Yu gɛt tumɔs insulin na yu bɔdi? Lɛ wi tɔk bɔt Hyperinsulinemia!

Yu dɔn ɛva yɛri bɔt aw yu blɔd shuga de bɔku, ɔr di tin dɛn we kin apin bifo yu gɛt dayabitis? Wɛl, wan sik de we dɛn kɔl Hyperinsulinemia we gɛt fɔ du wit di ɔmon insulin we wi bɔdi de mek, bɔt sɔntɛm wi nɔ kin tɔk bɔt am bɔku. Nɔ wɔri, wi go tɔk bɔt am simpul wan, di we we yu go ɔndastand.

Wetin na dis (Hyperinsulinemia)? Fɔ put am rili simpul wan...

Hyperinsulinemia na we yu gɛt mɔ insulin pas aw i kin bi na yu blɔd. So, wetin na insulin? Na wan rili impɔtant ɔmon we wi pankrias de mek ɛn pul am. I de ɛp fɔ kɔntrol di lɛvɛl we di glukɔs (shuga) de na wi blɔd, mɔ di glukɔs.

Bɔku tɛm, wan sik we dɛn kɔl insulin resistance . Dis na we yu mɔsul, fat, ɛn liva sɛl dɛn nɔ de ansa fayn to insulin. We dis insulin rεsistεns de apin, yu bכdi de rεspכnd bay we i de prodyuz mכr insulin fכ tray fכ kip yu bכdi shuga lεvεl hεlth. Dis na we di hyperinsulinemia kin apin.

Imajin se pɔsin gɛt tumɔs insulin na in bɔdi, bɔt i nɔ gɛt insulin rɛsistɛns. If dat apin, dɛn blɔd shuga kin go dɔŋ, ɛn dis kin mek dɛn gɛt wan sik we dɛn kɔl haypoglycemia . Bɔt pan aypa insulinɛmia, we kin mek dɛn nɔ gɛt insulin, di insulin we pasmak nɔ kin mek dɛn blɔd shuga go dɔŋ.

Bɔt if dis insulin resistance ɛn hyperinsulinemia kɔntinyu, i kin mek pɔsin gɛt wan sik we dɛn kɔl hyperglycemia, usay di shuga na di blɔd kin kɔntinyu fɔ bɔku. Dis na di praymar kɔz fɔ prɛdiabitis ɛn tayp 2 dayabitis leta.

Impɔtant: (Hyperinsulinemia) ɛn (Hyperinsulinism) na tu difrɛn tin dɛn!

Bɔku pipul dɛn kin kɔnfyus di tu kɔndishɔn dɛn bikɔs dɛn nem dɛn fiba. Hyperinsulinism na difrɛn kɔndishɔn. Na wan prɔblɛm we de na di pankrias we de mek di insulin we de na di blɔd bɔku. Dis kin bi bikɔs ɔf wan tumbu we de mek tumɔs insulin (we dɛn kɔl insulinoma), ɔ bikɔs ɔf wan jenɛtik muteshɔn we de na di bɔdi. difrεnt lεk di haypa insulin, di haypa insulin de mek di bכdi shuga dכn (haypoglycemia).

Udat kin gɛt dis sik pasmak (Hyperinsulinemia)?

Hyperinsulinemia, we kin kam bikɔs pɔsin nɔ gɛt insulin, kin afɛkt ɛnibɔdi. I kin bi fɔ sɔm tɛm ɔ fɔ lɔng tɛm. Tu men tin dɛn de we dɛn tink se kin mek dis apin:

1. Fat we pasmak na yu bɔdi: Ɛkstra fat, mɔ we de rawnd yu bɛlɛ.

2. Nɔ de du bɔku tin dɛn: Dis min se yu nɔ de du ɛksasaiz.

Aw kɔmɔn tin kin apin we pɔsin gɛt aypa insulin?

Bikɔs no patikyula tɛst nɔ de fɔ no if pɔsin gɛt aypa insulin, ɛn di we aw pɔsin nɔ gɛt insulin nɔ kin mek i gɛt ɛni big big sayn te i go bifo to pɔsin we gɛt dayabitis ɔ tayp 2 dayabitis, i nɔ kin izi fɔ tɔk klia wan aw i kɔmɔn. Bɔt wi kin gɛt wan aidia bɔt ɔmɔs pipul dɛn gɛt prɛdiabɛtis. Bɔku bɔku big pipul dɛn ɔlsay na di wɔl gɛt prɛdiabetes. Dis min se na prɔblɛm we kɔmɔn pas aw yu go tink.

