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Wi go tɔk bɔt dis spɛshal kayn dayabitis we kin kam pan big pipul dɛn (Latent Autoimmune Diabetes in Adults - LADA)?

Wi go tɔk bɔt dis spɛshal kayn dayabitis we kin kam pan big pipul dɛn (Latent Autoimmune Diabetes in Adults - LADA)?

Tide wi go tɔk bɔt wan kayn dayabitis we difrɛn smɔl. Sɔntɛm yu nɔ bin dɔn yɛri bɔku tin bɔt am. Dɛn kɔl am LADA, we tinap fɔ Latent Autoimmune Diabetes in Adults. Dis kin tan lɛk se i kɔmplikt smɔl, bɔt lɛ wi brok am to simpul ɛksplen bɔt wetin de apin.

Wetin na LADA rili?

Fɔ tɔk am simpul wan, pan dis dayabitis we dɛn kɔl LADA, yu kin si di sayn dɛm fɔ di fɔs kayn dayabitis (`Type 1 diabetes`) ɛn bak di simptom dɛm fɔ di sɛkɔn kayn dayabitis (`Type 2 diabetes`). Na dat mek sɔm pipul dɛn kin kɔl am bak `Type 1.5 diabetes` . fכ bi prεsis, dכkta dεn biliv se dis kin kכz bak fכ difεkt na wi bכdi in כwn imyun sistεm (dεn kכl dis `autoimmune condition`). Bɔt dɛn kin no am mɔ we pɔsin big, dat na bitwin 30 ɛn 50. Wan ɔda spɛshal tin na dat, lɛk `Type 2 diabetes`, i kin go ɔp bak smɔl smɔl pan di simptom dɛm. Fɔ dis rizin, bɔku tɛm dɛn kin misdiagnose am as `Type 2 diabetes`.

Aw kɔmɔn tin fɔ LADA?

Pan ɔl we bɔku pipul dɛn nɔ yɛri bɔt LADA, fɔ tru, i nɔ kin bɔku so. Stɔdi dɔn sho se bitwin 4% ɛn 12% pan di pipul dɛm we dɛn bin dɔn no fɔs se dɛn gɛt `Type 2 diabetes` kin rili gɛt LADA. Jɔs tink bɔt, lɛk 530 milyɔn big pipul dɛn ɔlsay na di wɔl de sɔfa wit `Type 2 diabetes`. Dat min se bɔku bɔku pipul dɛn kin gɛt LADA! So, i rili impɔtant fɔ mek wi no bɔt dis.

Wetin na di sayn dɛm fɔ LADA?

Di sayn dɛm fɔ LADA kin rili fiba di wan dɛm wae kin gɛt ɔda kayn shuga. Si if dɛn tin ya tan lɛk se yu sabi:

  • Fɔ fil fɔ tɔsti ɔltɛm (polydipsia).
  • Di nid fɔ pis ɔltɛm.
  • We yu nɔ go ebul fɔ imajin fɔ lɔs yu wet .
  • Di vishɔn we nɔ klia .
  • Fɔ fil taya ɔltɛm (taya).
  • Skin we dray ɛn we de it .

If yu gɛt wan ɔ mɔ pan dɛn sayn ya, i go fayn fɔ mek yu go to dɔktɔ.

Wetin kin mek pɔsin gɛt LADA?

Risach pipul dɛm tink se LADA kin apin bikɔs di antibodi dɛm na wi bɔdi kin mistek atak ɛn pwɛl di sɛl dɛm we de mek insulin na wi pankrias. As yu no, insulin na ɔmon we de ɛp wi sɛl dɛn fɔ tek di shuga (glucose) we de na wi blɔd ɛn yuz am fɔ gɛt ɛnaji. So, we insulin nכ de prodyuz fayn, shuga (glucose) de bכku na di bכdi. Dis we ya, i tan lɛk Tayp 1 dayabitis. Pipul dɛn we gɛt Tayp 1 dayabitis fɔ tek insulin injɛkshɔn fɔ mek dɛn kɔntinyu fɔ liv. Bɔku tɛm, dɛn kin no se dɛn gɛt tayp 1 dayabitis we dɛn smɔl ɔ we dɛn yɔŋ.

