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Wetin na glukɔs? Aw i gɛt fɔ du wit dayabitis?

Wetin na glukɔs? Aw i gɛt fɔ du wit dayabitis?

We yu de tɔk bɔt dayabitis, yu go mɔs de yɛri di wɔd dɛn ‘Blɔd Shuga’ ɔ ‘Glukɔs’ ɔltɛm, nɔto so? I rili impɔtant fɔ no gud gud wan wetin na glukɔs, di ɛnaji we wi bɔdi de gɛt frɔm di it dɛn we wi de it, mɔ if yu gɛt dayabitis ɔ yu de pan risk fɔ gɛt dayabitis. So tide, mek wi tok abaut am simpul, A to Z.

Wetin na Glukɔs ɛn Insulin?

Fɔ tɔk am simpul wan, glukɔs na wan kayn shuga . Wi kin gɛt am frɔm di it dɛn we wi de it, mɔ di it dɛn we gɛt bɔku kabɔhaydrɛt. Wi bɔdi de yuz dis glukɔs fɔ gi di sɛl dɛn ɛnaji. we dis glukoz de travul tru di bכdi, wi kin kכl am "blכd shuga" כ 'blכd shuga'.

Naw na dis say di wok we insulin de du de kam insay, insulin na ɔmon we dɛn kin mek na wi pankrias. Lɛk ki, dis insulin na in de opin di sɛl dɛn na wi bɔdi ɛn ɛp di glukɔs we de na di blɔd fɔ go insay dɛn sɛl dɛn de ɛn mek ɛnaji.

If dɛn dɔn no se yu gɛt Dayabitis Mɛlitɔs, dat min se yu blɔd glukɔs lɛvɛl pas aw i fɔ bi. Tu rizin dɛn de we kin mek wi du dis:

1. Either yu bכdi nכ de prodyuz inof insulin fכ muv di glukכs we de insay di bכdi insay di sεl dεm.

2. Ɔ, pan ɔl we insulin de, yu bɔdi in sɛl dɛn nɔ de ansa am fayn (Insulin Resistance).

If yu gɛt ay glukɔs na yu blɔd fɔ lɔng tɛm, dat kin pwɛl impɔtant pat dɛn lɛk yu kidni, yay, nerv, ɛn at. So i impɔtant fɔ no bɔt dis.

Aw wi kin gɛt glukɔs insay wi bɔdi?

Bɔku pan di it dɛn we wi kin it, mɔ di wan dɛn we gɛt bɔku kabɔhaydrɛt lɛk bred, rays, pɔt, ɛn frut, gɛt glukɔs. afta wi it, di it de go insay di bεlε εn di εnzym dεm εn asid dεm de brok am to sכm sכm pat dεm. dis prכsεs de rilis glukכs.

Dɔn, dis glukɔs kin go na di insay ɛn i kin go insay di blɔd. na da tεm de di כmon insulin de kam insay εn εp fכ sεnd dis glukכs insay di sεl dεm.

Enaji εn stכrej: Wetin kin apin to glukכs insay di bכdi?

Dɛn mek wi bɔdi fɔ mek di glukɔs we de na di blɔd kɔntinyu fɔ de ɔltɛm. we di glukoz na di bכdi go כp afta wi it, spεshal sεl dεm (beta sεl dεm) na di pankrias de mek insulin. Dis insulin de opin di domɔt fɔ mek glukɔs go insay di mɔsul, fat, ɛn liva sɛl dɛn.

Di men fiul fɔ di bren

Pan ɔl we bɔku pan di sɛl dɛn na wi bɔdi de yuz glukɔs, amino asid (di tin dɛn we de mek di prɔtin dɛn), ɛn fat fɔ gɛt ɛnaji, di men tin we wi bren de yuz na glukɔs.Glukose impɔtant fɔ prosɛs infɔmeshɔn fɔ di nɛv sɛl dɛn ɛn di kemikal mɛsenja dɛn na di bren. If glukɔs nɔ de, wi bren nɔ go ebul fɔ wok fayn.

