Yu gɛt bɔku bɔku triglisɛrɛyd na yu blɔd? Lɛ wi tɔk bɔt haypa triglisɛridemia!

Yu dɔn ɛva yɛri bɔt blɔd fat? Sɔntɛm dɔktɔ dɔn tɛl yu se "i tan lɛk se triglisɛrɛyd dɛn ay smɔl." Tide wi go tɔk bɔt wetin dɛn triglisayz ya rili bi, wetin kin apin we dɛn ay, ɛn wetin wi fɔ du bɔt am. Nɔ wɔri, a go ɛksplen dis rili simpul wan, di we we yu go ɔndastand.

Wetin na triglisɛrɛyd? Wetin mek dɛn impɔtant to wi?

Fɔ tɔk am simpul wan, triglisɛrɛyd na wan kayn fat we de na wi blɔd. Dɛn de gi wi bɔdi trɛnk. Wi kin gɛt triglisrayd frɔm di it dɛn we wi de it, mɔ frɔm tin dɛn lɛk bɔta ɛn ɔyl. Nɔto dat nɔmɔ, bɔt we wi it mɔ kalori pas wetin wi bɔdi nid, wi bɔdi kin chenj dɛn ɛkstra kalori dɛn de to triglisrayd ɛn kip dɛn. Leta, we wi nɔ gɛt bɛtɛ trɛnk, wi bɔdi kin yuz dɛn triglisɛrɛyd dɛn ya we dɛn dɔn kip.

So, triglisɛrɛyd na impɔtant tin we de gi wi ɛnaji. Bɔt if dɛn bɔku pasmak na di blɔd, dat kin mek pɔsin gɛt sik dɛn we de mek pɔsin in at ɛn blɔd. Na dat mek i impɔtant fɔ mek wi no bɔt dis sik we dɛn kɔl haypa triglisɛrɛydemia ɛn kɔntrol wi triglisɛrɛyd lɛvɛl.

So, ɔmɔs triglisɛrɛyd fayn?

di avrej adכlt bכdi triglisεrayd lεvεl fכ less dan 150 miligram pan wan dεsilita (150 mg/dL). infakt, less dan 100 mg/dL na di ideal. Di tin we impɔtant pas ɔl na fɔ mɛzhɔ yu triglisrayd lɛvɛl we yu de fast. Dis min se yu gɛt yu blɔd tɛst afta yu nɔ it ɔ drink ɛnitin (eksept wata) fɔ lɛk 10-12 awa so.

haypatriglisεridεmia na we yu triglisεrayd lεvεl dεm na 150 mg/dL כ pas dat.

Na dis na smɔl ɔvaviu fɔ di fasting triglisɛrɛyd lɛvɛl na big pipul dɛn:

• di hεlty lεvεl: less dan 150 mg/dL
• Bɔdalayn ay: 150 – 199 mg/dL
• Hεy lεvεl: 200 – 499 mg/dL
• I rili ay: 500 mg/dL ɔ pas dat

di hεlty triglisεrayd lεvεl fכ pikin dεm εn yכng pipul dεm (we ol 10-19 ia) fכ less dan 90 mg/dL. If yu pikin in triglisrayd lɛvɛl bɔku, i go fayn fɔ mek yu tɔk to dɔktɔ bɔt aw fɔ stɔp am.

Wetin na Sivεr Hypertriglyceridemia? Yu tink se i denja?

Sivεr haypa triglisεridεmia na we yu triglisεrayd lεvεl na 500 mg/dL כ pas dat. Pipul dɛm wae gɛt dis sik kin gɛt mɔr risk fɔ gɛt wan sik wae dɛn kɔl akyu pankriaytis . Dis na inflamɛns na di pankrias we nid fɔ gɛt mɛrɛsin.

