kin sɛl dɛn tin ya as natura l sɔpɔt we de mek di kɔlɔstrel nɔ bɔku. Bɔt yu tink se i rili simpul so? Lɛ wi tɔk klia wan bɔt dis tide.

Wetin na Rɛd Yist Rays?

Fɔ tɔk am simpul wan, rɛd yist rays na prɔdak we dɛn mek bay we dɛn de fɛmɛnt rays we dɛn de yuz wan spɛshal kayn yist we dɛn kɔl *Monascus purpureus*. Dɛn dɔn de yuz am as tradishɔnal mɛrɛsin na Eshia kɔntri dɛn lɛk Chaina fɔ lɔng lɔng tɛm. Dɛn kin yuz am bak fɔ mek it kɔlɔ ɛn fɔ mek tin nɔ pwɛl.

Dis rɛd yist rays kin gɛt bɔku tin dɛn we dɛn se kin ɛp fɔ kɔntrol kɔlɔstrel.

  • Monacolins: di mכst imכtant pan dεm na monacolin K. Dis na di men aktif ingridiεnt fכ dis.
  • Ɔda tin dɛn we dɛn kin yuz fɔ mek am: Apat frɔm dat, i gɛt bak sterol, isoflavones, ɛn "healthy fats."

Di wɔd "natural" kin mek yu fil fayn. Bɔt wi nid fɔ rili tek tɛm. Bikɔs dɛn nɔ dɔn du ɛni lɔng tɛm, bɔku risach bɔt dis yet. So i nɔr klia wetin na di rayt doz, ɔr if i sef fɔ tek lɔng tɛm. I kin ivin denja fɔ sɔm pipul dɛn.

Di impɔtant tin na dat di tin dɛn we dɛn kin yuz fɔ mek rɛd yist rays kin difrɛn bad bad wan frɔm wan brand to ɔda brand, so wetin dɛn kin tɔk bɔt wan brand nɔ kin apin to ɔda wan.

Dis na rili ‘supliment’? Ɔ na mɛrɛsin?

Dis na di kwɛstyɔn we kin mek bɔku pipul dɛn kɔnfyus. I sɔprayz fɔ no se di ansa na dat, i kin bi ɔl tu .

Memba aw a bin se di most impotant ingredient fo dis na monacolin K? Dis kemikal we dɛn kɔl monacolin K rili fiba di aktif kemikal we de insay di drɔg we dɛn kɔl lovastatin , we dɔktɔ dɛn kin se fɔ mek di kɔlɔstrel nɔ bɔku. Dat min se pan kemikal, dɛn na di sem.

Dis na di rizin we mek big prɔblɛm kin kam. na wan say, dεn de sכl am as "natural supliment". Bɔt institiushɔn dɛn lɛk FDA (Food and Drug Administration) na Amɛrika se bikɔs i gɛt wan tin we de insay wan mɛrɛsin, dɛn nɔ go ebul fɔ sɛl am if dɛn nɔ gɛt lɔ. Bikɔs dɛn kin sho se if dɛn tek dɛn tin ya we dɛn nɔ kɔntrol di rayt we, dɛn kin mek di mɔsul dɛn pwɛl bad bad wan ɛn ivin gɛt sik na di kidni.

Pan ɔl dɛn lɔ ya, bɔku pipul dɛn kin bay dɛn kayn tin dɛn de na di Intanɛt. BɔtDi men prɔblɛm na dat yu nɔ gɛt we fɔ no ɔmɔs aktif ingridiɛnt (monacolin K) de na di bɔtul we yu bay.

Di bɛnifit dɛn we rɛd yist rays gɛt

Bɔku impɔtant bɛnifit dɛn de we pipul dɛn we de tek rɛd yist rays kin ɛkspɛkt. Lɛ wi luk dɛn na wan tebul.

Yuzful tin Aw i kin apin (insay simpul wɔd dɛn) .
Fɔ ridyus "bad" kɔlɔstrel (LDL) . di kεmikכl we de insay, we dεn kכl monokolin K, de ridyus di kכlestכl we di liva de mek. Dis na di rayt we aw di statin mɛrɛsin dɛn we dɔktɔ dɛn kin gi kin wok.
Fɔ ridyus di risk fɔ gɛt strok We di kɔlɔstrel nɔ bɔku, di risk fɔ mek di blɔd klɔt na di blɔd vesel dɛn kin go dɔŋ. Bɔku tɛm, dɛn denja blɔd ya kin mek pɔsin gɛt strok ɛn at atak.
Fɔ mek di blɔd go fayn fayn wan we di kכlestכl dεposit (plak) dεm na di bכdi vεsul dεm dכn dכn, di bכdi fכ fכlכ kin izi, we min se di bכdi in כgan dεm εn mכsul dεm de gεt bεtε כksijεn.

Aw dis rili wok fɔ mek di kɔlɔstrel nɔ bɔku?

sכm risεch dεn sho se rεd yist rays prodakt dεm we gεt monacolin K kin ridyus di tכtal kכlestכl lεvεl bכku bכku wan, spεshal wan "bad" kכlestכl (LDL). Wan stכdi sho se if yu tek 2.4 gram pan de, i de ridyus di LDL lεvεl bay 22% εn di tכtal kכlestכl bay 16% ova 12 wik.