Bɔt lɛk Tayp 2 dayabitis, dɛn kin no se LADA dɔn big, ɛn di sayn dɛn kin sho smɔl smɔl. Dis na bikɔs yu pankrias de ridyus di insulin we i de mek smɔl smɔl. So di sayn dɛm nɔ kin apin wantɛm wantɛm. Yu nɔ go nid fɔ gɛt insulin injɛkshɔn fɔ mɔnt ɔ ia . Dis na di rizin we mek sɔm dɔktɔ dɛn kin tink se dɛn gɛt Tayp 2 dayabitis fɔs, nɔto LADA.

Ɛni spɛshal trig de fɔ LADA?

No patikyula ‘trig’ nɔ de fɔ LADA. Dis na bikɔs na sɔntin we yu nɔ ebul fɔ kɔntrol, we yu imyun sistɛm de mek. Sɔntɛnde yu nɔ kin ivin no se yu gɛt LADA. Dis na bikɔs yu pankrias stil de mek inof insulin fɔ mek i nɔ mek yu gɛt di sik.

Wetin na di risk factor dɛm fɔ LADA?

LADA gɛt wan strɔng jenɛtik kɔmpɔnɛnt. Dis min se if yu mama ɛn papa ɔ grani ɛn granpa gɛt dis sik, yu go gɛt dis sik. Pruf de bak se di tin dɛn we de apin na di envayrɔmɛnt ɛn di tin dɛn we de mek pɔsin liv in layf, lɛk we pɔsin fat, kin mek i gɛt LADA. Bɔt, nɔto dɛn tin ya nɔmɔ kin mek pɔsin gɛt dis sik.

Posisibul kɔmplikɛshɔn dɛn we LADA kin gɛt

De big prɔblɛm kin kam wae yu nɔr trit dis sik fayn fayn wan. Bikɔs i tan lɛk Tayp 2 dayabitis, yu nɔ go bigin fɔ tek insulin tritmɛnt kwik kwik wan. Dis kin mek di risk fɔ gɛt wɛlbɔdi prɔblɛm lɛk di kidni we de pwɛl .

Dayabitis-rilayt ketoacidosis (DKA) na wan rili siriɔs, we kin mek pɔsin in layf de pan denja fɔ LADA. I kin apin we yu liva bigin fɔ brok di bɔdi fat fɔ gɛt ɛnaji we yu sɛl dɛn nɔ gɛt inof glukɔs (blɔd shuga) fɔ yuz fɔ ɛnaji. Dis na mɛdikal imejensi.

Aw dɛn kin no bɔt LADA?

LADA kin afɛkt big pipul dɛn we dɛn bin dɔn no fɔs se dɛn gɛt Tayp 2 dayabitis. Bɔt we yu nɔ ebul fɔ kɔntrol yu blɔd shuga lɛvɛl wit mɛrɛsin dɛn we yu de it (lɛk mɛtfɔmin) ɛn chenj yu layf (lɛk fɔ it tin dɛn we gɛt fayn fayn tin dɛn fɔ it), dɔktɔ dɛn kin sɔprayz se yu gɛt Tayp 1.5 dayabitis (we dɛn kin kɔl bak LADA).

Yu dɔktɔ kin no LADA wit wan blɔd tɛst we dɛn kɔl GAD Antibodies Test (`GAD Antibodies Test` - `glutamic acid decarboxylase antibodies`) . dis de chεk fכ di prεsεns fכ `autoantibodies` dεm we de atak glutamik asid dεkarbכksilayz (`GAD`). If yu gɛt dɛn ɔtoantibodi ya na yu blɔd, dat min se yu gɛt ɔtoimyun sik we de afɛkt yu pankrias. If yu na big pɔsin, i go mɔs bi se na LADA.

Di dɔktɔ kin ɔda fɔ mek dɛn du C-peptide tɛst.Dɛn kin tɛl yu bak fɔ du tɛst. dis de mכsu aw bכku C-pεptida de insay yu bכdi. C-peptide kin tɛl yu dɔktɔ aw bɔku insulin yu pankrias de mek.

