Wetin na VLDL (Very-Low-Density Lipoprotein) we de insay yu kɔlɔstrel? Lɛ wi fɛn ɔndastand di rayt tin we i bi!

Yu go mɔs dɔn yɛri bɔt kɔlɔstrel , nɔto so? Yu go ivin si dis wɔd na yu blɔd ripɔt. Natin nɔ de fɔ fred we yu yɛri di wɔd kɔlɔstrel. Bɔt, difrɛn kayn am de, ɛn i impɔtant fɔ no bɔt dɛn. Tide wi go tɔk bɔt wan mɛmba na di kɔlɔstrel famili, ɛn dat na VLDL (Very-Low-Density Lipoprotein) . Dis nem kin bi nyu tin to yu smɔl, bɔt nɔ wɔri, lɛ wi ɛksplen am simpul wan.

Wetin na VLDL (Vɛri-Lɔ-Dɛnsity Lipoprotein), padi?

Fɔ tɔk am simpul wan, VLDL na spɛshal kayn patikyula tin we wi liva kin mek ɛn afta dat i kin kɔmɔt na wi blɔd. Dɛn tin ya de insay wan grup we gɛt prɔtin dɛn we dɛn kɔl lipoproteins .

Tink bɔt am, di fat dɛn we de na wi blɔd, lɛk kɔlɔstrel ɛn triglisɛrɛyd , nɔ de muv rawnd na di blɔd fɔ dɛnsɛf. Dɛn nɔ de sɔlv insay wata. So, dis fεt dεm nid motoka fכ transpכt dεm to difrεn pat dεm na di bכdi. Dɛn kɔl da motoka de lipoproteins. I tan lɛk bɔs!

di men wok we VLDL de du na fכ tek triglisεrayd εn sכm kכlestכl εn transpכt dεm to di ples dεm we dεn nid dεm na di bכdi. Dis we ya, VLDL de ɛp wi bɔdi fɔ gɛt ɛnaji, kip ɛnaji, ɛn bak fɔ kɔntrol blɔd prɛshɔn. So, dɛn rili impɔtant fɔ di nɔmal wok we wi bɔdi de du.

Bɔt na sɔntin we yu fɔ mɛmba: If di VLDL we de na di blɔd tumɔs , i kin denja. If dat apin, di risk fɔ gɛt sik na di at ɛn di blɔd kin go ɔp.

Wetin de insay dɛn VLDL patikyula dɛn ya?

wan VLDL patikyula de mek am mכst wit wan kayn fεt we dεn kכ l triglisεrayd . I gɛt bak kɔlɔstrel , fat asid, ɛn wan spɛshal kayn prɔtin we dɛn kɔl apolipoprotein B (apoB) . Risach dɔn sho se we dis apoB de go ɔp na di blɔd, dat kin mek i gɛt mɔ sik we de mek pɔsin gɛt di sik we de na di at ɛn di blɔd.

infakt, כl di lipoprotein dεm na fεt εn protin dεm. כltu, di כmכnt εn di kayn fεt εn protin dεm de difrεn frכm wan lipoprotein to כda wan. Bikɔs VLDL dɛn gɛt bɔku triglisɛrɛyd , sɔntɛnde dɔktɔ dɛn kin kɔl dɛn “triglisɛrɛyd-rich lipoproteins.”

VLDL ɛn VLDL kɔlɔstrel na tu difrɛn tin?

Dis kin tan lɛk se i kin kɔnfyus smɔl, bɔt i izi fɔ ɔndastand. כltεm, dεn kin yuz di tεm dεm "VLDL" εn "VLDL kכlestכl" fכ chenj. Bɔt, smɔl difrɛns de bitwin dɛn tu we impɔtant fɔ no.

• VLDL de tכk bכt di lipoprotein patikyula we wi bin mεnshכn bifo. dat min se di "vehicle" כ "bus" we di liva de mek εn we de kכri fεt εn protin dεm na di bכdi.
• VLDL kכlestכl de t כk bכt di kכlestכl pasenja dεm na di VLDL bכs. dat min se di amoun fכ di kכlestכl we di VLDL patikyula de transpכt tru di bכdi.

כda kayn lipoprotein dεm lεk LDL (Low-Density Lipoproteins) εn HDL (High-Density Lipoproteins) , lεk dεn כda kayn kכlestכl ya. Ɛni wan pan dɛn kin kɛr difrɛn amɔnt ɛn kayn fat ɛn prɔtin dɛn. Na dat mek wi kin tɔk bak bɔt LDL kɔlɔstrel ɛn HDL kɔlɔstrel.

So VLDL na wan kayn "bad" kɔlɔstrel?

