Ingabe nawe uzizwa womile ngaso sonke isikhathi? Ake sixoxe ngale polydipsia!

Kuvamile ukuzizwa womile uma sikhathele noma ngemva kokudla ukudla okunzima. Ukuphuza ingilazi eyodwa noma amabili amanzi kuzoqeda ukoma. Kodwa wake wazizwa yini ukuthi kungakhathaliseki ukuthi uphuza amanzi angakanani noma uhlobo luni loketshezi oluphuzayo, ukoma kwakho akuqedwa? Uyaqhubeka uphuza amanzi... nokuningi... kodwa usalokhu womile. Yilokho esikubiza ngezokwelapha ngokuthi i-polydipsia. Lokhu akukhona ukoma nje, kuyinto ejulile kunalokho. Ake sixoxe ngalokhu ngamagama alula nanemininingwane.

Iyini ngempela i-Polydipsia?

Kalula nje, i-polydipsia yigama lezokwelapha elisho ukoma ngokweqile noma isifiso sokuphuza amanzi ngokungalawuleki. Kunezinhlobo ezimbili eziyinhloko zalokhu.

1. I-Polydipsia Eyinhloko

Okwenzekayo kulesi simo ukuthi uzizwa womile kakhulu, kodwa akukho sizathu esibonakalayo, okungukuthi, asikho isifo, esibangela lokho. Lokhu kuhlukaniswe kabili futhi.

• I-Psychogenic polydipsia: Lokhu kuhlotshaniswa nezinkinga zempilo yengqondo. Kuvamile kakhulu kubantu abanezimo ezifana ne -schizophrenia . Odokotela bacabanga ukuthi laba bantu bangaphuza amanzi amaningi kakhulu ngoba ubuchopho babo abucubunguli kahle izimpawu zokoma noma ngoba bezama ukubhekana nokucindezeleka.
• I-Dipsogenic Polydipsia: Lokhu kubangelwa ukuphuza amanzi amaningi kakhulu, kungaba ngenxa yephutha eliku-hypothalamus, ingxenye yobuchopho bethu elawula ukoma, noma ngenxa yokuqiniseka ngokweqile ukuthi "ukuphuza amanzi amaningi kuhle emzimbeni."

2. I-Polydipsia Yesibili

Lolu uhlobo oluvame kakhulu. Kulesi simo, kunesizathu esithile somzimba sokoma ngokweqile. Ngokuvamile kuyisibonakaliso sesimo sezokwelapha esifana nesifo sikashukela. Kungabangelwa futhi yimithi ethile, njengemithi yokukhipha isisu, esetshenziselwa ukwelapha umfutho wegazi ophakeme.

Ziyini izimpawu zalesi simo?

Nakuba i-polydipsia iyisibonakaliso sesinye isifo, kunezimpawu eziningana ezihlukile kulesi simo. Futhi, izimpawu zingase zihluke kuye ngesifo esiyisisekelo.

Izimbangela eziyinhloko zokoma ngokweqile

Njengoba sixoxile ngaphambili, lokhu kuvame ukuba uphawu lwesinye isimo sezokwelapha.

• Isifo Sikashukela: Uma amazinga kashukela egazini lakho ephezulu kakhulu, izinso zakho zizama ukukhipha ushukela omningi emchameni wakho. Lokhu kubangela ukuthi uchame kaningi. Uma ulahlekelwa amanzi amaningi emzimbeni wakho, uba manzi emzimbeni futhi uzizwe womile kakhulu. Ngakho-ke uphuza amanzi amaningi. Lo mjikelezo uyaqhubeka.

• Isifo Sikashukela Insipidus: Lokhu kuhlukile kunesifo sikashukela esivame ukusazi. Asihlobene ne-pancreas. Lesi simo sibangelwa inkinga ngezinso noma i-pituitary gland. Lesi sifo sikhiqiza i-hormone elawula ukuthi izinso zigcina amanzi angakanani emzimbeni.

