Yu gɛt dayabitis? Ɔ yu mama, papa, ɔ ɔda pɔsin na yu famili gɛt dayabitis? Dɔn dis tɔpik we wi de tɔk bɔt tide go rili impɔtant to yu. Dayabitis na wan sik wae yu kin ebul fɔ kɔntrol fayn fayn wan, ɛn yu kin liv nɔrmal layf. Bɔt if dɛn nɔ kɔntrol am fayn, i kin mek i gɛt siriɔs imejensi we kin ivin put in layf pan denja. Dat na di imejensi we denja pas ɔl we wi de tɔk bɔt tide. Dɛn kɔl dis Hyperosmolar Hyperglycemic State , we wi kin kɔl HHS fɔ shɔt tɛm.
Fɔ tɔk am simpul wan, wetin na HHS?
Okay, mek wi put dis rili simpul. Imajin se yu blɔd shuga lɛvɛl (blɔd glukɔs) go so ay dat i nɔ pɔsibul fɔ kɔntrol am. we yu bכdi shuga lεvεl go pas 180 mg/dL, wi kidni dεm fכ pul da εksyכs shuga de insay di urine. Bɔt dis shuga nɔ jɔs de kɔmɔt fɔ insɛf, i de tek di bɔdi in valyu wata bak.
Naw, wetin go apin if yu blɔd shuga lɛvɛl go to wan rili ay lɛvɛl, lɛk 600 mg/dL, ɛn i de so fɔ sɔm dez, sɔntɛm wik? Yu kin bigin fɔ pis pasmak, ɛn yu bɔdi kin gɛt wata bad bad wan. As di wata we de na yu bɔdi de go dɔŋ, yu blɔd de tik smɔl smɔl. Jɔs lɛk we wi de mek ɔni, wata kin tik. We wi de tɔk bɔt mɛrɛsin, wi kin kɔl dis tik we di blɔd de tik hyperosmolarity .
So, HHS na imejensi we de mek pɔsin in layf de pan denja we kin apin we dɛn tri tin ya kam togɛda: di shuga we de na di blɔd we rili ay (haypa glycemia) , di wata we de kɔmɔt na di bɔdi bad bad wan , ɛn di haypa ɔsmolariti. Dɛn kin si dis sik mɔ pan pipul dɛm wae gɛt Tayp 2 Dayabitis. Dis na wan sik wae nid fɔ go na ɔspitul kwik kwik wan ɛn trit am. If dɛn nɔ trit am, i kin ivin mek pɔsin day.
HHS ɛn DKA: Wetin na di difrɛns bitwin di tu?
Wan ɔda denja imejensi we kin apin to pipul dɛn we gɛt dayabitis na DKA (Diabetic Ketoacidosis). Pan ɔl we HHS ɛn DKA ɔl tu na tin dɛn we kin mek pɔsin in layf de pan denja bikɔs ɔf di blɔd shuga we bɔku, klia difrɛns de bitwin dɛn tu. I impɔtant fɔ no dis difrɛns.
- DKA (Diabetic Ketoacidosis): Dis kin apin mɔ pan pipul dɛm wae gɛt Tayp 1 Dayabitis. Wetin kin apin ya na dat di bɔdi nɔ gɛt insulin ɔmon. Bikɔs insulin nɔ de, di bɔdi in sɛl dɛn nɔ kin ebul fɔ yuz shuga fɔ mek ɛnaji. Dɔn di bɔdi kin go ɔda we fɔ fɛn ɛnaji. Dat min se na bay we dɛn de bɔn di fat we de na di bɔdi. We dɛn bɔn fat dis kayn we, dɛn kin ad wan kemikal we dɛn kɔl ketɔn to di blɔd as bayprodukt. Dɛn ketɔn ya na asid. we di kεtכn dεm de bכku na di bכdi, di bכdi kin bi asid. Dis na di tin we denja pas ɔl bɔt DKA.
- HHS (Hayperosmolar Haypaglaysemik Stet): .Dis kin apin mɔ pan pipul dɛm wae gɛt tayp 2 dayabitis. Na ya, bɔku bɔku insulin de na di bɔdi. Bikɔs ɔf da smɔl insulin de, di bɔdi de bɔn fat ɛn i nɔ de mek di ketɔn dɛn. So, di blɔd nɔ kin bi asid na HHS. Bɔt di men prɔblɛm we de ya na di we aw di shuga de go ɔp we pɔsin nɔ ebul fɔ kɔntrol ɛn di bad bad wata we de kɔmɔt na di blɔd ɛn di blɔd de tik we kin apin.