Hypertriglyceridemia na wan sik wae nid fɔ pe atɛnshɔn ɛn trit am. Bɔt bɔku tɛm, i nɔ kin bi mɛdikal imejensi.Yu dɔktɔ kin ɛp yu fɔ lɛf dis lɛvɛl. If yu du dat, i go ɛp fɔ mek yu nɔ gɛt prɔblɛm dɛn we go afɛkt yu at ɔ yu pankrias.

Yu tink se aypa triglisɛridemia ɛn kɔlɔstrel na di sem tin?

Yɛs, dɛn tu tin ya nɔto di sem. Hypertriglyceridemia ɛn ay kɔlɔstrel na tu difrɛn tin dɛn. Bɔt dɛn ɔl tu gɛt fɔ du wit di fat tin dɛn we dɛn kɔl lipid we de na yu blɔd.

Lipid na fεt tin dεm we de du imכtant wok dεm na wi bכdi. Triglisεrayd εn kכlestכl na tu kayn lipid dεm. Dɛn tu de travul tru di blɔd insay di pat dɛn we de kɛr di tin dɛn we dɛn kɔl lipoproteins.

di haypa triglisεridεmia na di inkrεs pan di triglisεrayd dεm na di bכdi. Hypercholesterolemia na we di kɔlɔstrel de bɔku.

Bɔku tɛm, pipul dɛn we gɛt ay triglisɛrɛyd kin gɛt ay kɔlɔstrel lɛvɛl bak. di ay triglisεrayd kin bi denja, εspεshali if yu "gud" kכlestכl, HDL (High-Density Lipoprotein), lכw εn yu "bad" kכlestכl, LDL (Low-Density Lipoprotein), hכy.

Tɔk to yu dɔktɔ bɔt wetin dɛn lɛvɛl ya na yu blɔd min ɛn aw fɔ mek dɛn kam bak to wɛlbɔdi lɛvɛl.

Aw dis sik kin kɔmɔn?

Akɔdin to sɔv we dɛn du na Amɛrika, lɛk wan pan ɛvri fayv big pipul dɛn gɛt ay triglisrayd lɛvɛl (mɔ pas 150 mg/dL). Bɔt dis risk kin bɔku we pɔsin de ol. Risach dɔn sho se 42% pan pipul dɛm wae dɔn pas 60 ia gɛt dis sik. Dis sik kin bi kɔmɔn na Sri Lanka bak, bikɔs ɔf di we aw wi de it ɛn di we aw wi de liv wi layf.

Wetin na di sayn dɛm wae de sho se yu gɛt hypertriglyceridemia?

Bɔrku pipul dɛm wae gɛt hypertriglyceridemia nɔr kin sho ɛni sayn. Na dat mek i kin tan lɛk "silent enemy." Pipul wae gɛt siriɔs hypertriglyceridemia kin gɛt wan sik sɔmtɛm wae dɛn kɔl xanthomas. Dis na smɔl smɔl fat we kin mek ɔnda di skin. Dɛn kin si dɛn mɔs arawnd di aylid dɛn. Bɔt sɔntɛnde dɛn kin kam bak na say dɛn lɛk di ni, di ɛlb, ɛn di an.

Wetin mek di triglisɛrɛyd lɛvɛl na wi bɔdi kin go ɔp? Wetin na di rizin dɛn?

Bɔku tin dɛn de we kin mek pɔsin gɛt aypa triglisɛridemia. Dɛn na:

• Di tin dɛn we de mek pɔsin liv in layf
• Ɔda sik dɛn we kin mek pɔsin sik
• Sɔm mɛrɛsin dɛn
• Jɛnɛtik lipid dizayd

Lɛ wi luk ɛni wan pan dɛn tin ya smɔl mɔ.

Di inflɛns we di we aw pɔsin de liv in layf

Di we aw wi de liv wi layf gɛt big impak pan di triglisrayd lɛvɛl we wi gɛt. Bikɔs, lɛk aw a bin dɔn tɔk bifo tɛm, di mɔ we wi de it kalori, na di mɔ wi bɔdi kin chenj to triglisɛrɛyd. So, if yu drink mɔ shuga, fat, ɔ rɔm pas aw yu bɔdi nid, yu triglisrayd lɛvɛl kin go ɔp.