Bɔt di big prɔblɛm we de ya na...

dis rizulεt dεm de dipכnt pan aw mכch monacolin K de insay di prכdak we yu bay. Di amount kin difrɛn bad bad wan frɔm wan prɔdak to ɔda prɔdak. Insay wan stɔdi, we dɛn bin de tɛst difrɛn brand dɛn na di makit, sɔm nɔ bin gɛt monacolin K atɔl (0%), we ɔda wan dɛn bin gɛt lɛvɛl we ay lɛk 0.58%.

Dat min se di bɔtul we yu pe fɔ nɔ gɛt ɛnitin fɔ mek yu nɔ gɛt bɔku kɔlɔstrel atɔl, ɔ i kin gɛt di sem tin we di pil gɛt. I tan lɛk se na lɔtri.

Sayd ɛfɛkt ɛn risk dɛn we yu fɔ no bɔt!

Nɔ tek am se bikɔs na "natural" nɔr sayd ɛfɛkt nɔ de. Dat na big mistek. Rɛd yist rays kin gɛt di sem risk ɛn sayd ɛfɛkt lɛk di statin mɛrɛsin dɛn we dɔktɔ gi.

Smɔl smɔl sayd ɛfɛkt dɛn we dɛn kin ripɔt:

  • Ed de at
  • At bɔn
  • Bɛlɛ nɔ kin fil fayn

Bɔt wi nid fɔ no bak bɔt mɔ siriɔs risk dɛn . Dɛn tin ya na di sem tin wit di sayd ɛfɛkt dɛn we di mɛrɛsin we dɛn kɔl lovastatin kin gɛt.

  • Prɔblɛm dɛn na di mɔsul dɛn: Di mɔsul dɛn kin pen ɛn wikɛd.
  • Prɔblɛm dɛn na di liva: Di ɛnzaym dɛn na di liva kin bɔku.

So, di pipul dɛn we de kam biɛn ya ɛn di wan dɛn we de tek dɛn mɛrɛsin ya nɔ fɔ yuz rɛd yist rays atɔl .

Wonin: Nɔ tek rɛd yist rays pan dɛn kayn tin ya
If yu gɛt dɛn wɛlbɔdi prɔblɛm ya: If yu de tek dɛn mɛrɛsin ya:

  • Kidni sik
  • Liva sik
  • If yu gɛt bɛlɛ
  • If yu na mama we de gi pikin in bɛlɛ
  • If yu gɛt alɛji fɔ fɛns ɔ yist

  • Ɔda kɔlɔstrel mɛrɛsin dɛn (e.g., statin dɛn lɛk lovastatin, atorvastatin, simvastatin) .
  • Ɔda kɔlɔstrel mɛrɛsin dɛn lɛk Gemfibrozil, Fenofibrate
  • Drug dεm we de sכpres di imyun (e.g. cyclosporine) .
  • Antifungal mεdikeshכn dεm (e.g. kεtokonazכl, flukonazכl) .
  • Sɔm antibayɔtiks, lɛk ɛrythromycin
  • Protease inhibitors fɔ HIV

Dɛn kin intarakt bak wit mɛrɛsin fɔ blɔd prɛshɔn ɛn tayroyd disɔda. So, ilɛksɛf yu gɛt wɛlbɔdi, mek shɔ se yu tɔk to yu dɔktɔ bifo yu yuz rɛd yist rays ɔ ɛni ɔda supamakit .**

Mɛsej we dɛn kin kɛr go na os

  • Rɛd yist rays nɔto jɔs "nɛchral." Di kɔmpawnd we de insay, we na monokolin K, gɛt kemikal dɛn we fiba di mɛrɛsin we dɛn kɔl lovastatin, we dɔktɔ dɛn kin gi am.
  • Di amount of dis aktif ingridiɛnt na prodak dɛm na di makit nɔ de rigul, so yu nɔ gɛt ɛni we fɔ no ɔmɔs, ɔ if ɛni wan de, de insay di bɔtul we yu bay.
  • Risk de fɔ gɛt sayd ɛfɛkt dɛn we fiba di statin drɔgs (espɛshali to di liva ɛn mɔsul dɛm).
  • If yu gɛt ay kɔlɔstrel, instead fɔ disayd fɔ yusɛf ɛn yuz supamakit lɛk dis, di bɛst ɛn sef tin fɔ du na fɔ go to dɔktɔ we sabi du di wok ɛn gɛt di rayt advays ɛn tritmɛnt.

Rɛd Yist Rays, kɔlɔstrel, kɔlɔstrel, lovastatin, statin, at wɛlbɔdi, sayd ɛfɛkt dɛn

Frequently Asked Questions (FAQ)

Aw dis rili wok fɔ mek di kɔlɔstrel nɔ bɔku?

sכm risεch dεn sho se rεd yist rays prodakt dεm we gεt monacolin K kin ridyus di tכtal kכlestכl lεvεl bכku bכku wan, spεshal wan "bad" kכlestכl (LDL). Wan stכdi sho se if yu tek 2.4 gram pan de, i de ridyus di LDL lεvεl bay 22% εn di tכtal kכlestכl bay 16% ova 12 wik.

⚠️ Important: The medical articles and information on Nirogi Lanka are for general awareness only, and are by no means a substitute for professional medical advice, diagnosis, or treatment. For any medical problem you have, consult a qualified physician immediately.

💬 Comments (0)

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