Wetin na di tritmɛnt dɛm fɔ LADA?

Fɔ trit LADA na tin we kɔmplikt smɔl. We yu bigin, yu kin tek di mɛrɛsin dɛn we yu kin tek fɔ gɛt dayabitis. Dɛn kin ɛp fɔ kɔntrol yu blɔd shuga fɔ sɔm tɛm. Bɔt as di sik de wɔs, di we aw yu bɔdi de tek dɛn mɛrɛsin dɛn de go dɔŋ. Dɔn yu fɔ chenj to insulin injɛkshɔn fɔ kɔntrol yu blɔd shuga. Bikɔs yu pankrias kin lɔs in pawa fɔ mek insulin fɔ insɛf kpatakpata. Dis kin apin fɔ sɔm mɔnt ɔ sɔm ia.

No tritmɛnt nɔ de we dɛn dɔn gri pan fɔ LADA. Sɔm spɛshal pipul dɛn biliv se i bɛtɛ fɔ bigin tritmɛnt wit insulin wantɛm wantɛm. Ɔda pipul dɛn kin wet fɔ sɔm tɛm bifo dɛn bigin fɔ tek insulin injɛkshɔn. Yu dɔktɔ go disayd us tritmɛnt go fayn fɔ yu.

Bikɔs di doz ɛn di mɔnt fɔ insulin we yu nid kin chenj, yu nid fɔ kɔntinyu fɔ wach di shuga we yu gɛt na yu blɔd.

Wetin pɔsin we gɛt LADA kin ɛkspɛkt?

No mɛrɛsin nɔ de fɔ Tayp 1.5 dayabitis. Bɔt yu dɔktɔ kin ɛp yu fɔ fɛn tritmɛnt fɔ ɛp yu fɔ kɔntrol am. We yu bigin fɔ tek insulin, yu go ebul fɔ kɔntrol di shuga we de na yu blɔd bɛtɛ bɛtɛ wan. If yu de manej yu fayn fayn wan, yu go ebul fɔ liv nɔmal layf.

Wetin na di layf we pɔsin we gɛt LADA de liv?

Yu layf dipen pan bɔku tin dɛn. If yu gɛt Tayp 1.5 dayabitis, dat nɔ kin mek yu layf shɔt. Bɔt if yu gɛt LADA ɛn yu nɔ ebul fɔ kɔntrol yu glukɔs lɛvɛl, yu de pan risk fɔ gɛt wɛlbɔdi prɔblɛm dɛn we kin mek yu layf shɔt. Di men rizin fɔ dis na bikɔs if pɔsin gɛt bɔku shuga na in blɔd fɔ lɔng tɛm, i kin mek i gɛt prɔblɛm dɛn lɛk at sik . Dɛn tin ya kin afɛkt yu layf. Na dat mek i rili impɔtant fɔ kɔntrol di blɔd shuga.

Yu tink se dɛn kin mek dɛn nɔ gɛt LADA?

No we nɔ de fɔ mek yu nɔ gɛt LADA dayabitis. Bikɔs yu nɔr kin ebul fɔ kɔntrol ɔr prɛvɛnt di ɔtoimyun kɔndishɔn wae de kɔz am , fɔ no di sik kwik kwik wan ɛn trit am na di bɛst we fɔ mek yu nɔr gɛt prɔblɛm wit dis sik.

Aw a go tek kia ɔf misɛf if a gɛt LADA?

Di bɛst we fɔ kia fɔ yusɛf na fɔ kɔntrol di shuga we yu gɛt na yu blɔd. Bɔku tɛm, dɛn nɔ kin no di rɔŋ we fɔ gɛt LADA dayabitis. I kin mek yu at pwɛl we yu tink se yu de du ɔltin fayn, bɔt i stil at fɔ kɔntrol di shuga we yu gɛt na yu blɔd. If dɛn no se yu gɛt LADA, fala di tritmɛnt plan we yu dɔktɔ gi yu ɛksaktɔli.. Mek shɔ se yu kip ɔl di apɔntinmɛnt dɛn we yu dɔn mek fɔ fala yu. Na dɛn apɔntinmɛnt ya, yu dɔktɔ kin chɛk yu pankrias ɛn ɔl yu wɛlbɔdi. Dɛn kin ajɔst bak di insulin we yu nid.