Di tin dɛn we kin mek pɔsin gɛt bɔku triglisrayd:

• Drink tumɔs rɔm: Yu no, sɔm pan wi kin drink smɔl tumɔs we wi de pati. Dat kin afɛkt wi bak.
• Fɔ it pasmak wit rifin kabɔhaydrɛt ɛn shuga: Fɔ it we dɛn mek wit bred flawa (e.g. shɔt bred, bekeri it), drink we dɛn dɔn swit, kek, biskit, ɛn ɔda tin dɛn.
• If yu it bɔku bɔku fat: Dɛn tin ya de na it dɛn we gɛt bɔku fat, rɛd mit, ɛn it dɛn we dɛn mek wit kokonat ɔyl.
• Nɔr de du tin wae nɔr de du natin (sidɔm layf): Sidɔm na wan ples ɔl di de, woke, ɛn nɔr de du ɛksɛsayz.

Jɔs tink bɔt dat, if wi it wan fish we dɛn kuk wit ɔyl, wan brot we dɛn mek wit kokonat milk, ɛn wan swit drink apat frɔm di rays we wi kin it insay wan de, di ɛkstra kalori dɛn we wi kin gɛt frɔm ɔl dat kin de na di bɔdi as triglisrayd.

Bikɔs ɔf sɔm sik dɛn

di triglisεrayd lεvεl kin inkrεs bak bikoz fכ difrεn sik dεm. Na sɔm ɛgzampul dɛn ya:

• Shuga
• Di sik we dɛn kɔl haypotayroyd
• Insulin rɛsistɛns
• Kidni sik
• Liva sik
• Lupus we gɛt di sik
• Mεtabolik sεndrכm
• Nɛfrɔtik sindrom
• We pɔsin wet pasmak ɔ we fat pasmak
• Rimatɔyd at at sik

Uman dɛn gɛt ɔda patikyula tin dɛn we kin mek dɛn gɛt dis sik:

• Menopause we pɔsin kin gɛt
• bεlε - spεshal wan insay di tכd trimεst.

Na bikɔs ɔf sɔm pan di mɛrɛsin dɛn we wi kin tek

Di triglisɛrɛyd lɛvɛl kin go ɔp bak we yu de yuz sɔm mɛrɛsin dɛn. Ɛgzampul dɛn:

• Antisaykotik drɔgs - ɛgz. Klɔzapin, Ɔlanzapin
• Sɔm mɛrɛsin dɛn we dɛn kin yuz fɔ mɛn HIV infɛkshɔn (Antiretroviral (ART) protease inhibitors) .
• Sɔm mɛrɛsin dɛn we de mek pɔsin gɛt ay blɔd prɛshɔn (Beta-blockers - nonselective) .
• Kɔtikosterɔyd dɛn
• Sayklɔfosfamid
• Ɛstrojen we dɛn kin tek bay mɔt
• Tamoksifen we gɛt di sik
• Thiazides - dεn dεm we de mek yu de pis bכku

Di tin we impɔtant pas ɔl: Nɔ ɛva stɔp fɔ tek di mɛrɛsin we dɔktɔ gi yu if yu nɔ tɔk to am fɔs. If wan mɛrɛsin de mek yu triglisɛrɛyd go ɔp, yu dɔktɔ go ebul fɔ ɛp yu. Dɛn kin chenj di doz ɔ gi dɛn ɔda mɛrɛsin.