Aw di haypa insulin kin afɛkt mi bɔdi?

Apat frɔm prɛdiabɛtis ɛn tayp 2 dayabitis, haypa insulinemia gɛt fɔ du bak wit dɛn tin ya:

• Fat pasmak: Dis min se yu fɔ fat pasmak.
• Metabolic Syndrome: Dis na wan kɔlekɛshɔn fɔ sɔm kayn simptom dɛm wae gɛt fɔ du wit dis.
• Polycystic Ovary Syndrome (PCOS): Na di ɔmon we nɔ de balans we kin afɛkt uman dɛn.
• di triglisεrayd lεvεl dεm we de εlevεt: Dis na wan kayn fεt we de insay di bכdi.
• di yurik asid lεvεl we de go כp: Dis kin mek yu gεt kכndyushכn lεk gaut.
• Atherosclerosis: Dis kin mek yu gɛt at sik mɔ ɛn mɔ.
• Haypatɛnshɔn: Dis min se yu blɔd prɛshɔn go go ɔp .

Wetin na di sayn dɛm fɔ (Hyperinsulinemia)?

Hyperinsulinemia, we kin kam bikɔs yu nɔ gɛt insulin, nɔ kin mek yu gɛt ɛni klia sayn. Dis na bikɔs yu pankrias kin mek inof insulin fɔ mek i ebul fɔ win di resistans. Bɔt we yu kɔntinyu fɔ gɛt insulin ɛn we yu gɛt bɔku insulin, dat kin mek yu gɛt prɛdayabitis ɛn tayp 2 dayabitis.

Bɔrku pipul nɔr kin sho sayn dɛm fɔ prɛdiabetes fɔ lɔng lɔng tɛm. Bɔt sɔm pipul dɛn kin gɛt sɔm kayn sik lɛk:

• di skin we de dak na di armpit, bak pan di nεk, εn di sayd dεm: dεn kכl dis `(Acanthosis Nigricans)`. I tan lɛk dɔti, bɔt nɔto dɔti.
• Skin tag: Sɔm pipul dɛn kin kɔl dɛn skin tag ya bak.
• Di chenj dɛn we de apin na di yay:Dɛn tin ya kin go bifo leta to dayabitik retinopathy, we na wan sik we kin mek di yay dɛn pwɛl bikɔs ɔf dayabitis.

Di sayn dɛm fɔ Tayp 2 Dayabitis na:

• Fɔ tɔsti pasmak.
• Fɔ pis bɔku tɛm.
• Angri pasmak.
• Di vishɔn we nɔ klia.
• Ed de at.
• Infεkshכn dεm na di vagina εn skin.
• Dilay we wund de wɛl.

If yu gɛt wan ɔ mɔ pan dɛn sik ya, i rili impɔtant fɔ go to dɔktɔ wantɛm wantɛm.

Wetin na di tin dɛn we kin mek pɔsin gɛt aypa insulin?

Naw wi no se di men tin we kin mek pɔsin gɛt aypa insulin na we pɔsin nɔ ebul fɔ gɛt insulin . We yu gɛt insulin rɛsistɛns, yu bɔdi nɔ de yuz insulin fayn, so yu pankrias gɛt fɔ mek ɛkstra insulin fɔ tray fɔ mek yu blɔd shuga lɛvɛl wɛl.

Aw fɔ no if pɔsin gɛt (Hyperinsulinemia)?

Hyperinsulinemia kin tranga smɔl fɔ no, as i nɔ de sho di sayn dɛm te i go bifo to prɛdiabetes ɔ tayp 2 dayabitis. Nɔ kɔmɔn tɛst nɔ de bak fɔ no di insulin lɛvɛl kɔrɛkt wan na di blɔd, bikɔs di insulin lɛvɛl kin chenj bad bad wan ɔl di de.

So, bikɔs no wan tɛst nɔ de we go ebul fɔ no if pɔsin gɛt aypa insulin, yu dɔktɔ go tink bɔt sɔm tin dɛn we i de chɛk dis sik:

• Yu mεdikal histri: tin dεm lεk sik dεm wae yu bin dɔn gɛt bifo εn mεdikal dεm wae yu bin dɔn tek.
• Famili mɛdikal histri: Ɛnibɔdi na di famili gɛt sik lɛk dayabitis?
• Fɔ chɛk yu bɔdi.
• Di sayn dɛm wae yu de sho.
• Di rizɔlt fɔ blɔd tɛst: Fɔ ɛgzampul, fasting plasma glucose (FPG) tɛst.

Aw dɛn kin trit aypa insulinemia?