Fɔ kip ɛkstra ɛnaji

Afta di bɔdi dɔn yuz di ɛnaji we i nid, di glukɔs we lɛf de na di liva ɛn di mɔsul dɛn as smɔl smɔl bɔndɛl dɛn we dɛn kɔl glycogen . Wi bɔdi kin kip lɛk di ɛnaji we i nid fɔ wan de dis we.

Imajin se yu nɔ it fɔ sɔm awa. Yu blɔd glukɔs lɛvɛl de go dɔŋ. Da tɛm de, di pankrias kin stɔp fɔ mek insulin ɛn bigin fɔ mek ɔda ɔmon we dɛn kɔl glukagon . dis glukagכn de gi signal fכ di liva fכ kכnvכlt di glycogen we dεn stכr bak to glukכs. Dɔn da glukɔs de go insay di blɔd, ɛn i de gi di bɔdi trɛnk te yu it bak.

Blɔd Glukɔs ɛn Dayabitis

I nɔmal fɔ mek di blɔd shuga go ɔp afta yu dɔn it ɛn afta dat i go fɔdɔm bak afta sɔm awa. di blɔd shuga lɛvɛl we pɔsin we gɛt wɛlbɔdi de fast fɔ smɔl pas 100 mg/dL.

Bɔt if yu gɛt dayabitis, yu bɔdi nɔ go ebul fɔ du dis fayn fayn wan.

Tu men kayn dayabitis de:

  • Tayp 1 Dayabitis: Insay dis kayn dayabitis, di bɔdi in imyun sistɛm kin mistek pwɛl di sɛl dɛn we de mek insulin na di pankrias, ɛn dis kin mek di bɔdi nɔ de mek inof insulin.
  • Tayp 2 Dayabitis: Insay dis sik, di bɔdi in sɛl dɛn nɔ de ansa fayn fayn wan to insulin. dis de mek di pankrias gεt fכ mek mכr εn mכr insulin fכ alaw glukכs fכ go insay di sεl dεm. As tɛm de go, di pankrias kin wik ɛn i nɔ kin ebul fɔ mek insulin.

We insulin nɔ de, di glukɔs kin bɔku na di blɔd instead fɔ go insay di sɛl dɛn. If yu blɔd glukɔs lɛvɛl pas 200 mg/dL 2 awa afta yu it ɔ pas 125 mg/dL we yu de fast, dɛn kin kɔl am haypa glycemia .

Risk fɔ mek yu gɛt ay blɔd shuga fɔ lɔng tɛm
❤️ Hat sik, at atak ɛn strok
Kidni sik
🧠 Nyurolɔjik dizayd
👁️ Retinopathy, na sik we de na di yay

Aw fɔ no di blɔd shuga lɛvɛl?

Di tu we dɛn we dɛn kin yuz mɔ na:

1. Fingastik Tɛst: Dɛn kin tek smɔl blɔd frɔm di tip na di finga bay we dɛn de yuz lans, put am pan tɛst strip, ɛn mɛzhɔ am wit glukɔs mita. Yu kin si di shuga we yu gɛt naw na yu blɔd insay sɔm sɛkɔn.

2. Kɔntinyu Blɔd Glukɔs Monitor (CGM): Dɛn kin put smɔl sɛns ɔnda di skin, bɔku tɛm na di an ɔ di bɛlɛ. I de kɔntinyu fɔ mɛzhɔ yu blɔd shuga lɛvɛl bay we i de wach di wata we de bitwin di sɛl dɛn ɛn transmit di data to wan divays lɛk fon ap.

Wetin na di A1c tɛst?

Di A1c test (HbA1c test) na blɔd tɛst we de sho yu avɛrej blɔd shuga lɛvɛl fɔ di pas 2-3 mɔnt.

We glukɔs go insay di blɔd, i kin tay pan ɛmoglobin, we na di pat pan di rɛd blɔd sɛl dɛn we kin kɛr ɔksijɛn insay di blɔd. di A1c tεst de mכsu aw bכku pan di εmoglobin we de na yu bכdi kכba wit shuga (glycated). Di ay glukɔs na di blɔd, na di mɔ yu A1c valyu go ay.