Yes, in shot, if di amount of VLDL kכlestכl na di bכdi hכy pas aw i nid, dεn kin se i "bad". Wi bɔdi nid sɔm kayn VLDL fɔ wetin i nid. כltu, if di lεvεl fכ VLDL inkrεs, i de inkrεs di risk fכ atεrosklεrosis , dat na di fכmeshכn fכ fεt dεposit (plek) insay di bכdi vεsul dεm (atεri dεm). Dis na di rut wae de mek bɔrku at sik.

Aw VLDL kin ɛp fɔ mek di atrɔsklerɔsis?

we wi liva de prodyuz mכr VLDL pas aw wi bכdi nid, di bכdi de mεtabolayz (brεk dכn) di εkstra VLDL. dis we ya, dεn kin kכnvכlt VLDL fכs to IDL (Intermediate-Density Lipoproteins) εn afta dat LDL (Low-Density Lipoproteins) . dis IDL εn LDL na di kayn lipoprotein dεm we di bכdi nid.

Bɔt, di prɔblɛm na dat, if dis LDL bi mɔ pas wetin di bɔdi nid, i kin bil. we di LDL pasmak de na di bכdi, i kin stכk na di wכl dεm na di at dεm, usay fεt dεposit (plek) kin bigin fכ fכm. As dɛn fat dɛn ya de gro smɔl smɔl, dɛn kin mek di at dɛn smɔl ɛn mek di blɔd nɔ flɔ.

Risach dɔn sho se we di VLDL lɛvɛl go ɔp wit di LDL lɛvɛl we de go ɔp , i denja pas fɔ inkris pan ɛni wan pan dɛn wan ya nɔmɔ.

nכto dat nכmכ, we VLDL brok dכn εn LDL fכm, sכm "rεmnant patikyula dεm " de lεf na di bכdi. Dɛn pat dɛn ya we lɛf kin gɛt bɔku kɔlɔstrel bak. Bikɔs dɛn smɔl, i kin izi fɔ lɛ dɛn stɔp na di at in wɔl dɛn ɛn mek dɛn gɛt atɛrosklɛrosis (I de mek di atɛrosklɛrosis prɔses kwik kwik wan.

Atherosclerosis na wan denja sik bikɔs i kin mek yu at dɛn smɔl, ɛn dis kin mek yu gɛt difrɛn kayn sik dɛn we de mek yu at ɛn blɔd. Na sɔm ɛgzampul dɛn ya:

• Koronari atεri sik ( sik dεm na di at dεm we de gi blɔd to di at) .
• Sεribrovaskul sik ( sik dεm na di bכdi we de gi bכdi to di bren, we kin mek i gεt kכndyushכn lεk strכk) .
• Pεriferal atεri sik ( sik dεm na di vein dεm we de gi bכdi to di an εn leg) .
• di aorta anεrizm (we di aorta, we na di men bכdi in bכdi de wik εn bulg) .

Uswan we wɔs pas ɔl, LDL ɔ VLDL?

infakt, כl tu di LDL kכlestכl εn VLDL kכlestכl de kכntribyut fכ di bכku fεt dεposit (plak) na wi at. So, i at fɔ se wan spɛshal "wɔs" pas di ɔda wan. di tu dεm na ikwal atherogenic, we min se dεn de kכntribyut to di prכsεs fכ fεt dεposit dεm na di at dεm. So i impɔtant fɔ no bɔt dɛn tu tin ya.

Aw dɛn kin mɛzhɔ VLDL kɔlɔstrel?

Dɔktɔ dɛn kin mɛzhɔ yu VLDL kɔlɔstrel lɛvɛl tru wan blɔd tɛst we dɛn kɔl lipid panɛl . Dis na blɔd tɛst we dɛn kin du rili kɔmɔn.

dis lipid panεl de sho di tכtal kכlestכl lεvεl na yu bכdi. Nɔto dat nɔmɔ, bɔt i de sho bak di lɛvɛl dɛn fɔ difrɛn kayn kɔlɔstrel we dɛn kɔl HDL (gud kɔlɔstrel), LDL (bad kɔlɔstrel), ɛn VLDL sɛpret wan. Dɔn bak, dis tɛst kin no di lɛvɛl we di triglisɛrɛyd dɛn de na yu blɔd. Mɛmba se wi bin se VLDL gɛt di mɔs triglisɛrɛyd.

Tipikli, lab dεm nכ de mεzj yu VLDL lεvεl dεm dairekt wan. Bifo dat, dεn de yuz yu triglisεrayd lεvεl dεm fכ kכl yu VLDL lεvεl wit mεtematכk fכmula. in jεnarכl, yu triglisεrayd dεm na lεk fayv tεm yu VLDL lεvεl.

Bɔt if yu gɛt wan bad bad sik we dɛn kɔl hypertriglyceridemia , we na we yu triglyceride lɛvɛl rili bɔku, dat mats nɔ go kɔrɛkt lɛk dat. If na so i bi, yu dɔktɔ kin yuz difrɛn we fɔ no di VLDL lɛvɛl we yu gɛt.