• I-Diabetic Ketoacidosis (DKA): Lokhu kuyinkinga eyingozi yesifo sikashukela. Uma umzimba ungenayo i-insulin eyanele yokusebenzisa i-glucose, umzimba uqala ukushisa amafutha ukuze uthole amandla. Lokhu kubangela ukuthi amakhemikhali abizwa ngokuthi ama-ketone aqongeleleke egazini. Uma lawa eqongeleleka egazini, igazi liba ne-asidi futhi lingaba yingozi emzimbeni. Izimpawu eziyinhloko ze-DKA ukoma ngokweqile kanye nokuchama ngokweqile.

• I-Psychogenic Polydipsia: Ezinye izimo zempilo yengqondo zingabangela isifiso esingalawuleki sokuphuza amanzi. Izibonelo zifaka phakathi i-schizophrenia , ukucindezeleka, kanye ne-anorexia.

• Ezinye izizathu:Ukoma ngokweqile kungabangelwa nayizimo ezifana nokuphelelwa amanzi emzimbeni okubangelwa ukuhlanza noma uhudo ngokweqile, ukuphuza utshwala ngokweqile, isifo sika-Addison, ukwehluleka kwezinso, kanye ne-sickle cell anemia.

Ingabe ukuphuza amanzi amaningi kakhulu kuyingozi? (Izinkinga)

Yebo, kuthiwa into eningi kakhulu ayilungile. Ngisho namanzi awahlukile. Ukuphuza amanzi amaningi kungaphazamisa ibhalansi yamakhemikhali emzimbeni futhi kubangele izinkinga ezinkulu.

I-Hyponatremia

Lokhu kusho ukuthi izinga le-sodium egazini liphansi ngendlela eyingozi. Ngokuvamile, amanzi amaningi esiwaphuzayo akhishwa emchameni. Kodwa uma siphuza amanzi amaningi kunalawo umzimba wethu ongawaphatha, igazi lethu liyancibilika. Cabanga ngakho njengokufaka amanzi amaningi engilazini yamanzi ancibilikiswe usawoti. I-sodium iyi-electrolyte ebalulekile elawula umfutho wegazi lethu, ukusebenza kwezinzwa, kanye nenani lamanzi azungeze amaseli ethu. Uma iphansi, kungase kube nezimpawu ezilandelayo:

• Ikhanda elibuhlungu
• Isicanucanu nokuhlanza
• Ukuqaqamba kwemisipha
• Impendulo ehamba kancane
• Ubunzima bokukhuluma (ukungingiza)
• Ukuntuleka kwamandla
• Ukudideka
• Ukuquleka

Uma lesi simo singalashwa, singaholela ekulimaleni kwezitho zomzimba, ekukhomeni, ngisho nasekufeni.

Ukudakwa ngamanzi kuyindlela embi kakhulu ye-hyponatremia. Akuvamile kakhulu, kodwa kungenzeka ngemva kokuzivocavoca kakhulu, ukuphuza amanzi amaningi ngaphandle kokuphuza isiphuzo esine-sodium, ukugula ngengqondo, noma ukuhlanza/uhudo olunzima.

Iphathwa kanjani?

Ukwelashwa kuncike ekutheni yini ebangela i-polydipsia yakho. Udokotela wakho uzoqala azame ukuthola imbangela.

• Ukuhlolwa kwezokwelapha: Ukuhlolwa kwegazi ukuhlola amazinga akho e-sodium kanye noshukela, kanye nokuhlolwa komchamo ukuhlola ushukela emchameni wakho. Ungase futhi ucelwe ukuthi ulinganise ukuthi uphuza uketshezi olungakanani nokuthi ukhiqiza umchamo ongakanani esikhathini samahora angama-24.
• Ngesifo Sikashukela:Ukwelashwa okudingekayo (imithi, i-insulin, ukudla okunempilo) kuzonikezwa ukuze kulawulwe kahle amazinga kashukela egazini.
• Ngesifo Sikashukela i-Insipidus: Kungase kunikezwe ama-hormone noma eminye imithi.
• Ngezizathu zengqondo (i-Psychogenic Polydipsia): Ukunciphisa inani lamanzi owaphuzayo, ukwelulekwa kwengqondo, futhi, uma kudingeka, ukwelashwa ngezidakamizwa kuyanconywa.

Into ebaluleke kakhulu ukubona udokotela bese uthola ukuxilongwa okufanele uma unalezi zimpawu, kunokuba wenze izinqumo ngokwakho.