A1c rizulyt Minin
I nɔ rich 5.7% . Hεlth (nכmal) lεvεl
5,7% - 6,4% . Prediabetes (bifo yu gɛt dayabitis) .
6.5% ɔ mɔ Shuga

Impɔtant:Dɛn valyu ya na jɔs wan jenɛral gayd. I impɔtant fɔ tɔk to yu dɔktɔ fɔ ɔndastand yu rizɔlt ɛn fɔ gɛt kɔrɛkt diagnosis. insay sכm kes dεm (fכ egzampl, sכm jεnεtik kכndishכn dεm), di A1c valyu dεm kin difrεn. If na so i bi, yu dɔktɔ kin tɛl yu fɔ du ɔda tɛst.

Wetin na di tin dɛm we kin mek yu blɔd shuga nɔ bɔku (Hypoglycemia)?

haypoglycemia na wan kכndyushכn we di bכdi shuga lεvεl dכn dכn dכn 70 mg/dL. Dis kin bi kwik kwik wan wit mɛrɛsin. Bɔku rizin dɛn de we mek dis kin apin.

  • Fɔ drink rɔm we yu ɛmti bɛlɛ.
  • Fɔ tek mɔ insulin ɔ ɔda mɛrɛsin fɔ dayabitis pas aw i nid.
  • Fɔ skip fɔ it.
  • Fɔ ɛksesaiz pas aw i kin du.
  • di chenj dεm na di כmon dεm (e.g. di mכnt mεnstrashכn).

Wetin yu kin du we yu shuga lɛvɛl go dɔŋ?

Yu kin fala di "15-15 rule" we di American Diabetes Association dɔn se:

1. It ɔ drink 15 gram fast-aktin kabɔhaydrɛt (e.g. 3 ti spɔnj shuga/glukɔs, 1 tebul spɔnj ɔni).

2. Wet fɔ 15 minit .

3. Chek yu blɔd shuga bak. If i stil de dɔŋ 70 mg/dL, tek ɔda 15 gram kabɔhaydrɛt.

4. Ripit dis te di shuga lɛvɛl kam bak to nɔmal.

Wetin na di tin dɛm wae kin mek pɔrsin gɛt bɔrku shuga na yu blɔd (Hyperglycemia)?

If di blɔd shuga lɛvɛl pas 180 mg/dL, dɛn kɔl am haypa glycemia. Bɔku rizin dɛn de fɔ dis.

  • Nɔ tek insulin ɔ ɔda mɛrɛsin dɛn we go du fɔ yu.
  • Fɔ nɔ ebul fɔ du bɔku tin dɛn fɔ du.
  • Wan sik (e.g. kol, flu) ɔr infekshɔn.
  • Strɛs.
  • Fɔ it mɔ pas aw dɛn bin dɔn plan.

Ustɛm yu fɔ go na di Imejɛnsi Tritmɛnt Yunit (ETU)?

If yu blɔd shuga lɛvɛl na 300 mg/dL ɔ pas dat tu tɛm insay wan row , kɔl yu dɔktɔ wantɛm wantɛm.

If yu nɔ trit yu ay blɔd blɔd, dat kin mek yu gɛt ketoacidosis , ɔ dayabitik kɔma we kin mek yu layf de pan denja. Go to ɔspitul in imejensi dipatmɛnt (ETU) wantɛm wantɛm if yu gɛt ɛni wan pan dɛn sik ya:

  • Wan frut smel pan di briz.
  • Nɔs ɛn vɔmit.
  • Di mɔt we de dray pasmak.
  • I nɔ kin izi fɔ yu fɔ blo.