Lɛ wi luk dis tebul fɔ mek wi no mɔ bɔt dis difrɛns.
| Tin | HHS (Hayperosmolar Haypaglaysemik Stet) . | DKA (Dayabɛtik Kɛtoasidɔs) . |
|---|---|---|
| Di wan dɛn we dɛn kin si mɔ | Na pipul dɛm wae gɛt Tayp 2 Dayabitis, mɔr di wan dɛm wae dɔn pas 65 ia. | Na di wan dɛn we gɛt Tayp 1 Dayabitis. |
| Di blɔd shuga lɛvɛl | I rili ay (bɔku tɛm >600 mg/dL). | I ay (bɔku tɛm >250 mg/dL), bɔt i nɔ kin ay lɛk HHS. |
| Prɛzɛns fɔ di ketɔn dɛn | Nɔ, ɔ i rili smɔl. | I de insay bɔku bɔku wan na di blɔd ɛn urine. |
| Di asid we de na di blɔd | Di blɔd nɔ kin bi asid. | Di blɔd kin bi asid we denja. |
| Men prɔblɛm | Di wata we de kɔmɔt na di bɔdi bad bad wan ɛn di blɔd de tik (hyperosmolarity). | Asidכsis na di bכdi bikoz fכ di kεtכn dεm (Ketoacidosis). |
Wetin na di sayn dɛm fɔ HHS? Wach ɔt fɔ dɛn tin ya!
Di tin we denja pas ɔl bɔt HHS na dat di sayn dɛm nɔ kin apin wan tɛm . Dɛn kin divɛlɔp smɔl smɔl, fɔ sɔm dez ɔr wik. So pɔsin nɔ go no am te i rili siriɔs. Na dat mek i impɔtant fɔ no bɔt dɛn sik ya.
- di blכd shuga lεvεl we rili hכy: Na di hכm glukכs mita ridin we hכy pas 600 mg/dL (כ 33 mmol/L).
- Kɔnfyushɔn na yu maynd: I nɔ kin no natin, i nɔ kin ebul fɔ pe atɛnshɔn, i kin kɔnfyus we i de tɔk, i nɔ kin gɛt tɛm, ples, ɛn pipul dɛn we i sabi.
- Fɔ si ɔ yɛri tin dɛn we nɔ de (Hallucinations): Dɛn kin se yu de si ɔ yɛri tin dɛn we nɔ rili de.
- Nɔr kin no natin: If i sik bad bad wan, i kin kam wit kɔma.
- Dray mɔt, tɔŋ, ɛn tɔsti pasmak: Na filin fɔ tɔsti we nɔ de go ilɛksɛf yu drink bɔku wata.
- Yu kin pis bɔku tɛm: Pan ɔl we yu kin pis pasmak fɔs, yu kin pis smɔl as yu nɔ gɛt wata na yu bɔdi i kin rili bad.
- Yu nɔ de si fayn ɔ yu nɔ de si fayn: Smɔl smɔl yu nɔ de si fayn.
- Bɔdi wik: Na filin fɔ wikɛd na di bɔdi. Sɔntɛnde, wan say na di bɔdi kin paralayz, lɛk se i gɛt strok.
If yu ɔ sɔmbɔdi na yu famili gɛt ɛni wan pan dɛn sik ya, duya nɔ wet igen. Nɔ tink se na nɔmal tin. Mek dɛn go na di ɔspitul Imejɛnsi Dipatmɛnt (ETU) we de nia yu kwik kwik wan. Dis kin bi tin we go mek pɔsin day ɔ day.
Wetin mek dis HHS kɔndishɔn kin apin? Wetin na di men tin dɛn we kin mek pɔsin gɛt dis sik?
Pɔsin we gɛt dayabitis we dɛn kin kɔntrol fayn fayn wan nɔ kin gɛt HHS. Bɔt pan pɔrsin wae nɔr kin kɔntrol in dayabitis fayn fayn wan, dis kin apin bikɔs ɔf wan "trigger." Dat min se, wan tin we apin kin mek di blɔd shuga lɛvɛl tumɔs fɔ mek dɛn kɔntrol, we kin mek dɛn gɛt HHS.