Di tin dɛn we de apin na di jɛnɛtiks bin de pas tru bɔku bɔku jɛnɛreshɔn dɛn

Sɔm pipul dɛn kin gɛt abnɔmal triglisɛrɛyd ɛn kɔlɔstrel lɛvɛl bak bikɔs ɔf di jɛnɛtik tin dɛn. Ɛgzampul dɛn:

• Famili kɔmbayn haypa lipidemia: Di triglisɛrɛyd dɛn we de go ɔp, di LDL kɔlɔstrel we de go ɔp, ɛn di HDL kɔlɔstrel we de go dɔŋ.
• Famili haypatriglisεridεmia (tayp IV famili dislipidεmia): Triglisεrayd dεm nכmכ de inkrεs.
• di famili disbetalipoproteinemia (tayp III haypalipoproteinemia): di triglisεrayd εn di tכtal kכlestכl lεvεl dεm de inkrεs.
• famili chylomicronemia syndrome (FCS): di triglisεrayd lεvεl dεm we de εlevεt pas 1000 mg/dL.

Bɔku tɛm, wan ɔ mɔ pan dɛn tin ya kin mek pɔsin gɛt aypa triglisɛrɛydemia. Fɔ ɛgzampul, pɔsin kin gɛt prɔblɛm wit in jɛnɛtiks ɛn i kin bi se i nɔ de liv fayn layf. Yu dɔktɔ go no wetin na dɛn tin ya ɛn tɛl yu di bɛst tritmɛnt.

Aw yu go no if yu gɛt hypertriglyceridemia?

Tu men we dɛn de fɔ no dis: fɔ mek dɔktɔ chɛk yu bɔdi ɛn fɔ chɛk yu blɔd.

Fizik Ɛgzam we Dɔktɔ de du

Di dɔktɔ go du wan ɛgzam fɔ yu bɔdi fɔ no bɔt yu wɛlbɔdi. I go aks yu bak bɔt dɛn tin ya:

• Ɛnibɔdi na di famili gɛt at sik, ay kɔlɔstrel, ɔ ay triglisɛrɛyd?
• Yu layf stayl (e.g. rɔm, yuz tabak).
• Ɔda sik dɛn we yu gɛt.
• Di mɛrɛsin dɛn we yu de yuz.

Wit dis infɔmeshɔn, yu dɔktɔ kin ɔndastand wetin de mek yu gɛt ay triglisɛrɛyd ɛn yu risk fɔ gɛt at sik.

Blɔd Tɛst

Wan simpul blɔd tɛst we dɛn kɔl lipid panɛl na di wangren we fɔ no di rayt we fɔ no if pɔsin gɛt di sik we dɛn kɔl hypertriglyceridemia. Dis tɛst de mɛzhɔ yu triglisrayd lɛvɛl ɛn bak yu kɔlɔstrel lɛvɛl.

As a bin don tok bifo, di dokta go tel yu fo fast fo lek 10-12 awa bifo dis blod tehst. Dat min se yu nɔ fɔ it ɔ drink ɛnitin pas wata. Fɔ gɛt kɔrɛkt rizɔlt, i rili impɔtant fɔ fast jɔs lɛk aw di dɔktɔ tɛl yu.

Wetin na di bɛst tritmɛnt fɔ dis? Aw a go ridyus am?

Tri men tritmɛnt dɛn de fɔ di haypa triglisɛridemia:

• Di we aw pipul dɛn de liv dɛn layf kin chenj
• Mɛrɛsin dɛn (drɔg dɛn) .
• Fɔ trit di ɔndalayn kɔz dɛm fɔ ay triglisɛrɛyd.

Yu kin nid wan ɔ mɔ pan dɛn we ya. Yu dɔktɔ go disayd di bɛst tritmɛnt plan fɔ yu.

Laif stayl chenj - dis na di fɔs tin dɛm wae impɔtant!