I rili impɔtant fɔ kip yu blɔd shuga lɛvɛl insay di rayt rɛnj. Du wetin yu dɔktɔ tɛl yu fɔ du. Chek yu blɔd shuga lɛvɛl ɔltɛm. If yu de du ɛksɛsayz, yu de it tin dɛn we go mek yu it fayn, ɛn yu de tek mɛrɛsin dɛn we dɛn gi yu, yu go ebul fɔ liv fayn layf.

Mɛsej we dɛn kin kɛr go na os

  • Glukɔs na di men kayn shuga we de gi wi bɔdi ɛnaji. di כmon insulin de εp fכ muv dis glukכs insay di sεl dεm.
  • Dayabitis na wan sik wae de mek di glukɔs na di blɔd go ɔp. As tɛm de go, dis kin pwɛl di ɔgan dɛn lɛk di kidni, di yay, ɛn di at.
  • If yu gɛt dayabitis, i impɔtant fɔ chɛk yu blɔd shuga ɔltɛm lɛk aw yu dɔktɔ tɛl yu.
  • If yu blɔd shuga nɔ bɔku (haypoglycemia) ɔ yu blɔd shuga (hyperglycemia) ɔl tu kin denja. Yu fɔ no wetin fɔ du pan dɛn tin dɛn de.
  • Di A1c test de sho yu avrej blɔd shuga lɛvɛl fɔ di pas 2-3 mɔnt. Dis kin ɛp yu dɔktɔ fɔ mek wan tritmɛnt plan fɔ yu.

Glukɔs, dayabitis, blɔd shuga, insulin, A1c tɛst, aypa glycemia, haypoglycemia

Frequently Asked Questions (FAQ)

Aw fɔ no di blɔd shuga lɛvɛl?

Di tu we dɛn we dɛn kin yuz mɔ na:

Wetin yu kin du we yu shuga lɛvɛl go dɔŋ?

Yu kin fala di "15-15 rule" we di American Diabetes Association dɔn se:

⚠️ Important: The medical articles and information on Nirogi Lanka are for general awareness only, and are by no means a substitute for professional medical advice, diagnosis, or treatment. For any medical problem you have, consult a qualified physician immediately.

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Wetin na glukɔs? Aw i gɛt fɔ du wit dayabitis?

Wetin na glukɔs? Aw i gɛt fɔ du wit dayabitis?

We yu de tɔk bɔt dayabitis, yu go mɔs de yɛri di wɔd dɛn ‘Blɔd Shuga’ ɔ ‘Glukɔs’ ɔltɛm, nɔto so? I rili impɔtant fɔ no gud gud wan wetin na glukɔs, di ɛnaji we wi bɔdi de gɛt frɔm di it dɛn we wi de it, mɔ if yu gɛt dayabitis ɔ yu de pan risk fɔ gɛt dayabitis. So tide, mek wi tok abaut am simpul, A to Z.

Wetin na Glukɔs ɛn Insulin?

Fɔ tɔk am simpul wan, glukɔs na wan kayn shuga . Wi kin gɛt am frɔm di it dɛn we wi de it, mɔ di it dɛn we gɛt bɔku kabɔhaydrɛt. Wi bɔdi de yuz dis glukɔs fɔ gi di sɛl dɛn ɛnaji. we dis glukoz de travul tru di bכdi, wi kin kכl am "blכd shuga" כ 'blכd shuga'.

Naw na dis say di wok we insulin de du de kam insay, insulin na ɔmon we dɛn kin mek na wi pankrias. Lɛk ki, dis insulin na in de opin di sɛl dɛn na wi bɔdi ɛn ɛp di glukɔs we de na di blɔd fɔ go insay dɛn sɛl dɛn de ɛn mek ɛnaji.

If dɛn dɔn no se yu gɛt Dayabitis Mɛlitɔs, dat min se yu blɔd glukɔs lɛvɛl pas aw i fɔ bi. Tu rizin dɛn de we kin mek wi du dis:

1. Either yu bכdi nכ de prodyuz inof insulin fכ muv di glukכs we de insay di bכdi insay di sεl dεm.

2. Ɔ, pan ɔl we insulin de, yu bɔdi in sɛl dɛn nɔ de ansa am fayn (Insulin Resistance).

