Yu bɔdi nɔ de ansa fayn to insulin? (Insulin Resistance) Lɛ wi lan bɔt am jɔs!

Tide wi go tok abaut wan wod we plenti pipul de tok abaut, and i de mek pipul de fred likl tu. Dat na ‘Insulin Resistance’. Sɔntɛm yu dɔktɔ dɔn tɛl yu bɔt am, ɔ yu dɔn rid bɔt am sɔnsay. Wetin na in rili? Wetin mek i rili impɔtant? Lɛ wi ɔndastand ɔl dis simpul wan.

So, wetin na ‘Insulin Resistance’?

Fɔ tɔk am simpul wan, insulin rɛsistɛns na we di sɛl dɛn na yu bɔdi, mɔ di wan dɛn we de na yu mɔsul, fat sɛl, ɛn liva, nɔ de ansa di ɔmon insulin fayn fayn wan. Dɛn kin kɔl dis bak impaired insulin sensitivity.

Tink bɔt am, insulin na impɔtant pat na wi bɔdi. I tan lɛk pɔsin we de kɔntrol trafik na wi bɔdi. Na wetin de ɛp wi fɔ sɛn glukɔs, ɔ di shuga we wi de it frɔm wi blɔd, insay wi sɛl dɛn fɔ mek ɛnaji. Naw, wetin kin apin we di sɛl dɛn nɔ de lisin to dis insulin fayn fayn wan? di shuga we de insay di bכdi nכ kin ebul fכ go insay di sεl dεm εn i kin gɛda na di bכdi.

Wetin kin apin da tɛm de? Wetin kin apin to di pankrias?

Na da tɛm de di prɔblɛm kin bigin. wi pankrias – we na wi bכdi in insulin fכktכri – de no se, "O, di bכdi shuga de go tu hכy, εn i nכ de go insay di sεl dεm." So wetin di pankrias de du? I kin bigin fɔ mek mɔ ɛn mɔ insulin fɔ kɔntrol di ay shuga lɛvɛl. I tan lɛk se yu de push domɔt fɔ opin we i nɔ ebul fɔ opin. Wi kin kɔl dis haypa insulinemia.

Fɔ sɔm tɛm, if di pankrias ebul fɔ mek mɔ insulin dis we ɛn kɔntrol di blɔd shuga, no big prɔblɛm nɔ de. Bɔt, smɔl smɔl, as di sɛl dɛn de rɛsist to insulin, ilɛksɛf di pankrias de mek insulin, i nɔ go du fɔ am. Na da tɛm de di blɔd shuga kin bigin fɔ bɔku ɔltɛm. Wi kin kɔl dis ‘haypa glycemia’.

Dis ‘insulin resistance’ denja? Wetin na di bad tin dɛn we kin apin?

Yɛs, if yu kɔntinyu fɔ du dis, i kin denja smɔl. Bikɔs, we dɛn ɛlevɛt blɔd shuga lɛvɛl ya de fɔ lɔng tɛm, yu kin gɛt ‘prediabetes’. Dat min se yu de nia fɔ gɛt dayabitis. Ɛn afta dat, yu go gɛt Tayp 2 dayabitis pasmak.

Nɔto dat nɔmɔ, dis sik gɛt fɔ du wit sɔm ɔda wɛlbɔdi prɔblɛm dɛn:

• Fat , mɔ di fat we pasmak na di bɛlɛ eria.
• Cardiovascular disease , we min hat sik ɛn blɔd vesel prɔblɛm.
• Metabolic dysfunction-associated steatotic liver disease , we dɛn bin de kɔl fɔs nɔ-alkohol fat liva sik.
• Mεtabolik sεndrכm.Dis nɔto wan sik, bɔt na wan kɔlekɛshɔn fɔ sɔm sayn dɛm wae kin kam togɛda. Fɔ ɛgzampul, tin dɛn lɛk ay blɔd prɛshɔn, ay blɔd shuga, bɛlɛ fat we de bɔku, ɛn kɔlɔstrel we nɔ balans.
• Polisistik Ɔvari Sindrom (PCOS). Dis na di ɔmon imbalans we kin apin pan uman dɛn.

Udat kin gɛt ‘insulin resistance’?