Ustɛm yu fɔ go to dɔktɔ?

If yu gɛt sɔm sayn dɛn fɔ gɛt dayabitis, lɛk fɔ tɔsti pasmak ɛn fɔ pis ɔltɛm, go to dɔktɔ wantɛm wantɛm. Nɔ fɔgɛt fɔ sheb yu kɔmplit wɛl bɔdi histri wit yu dɔktɔ.

Na mɔtalman, if dɛn dɔn no se yu gɛt `Type 2 diabetes` ɛn yu simptom nɔ de bɛtɛ afta yu dɔn trit yu, aks yu dɔktɔ fɔ tɛst yu fɔ `Type 1.5 diabetes` (LADA).

Tayp 1.5 dayabitis, ɔ LADA, na wan sik we bɔku tɛm dɛn nɔ kin no di rayt we. I gɛt sayn dɛm fɔ ɔl tu di Tayp 1 ɛn Tayp 2 dayabitis. Bɔt bɔku tɛm, di sayn dɛm kin sho se yu gɛt Tayp 2. So i kin mek yu at pwɛl ɛn kɔnfyus fɔ no se yu gɛt dis kayn dayabitis. Yu kin bigin fɔ tek mɛrɛsin dɛn we yu kin it, bɔt yu blɔd shuga stil bɔku. Ɔ yu tritmɛnt kin wok fɔ sɔm tɛm, bɔt afta dat i kin stɔp fɔ wok. Yu dɔktɔ dɛn go ɛp yu fɔ fɛn di bɛst we fɔ kɔntrol di shuga we yu gɛt na yu blɔd ɛn fɔ mek yu gɛt wɛlbɔdi. Nɔr frayd fɔ aks kwɛstyɔn bɔt yu diagnosis ɛn tritmɛnt plan.

Wetin na di impɔtant tin dɛn we wi go kɛr go na os frɔm dis atikul?

LADA na spɛshal kayn dayabitis we kin apin to big pipul ɛn i gɛt sayn dɛm fɔ ɔl tu di `Type 1` ɛn `Type 2` dayabitis. Sɔntɛnde dɛn kin misdiagnose am as `Type 2`.

  • Di sayn dɛm na fɔ tɔsti pasmak, fɔ pis ɔltɛm, fɔ lɛf fɔ gɛt bɔku bɔku bɔdi, ɛn fɔ taya.
  • dis kin kכz fכ wan כtoimyun kכndishכn, εn i kin gεt jεnεtik kכmכpכnt bak.
  • If dɛn nɔ no di sik kɔrɛkt wan ɛn trit am kwik, prɔblɛm dɛn lɛk di kidni we dɔn pwɛl ɛn DKA kin apin.
  • Dis na difinitiv wan we dɛn kin no bay di GAD Antibodies Test.
  • Pan ɔl we dɛn kin gi mɛrɛsin fɔs as tritmɛnt, as tɛm de go, dɛn kin nid fɔ gi insulin injɛkshɔn.
  • If dɛn dɔn no se yu gɛt Tayp 2 dayabitis ɛn yu tritmɛnt nɔ de kɔntrol am, tɔk to yu dɔktɔ bɔt LADA. Nɔ ɛva stɔp ɔ chenj yu tritmɛnt fɔ yusɛf. Ɔltɛm fala wetin yu dɔktɔ tɛl yu fɔ du.

` LADA, dayabitis, dayabitis we kin bigin fɔ big pipul, tayp 1.5 dayabitis, ɔtoimyun dayabitis, insulin, blɔd shuga

Frequently Asked Questions (FAQ)

Ɛni spɛshal trig de fɔ LADA?