If yu gɛt ay glukɔs na yu blɔd fɔ lɔng tɛm, dat kin pwɛl impɔtant pat dɛn lɛk yu kidni, yay, nerv, ɛn at. So i impɔtant fɔ no bɔt dis.

Aw wi kin gɛt glukɔs insay wi bɔdi?

Bɔku pan di it dɛn we wi kin it, mɔ di wan dɛn we gɛt bɔku kabɔhaydrɛt lɛk bred, rays, pɔt, ɛn frut, gɛt glukɔs. afta wi it, di it de go insay di bεlε εn di εnzym dεm εn asid dεm de brok am to sכm sכm pat dεm. dis prכsεs de rilis glukכs.

Dɔn, dis glukɔs kin go na di insay ɛn i kin go insay di blɔd. na da tεm de di כmon insulin de kam insay εn εp fכ sεnd dis glukכs insay di sεl dεm.

Enaji εn stכrej: Wetin kin apin to glukכs insay di bכdi?

Dɛn mek wi bɔdi fɔ mek di glukɔs we de na di blɔd kɔntinyu fɔ de ɔltɛm. we di glukoz na di bכdi go כp afta wi it, spεshal sεl dεm (beta sεl dεm) na di pankrias de mek insulin. Dis insulin de opin di domɔt fɔ mek glukɔs go insay di mɔsul, fat, ɛn liva sɛl dɛn.

Di men fiul fɔ di bren

Pan ɔl we bɔku pan di sɛl dɛn na wi bɔdi de yuz glukɔs, amino asid (di tin dɛn we de mek di prɔtin dɛn), ɛn fat fɔ gɛt ɛnaji, di men tin we wi bren de yuz na glukɔs.Glukose impɔtant fɔ prosɛs infɔmeshɔn fɔ di nɛv sɛl dɛn ɛn di kemikal mɛsenja dɛn na di bren. If glukɔs nɔ de, wi bren nɔ go ebul fɔ wok fayn.

Fɔ kip ɛkstra ɛnaji

Afta di bɔdi dɔn yuz di ɛnaji we i nid, di glukɔs we lɛf de na di liva ɛn di mɔsul dɛn as smɔl smɔl bɔndɛl dɛn we dɛn kɔl glycogen . Wi bɔdi kin kip lɛk di ɛnaji we i nid fɔ wan de dis we.

Imajin se yu nɔ it fɔ sɔm awa. Yu blɔd glukɔs lɛvɛl de go dɔŋ. Da tɛm de, di pankrias kin stɔp fɔ mek insulin ɛn bigin fɔ mek ɔda ɔmon we dɛn kɔl glukagon . dis glukagכn de gi signal fכ di liva fכ kכnvכlt di glycogen we dεn stכr bak to glukכs. Dɔn da glukɔs de go insay di blɔd, ɛn i de gi di bɔdi trɛnk te yu it bak.

Blɔd Glukɔs ɛn Dayabitis

I nɔmal fɔ mek di blɔd shuga go ɔp afta yu dɔn it ɛn afta dat i go fɔdɔm bak afta sɔm awa. di blɔd shuga lɛvɛl we pɔsin we gɛt wɛlbɔdi de fast fɔ smɔl pas 100 mg/dL.

Bɔt if yu gɛt dayabitis, yu bɔdi nɔ go ebul fɔ du dis fayn fayn wan.

Tu men kayn dayabitis de:

  • Tayp 1 Dayabitis: Insay dis kayn dayabitis, di bɔdi in imyun sistɛm kin mistek pwɛl di sɛl dɛn we de mek insulin na di pankrias, ɛn dis kin mek di bɔdi nɔ de mek inof insulin.
  • Tayp 2 Dayabitis: Insay dis sik, di bɔdi in sɛl dɛn nɔ de ansa fayn fayn wan to insulin. dis de mek di pankrias gεt fכ mek mכr εn mכr insulin fכ alaw glukכs fכ go insay di sεl dεm. As tɛm de go, di pankrias kin wik ɛn i nɔ kin ebul fɔ mek insulin.