I kin rili apin to ɛnibɔdi. Yu nɔ nid fɔ gɛt dayabitis. Sɔntɛnde, i kin bi fɔ shɔt tɛm nɔmɔ. Fɔ ɛgzampul, if yu tek stɛroyd fɔ sɔm tɛm fɔ sik, yu kin gɛt insulin rɛsistɛns. E kin bi bak wae de te, min se na sik wae de teik lɔng tɛm.

Tu men tin dɛn de we dɛn tink se kin afɛkt dis:

1. Fat we pasmak na yu bɔdi , mɔ we de rawnd yu bɛlɛ.

2. Nɔ de du bɔku tin dɛn , we min se yu nɔ fɔ du ɛksasaiz.

Dɔn bak, if pɔsin na yu famili gɛt dayabitis (espɛshali tayp 2 dayabitis), prɛdiabɛtis, ɔ kɔndishɔn lɛk polycystic ovary syndrome (PCOS), yu kin gɛt mɔr sik bak fɔ gɛt insulin rɛsistɛns.

Insulin resistance na kɔmɔn tin fɔ pipul dɛm wae gɛt prediabetes ɛn tayp 2 dayabitis. Pipul wae gɛt tayp 1 dayabitis kin gɛt insulin rɛsistɛns bak sɔmtɛm.

Wetin na di sayn dɛm fɔ dis? Aw wi go no?

Na dis na di prɔblɛm. Bɔrku tɛm, if yu pankrias kin mek mɔ insulin ɛn kɔntrol yu blɔd shuga lɛvɛl, yu nɔ kin gɛt ɛni sayn. Sɔntɛm yu nɔ ivin no am.

Bɔt as tɛm de go, dis sik kin wɔs smɔl smɔl. Di sɛl dɛn na di pankrias we de mek insulin sɛf kin bigin fɔ wik smɔl smɔl. Afta sɔm tɛm, di pankrias nɔ kin ebul fɔ mek insulin we go du fɔ am. Na da tɛm de di blɔd shuga kin bigin fɔ go ɔp ɛn di sayn dɛn kin bigin fɔ sho.

We yu blɔd shuga kɔntinyu fɔ bɔku, yu kin gɛt sɔm sayn dɛn lɛk:

• We yu de lɔs yu wet fɔ natin
• Fɔ fil taya ɔltɛm
• Fɔ fil se yu tɔsti pas aw yu kin fil
• Fɔ pis bɔku tɛm
• Fɔ fil angri pas aw i kin fil
• Di vishɔn we nɔ klia
• Yist infεkshכn we kin de fכm (especially na di vagina εn sכmtεm na di skin fold dεm na uman dεm) .

Bɔrku pipul dɛm wae gɛt prediabetes nɔr kin gɛt ɛni sayn, sɔmtɛm fɔ lɔng tɛm. Prediabetes nɔ kin sho ɛni sayn te i go bifo to tayp 2 dayabitis. Bɔt sɔm pipul dɛn we gɛt prɛdiabɛtis kin sho sɔm sayn dɛn lɛk:

• Yu armpit dɛn, di bak pat na yu nɛk, ɛn di sayd dɛn na yu bɔdi we dak . Dɛn kɔl dis acanthosis nigricans . I tan lɛk dɔti, bɔt nɔto dɔti.
• Smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl smɔl .
• Di chenj dɛn we de apin na di yay . Dɛn tin ya kin mek yu yay pwɛl bikɔs ɔf dayabitis (dayabitis-rilayt retinopathy).

Impɔtant: If yu gɛt wan ɔ mɔ pan dɛn sik ya, mek shɔ se yu go to yu dɔktɔ. I rili impɔtant.

Wetin mek wi kin gɛt ‘insulin resistance’? Wetin na di tin dɛn we kin mek i apin?

Sayɛnsman dɛn stil de wok fɔ no di rayt we aw dis kin apin. Naw dɛn no se sɔm jin dɛn kin mek pɔsin gɛt mɔ ɔ smɔl chans fɔ gɛt insulin rɛsistɛns. Dɔktɔ dɛn no bak se di sik kin wɔs we pɔsin de gɛt mɔ wet, ɛn if pɔsin lɛf fɔ it bɔku bɔku it, dat kin ɛp fɔ kɔntrol am te to sɔm mak.

Apat frɔm dat, bɔku ɔda rizin dɛn de.

Kɔz dɛn we dɛn dɔn gɛt

Dis nɔto di rizin dɛn we dɛn bɔn wi wit, bɔt na di rizin dɛn we kin apin insay wi layf.