Bɔrku tɛm, di tritmɛnt kin bigin wit di chenj wae yu de liv yu layf. Dis na di men tin dɛn:

• Nɔ drink rɔm: ridyus am as yu ebul, ɔ stɔp am kpatakpata.
• Nɔ it tin dɛn we gɛt kabɔhaydrɛt ɛn shuga we dɛn dɔn rifin: Nɔ it bred, swit, ɛn drink dɛn as yu ebul. Bifo dat, it ful gren, vɛjitebul, ɛn frut.
• It mɔ sifud, mɔ fish we gɛt omega-3 fat asid: fish lɛk salmɔn, makrɛl, ɛring, ɛn tuna na fayn tin fɔ pik.
• Ɛksesaiz mɔ:Du sɔntin lɛk fɔ waka, rɔn, swim fɔ at le 30 minit insay di de. Bi aktif pan yu ɛvride aktiviti dɛn bak.
• Kɔntrol di kalori we yu de it fɔ lɛ yu nɔ gɛt bɔku bɔku bɔdi: Tray fɔ gɛt wɛlbɔdi wet fɔ yu ayt.
• Pik fayn fayn fat dɛn instead ɔf sɛtyuret ɛn trans fat: stɔp tin dɛn lɛk kokonat ɔyl, pam ɔyl, bɔta, ɛn majarin, ɛn yuz tin dɛn lɛk ɔliv ɔyl, kanola ɔyl, ɛn sɔm nɛt.

"Smɔl chenj lɛk fɔ yuz smɔl ɔyl we wi de kuk na os, fɔ kɔt di fat it dɛn we wi de it na di stoa, ɛn fɔ drink ti we nɔ swit instead ɔf ti we gɛt shuga kin mek big difrɛns."

Di mɛrɛsin dɛn we dɛn kin yuz

If di chenj we yu de chenj yu layf nɔmɔ nɔ de kɔntrol di triglisrayd lɛvɛl, yu dɔktɔ kin gi yu mɛrɛsin. Mɛrɛsin impɔtant mɔ fɔ pipul dɛn we gɛt siriɔs haypa triglisɛrɛydemia. Bɔku kayn mɛrɛsin dɛn de we yu dɔktɔ kin gi yu:

• Fibrates - fכ egzampl. Fenofibrate we dɛn kɔl
• Prɛskrishɔn omega-3 fat asid dɛn - ɛgz. Ikosapɛnt ɛtil (IPE) .
• Statin dεm - pan כl we dεn kin fכs fכ lכs kכlestכl, sכmtεm dεn kin afekt triglisεrayd dεm bak.

Fɔ kɔntrol ɔda sik dɛn

If yu gɛt ɔda mɛrɛsin (e.g. dayabitis) we kin de mek yu gɛt ay triglisɛrɛyd lɛvɛl, i impɔtant bak fɔ kɔntrol da sik de fayn fayn wan. Yu dɔktɔ go gi yu di advays ɛn tritmɛnt we yu nid.

Yu tink se dɛn kin mek dɛn nɔ gɛt di sik we dɛn kɔl hypertriglyceridemia?

Sɔm tin wae kin mek pɔrsin kin gɛt dis sik (we kin ol, sɔm sik dɛm) nɔr kin ebul fɔ kɔntrol. Bɔt, bɔku tin dɛn de we wi kin du na wi layf ɛvride fɔ mek wi nɔ gɛt dis sik. Na sɔm pan dɛn:

• It tin dɛn we go ɛp yu at: It mɔ vɛjitebul, frut, ɔl gren, ɛn tin dɛn we nɔ gɛt bɛtɛ prɔtin. Ridyus ɔyl, shuga, ɛn sɔl.
• Bi aktif: Apat frɔm di ɛksesaiz we yu dɔn plan fɔ du, muv yu bɔdi as mɔ as yu ebul we yu de du yu wok ɛvride. Tin dɛn lɛk fɔ tek di stej insted ɔf di ɛlifant ɛn waka go na say dɛn we de nia de.
• Mek yu gɛt wɛlbɔdi wet.
• Limit alcohol: Di maksimam na wan standad drink fɔ wan de fɔ uman ɛn tu standad drink fɔ wan de fɔ man.
• Ridyus di shuga ɛn rifin kabɔhaydrɛt.

Aks yu dɔktɔ fɔ ɔda advays fɔ ɛp fɔ kɔntrol yu triglisrayd lɛvɛl.

Wetin na de luk fɔ pɔrsin wae gɛt dis sik? (Autluk) .