Wetin na di nɔmal rɛnj fɔ VLDL kɔlɔstrel?

wan nכmal, כ hεlty, lεvεl fכ VLDL na less dan 30 miligram pan wan desilita bכdi ( < 30 mg/dL ). If yu VLDL kɔlɔstrel lɛvɛl na 30 mg/dL ɔ pas dat , dɛn kin tek am se i ay.

Wetin kin apin if di VLDL kɔlɔstrel go ɔp?

di men tin na dat di εlevεt VLDL kכlestכl lεvεl de inkrεs di risk f כ kכdivaskyul sik .

Bɔt bikɔs dɛn kin kɔl di VLDL lɛvɛl bay we dɛn de yuz fɔmula we gɛt fɔ du wit matematiks pas fɔ mek dɛn mɛzhɔ am dairekt wan, ɛn bikɔs VLDL nɔto di men tin we dɔktɔ dɛn kin want fɔ du we dɛn de trit yu, yu dɔktɔ nɔ kin tɔk to yu spɛshal wan bɔt yu VLDL lɛvɛl. Bifo dat, i go pe atɛnshɔn mɔ pan ɔda valyu dɛn, lɛk yu LDL lɛvɛl ɛn triglisɛrɛyd , bikɔs na dɛn valyu dɛn de dɛn kin pe atɛnshɔn mɔ pan fɔ tritmɛnt.

Bɔrku pipul dɛm wae gɛt ay VLDL kɔlɔstrel lɛvɛl kin gɛt wan ɔr mɔr pan dɛn tin ya bak di sem tɛm:

• di triglisεrayd lεvεl dεm we de go כp (Hypertriglyceridemia) .
• Di LDL kɔlɔstrel lɛvɛl we de go ɔp
• Inkrεs pan di tכtal kכlestכl lεvεl (Hyperlipidemia) .

Di gud nyus na dat, sɔm tin dɛn de we yu kin du fɔ ridyus dɛn kayn fat ya (lipid) we de na yu blɔd:

• Fɔ fala di it we go ɛp yu at , lɛk di it we de na di Mɛditarenian , we de pe atɛnshɔn mɔ pan vɛjitebul, frut, ligɛm, ɔl gren, fish, ɛn fayn fayn fat lɛk ɔliv ɔyl.
• Ɛksesaiz ɔltɛm . Bɔt i rili impɔtant fɔ tɔk to yu dɔktɔ ɛn gɛt advays bifo yu bigin fɔ du nyu ɛksɛsayz program.
• Mek yu gɛt wɛlbɔdi wet we fit yu ayt ɛn di kayn bɔdi we yu gɛt.
• If yu dɔktɔ tɛl yu fɔ tek am, tek mɛrɛsin dɛn we de mek yu nɔ gɛt bɔku kɔlɔstrel lɛk statin .

Sɔntɛnde, yu kin gɛt wan ɔndalayn mɛdikal kɔndishɔn we kin mek yu triglisɛrɛyd dɛn go ɔp, we kin mek bak yu VLDL kɔlɔstrel lɛvɛl go ɔp. Dɔn bak, sɔm mɛrɛsin dɛn we yu kin tek kin mek dɛn tu mɛrɛsin ya go ɔp. So, i impɔtant fɔ tɔk opin wan wit yu dɔktɔ bɔt wetin de mek yu VLDL kɔlɔstrel lɛvɛl go ɔp ɛn wetin yu kin du fɔ adrɛs dɛn.

Insay yu ɛvride layf, yu nɔ go tink bɔku bɔt dɛn lipoprotein ya we de travul rawnd na yu blɔd. Bɔt if dɔktɔ tɛl yu se yu kɔlɔstrel bɔku, yu kin bigin tink bɔt dɛn tin ya ɛn aw dɛn kin afɛkt yu wɛlbɔdi.

Fɔ dɔn, sɔntin fɔ mɛmba...

Di tin we impɔtant pas ɔl na fɔ nɔ panik if yu kam fɔ no se yu kɔlɔstrel lɛvɛl bɔku. Dis na wan sik wae dɛn kin kɔntrol bɔrku tɛm wit chenj na yu layf ɛn if nid de, yu kin tek mɛrɛsin.

Tɔk to yu dɔktɔ bɔt wetin yu VLDL lɛvɛl min fɔ yu ɛn wetin yu kin du fɔ mek i bɛtɛ. I go gi yu di bɛst advays. Fɔ mek yu gɛt wɛlbɔdi, i go fayn fɔ mek yu no bɔt dɛn tin ya ɛn fala yu dɔktɔ in advays!

` Kכlestכl, VLDL, Triglisεrayd, At Sik, Lipid Panεl, Atεrosklεrosis, Wεlth