Yu dɔn ɛva gɛt dis apin to yu, we gɛt dayabitis? Pan ɔl we yu dɔn de tek yu mɛrɛsin na nɛt ɛn dɛn dɔn kɔntrol yu blɔd shuga fayn fayn wan, we yu wek na mɔnin ɛn chɛk yu glukomita, yu blɔd shuga kin rili bɔku. Yu kin de tink se, "O, a nɔ no if a tek inof insulin na nɛt." Bɔt di rizin fɔ dis kin bi sɔntin we rili difrɛn frɔm wetin yu tink. Tide wi de tɔk bɔt wan kɔndishɔn wae dɛn kin sɔprayz pan tɛm lɛk dis, bɔt nɔr kin no bɔt am bad bad wan. Dat na di ‘Somogyi ifɛkt.’
Wetin na di Somogyi Ifɛkt?
Fɔ tɔk am simpul wan, di Somogyi Ifɛkt na we yu blɔd shuga lɛvɛl go dɔŋ tumɔs (haypoglycemia) na midul nɛt we yu de slip, ɛn yu bɔdi de pul ɔmon dɛn fɔ ansa, we de mek yu blɔd shuga lɛvɛl go ɔp tumɔs (haypa glycemia) we yu wek na mɔnin. I tan lɛk rɔba band we kin snap bak na wan say we yu lɛf am. sכmtεm dεn kin kכl dis "ribaund haypa glycemia."
Dis na fɔ di fɔstɛm insay di 1930 dɛm bay wan dɔktɔ we nem Maykɛl Somogyi, na dat mek dɛn gi di sik in nem.
Bɔt na sɔntin we impɔtant fɔ ɔndastand. Sayɛnsman dɛn stil nɔ de tek di Somogyi Effect as 100% pruf, na ‘tiori’. Dat min se pan ɔl we sayɛns pruf de fɔ se i kin apin, nɔto ɔlman kin gri wit am. Trade, dɔktɔ dɛn bin de tink se na dis na di men rizin we mek pɔsin gɛt bɔku blɔd shuga na mɔnin, bɔt nyu tin dɛn we dɛn dɔn mek, mɔ di we aw dɛn de yuz kɔntinyu fɔ wach di glukɔs (CGM), dɔn mek pipul dɛn aks kwɛstyɔn bɔt dis aidia.
Bɔt i rili impɔtant fɔ no dis as wan pan di bɔku rizin dɛn we kin mek pɔsin gɛt bɔku blɔd shuga na mɔnin.
Wetin na ɔda rizin dɛn we kin mek pɔsin gɛt bɔku blɔd shuga na mɔnin?
Bifo yu pɔynt di finga nɔmɔ pan di Somogyi ifɛkt, sɔm ɔda tin dɛn de we kin mek yu gɛt ay blɔd shuga na mɔnin.
- Nɔ di mɛrɛsin we yu fɔ tek: If yu tek tumɔs smɔl pan yu dayabitis mɛrɛsin ɔ insulin na nɛt, i kin lɛf fɔ wok igen bay mɔnin ɛn mek yu blɔd shuga bɔku.
- Dawn Phenomenon: Dis na di rizin we kin mek yu gɛt bɔku blɔd shuga na mɔnin. Lɛ wi luk dis smɔl mɔ.
- Mismatch bitwin dina ɛn di mɛrɛsin doz: If dɛn nɔ kɔl di insulin doz fɔ mach di amount of carbohydrates we dɛn it na dina, di blɔd shuga lɛvɛl kin go ɔp na mɔnin.
- Insulin Resistance: Na tin we di bɔdi in sɛl dɛn nɔ de ansa fayn fayn wan to insulin.
Wetin na di difrɛns bitwin di Somogyi Effect ɛn di Dawn Phenomenon?
Pan ɔl we dɛn tu tin ya kin mek di blɔd shuga go ɔp na mɔnin, big difrɛns de bitwin dɛn tu tin ya. As pɔrsin wae gɛt shuga, fɔ ɔndastand dis difrɛns go ɛp yu bad bad wan.
di Dawn Fenomen na we wi bכdi de rilis sכm כmon dεm (lεk kכtisol εn growth כmon) na di ali mכnin (arawnd 3-4 am). Dɛn ɔmon dɛn ya kin mek di blɔd shuga go ɔp. Dis na tru bak fɔ pipul dɛn we nɔ gɛt dayabitis, bɔt dɛn bɔdi de mek ɛn kɔntrol insulin. Pipul wae gɛt shuga nɔr kin ebul fɔ du dis, so dɛn kin gɛt bɔrku shuga na dɛn blɔd na mɔnin.