Di men tin dɛn we kin mek pɔsin gɛt HHS na:
- Infεkshכn: 50% to 60% pan di pipul dεm we gεt HHS gεt sכm kayn infεkshכn. Ɛspɛshali nyumonia , urinary tract infection (UTI ), ɛn sɛpsis .) infεkshכn na di men wan dεm. Imajin, wan ol mama gɛt dayabitis, i kin gɛt infɛkshɔn na in chɛst wit bɔku bɔku mucus. Di strɛs we dis infɛkshɔn kin mek kin mek di blɔd shuga go ɔp to di lɛvɛl we pɔsin nɔ kin ebul fɔ kɔntrol.
- Nɔ tek yu dayabitis mɛrɛsin lɛk aw dɛn tɛl yu fɔ tek am: Dis na rizin bak we kin rili apin. Sɔm pipul dɛn kin bost ɛn stɔp fɔ tek dɛn mɛrɛsin, ɔ dɛn nɔ kin tek am di rayt tɛm. If yu tek insulin, if yu nɔ tek am fayn, yu blɔd shuga lɛvɛl kin go ɔp bad bad wan.
- Ɔda mɛrɛsin dɛn: Sɔm mɛrɛsin dɛn lɛk kɔtikosterɔyd (we dɛn kin yuz fɔ mɛn asma ɛn at at), thiazide diuretics (we dɛn kin yuz fɔ mɛn ay blɔd prɛshɔn), ɛn sɔm mɛrɛsin dɛn fɔ mek pɔsin gɛt maynd sik, kin mek bak di blɔd shuga bɔku ɛn mek i gɛt HHS.
- Di imejensi we kin kam pan di at ɛn di blɔd: If siriɔs tin apin lɛk strok , at atak , ɔ pulmonary embolism , di bɔdi kin gɛt bɔku strɛs. Di strɛs ɔmon dɛm we di bɔdi kin pul dis tɛm kin mek di blɔd shuga go ɔp wantɛm wantɛm.
Udat de pan ay risk fɔ gɛt HHS?
As wi bin dɔn tɔk, dis risk nɔ kin bɔku if dɛn kɔntrol dayabitis fayn fayn wan. Bɔt dɛn pipul ya kin gɛt bɔku risk fɔ gɛt HHS:
- Pipul dɛn we nɔ de kɔntrol dayabitis fayn fayn wan.
- Big pipul dɛn we dɔn pas 65 ia: As dɛn de ol, dɛn nɔ kin ebul fɔ no se dɛn tɔsti, so dɛn nɔ kin no se dɛn nɔ gɛt wata na dɛn bɔdi.
- Dɛn wan wae gɛt ɔda sik (infɛkshɔn, sik) ɔr at sik.
Us kɔmplikɛshɔn kin apin bikɔs ɔf HHS?
If dɛn nɔ trit HHS insay di tɛm, i kin mek i gɛt siriɔs prɔblɛm dɛn.
- Di sik dɛn we kin mek pɔsin sik
- Kɔma
- Ɔgan dɛn we nɔ de wok fayn
- Day
Bitwin 10% ɛn 20% pan di pipul dɛm wae gɛt HHS kin day. Dat min se pan ɛvri 10 pipul dɛn, wan ɔ tu pipul dɛn go day. Na so dis kɔndishɔn siriɔs.
Aw dɛn kin no ɛn trit HHS na ɔspitul?
We dɛn admit yu na di Imejɛnsi Dipatmɛnt (ETU), dɔktɔ go chɛk yu ɛn aks yu bɔt di sik dɛn we yu gɛt. Dɔn, dɛn go ɔda fɔ mek dɛn du bɔku blɔd tɛst. Dɛn tin ya go chɛk yu blɔd shuga lɛvɛl , blɔd osmolality, kidni wok, ɛn if ketɔn dɛn de na yu blɔd.
If di blɔd shuga lɛvɛl pas 600 mg/dL ɛn di blɔd ketɔn lɛvɛl smɔl, dɛn kin no se na HHS.