We insulin nɔ de, di glukɔs kin bɔku na di blɔd instead fɔ go insay di sɛl dɛn. If yu blɔd glukɔs lɛvɛl pas 200 mg/dL 2 awa afta yu it ɔ pas 125 mg/dL we yu de fast, dɛn kin kɔl am haypa glycemia .

Risk fɔ mek yu gɛt ay blɔd shuga fɔ lɔng tɛm
❤️ Hat sik, at atak ɛn strok
Kidni sik
🧠 Nyurolɔjik dizayd
👁️ Retinopathy, na sik we de na di yay

Aw fɔ no di blɔd shuga lɛvɛl?

Di tu we dɛn we dɛn kin yuz mɔ na:

1. Fingastik Tɛst: Dɛn kin tek smɔl blɔd frɔm di tip na di finga bay we dɛn de yuz lans, put am pan tɛst strip, ɛn mɛzhɔ am wit glukɔs mita. Yu kin si di shuga we yu gɛt naw na yu blɔd insay sɔm sɛkɔn.

2. Kɔntinyu Blɔd Glukɔs Monitor (CGM): Dɛn kin put smɔl sɛns ɔnda di skin, bɔku tɛm na di an ɔ di bɛlɛ. I de kɔntinyu fɔ mɛzhɔ yu blɔd shuga lɛvɛl bay we i de wach di wata we de bitwin di sɛl dɛn ɛn transmit di data to wan divays lɛk fon ap.

Wetin na di A1c tɛst?

Di A1c test (HbA1c test) na blɔd tɛst we de sho yu avɛrej blɔd shuga lɛvɛl fɔ di pas 2-3 mɔnt.

We glukɔs go insay di blɔd, i kin tay pan ɛmoglobin, we na di pat pan di rɛd blɔd sɛl dɛn we kin kɛr ɔksijɛn insay di blɔd. di A1c tεst de mכsu aw bכku pan di εmoglobin we de na yu bכdi kכba wit shuga (glycated). Di ay glukɔs na di blɔd, na di mɔ yu A1c valyu go ay.

A1c rizulyt Minin
I nɔ rich 5.7% . Hεlth (nכmal) lεvεl
5,7% - 6,4% . Prediabetes (bifo yu gɛt dayabitis) .
6.5% ɔ mɔ Shuga

Impɔtant:Dɛn valyu ya na jɔs wan jenɛral gayd. I impɔtant fɔ tɔk to yu dɔktɔ fɔ ɔndastand yu rizɔlt ɛn fɔ gɛt kɔrɛkt diagnosis. insay sכm kes dεm (fכ egzampl, sכm jεnεtik kכndishכn dεm), di A1c valyu dεm kin difrεn. If na so i bi, yu dɔktɔ kin tɛl yu fɔ du ɔda tɛst.

Wetin na di tin dɛm we kin mek yu blɔd shuga nɔ bɔku (Hypoglycemia)?

haypoglycemia na wan kכndyushכn we di bכdi shuga lεvεl dכn dכn dכn 70 mg/dL. Dis kin bi kwik kwik wan wit mɛrɛsin. Bɔku rizin dɛn de we mek dis kin apin.

  • Fɔ drink rɔm we yu ɛmti bɛlɛ.
  • Fɔ tek mɔ insulin ɔ ɔda mɛrɛsin fɔ dayabitis pas aw i nid.
  • Fɔ skip fɔ it.
  • Fɔ ɛksesaiz pas aw i kin du.
  • di chenj dεm na di כmon dεm (e.g. di mכnt mεnstrashכn).

Wetin yu kin du we yu shuga lɛvɛl go dɔŋ?