Bɔt di Somogyi Ifɛkt difrɛn. I kin apin we di blɔd shuga dɔn drɔp (haypoglycemia) na di midul nɛt. Insay di Dawn Phenomenon, di blɔd shuga nɔ de drɔp na nɛt.
Jɔs mɛmba se: Di tin we kin apin to di mɔnin na di natura l ɔmon prɔses. di Somogyi ifekt na di bכdi in rεspכns we di shuga lεvεl dכn dכn na nɛt.
Lɛ wi luk di men tin dɛn we difrɛn bitwin dɛn tu.
| Tin | Somogyi Ifɛkt we pɔsin kin gɛt | Dawn Fenomen we de apin |
|---|---|---|
| Di rut we de mek pɔsin gɛt dis sik | Di blɔd shuga we nɔ bɔku na di midul nɛt (haypoglycemia) . | Di bɔdi kin pul ɔmon dɛn na mɔnin bay insɛf . |
| Shuga lɛvɛl na 2-3 am | Bɔku tɛm na wan lɔw lɛvɛl . | Bɔku tɛm na nɔmal ɔ ɛlevɛt lɛvɛl. |
| Wetin mek dis de apin? | di bכdi de rilis כmon dεm f כ ansa di shuga lεvεl lכw. | Na nɔmal tin we kin mek di bɔdi rɛdi fɔ di wok we i de du fɔ di de. |
| Bɔku tɛm we dɛn kin si am | Na tin we nɔ kin apin so ɔltɛm. | Rili kɔmɔn tin. |
Wetin na di sayn dɛm fɔ di Somogyi ifɛkt?
Di men sayn fɔ dis na di ay blɔd shuga lɛvɛl we yu wek na mɔnin. Bɔt yu kin gɛt sayn dɛn bak we de sho se yu blɔd shuga nɔ bɔku na nɛt.
Di sayn dɛm we de sho se yu gɛt bɔku blɔd shuga na mɔnin (Hyperglycemia) .
- Tɔsti mɔ: Fɔ fil lɛk se yu nɔ de gɛt bɛtɛ wata ilɛksɛf yu drink bɔku.
- Yu fɔ pis bɔku tɛm: Yu nid fɔ pis bɔku tɛm pas aw yu kin pis.
- Angri we de go ɔp: Fɔ fil angri pas aw i kin fil na mɔnin.
- Ɛd we de at: Ɛd we de at we yu wek na mɔnin.
- Fɔ vɛks: Fɔ vɛks pan smɔl smɔl tin dɛn.
Di sayn dɛm we de sho se yu blɔd shuga nɔ bɔku na nɛt (Nocturnal Hypoglycemia) .
Yu nɔ go notis dɛn sayn ya bikɔs yu de slip.
- Swet we yu de slip: We yu de swet sote di shit ɛn di pajama wet.
- Bad drim: Fɔ wek frɔm slip afta yu dɔn drim we de mek yu fred.
- Slip we nɔ de rɛst: Fɔ tos ɛn tɔn, slip we nɔ de rɛst.
- Fɔ fil taya we yu wek na mɔnin: Fɔ fil taya ɛn grog na mɔnin, ivin afta yu dɔn slip fayn.
If yu gɛt sɔm kayn sik lɛk dis na nɛt ɛn yu blɔd shuga bɔku na mɔnin, yu kin sɔprayz se di Somogyi ifɛkt de.
Wetin mek dis de apin? Wetin na di rizin?
Imajin, yu tek insulin injɛkshɔn na nɛt, ɔ yu bin de du ɛksɛsayz bifo yu it dina. Fɔ dis rizin, na di midul nɛt we yu de slip, we yu nɔ ivin no, yu blɔd shuga lɛvɛl de drɔp denja wan (`haypoglycemia`).
Na dis tɛm, yu bɔdi de tink se, "O, sɔntin bad go apin!" So di bɔdi de tek imejensi akshɔn fɔ "sev" yu. Dat min se, i kin pul sɔm ɔmon dɛn wantɛm wantɛm na di blɔd we kin wok we tin nɔ izi.