No patikyula ‘trig’ nɔ de fɔ LADA. Dis na bikɔs na sɔntin we yu nɔ ebul fɔ kɔntrol, we yu imyun sistɛm de mek. Sɔntɛnde yu nɔ kin ivin no se yu gɛt LADA. Dis na bikɔs yu pankrias stil de mek inof insulin fɔ mek i nɔ mek yu gɛt di sik.

⚠️ Important: The medical articles and information on Nirogi Lanka are for general awareness only, and are by no means a substitute for professional medical advice, diagnosis, or treatment. For any medical problem you have, consult a qualified physician immediately.

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Wi go tɔk bɔt dis spɛshal kayn dayabitis we kin kam pan big pipul dɛn (Latent Autoimmune Diabetes in Adults - LADA)?

Wi go tɔk bɔt dis spɛshal kayn dayabitis we kin kam pan big pipul dɛn (Latent Autoimmune Diabetes in Adults - LADA)?

Tide wi go tɔk bɔt wan kayn dayabitis we difrɛn smɔl. Sɔntɛm yu nɔ bin dɔn yɛri bɔku tin bɔt am. Dɛn kɔl am LADA, we tinap fɔ Latent Autoimmune Diabetes in Adults. Dis kin tan lɛk se i kɔmplikt smɔl, bɔt lɛ wi brok am to simpul ɛksplen bɔt wetin de apin.

Wetin na LADA rili?

Fɔ tɔk am simpul wan, pan dis dayabitis we dɛn kɔl LADA, yu kin si di sayn dɛm fɔ di fɔs kayn dayabitis (`Type 1 diabetes`) ɛn bak di simptom dɛm fɔ di sɛkɔn kayn dayabitis (`Type 2 diabetes`). Na dat mek sɔm pipul dɛn kin kɔl am bak `Type 1.5 diabetes` . fכ bi prεsis, dכkta dεn biliv se dis kin kכz bak fכ difεkt na wi bכdi in כwn imyun sistεm (dεn kכl dis `autoimmune condition`). Bɔt dɛn kin no am mɔ we pɔsin big, dat na bitwin 30 ɛn 50. Wan ɔda spɛshal tin na dat, lɛk `Type 2 diabetes`, i kin go ɔp bak smɔl smɔl pan di simptom dɛm. Fɔ dis rizin, bɔku tɛm dɛn kin misdiagnose am as `Type 2 diabetes`.

Aw kɔmɔn tin fɔ LADA?

Pan ɔl we bɔku pipul dɛn nɔ yɛri bɔt LADA, fɔ tru, i nɔ kin bɔku so. Stɔdi dɔn sho se bitwin 4% ɛn 12% pan di pipul dɛm we dɛn bin dɔn no fɔs se dɛn gɛt `Type 2 diabetes` kin rili gɛt LADA. Jɔs tink bɔt, lɛk 530 milyɔn big pipul dɛn ɔlsay na di wɔl de sɔfa wit `Type 2 diabetes`. Dat min se bɔku bɔku pipul dɛn kin gɛt LADA! So, i rili impɔtant fɔ mek wi no bɔt dis.

Wetin na di sayn dɛm fɔ LADA?

Di sayn dɛm fɔ LADA kin rili fiba di wan dɛm wae kin gɛt ɔda kayn shuga. Si if dɛn tin ya tan lɛk se yu sabi:

  • Fɔ fil fɔ tɔsti ɔltɛm (polydipsia).
  • Di nid fɔ pis ɔltɛm.
  • We yu nɔ go ebul fɔ imajin fɔ lɔs yu wet .
  • Di vishɔn we nɔ klia .
  • Fɔ fil taya ɔltɛm (taya).
  • Skin we dray ɛn we de it .

If yu gɛt wan ɔ mɔ pan dɛn sayn ya, i go fayn fɔ mek yu go to dɔktɔ.

Wetin kin mek pɔsin gɛt LADA?