Di tritmɛnt na fɔ kip yu na wɔd ɔ intensiv kia yunit (ICU) ɛn gi yu IV wata.
Tri men gol dɛn de fɔ tritmɛnt:
1. Rihaydreshɔn: Dɛn kin gi salin insay di bɛlɛ fɔ mek yu nɔ gɛt wata.
2. Balancing Electrolytes: We yu nɔ gɛt wata na yu bɔdi, bɔku valyu minral dɛm lɛk potassium de lɔs frɔm di bɔdi. Dɛn kin riples dɛn tru salin.
3. Fɔ kɔntrol di blɔd shuga lɛvɛl: Dɛn kin gi insulin insay di bɛlɛ fɔ mek di blɔd shuga lɛvɛl smɔl smɔl ɛn kɔntrol am.
Apat frɔm dɛn tritmɛnt ya, if ɔndalayn infɛkshɔn de we mek di HHS kɔndishɔn, dɛn kin trit dat bak.
Yu tink se kɔmplikeshɔn kin apin we dɛn de trit am?
Yɛs, pan ɔl we i nɔ kin apin so ɔltɛm, prɔblɛm kin apin we pɔsin gɛt tritmɛnt. Dat min se if di blɔd shuga lɛvɛl go dɔŋ wantɛm wantɛm ɛn kwik kwik wan, di bren sɛl dɛn kin ful-ɔp wit wata wantɛm wantɛm ɛn di bren kin swel (sɛribra ɛdima). Bɔt dis nɔ kin apin so ɔltɛm. Dɔktɔ dɛn no aw fɔ mek di blɔd shuga go dɔŋ sef wan ɛn sloslo. So nid nɔ de fɔ wɔri bɔt am.
Aw fɔ protɛkt yusɛf frɔm HHS?
Di bɛst ɛn wangren we fɔ avɔyd dis denja na fɔ kɔntrol yu dayabitis fayn fayn wan. Fɔ du dis, mek shɔ se yu du dɛn tin ya.
- Chek yu blɔd shuga lɛvɛl ɔltɛm ɛn tray fɔ kip am insay di target rɛnj we yu dɔktɔ dɔn gi yu.
- Tek di mɛrɛsin (pil ɔ insulin) we yu dɔktɔ tɛl yu fɔ tek di rayt tɛm ɛn di rayt doz, ɛn nɔ mis wan de.
- If i nɔ izi fɔ yu fɔ kɔntrol yu dayabitis, tɔk to yu dɔktɔ bɔt am ɛn gɛt di advays we yu nid.
- Fɔ fala di it we fayn fɔ yu.
- We yu gɛt sik lɛk kol ɔ flu, tek tɛm pasmak. Drink bɔku wata, rɛst bɔku, ɛn chɛk yu blɔd shuga bɔku tɛm pas aw yu kin du am. Dis na bikɔs di strɛs we de pan yu bɔdi we yu sik kin mek yu blɔd shuga go ɔp.
- Yu fɔ no di sayn dɛm fɔ HHS. If yu notis ɛni wan pan dɛn sayn ya, tek akshɔn wantɛm wantɛm.
Mɛsej we dɛn kin kɛr go na os
- HHS na siriɔs, layf de pan denja mɛdikal imejensi wae kin apun pan pipul dɛm wae gɛt shuga.
- Di men rizin fɔ dis na di ay blɔd shuga lɛvɛl we dɛn nɔ kin ebul fɔ kɔntrol ɛn di bad bad wata we kin kɔmɔt na di bɔdi we kin mek i nɔ gɛt wata.
- Kɔnfyushɔn na yu maynd, yu tɔsti pasmak, ɛn yu gɛt bɔku bɔku shuga (we pas 600 mg/dL) na di men sayn dɛm.
- Bɔrku tɛm, HHS kin kam wae pɔrsin gɛt infekshɔn ɔr wae dɛn stɔp fɔ tek dayabitis mɛrɛsin.
- Di bɛst we fɔ avɔyd dis na fɔ kɔntrol yu dayabitis gud gud wan.
- If yu tink se yu ɔ pɔsin we yu sabi gɛt di sayn dɛm fɔ HHS, go na ɔspitul in Imejɛnsi Dipatmɛnt (ETU) wantɛm wantɛm. Taym na di men tin.
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