Yu gɛt dayabitis? Yu fɔ no bɔt di denja sik we dɛn kɔl DKA (Diabetic Ketoacidosis) .

Yu na pɔsin we gɛt dayabitis? Dɔn yu fɔ de wɔri ɔltɛm bɔt di shuga we yu gɛt na yu blɔd, nɔto so? Bɔt yu dɔn yɛri bɔt wan sik we rili siriɔs we kin ivin put yu layf pan denja we kin kam wit dayabitis? Na dat wi de tok abaut tide. Dɛn kin kɔl dis sik we dɛn kɔl Dayabetic Ketoacidosis , ɔ (DKA) fɔ shɔt tɛm. I rili impɔtant fɔ mek yu sef no bɔt dis gud gud wan.

Fɔ tɔk am simpul wan, wetin na DKA?

Okay, mek wi put am dis we. Insulin na ɔmon we de ɛp wi bɔdi in sɛl dɛn fɔ yuz glukɔs ɔ shuga fɔ gɛt pawa. We pɔsin we gɛt dayabitis nɔ gɛt inof insulin, ɔ we di insulin we i gɛt nɔ de wok fayn, di sɛl dɛn nɔ kin ebul fɔ yuz di shuga we de na di blɔd.

Dɔn wi bɔdi kin tink se, "O, a de dɔn wit ɛnaji." So di bɔdi de yuz ɔda we fɔ mek ɛnaji. Dat na bay we yu de bɔn di fat we de na di bɔdi. We dɛn bɔn di fat dis kayn we, wan kemikal we dɛn kɔl ketɔn kin kɔmɔt na di blɔd as tin we dɛn kin yuz fɔ mek di blɔd. Na nɔmal tin fɔ gɛt sɔm ketɔn. Bɔt we dɛn nɔ chɛk dis prɔses bikɔs insulin nɔ de, bɔku kitɔn dɛn kin gɛda na di blɔd. dis kεtכn dεm na wan kayn asid. So, we dɛn gɛda na di blɔd, wi blɔd kin bi asid.

If yu blɔd shuga lɛvɛl kin pas 240 mg/dL , i fayn fɔ chɛk yu ketɔn lɛvɛl ɛvri 4-6 awa. If yu blɔd shuga lɛvɛl pas 250 mg/dL, dɛn kin tek am se na di biginin fɔ DKA. If yu blɔd shuga nɔ de nia di nɔmal rɛnj, tɔk to yu dɔktɔ wantɛm wantɛm .

Ustɛm yu fɔ go na imejensi ɔspitul (ETU)?

If yu blɔd shuga lɛvɛl na 300 mg/dL ɔ pas dat ɛn i nɔ de kam dɔŋ, ɔ if yu kɔntinyu fɔ vɔmit, fil taya pasmak, ɔ gɛt sayn dɛn fɔ DKA lɛk fɔ at fɔ blo, go to di Imejɛnsi Dipatmɛnt (ETU) we de nia yu wantɛm wantɛm.

Yu tink se Ketoacidosis ɛn Ketosis na tu difrɛn tin?

Yɛs, pan ɔl we dɛn tu wɔd ya tan lɛk se dɛn fiba, dɛn na tu tin dɛn we rili difrɛn.

• Ketosis: Dis kin apin we yu it it we nɔ gɛt bɔku kabɔhaydrɛt (lɛk bred, rays, pasta) (e.g., di keto it). Dɔn di bɔdi kin bɔn fat fɔ gɛt ɛnaji. na ya bak, dεn de mek di kεtכn dεm, bכt i de apin insay wan we we dεn kכntrol. Na bikɔs inof insulin de na di bɔdi. Ketosis nɔto tin we de mek pɔsin denja.
• Kεtoasidכsis (DKA): .Dis kin apin bikɔs dɛn nɔ gɛt insulin. di kεtכn dεm kin bכku we dεn nכ kin kכntro, we kin mek di blכd bi asid. Dis kin mek pɔsin in layf de pan denja.

Wetin na di sayn dɛm fɔ DKA?

Di sayn dɛm fɔ DKA kin kam kwik kwik wan. Sɔntɛnde, dɛn kin fɔs no se pɔsin gɛt dayabitis afta i dɔn gɛt DKA. E fayn fɔ no bɔt dɛn sik ya.

Wetin na di men tin dɛn we kin mek pɔsin gɛt DKA?

Pan ɔl we di men tin we kin mek pɔsin gɛt DKA na we i nɔ gɛt insulin, bɔku tin dɛn de we kin mek i gɛt dis sik.