Risach pipul dɛm tink se LADA kin apin bikɔs di antibodi dɛm na wi bɔdi kin mistek atak ɛn pwɛl di sɛl dɛm we de mek insulin na wi pankrias. As yu no, insulin na ɔmon we de ɛp wi sɛl dɛn fɔ tek di shuga (glucose) we de na wi blɔd ɛn yuz am fɔ gɛt ɛnaji. So, we insulin nכ de prodyuz fayn, shuga (glucose) de bכku na di bכdi. Dis we ya, i tan lɛk Tayp 1 dayabitis. Pipul dɛn we gɛt Tayp 1 dayabitis fɔ tek insulin injɛkshɔn fɔ mek dɛn kɔntinyu fɔ liv. Bɔku tɛm, dɛn kin no se dɛn gɛt tayp 1 dayabitis we dɛn smɔl ɔ we dɛn yɔŋ.

Bɔt lɛk Tayp 2 dayabitis, dɛn kin no se LADA dɔn big, ɛn di sayn dɛn kin sho smɔl smɔl. Dis na bikɔs yu pankrias de ridyus di insulin we i de mek smɔl smɔl. So di sayn dɛm nɔ kin apin wantɛm wantɛm. Yu nɔ go nid fɔ gɛt insulin injɛkshɔn fɔ mɔnt ɔ ia . Dis na di rizin we mek sɔm dɔktɔ dɛn kin tink se dɛn gɛt Tayp 2 dayabitis fɔs, nɔto LADA.

Ɛni spɛshal trig de fɔ LADA?

No patikyula ‘trig’ nɔ de fɔ LADA. Dis na bikɔs na sɔntin we yu nɔ ebul fɔ kɔntrol, we yu imyun sistɛm de mek. Sɔntɛnde yu nɔ kin ivin no se yu gɛt LADA. Dis na bikɔs yu pankrias stil de mek inof insulin fɔ mek i nɔ mek yu gɛt di sik.

Wetin na di risk factor dɛm fɔ LADA?

LADA gɛt wan strɔng jenɛtik kɔmpɔnɛnt. Dis min se if yu mama ɛn papa ɔ grani ɛn granpa gɛt dis sik, yu go gɛt dis sik. Pruf de bak se di tin dɛn we de apin na di envayrɔmɛnt ɛn di tin dɛn we de mek pɔsin liv in layf, lɛk we pɔsin fat, kin mek i gɛt LADA. Bɔt, nɔto dɛn tin ya nɔmɔ kin mek pɔsin gɛt dis sik.

Posisibul kɔmplikɛshɔn dɛn we LADA kin gɛt

De big prɔblɛm kin kam wae yu nɔr trit dis sik fayn fayn wan. Bikɔs i tan lɛk Tayp 2 dayabitis, yu nɔ go bigin fɔ tek insulin tritmɛnt kwik kwik wan. Dis kin mek di risk fɔ gɛt wɛlbɔdi prɔblɛm lɛk di kidni we de pwɛl .

Dayabitis-rilayt ketoacidosis (DKA) na wan rili siriɔs, we kin mek pɔsin in layf de pan denja fɔ LADA. I kin apin we yu liva bigin fɔ brok di bɔdi fat fɔ gɛt ɛnaji we yu sɛl dɛn nɔ gɛt inof glukɔs (blɔd shuga) fɔ yuz fɔ ɛnaji. Dis na mɛdikal imejensi.

Aw dɛn kin no bɔt LADA?

LADA kin afɛkt big pipul dɛn we dɛn bin dɔn no fɔs se dɛn gɛt Tayp 2 dayabitis. Bɔt we yu nɔ ebul fɔ kɔntrol yu blɔd shuga lɛvɛl wit mɛrɛsin dɛn we yu de it (lɛk mɛtfɔmin) ɛn chenj yu layf (lɛk fɔ it tin dɛn we gɛt fayn fayn tin dɛn fɔ it), dɔktɔ dɛn kin sɔprayz se yu gɛt Tayp 1.5 dayabitis (we dɛn kin kɔl bak LADA).