- Adrenaline we gɛt di sik
- Kɔtikosterɔyd dɛn
- Ɔmon we de mek pɔsin gro
- Glukagɔn we dɛn kɔl Glucagon
dis כmon dεm de kam togεda fכ sεnd signal to yu liva fכ "Kwik kwik wan rilis shuga insay yu bכdi!" Dɔn di liva kin pul bɔku bɔku glukɔs we dɛn dɔn kip de insay yu blɔd wantɛm wantɛm.
If dis apin to pɔsin we nɔ gɛt dayabitis, in bɔdi kin mek insulin fɔ kɔntrol di shuga we de go ɔp. Bɔt bikɔs yu bɔdi we gɛt dayabitis nɔ kin ebul fɔ du dat, di shuga we de bɔku kin jɔs kɔntinyu fɔ go ɔp, ɛn we yu wek na mɔnin, yu kin si se yu gɛt bɔku shuga (haypa glycemia) . Na dat kin apin, kwik kwik wan.
Aw ɛksaktɔli yu kin si dis sityueshɔn?
Bikɔs bɔku rizin dɛn de fɔ mek yu gɛt bɔku blɔd shuga na mɔnin, i kin at smɔl fɔ no if dis na di Somogyi ifɛkt. Dɔn bak, lɛk aw wi bin dɔn tɔk, bikɔs dɛn nɔ kin tek dis as sɔntin we rili kɔmɔn igen, i kin bi chalenj ivin fɔ dɔktɔ fɔ "diagnose" am.
Di bɛst tin fɔ du, if yu kɔntinyu fɔ gɛt ay blɔd shuga na mɔnin, na fɔ tɔk to yu dayabitis dɔktɔ bɔt am. I go tray fɔ no di rizin bay we i de luk aw yu blɔd shuga de chenj.
Di bɛst we fɔ du dis na fɔ yuz wan Kɔntinyu Glukɔs Monitoring (CGM) divays. Dis kin mek yu si klia grafik fɔ aw yu blɔd shuga dɔn chenj ɔl di nɛt.
If yu nɔ de yuz CGM, yu dɔktɔ kin tɛl yu fɔ du sɔntin lɛk dis:
| Tɛm | Wetin fɔ du |
|---|---|
| Tu awa afta dɛn dɔn it ivintɛm it | Mekɔp ɛn rayt yu blɔd shuga lɛvɛl. |
| Bifo yu go slip | Mekɔp yu blɔd shuga lɛvɛl bak ɛn rayt am. |
| Na di midul nɛt (e.g. 2-3 am) . | Set alam, grap, chɛk yu blɔd shuga lɛvɛl ɛn rayt am dɔŋ. (Dis na di tɛst we impɔtant pas ɔl.) |
| We yu wek na mɔnin | Mekɔp yu blɔd shuga lɛvɛl wan las tɛm ɛn rayt am. |
If yu du dis fɔ sɔm dez ɛn sho di tin dɛn we yu dɔn rid to yu dɔktɔ, i go ebul fɔ no gud gud wan bɔt wetin de apin na nɛt. If yu blɔd shuga smɔl na midul nɛt ɛn ay na mɔnin, i kin bi di Somogyi ifɛkt.
Us sɔlv dɛn kin du fɔ dis?
We yu dɔn no di rizin, yu ɛn yu dɔktɔ fɔ wok togɛda fɔ fɛn sɔlv. Ɔlman difrɛn, so no wan saiz-fit-ɔl sɔlv nɔ de. I kin tek sɔm tɛm fɔ fɛn wetin go wok fɔ yu.
Wonin: Nɔ ɛva chenj yu insulin doz ɔ ɔda mɛrɛsin fɔ dayabitis if yu nɔ go to yu dɔktɔ fɔs. If yu du dat, dat kin mek yu blɔd shuga go dɔŋ ɔ i go bɔku.
Yu dɔktɔ kin tɛl yu fɔ sɔlv prɔblɛm dɛn lɛk dɛn tin ya:
- Chenj yu insulin ɔ mɛrɛsin doz: Dɛn kin aks yu fɔ ridyus yu insulin doz smɔl na nɛt.
- Chenj yu dina: Dɛn kin advays yu fɔ chenj di kabɔhaydrɛt, prɔtin, ɔ fat we yu de it fɔ dina. Dɛn kin advays yu bak fɔ it smɔl snek bifo yu go slip.
- Fɔ chenj to insulin Pɔmp: If i pɔsibul, insulin pɔmp kin ɛp fɔ kɔntrol di insulin doz dɛn kɔrɛkt wan.