• Nɔ tek insulin kɔrɛkt wan: Dis risk kin bɔku if yu nɔ tek di insulin doz we yu dɔktɔ tɛl yu fɔ tek di tɛm ɔ if yu skip wan doz. Sɔntɛnde, fɔ yuz insulin we dɔn pas kin bi bak wan kɔz.
• Sik: We yu gɛt fiva, kol, urinary tract infection (UTI), ɔ nyumonia, yu bɔdi kin mek ɔmon dɛn we de mek yu strɛs (lɛk kɔtisol ɛn adrenaline). Dɛn ɔmon dɛn ya kin ambɔg di wok we insulin de du. So, yu kin gɛt DKA pan de dɛm wae yu sik.
• Ɔda tin dɛn we kin apin to di bɔdi: Di ​​risk fɔ gɛt DKA kin bɔku bak bikɔs ɔf di strɛs we de bɔku pan di bɔdi, lɛk fɔ du ɔpreshɔn, fɔ gɛt aksidɛnt, ɔ fɔ gɛt at atak ɔ strok.
• Dayabitis we dɛn nɔ no: Pipul dɛn we nɔ no se dɛn gɛt dayabitis, mɔ di wan dɛn we gɛt Tayp 1 dayabitis, kin gɛt DKA.
• Ɔda tin dɛn we kin mek yu gɛt: If yu drink pasmak, yu de yuz sɔm drɔgs, yu gɛt bɛlɛ, ɛn sɔm mɛrɛsin dɛn (e.g. kɔtikosterɔyd) kin mek yu gɛt DKA bak. If yu gɛt dayabitis, i impɔtant fɔ tɛl yu dɔktɔ bifo yu tek ɛni nyu mɛrɛsin.

Aw yu go mek DKA nɔ de divɛlɔp?

Bɔt gud tin na dat, DKA na sik we pɔsin kin ebul fɔ avɔyd, ɛn di men tin na fɔ kɔntrol yu dayabitis fayn fayn wan.

Na sɔm tin dɛn we yu kin du bɔt am:

• Chek yu blɔd shuga lɛvɛl ɔltɛm: Chek yu blɔd shuga lɛvɛl bɔku tɛm insay di de, lɛk aw yu dɔktɔ tɛl yu. Tray fɔ kip dɛn insay di say we dɛn kin se.
• Tek di mɛrɛsin ɛksaktɔli:Tek insulin ɔ ɔda mɛrɛsin fɔ dayabitis di rayt tɛm, di rayt doz. Nɔ ɛva skip di doz dɛn.
• It fayn: Fɔ fala di it plan we dɛn gi yu di rayt we.
• Drink bɔku wata: Drink bɔku wata we nɔ gɛt shuga ɛn wata.
• Mek plan fɔ sik de: Tɔk to yu dɔktɔ bifo tɛm bɔt aw fɔ chɛk yu blɔd shuga ɛn kitɔn lɛvɛl we yu gɛt fiva ɔ kol, ɛn if yu nid fɔ chenj yu insulin doz.
• Chek fכ kεtכn: Chεk fכ kεtכn εni tεm we yu bכdi shuga hכy pas 240 mg/dL. Yu kin du dis yuz urine strips ɔ sɔm glukomita.
• Rɛdi fɔ ɛni imejensi: Plan bifo tɛm wetin fɔ du ɛn usay fɔ go if DKA simptom dɛn sho.

Wetin na di tritmɛnt fɔ DKA na di ɔspitul?

We yu go na ETU wit di sayn dɛm fɔ DKA, dɔktɔ dɛm go tek kwik step fɔ kɔntrol yu kɔndishɔn. Dɛn kin gi dis tritmɛnt na ɔspitul.

1. Fluid we de insay di blɔd: Dɛn kin gi salin insay di bɛlɛ fɔ kɔntrol di wata we de kɔmɔt na di bɔdi ɛn fɔ kɔrɛkt di kemikal balans na di blɔd.

2. Insulin: Dɛn kin gi insulin insay di bɛlɛ fɔ stɔp di fɔmɛshɔn fɔ di ketɔn dɛn ɛn fɔ kɔntrol di blɔd shuga lɛvɛl.

3. Ilektrolayt Riplesmεnt: DKA de mek di bכdi lכs di imכtant minral dεm (ilεktrolayt) lεk sכdiכm εn potashכm. Bikɔs dɛn tin ya impɔtant fɔ mek di at, di mɔsul dɛn, ɛn di nerv dɛn wok, dɛn kin yuz salin fɔ chenj dɛn.

4. Tritmɛnt fɔ infɛkshɔn: If di tin we kin mek pɔsin gɛt DKA na infɛkshɔn, dɛn kin gi am antibayɔtik.

Bɔrku tɛm, dɛn kin kɔntrol de sik insay 24 awa, bɔt if de sik siriɔs, i kin tek lɔng tɛm.