• Fat we pasmak na di bɔdi: Sayɛnsman dɛn biliv se we pɔsin fat pasmak na in kin mek pɔsin nɔ ebul fɔ yuz insulin. di fεt we de dכn na di bכdi εria εn rawnd di intanεt כgan dεm (visceral fat) de inkrεs di risk bכku bכku wan.
• Nɔr de du tin wit yu bɔdi: We yu de du ɛksɛsayz, yu bɔdi kin fil lɛk insulin. Ɛksesaiz kin ɛp bak fɔ mek yu mɔsul dɛn tek glukɔs na yu blɔd, ɛn dis kin mek yu gɛt dis sik.
• Di it: Dɛn dɔn si se fɔ it tin dɛn we dɛn dɔn prosɛs bɔku, it dɛn we gɛt bɔku kabɔhaydrɛt, ɛn it dɛn we gɛt bɔku sɛtyuret fat, dat kin mek pɔsin nɔ gɛt insulin.
• Sɔm mɛrɛsin dɛn: Sɔm mɛrɛsin dɛn lɛk stɛroyd, sɔm mɛrɛsin fɔ ay blɔd prɛshɔn, ɛn mɛrɛsin dɛn we dɛn kin yuz fɔ mɛn HIV kin mek pɔsin nɔ gɛt insulin bak.

Ɔmon dizayd we kin mek pɔsin nɔ gɛt insulin

Sɔm ɔmon prɔblɛm dɛn kin afɛkt bak di we aw di bɔdi de yuz insulin.

• Cushing’s syndrome: Dis na wan sik wae nɔr kin bɔrku. I kin apin we wan ɔmon we dɛn kɔl kɔtisol bɔku pasmak na di bɔdi. di kכtisol we pasmak de ambɔg di wok we insulin de du.
• Acromegaly: Dis na sik bak we nɔ kin apin so ɔltɛm bɔt we kin rili siriɔs. I kin apin we di ɔmon we de gro tumɔs (GH) de na di bɔdi. Dis kin mek bak yu nɔ gɛt insulin.
• Hypothyroidism: Dis min se yu tayroyd gland nɔ de wok fayn, we min se i nɔ de mek inof tayroyd ɔmon. Dis kin mek yu mɛtabolism slo, we kin mek bak yu nɔ gɛt insulin.

Jεnεtik kכndishכn dεm we de mek insulin rεsistεns

Sɔm sik dɛn de we pɔsin kin gɛt frɔm in mama ɛn papa ɛn we kin mek pɔsin nɔ gɛt insulin bak. Dɛn tin ya nɔ kin apin so ɔltɛm. Sɔm ɛgzampul dɛn na tin dɛn we dɛn kɔl Tayp A insulin rɛsistɛns sindrom ɛn Donohue sindrom.

Ɛn sɔm ɔda tin dɛn we kin mek pɔsin gɛt jɛnɛtiks:

• Myotonic dystrophy: Dis na wan kayn we we di mɔsul dɛn wik we de afɛkt di mɔsul dɛm, yay dɛm, ɛn di ɔgan dɛm na di ɛndokrin sistɛm.
• Alström syndrome: Dis sik kin mek pɔsin fat ɛn gɛt tayp 2 dayabitis. I kin mek bak yu nɔ de si ɛn yɛri, yu gɛt dilated cardiomyopathy, ɛn yu nɔ de blo fayn.
• Werner syndrome: Dis kin mek pɔsin ol kwik kwik wan (progeria). I kin afɛkt bɔku tin dɛn na di bɔdi, lɛk di wan dɛn we nɔ kin gɛt insulin.
• Inherited lipodystrophy: Insay dis kכndyushכn, yu bכdi nכ de yuz כ stכr fεt fayn fayn wan.

Aw dɔktɔ dɛn kin no bɔt dis?

Insulin resistance na smɔl triki fɔ no prɛsishɔn, as nɔ spɛshal, yunivasal tɛst nɔ de fɔ am. As wi bin dɔn tɔk, if yu pankrias ebul fɔ mek insulin fɔ kɔntrol di sik, yu nɔ go gɛt ɛni sayn.

Bɔt we yu dɔktɔ tink se dis sik, dɛn go tink bɔt sɔm tin dɛn:

• Yu mɛdikal histri (lɛk di sik dɛn we yu bin dɔn gɛt trade ɛn di mɛrɛsin dɛn we yu bin de tek) .
• Yu famili wɛl bɔdi histri (ɛnibɔdi na yu famili gɛt dayabitis ɔ at sik?)
• Fɔ chɛk yu bɔdi
• Di sayn dɛm wae yu de tɔk bɔt

Us tɛst dɛn de du fɔ dis?