Bikɔs nɔto ɔl di tin dɛn we kin mek pɔsin gɛt aypa insulin (e.g., jɛnɛtik tin dɛn, ej) dɛn kin trit, chenj dɛn layf na di men tritmɛnt. Dɛn chenj ya we wi kin chenj di we aw pɔsin de liv in layf na:

• Lɛ yu bɔdi kam dɔŋ:
• Yu dɔktɔ ɔ pɔsin we sabi bɔt it kin advays yu fɔ stɔp fɔ it tin dɛn we gɛt bɔku kabɔhaydrɛt (e.g. rays, bred, nuts, pɔt, ɛn swit) bikɔs dɛn tin ya kin mek dɛn mek insulin.
• I se bak fɔ ridyus di fat we nɔ fayn, shuga, rɛd mit, ɛn stach dɛn we dɛn dɔn prosɛs .
• Bifo dat, it wan it we gɛt bɔku vɛjitebul, frut, ful gren, fish, ɛn bɔd we nɔ gɛt bɔdi.Dɛn kin advays am fɔ du am. Tink bɔt am lɛk fɔ sheb wan plet rays, ridyus di rays smɔl ɛn ad mɔ vɛjitebul, ɔyl, ɛn spays.

• Ɛksasayz:
• We yu du ɛksɛsayz, i de mek di insulin nɔ bɔku ɛn smɔl smɔl i de mek di insulin sɛnsitiviti go ɔp. Dis kin ɛp fɔ trit di hyperinsulinemia. I fayn if yu ebul fɔ du sɔntin lɛk fɔ waka, rɔn, ɔ bayk fɔ at le 30 minit ɛvride.

• We yu de lɔs yu wet:
• If yu lɔs yu wet, dat kin ɛp fɔ mek di sik bɛtɛ (Hyperinsulinemia). Bɔt we yu gɛt bɔku bɔku bɔdi, dat kin mek dis sik wɔs.
• Hyperinsulinemia kin bɛtɛ bak we dɛn trit pɔsin we fat wit chenj in layf, it, mɛrɛsin, ɔ bariatric ɔpreshɔn (dis na ɔpreshɔn fɔ ridyus fat).

Fɔ pipul dɛn we fat bad bad wan we dɛn du bariatric ɔpreshɔn, di haypa insulin kin bɛtɛ kwik kwik wan insay wan wik afta dɛn du di ɔpreshɔn. di insulin sεnsitiviti de impruv bak bitwin 6 εn 24 mכnt afta dεn כpεrayshכn. Bɔt dis ɔpreshɔn nɔ fayn fɔ ɔlman. Tɔk to yu dɔktɔ bɔt us tritmɛnt opshɔn dɛn we fayn fɔ yu.

Yu tink se dɛn kin rivɛns di haypa insulin?

difrεn kכz dεm εn fכs tin dεm de we de kכntribyut fכ insulin rεsistεns (i.e., haypa insulinεmia). We yu chenj di we aw yu de liv yu layf lɛk fɔ it fayn it, fɔ du ɛksɛsayz ɔltɛm , ɛn fɔ lɛf fɔ it bɔku bɔku it kin ɛp fɔ mek yu gɛt mɔ insulin ɛn fɔ mek yu nɔ gɛt insulin ɛn fɔ mek yu nɔ gɛt bɔku insulin. Dat min se, yes, dɛn kin kɔntrol am bɔku bɔku wan, ɛn sɔntɛnde dɛn kin ivin chenj am! כltu, nכto כl di kכz dεm kin rivεs (e.g., jεnεtik inflכεns).

I bεst fɔ tɔk to yu dɔktɔ bɔt wetin yu kin du fɔ mek yu ebul fɔ kɔntrol yu insulin rεsistεns εn haypa insulinεmia fayn fayn wan.

Wetin na di tin dɛm wae kin mek yu gɛt hyperinsulinemia?

Sɔm tin dɛn we kin mek yu gɛt prɔblɛm wit yu jɛnɛtik ɛn aw yu de liv yu layf kin mek yu gɛt mɔ insulin. Dɛn tin ya we kin mek pɔsin gɛt prɔblɛm na:

• Fɔ gɛt bɔku bɔku bɔdi ɔ fɔ fat pasmak: Fɔ gɛt bɔku fat, mɔ we yu de rawnd yu bɛlɛ.
• Ej 45 ia ɔ pas dat.
• Fɔ gɛt pɔsin we de nia yu fambul (mama ɔ papa ɔ brɔda ɛn sista) we gɛt dayabitis.
• Fɔ liv yu layf we yu nɔ de sidɔm ɛn we yu nɔ de du bɛtɛ tin fɔ du wit yu bɔdi.
• Fɔ gɛt wɛl bɔdi prɔblɛm lɛk ay blɔd prɛshɔn ɛn abnɔmal kɔlɔstrel lɛvɛl.
• Fɔ dɔn gɛt at atak ɔ strok bifo.
• Fɔ gɛt prɔblɛm dɛn we gɛt fɔ du wit slip lɛk slip apnea.
• Fɔ smok.