Yu kin fala di "15-15 rule" we di American Diabetes Association dɔn se:

1. It ɔ drink 15 gram fast-aktin kabɔhaydrɛt (e.g. 3 ti spɔnj shuga/glukɔs, 1 tebul spɔnj ɔni).

2. Wet fɔ 15 minit .

3. Chek yu blɔd shuga bak. If i stil de dɔŋ 70 mg/dL, tek ɔda 15 gram kabɔhaydrɛt.

4. Ripit dis te di shuga lɛvɛl kam bak to nɔmal.

Wetin na di tin dɛm wae kin mek pɔrsin gɛt bɔrku shuga na yu blɔd (Hyperglycemia)?

If di blɔd shuga lɛvɛl pas 180 mg/dL, dɛn kɔl am haypa glycemia. Bɔku rizin dɛn de fɔ dis.

  • Nɔ tek insulin ɔ ɔda mɛrɛsin dɛn we go du fɔ yu.
  • Fɔ nɔ ebul fɔ du bɔku tin dɛn fɔ du.
  • Wan sik (e.g. kol, flu) ɔr infekshɔn.
  • Strɛs.
  • Fɔ it mɔ pas aw dɛn bin dɔn plan.

Ustɛm yu fɔ go na di Imejɛnsi Tritmɛnt Yunit (ETU)?

If yu blɔd shuga lɛvɛl na 300 mg/dL ɔ pas dat tu tɛm insay wan row , kɔl yu dɔktɔ wantɛm wantɛm.

If yu nɔ trit yu ay blɔd blɔd, dat kin mek yu gɛt ketoacidosis , ɔ dayabitik kɔma we kin mek yu layf de pan denja. Go to ɔspitul in imejensi dipatmɛnt (ETU) wantɛm wantɛm if yu gɛt ɛni wan pan dɛn sik ya:

  • Wan frut smel pan di briz.
  • Nɔs ɛn vɔmit.
  • Di mɔt we de dray pasmak.
  • I nɔ kin izi fɔ yu fɔ blo.

I rili impɔtant fɔ kip yu blɔd shuga lɛvɛl insay di rayt rɛnj. Du wetin yu dɔktɔ tɛl yu fɔ du. Chek yu blɔd shuga lɛvɛl ɔltɛm. If yu de du ɛksɛsayz, yu de it tin dɛn we go mek yu it fayn, ɛn yu de tek mɛrɛsin dɛn we dɛn gi yu, yu go ebul fɔ liv fayn layf.

Mɛsej we dɛn kin kɛr go na os

  • Glukɔs na di men kayn shuga we de gi wi bɔdi ɛnaji. di כmon insulin de εp fכ muv dis glukכs insay di sεl dεm.
  • Dayabitis na wan sik wae de mek di glukɔs na di blɔd go ɔp. As tɛm de go, dis kin pwɛl di ɔgan dɛn lɛk di kidni, di yay, ɛn di at.
  • If yu gɛt dayabitis, i impɔtant fɔ chɛk yu blɔd shuga ɔltɛm lɛk aw yu dɔktɔ tɛl yu.
  • If yu blɔd shuga nɔ bɔku (haypoglycemia) ɔ yu blɔd shuga (hyperglycemia) ɔl tu kin denja. Yu fɔ no wetin fɔ du pan dɛn tin dɛn de.
  • Di A1c test de sho yu avrej blɔd shuga lɛvɛl fɔ di pas 2-3 mɔnt. Dis kin ɛp yu dɔktɔ fɔ mek wan tritmɛnt plan fɔ yu.

Glukɔs, dayabitis, blɔd shuga, insulin, A1c tɛst, aypa glycemia, haypoglycemia

Frequently Asked Questions (FAQ)

Aw fɔ no di blɔd shuga lɛvɛl?

Di tu we dɛn we dɛn kin yuz mɔ na:

Wetin yu kin du we yu shuga lɛvɛl go dɔŋ?

Yu kin fala di "15-15 rule" we di American Diabetes Association dɔn se:

⚠️ Important: The medical articles and information on Nirogi Lanka are for general awareness only, and are by no means a substitute for professional medical advice, diagnosis, or treatment. For any medical problem you have, consult a qualified physician immediately.

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