Di dɔktɔ kin du bɔku blɔd tɛst dɛn, lɛk:

• Glukɔs tɛst: If yu de fast plasma glukɔs (FPG) ɔ yu blɔd shuga tɛst ɔltɛm, dat kin ɛp fɔ no if yu gɛt prɛdiabɛtis, tayp 2 dayabitis, ɔ gestational dayabitis.
• A1c test: Dis kin tɛl yu aw yu blɔd shuga lɛvɛl dɔn tan lɛk fɔ di pas tri mɔnt.
• Lipid panɛl: Dis de mɛzhɔ di difrɛn kayn fat dɛn (lɛk kɔlɔstrel ɛn triglisɛrɛyd) we de na yu blɔd.

So, aw yu de trit dis? (Tritmɛnt)

Nɔto ɔl di tin dɛn we kin mek pɔsin nɔ gɛt insulin, dɛn kin trit, lɛk di jɛnɛtiks ɛn di ej. So, di men tritmɛnt na fɔ chenj di we aw yu de liv yu layf.

Dɛn chenj ya we wi kin chenj di we aw pɔsin de liv in layf na:

• It tin dɛn we gɛt fayn fayn tin dɛn fɔ it: Yu dɔktɔ go tɛl yu fɔ stɔp fɔ it tin dɛn we gɛt kabɔhaydrɛt ɛn fat we nɔ fayn fɔ yu. Dis min se yu fɔ kɔt di shuga, rɛd mit, ɛn it dɛn we dɛn dɔn prosɛs we gɛt stach. Bifo dat, dɛn kin tɛl yu fɔ it mɔ vɛjitebul, frut, ligɛm, ɛn ɔl gras. Fɔ gɛt prɔtin, fish ɛn chukchuk na gud opshɔn.
• Fɔ du bɔdi wok: We yu de du mɔdaret ɛksesaiz ɛvride, dat kin ɛp bɔku.dis de mek di yus fכ glukכs na di bכdi fכ εnεji, εn i de mek di insulin sεnsitiviti na di mכsul dεm bכku.
• Fɔ lɔs bɔku bɔku wet: Sɔntɛnde, we yu lɔs yu wet kin ɛp fɔ mek yu nɔ gɛt insulin. Yu dɔktɔ kin tɔk to yu bɔt aw fɔ ɛp yu fɔ lɛf fɔ it bɔku bɔku it dɛn.

As tɛm de go, dɛn chenj ya we kin apin to pɔsin in layf kin mek wi gɛt tin dɛn lɛk dɛn tin ya:

• Di insulin rɛsistɛns we de go dɔŋ
• Di shuga we de na di blɔd we de go dɔŋ
• Lɔw blɔd prɛshɔn
• di triglisεrayd εn LDL (bad kכlestכl) lεvεl dεm dכn dכn
• di HDL (gud kכlestכl) lεvεl dεm we de go כp

Yu dɔktɔ kin sɛn yu bak to spɛshal dɔktɔ, lɛk pɔsin we sabi bɔt it ɔ pɔsin we de stɔdi bɔt di bɔdi. Dɛn go wok wit yu fɔ mek wan tritmɛnt plan we go fayn fɔ yu.

Yu tink se spɛshal mɛrɛsin de fɔ dis?

Naw, no patikyula tritmɛnt nɔ de fɔ insulin rɛsistɛns. Bɔt yu dɔktɔ kin gi yu mɛrɛsin fɔ ɔda wɛlbɔdi prɔblɛm dɛn we yu go gɛt. Sɔm ɛgzampul dɛn na:

• Mɛrɛsin fɔ ay blɔd prɛshɔn
• Mεdikeshכn dεm lεk Metformin fכ dayabεtis כ prεdiabetes
• Mεdikeshכn dεm lεk statin fכ lכs LDL kכlestכl

Yu tink se dɛn kin chenj di we aw di insulin nɔ de wok? (Rivasibiliti)

Bikɔs bɔku tin dɛn de we kin mek dis apin, nɔto ɔl di tin dɛn we kin mek pɔsin du dis kin chenj. Bɔt, we pɔsin chenj in layf, dat kin mek big chenj. If yu it tin dɛn we gɛt fayn fayn tin dɛn fɔ du, ɛksesaiz ɔltɛm, ɛn lɛf fɔ it bɔku bɔku it dɛn, dat kin ɛp fɔ mek yu nɔ gɛt bɔku insulin. Tɔk to yu dɔktɔ bɔt wetin yu kin du fɔ mek yu ebul fɔ kɔntrol dis fayn fayn wan.