Wetin na di prɔgnosis fɔ hyperinsulinemia?

Di luk fɔ hyperinsulinemia, dat na aw di sik go divɛlɔp, de dipen pan sɔm tin dɛm:

• Insulin Resistance: Dis na di praymar kɔz fɔ hyperinsulinemia.
• Di kayn we aw insulin rɛsistɛns ɛn di haypa insulinemia.
• Aw yu kin gɛt sɛkɔndari kɔmplikeshɔn dɛn we kin apin bikɔs ɔf insulin rɛsistɛns ɛn aypa insulinemia.
• Aw yu de adap to di tritmɛnt ɛn aw yu bɔdi de du am.

Sɔm pipul dɛn kin gɛt smɔl insulin resistance ɛn hyperinsulinemia, we nɔ kin ɛva go bifo to prɛdiabetes ɔ tayp 2 dayabitis. Dɔn bak, sɔm pipul dɛn kin rivεs ɔr mεnεj insulin rεsistεns εn haypa insulinεmia wit layf stεyl chenj.

Bɔt if yu gɛt Tayp 2 Dayabitis bikɔs ɔf Hyperinsulinemia, yu fɔ tray yu bɛst fɔ manej di sik fayn fayn wan fɔ avɔyd prɔblɛm dɛn we kin apin.

Ustɛm a fɔ go to dɔktɔ bɔt aypa insulinemia?

If dɛn dɔn no se yu gɛt aypa insulin ɔ kɔndishɔn dɛn we gɛt fɔ du wit insulin rɛsistɛns, i rili impɔtant fɔ go to dɔktɔ ɔltɛm fɔ mek shɔ se yu blɔd shuga lɛvɛl de na di wɛlbɔdi lɛvɛl ɛn yu tritmɛnt de wok.

If yu gɛt sayn dɛm fɔ ay blɔd shuga (di wan dɛm wae wi bin dɔn tɔk bɔt fɔs) ɔr sayn dɛm fɔ prɛdiabɛtis, go to yu dɔktɔ wantɛm wantɛm. Dɛn kin du simpul tɛst fɔ chɛk yu blɔd shuga.

Hyperinsulinemia na wan sik wae kin ambɔg yu. Bɔku tɛm, na di rizin we mek dɛn nɔ gɛt insulin. Bikɔs i nɔ de sho di sayn dɛn te i go bifo to prɛdiabɛtis ɔ tayp 2 dayabitis, di bɛst tin we yu go du na fɔ mek yu gɛt wɛlbɔdi wet, ɛksesaiz ɔltɛm , ɛn it fayn it. Dɛn tin ya kin ɛp fɔ mek yu nɔ gɛt ɛn rivɛns insulin rɛsistɛns ɛn aypa insulinemia.

Di impɔtant tin dɛn we wi nid fɔ mɛmba (Take-Home Message) .

Okay, so a op se yu naw geht gud aidia boht wetin wi bin de tok boht (Hyperinsulinemia). Fɔ sɔmtin:

• Hyperinsulinemia na we insulin de go ɔp na di blɔd, bɔku tɛm na bikɔs di insulin nɔ de wok.
• Pan ɔl we dis nɔ kin sho ɛni sayn fɔs, leta i kin mek pɔsin gɛt prɛdiabɛtis ɛn tayp 2 dayabitis, ɛn bɔku ɔda wɛlbɔdi prɔblɛm dɛn.
• Fɔ liv fayn layf (gud it, ɛksesaiz, kɔntrol yu wet) na di bɛst shild ɛn di bɛst mɛrɛsin fɔ dis.
• If yu gɛt risk factor ɔr fil lɛk yu gɛt ɛni sayn, nɔr shem fɔ go to dɔktɔ. If dɛn no am kwik, i kin izi fɔ kɔntrol am.

Mɛmba se yu wɛlbɔdi de na yu an. Smɔl chenj dɛn kin mek big difrɛns!

` Hyperinsulinemia, Insulin Resistance, Dayabitis, Blɔd Shuga, Pankrias, Nutrishɔn, Ɛksesaiz