Yu dɔktɔ kin no LADA wit wan blɔd tɛst we dɛn kɔl GAD Antibodies Test (`GAD Antibodies Test` - `glutamic acid decarboxylase antibodies`) . dis de chεk fכ di prεsεns fכ `autoantibodies` dεm we de atak glutamik asid dεkarbכksilayz (`GAD`). If yu gɛt dɛn ɔtoantibodi ya na yu blɔd, dat min se yu gɛt ɔtoimyun sik we de afɛkt yu pankrias. If yu na big pɔsin, i go mɔs bi se na LADA.

Di dɔktɔ kin ɔda fɔ mek dɛn du C-peptide tɛst.Dɛn kin tɛl yu bak fɔ du tɛst. dis de mכsu aw bכku C-pεptida de insay yu bכdi. C-peptide kin tɛl yu dɔktɔ aw bɔku insulin yu pankrias de mek.

Wetin na di tritmɛnt dɛm fɔ LADA?

Fɔ trit LADA na tin we kɔmplikt smɔl. We yu bigin, yu kin tek di mɛrɛsin dɛn we yu kin tek fɔ gɛt dayabitis. Dɛn kin ɛp fɔ kɔntrol yu blɔd shuga fɔ sɔm tɛm. Bɔt as di sik de wɔs, di we aw yu bɔdi de tek dɛn mɛrɛsin dɛn de go dɔŋ. Dɔn yu fɔ chenj to insulin injɛkshɔn fɔ kɔntrol yu blɔd shuga. Bikɔs yu pankrias kin lɔs in pawa fɔ mek insulin fɔ insɛf kpatakpata. Dis kin apin fɔ sɔm mɔnt ɔ sɔm ia.

No tritmɛnt nɔ de we dɛn dɔn gri pan fɔ LADA. Sɔm spɛshal pipul dɛn biliv se i bɛtɛ fɔ bigin tritmɛnt wit insulin wantɛm wantɛm. Ɔda pipul dɛn kin wet fɔ sɔm tɛm bifo dɛn bigin fɔ tek insulin injɛkshɔn. Yu dɔktɔ go disayd us tritmɛnt go fayn fɔ yu.

Bikɔs di doz ɛn di mɔnt fɔ insulin we yu nid kin chenj, yu nid fɔ kɔntinyu fɔ wach di shuga we yu gɛt na yu blɔd.

Wetin pɔsin we gɛt LADA kin ɛkspɛkt?

No mɛrɛsin nɔ de fɔ Tayp 1.5 dayabitis. Bɔt yu dɔktɔ kin ɛp yu fɔ fɛn tritmɛnt fɔ ɛp yu fɔ kɔntrol am. We yu bigin fɔ tek insulin, yu go ebul fɔ kɔntrol di shuga we de na yu blɔd bɛtɛ bɛtɛ wan. If yu de manej yu fayn fayn wan, yu go ebul fɔ liv nɔmal layf.

Wetin na di layf we pɔsin we gɛt LADA de liv?

Yu layf dipen pan bɔku tin dɛn. If yu gɛt Tayp 1.5 dayabitis, dat nɔ kin mek yu layf shɔt. Bɔt if yu gɛt LADA ɛn yu nɔ ebul fɔ kɔntrol yu glukɔs lɛvɛl, yu de pan risk fɔ gɛt wɛlbɔdi prɔblɛm dɛn we kin mek yu layf shɔt. Di men rizin fɔ dis na bikɔs if pɔsin gɛt bɔku shuga na in blɔd fɔ lɔng tɛm, i kin mek i gɛt prɔblɛm dɛn lɛk at sik . Dɛn tin ya kin afɛkt yu layf. Na dat mek i rili impɔtant fɔ kɔntrol di blɔd shuga.

Yu tink se dɛn kin mek dɛn nɔ gɛt LADA?

No we nɔ de fɔ mek yu nɔ gɛt LADA dayabitis. Bikɔs yu nɔr kin ebul fɔ kɔntrol ɔr prɛvɛnt di ɔtoimyun kɔndishɔn wae de kɔz am , fɔ no di sik kwik kwik wan ɛn trit am na di bɛst we fɔ mek yu nɔr gɛt prɔblɛm wit dis sik.