- Chenj di tɛm we yu de wok: If yu de ɛksesaiz na ivintɛm ɔ na nɛt, yu go want fɔ chenj di tɛm ɔ di kayn we aw yu de wok.
Wetin na di prɔblɛm dɛn we kin apin if dɛn nɔ tek dis?
I nɔ mata if di Somogyi ifɛkt rili apin ɔ nɔ apin, dis sityueshɔn de rayz tu men pɔynt dɛm we wi wit dayabitis fɔ no bɔt:
1. Kɔntinyu fɔ mek di blɔd shuga go ɔp.
2. Risk fɔ mek yu blɔd shuga nɔ bɔku na nɛt.
1. Kɔntinyu fɔ mek di blɔd shuga go ɔp
Fɔ gɛt ay blɔd shuga lɛvɛl fɔ sɔm awa ɛvri mɔnin nɔr fayn fɔ yu ɔl yu shuga mɛnejɛmɛnt. I kin inkrisayz yu `A1C` valyu. `A1C` na tɛst we de gi yu aydia bɔt yu avɛrej blɔd shuga lɛvɛl fɔ di pas tri mɔnt. If yu gɛt ay `A1C` valyu fɔ lɔng tɛm i kin mek yu gɛt kɔmplikeshɔn frɔm dayabitis.
- Ay damej (Retinopathy) .
- Kidni we de pwɛl (Nephropathy) .
- Nyuropati (we de afɛkt di nerv dɛn) .
- At sik
2. Risk fɔ mek yu blɔd shuga nɔ bɔku na nɛt
Bɔrku pipul dɛn kin gɛt sayn dɛm fɔ haypoglycemia we dɛn blɔd shuga kin drɔp dɔŋ 70 mg/dL. Dɛn kin gɛt shek shek, angri, ɛn swet. Bɔt if dis kin apin we yu de slip, i kin dɔn let fɔ notis di sayn dɛm ɛn wek yu. Siriɔs haypoglycemia kin mek pɔsin in layf de pan denja.
If bɔku tɛm yu kin gɛt smɔl blɔd shuga na nɛt, mek shɔ se yu tɔk to yu dɔktɔ bɔt dat. Yu kin nid fɔ chenj yu tritmɛnt plan fɔ mek i nɔ apin.
Ustɛm yu fɔ rili go to dɔktɔ?
If yu kin gɛt ay blɔd shuga ɔltɛm we yu wek na mɔnin, nɔ ignore am. I impɔtant fɔ tɛl yu dɔktɔ we gɛt dayabitis. I kin luk yu ridin, no if di kɔz na di Somogyi ifɛkt, di Dawn phenomenon, ɔ ɔda tin, ɛn gi yu di bɛst tritmɛnt fɔ yu.
If yu gɛt bɔku blɔd shuga na mɔnin, dat kin rili mek yu at pwɛl. Bɔt bikɔs no wan say nɔ de fɔ sɔlv di prɔblɛm, i kin tek sɔm tɛm fɔ fɛn di rayt sɔlv fɔ yu. Peshɛnt ɛn wok wit yu dɔktɔ. If yu tek akshɔn naw, dat kin ɛp yu fɔ avɔyd prɔblɛm dɛn we go mit yu fɔ shɔt tɛm ɛn fɔ lɔng tɛm.
Mɛsej we dɛn kin kɛr go na os
- If yu gɛt dayabitis, bɔku rizin dɛn de we mek yu blɔd shuga kin bɔku na mɔnin. Di Somogyi Effect na wan pan dɛn.
- di Somogyi ifekt de apin as di bכdi in rεspכns to di lכw bכdi shuga lεvεl (hypoglycemia) insay di nεt.
- Yu kin saspek dis if yu gɛt ay blɔd shuga na mɔnin wit tin dɛm lɛk fɔ swet na nɛt ɛn fɔ drim bad.
- Di bɛst we fɔ no di rayt tin we mek yu gɛt am na fɔ mɛzhɔ di shuga ivin midul nɛt ɔ yuz CGM mashin.
- Nɔ ɛva chenj yu insulin ɔ dayabitis mɛrɛsin fɔ yusɛf we yu nɔ aks yu dɔktɔ.
- Yu kin kɔntrol dis sik fayn fayn wan bay we yu nɔr de wɔri bɔt am, bɔt yu kin tɔk to yu dɔktɔ ɛn gɛt di rayt tritmɛnt plan.
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