Aw di it kin afɛkt di we aw pɔsin kin gɛt insulin?

Di it dɛm wae yu de it kin gɛt big impak pan yu blɔd shuga ɛn insulin lɛvɛl. Di Glycemic Index (GI) na fɔ no aw kwik di it dɛn we yu de it de mek yu blɔd shuga go ɔp.

Fɔd dɛn we gɛt ay GI kin gɛt bɔku bɔku kabɔhaydrɛt ɛn/ɔ shuga. We yu it dɛn, yu blɔd shuga de go ɔp kwik kwik wan, ɛn yu nid bɔku insulin fɔ mek dɛn go dɔŋ bak. So, yu fɔ avɔyd fɔ it bɔku drink dɛn we gɛt shuga (soda, sɔft drink, atifishal frut drink) ɛn it dɛn lɛk:

• Bred (wayt bred) .
• Pɛtetɛ
• Di kayn siriɔs tin dɛn we yu fɔ it fɔ brɛf
• Swit it dɛn lɛk kek ɛn biskit
• Sɔm frut dɛn lɛk watamɛlɔn ɛn dɛti (dɛn tin ya gɛt bɔku shuga) .

We yu it it dɛn we gɛt smɔl GI , yu blɔd shuga de go ɔp sloslo. Dis min se yu pankrias nɔ nid fɔ wok tranga wan fɔ mek insulin. Jɛnɛral wan i bɛtɛ fɔ pipul dɛn we gɛt insulin rɛsistɛns fɔ it it dɛn we gɛt smɔl GI, lɛk:

• Ligim lɛk bins, chikip, ɛn lentil
• Frut dɛn lɛk apul ɛn bɛri
• Vɛgjetabul dɛn we nɔ gɛt bɔku stach - ɛgzampul dɛn: asparagus, kɔliflawa, vɛjitebul dɛn we gɛt grɛn lif
• Nat dɛn
• Dairy fud, fish, mit

Mɛmba se: Tɔk to yu dɔktɔ ɔltɛm bifo yu mek ɛni big chenj na di it we yu de it. I kin advays yu bɔt di bɛst we fɔ du tin fɔ yu.

Wetin na di tin dɛn we kin mek pɔsin gɛt insulin rɛsistɛns? (Di tin dɛn we kin mek pɔsin gɛt prɔblɛm)

Sɔm tin dɛn we kin mek yu gɛt prɔblɛm wit yu jɛnɛtik ɛn aw yu de liv yu layf kin mek yu gɛt mɔ chans fɔ gɛt insulin rɛsistɛns ɔ prɛdiabɛtis. Dɛn tin ya we kin mek pɔsin gɛt prɔblɛm na:

• Ɔvaweit ɔ fat , mɔ di bɔku bɔku fat we de rawnd di bɛlɛ.
• Ej 45 ia ɔ pas dat.
• Fɔ gɛt dayabitis pan pɔsin we de nia yu fambul (mama ɛn papa, brɔda ɛn sista).
• We pɔsin nɔ de du ɛnitin we i de du na in bɔdi.
• Fɔ gɛt wɛl bɔdi prɔblɛm lɛk ay blɔd prɛshɔn ɛn ay kɔlɔstrel.
• Fɔ gɛt dayabitis we yu gɛt bɛlɛ (jestational diabetes).
• Fɔ gɛt at sik ɔ strok.
• Slip dizayd , fɔ ɛgzampul, slip apnea.
• Fɔ smok.

Dɔn bak, di risk kin pas smɔl if yu kɔmɔt na dɛn etnik grup ya:

• Blak
• Hispanik/Latino pipul dɛn
• Eshian pipul dɛn
• Midul Ist pipul dɛn

Wi nɔr kin chenj sɔm tin dɛm wae kin mek wi gɛt dis sik, lɛk aw wi de na wi famili ɛn aw pɔrsin dɔn ol. Bɔt yu kin tray fɔ ridyus yu risk fɔ gɛt dis sik bay we yu gɛt wɛlbɔdi wet we fit yu ɛn ɛksesaiz ɔltɛm.