Dis kin dipen pan bɔku tin dɛn. Fɔ ɛgzampul:

• Wetin na di tin dɛm wae kin mek yu gɛt yu hypertriglyceridemia?
• Wetin na yu triglisɛrɛyd lɛvɛl?
• Us ɔda sik dɛn yu gɛt?

So, tɔk to yu dɔktɔ fɔ no ustɛm yu sik.

Bɔt mɛmba se, hypertriglyceridemia na kɔmɔn sik, ɛn nɔto yu wan de. Bɔrku pipul kin mɛn dis sik fayn fayn wan bay wae dɛn de fala dɛn dɔktɔ in advays, chenj dɛn layf, ɛn tek mɛrɛsin if nid de.

Ustɛm a fɔ go to di dɔktɔ?

Mek shɔ se yu go fɔ yu dɔktɔ chɛk-ap ɛvri ia. Apat frɔm dat, atɛnd ɛni ɔda apɔntinmɛnt lɛk aw yu dɔktɔ tɛl yu. If yu gɛt ɛni kwɛstyɔn ɔ tin we de mɔna yu, nɔ fred fɔ kɔntak yu dɔktɔ ɛnitɛm.

Us kwɛstyɔn dɛn yu fɔ aks di dɔktɔ?

E fayn fɔ lan bɔku tin bɔt yu sik, wetin kin mek yu gɛt am, ɛn aw fɔ trit am. Yu kin aks yu dɔktɔ kwɛstyɔn dɛn lɛk:

• Wetin mi triglisɛrɛyd lɛvɛl de tɛl mi?
• A gɛt ay kɔlɔstrel bak?
• Wetin de mek mi triglisɛrɛyd dɛn de go ɔp?
• Aw a go ebul fɔ stɔp mi triglisrayd lɛvɛl?
• Wetin na di risk we a kin gɛt fɔ gɛt at sik?
• Us kayn ɛksesaiz fayn fɔ mi?
• A fɔ go to pɔsin we sabi bɔt it?

Ustɛm yu nid fɔ go na di Imejɛnsi Tritmɛnt Yunit (ETU) ?

If yu gɛt ɛni wan pan dɛn sik ya , kɔl 911 (1990 na Sri Lanka) wantɛm wantɛm, ɔ go na di imejensi rum we de nia yu:

• Akyu pankrɛyayt - (bɔdi de pen bad bad wan, vɔmit, fiva) .
• At atak - (chɛst de pen, yu nɔ de blo shɔt, yu de swet) .
• Strɔk - (we yu fes, yu an ɔ yu leg, yu nɔ go ebul fɔ tɔk ɔ yu wik, yu nɔ go ebul fɔ tɔk, yu nɔ go ebul fɔ waka) .

Hypertriglyceridemia de inkrisayz di risk fɔ gɛt dɛn siriɔs kɔmplikeshɔn ya. So, i rili impɔtant fɔ no bɔt dɛn sik ya ɛn go to dɔktɔ kwik kwik wan if dɛn apin.

Fɔ dɔn, tin dɛn fɔ mɛmba (Take-Home Message) .

Fɔ kɔntrol di haypa triglisɛridemia kin tek sɔm tray. Bɔt i fayn fɔ yuz yu tɛm. Yu go nid fɔ mek sɔm chenj dɛn na di tin dɛn we yu de du ɛvride, ɛn sɔntɛnde i kin tan lɛk se dɛn chenj dɛn de nɔ izi. Bɔt bigin smɔl. Ivin smɔl chenj dɛn kin mek big difrɛns pan yu at wɛlbɔdi. Aks yu dɔktɔ bɔt di tin dɛn we yu nid ɛn sɔpɔt grup dɛn fɔ ɛp yu fɔ rich di tin dɛn we yu dɔn plan fɔ du. Yu wɛlbɔdi de na yu an!

` haypa triglisεridemia, triglisεrayd, kכlestכl, at sik, it, εksεsayz, layf stayl