Wetin na de luk fɔ pɔrsin wae gɛt insulin resistance?

If yu gɛt dis sik, aks yu dɔktɔ wetin yu go ɛkspɛkt ɛn aw fɔ mɛn am fayn fayn wan.

Di we aw yu de si tin dipen pan bɔku tin dɛn:

• Di tin we kin mek pɔsin nɔ gɛt insulin
• Di kayn we aw insulin rɛsistɛns kin tranga
• Aw yu ɛn yu dɔktɔ kin manej dis sik fayn fayn wan

Sɔm pipul dɛn kin gɛt smɔl insulin rɛsistɛns, we nɔ kin ɛva go bifo to prɛdayabitis ɔ tayp 2 dayabitis. Dɛn kin gɛt insulin rɛsistɛns bak we dɛn kin rivɛns ɔ manej wit chenj na dɛn layf.

Us kɔmplikeshɔn dɛn kin apin bikɔs ɔf di insulin rɛsistɛns?

If dɛn nɔr de manej dis kɔndishɔn fayn fayn wan, kɔmplikeshɔn kin apin. Di prɔblɛm dɛn we kin apin na:

• Tayp 2 dayabitis
• Mεtabolik sεndrכm
• At sik

Nɔto ɔlman we gɛt insulin rɛsistɛns go gɛt prɔblɛm dɛn. If dɛn dɔn no se yu gɛt dis sik, i impɔtant fɔ tray fɔ mek dɛn nɔ gɛt dɛn prɔblɛm ya. Mek shɔ se yu de go to yu dɔktɔ ɔltɛm, ɛn fala yu tritmɛnt plan gud gud wan.

Ustɛm a fɔ go to dɔktɔ bɔt insulin rɛsistɛns?

If dɛn dɔn no se yu gɛt dis sik, i rili impɔtant fɔ go to yu dɔktɔ ɔltɛm. I kin chɛk if yu blɔd shuga lɛvɛl de na wɛlbɔdi lɛvɛl ɛn if yu tritmɛnt de wok.

If yu gɛt sayn dɛn we de sho se yu gɛt bɔku blɔd shuga ɔ yu gɛt prɛdiabɛtis, go to yu dɔktɔ wantɛm wantɛm. I kin du simpul tɛst fɔ chɛk yu blɔd shuga lɛvɛl.

If pɔsin na yu famili gɛt dayabitis ɔ insulin rɛsistɛns, tɔk to yu dɔktɔ bɔt yu risk fɔ gɛt dis sik.

Us kwɛstyɔn dɛn a fɔ aks di dɔktɔ?

If dɛn no se yu gɛt dis sik, i go fayn fɔ aks yu dɔktɔ dɛn kwɛstyɔn ya:

• Wetin na di tin we kin mek a nɔ gɛt insulin?
• Wetin na mi risk fɔ gɛt prɛdiabɛtis ɔ tayp 2 dayabitis?
• Ɛni mɛrɛsin de we a go tek?
• A fɔ go to spɛshal pɔsin fɔ dis sik?

Fɔ dɔn, tin dɛn we wi nid fɔ mɛmba (Take-Home Message) .

So, wi dɔn si se insulin rɛsistɛns na smɔl kɔmpleks kɔndishɔn, ɛn i kin afɛkt wi wɛlbɔdi pan bɔku we dɛn. Bɔrku tɛm, i nɔr kin sho ɛni sayn te i tɔn to prɛdiabetes ɔr tayp 2 dayabitis.

So, di bɛst tin we yu go du na fɔ tray fɔ mek dis sik nɔ kam, ɔ at ɔl fɔ rivɛns am if i apin. Fɔ du dis , mek yu gɛt wɛlbɔdi we go fayn fɔ yu, ɛksesaiz ɔltɛm, ɛn it tin dɛn we gɛt fayn fayn tin dɛn fɔ du.

Bɔt sɔmtɛm, i nɔr kin pɔsibul fɔ protɛkt ɔ trit ɔl di tin dɛm wae kin mek dis sik kam. If yu gɛt ɛni kwɛstyɔn bɔt yu risk ɔ bɔt dis sik, mek shɔ se yu tɔk to yu dɔktɔ. Dɛn de de fɔ ɛp yu.

` Insulin resistance, dayabitis, prediabetes, blɔd shuga, pankrias, fat, it fayn, ɛksesaiz, ɔmon