Aw a go tek kia ɔf misɛf if a gɛt LADA?

Di bɛst we fɔ kia fɔ yusɛf na fɔ kɔntrol di shuga we yu gɛt na yu blɔd. Bɔku tɛm, dɛn nɔ kin no di rɔŋ we fɔ gɛt LADA dayabitis. I kin mek yu at pwɛl we yu tink se yu de du ɔltin fayn, bɔt i stil at fɔ kɔntrol di shuga we yu gɛt na yu blɔd. If dɛn no se yu gɛt LADA, fala di tritmɛnt plan we yu dɔktɔ gi yu ɛksaktɔli.. Mek shɔ se yu kip ɔl di apɔntinmɛnt dɛn we yu dɔn mek fɔ fala yu. Na dɛn apɔntinmɛnt ya, yu dɔktɔ kin chɛk yu pankrias ɛn ɔl yu wɛlbɔdi. Dɛn kin ajɔst bak di insulin we yu nid.

Ustɛm yu fɔ go to dɔktɔ?

If yu gɛt sɔm sayn dɛn fɔ gɛt dayabitis, lɛk fɔ tɔsti pasmak ɛn fɔ pis ɔltɛm, go to dɔktɔ wantɛm wantɛm. Nɔ fɔgɛt fɔ sheb yu kɔmplit wɛl bɔdi histri wit yu dɔktɔ.

Na mɔtalman, if dɛn dɔn no se yu gɛt `Type 2 diabetes` ɛn yu simptom nɔ de bɛtɛ afta yu dɔn trit yu, aks yu dɔktɔ fɔ tɛst yu fɔ `Type 1.5 diabetes` (LADA).

Tayp 1.5 dayabitis, ɔ LADA, na wan sik we bɔku tɛm dɛn nɔ kin no di rayt we. I gɛt sayn dɛm fɔ ɔl tu di Tayp 1 ɛn Tayp 2 dayabitis. Bɔt bɔku tɛm, di sayn dɛm kin sho se yu gɛt Tayp 2. So i kin mek yu at pwɛl ɛn kɔnfyus fɔ no se yu gɛt dis kayn dayabitis. Yu kin bigin fɔ tek mɛrɛsin dɛn we yu kin it, bɔt yu blɔd shuga stil bɔku. Ɔ yu tritmɛnt kin wok fɔ sɔm tɛm, bɔt afta dat i kin stɔp fɔ wok. Yu dɔktɔ dɛn go ɛp yu fɔ fɛn di bɛst we fɔ kɔntrol di shuga we yu gɛt na yu blɔd ɛn fɔ mek yu gɛt wɛlbɔdi. Nɔr frayd fɔ aks kwɛstyɔn bɔt yu diagnosis ɛn tritmɛnt plan.

Wetin na di impɔtant tin dɛn we wi go kɛr go na os frɔm dis atikul?

LADA na spɛshal kayn dayabitis we kin apin to big pipul ɛn i gɛt sayn dɛm fɔ ɔl tu di `Type 1` ɛn `Type 2` dayabitis. Sɔntɛnde dɛn kin misdiagnose am as `Type 2`.

  • Di sayn dɛm na fɔ tɔsti pasmak, fɔ pis ɔltɛm, fɔ lɛf fɔ gɛt bɔku bɔku bɔdi, ɛn fɔ taya.
  • dis kin kכz fכ wan כtoimyun kכndishכn, εn i kin gεt jεnεtik kכmכpכnt bak.
  • If dɛn nɔ no di sik kɔrɛkt wan ɛn trit am kwik, prɔblɛm dɛn lɛk di kidni we dɔn pwɛl ɛn DKA kin apin.
  • Dis na difinitiv wan we dɛn kin no bay di GAD Antibodies Test.
  • Pan ɔl we dɛn kin gi mɛrɛsin fɔs as tritmɛnt, as tɛm de go, dɛn kin nid fɔ gi insulin injɛkshɔn.
  • If dɛn dɔn no se yu gɛt Tayp 2 dayabitis ɛn yu tritmɛnt nɔ de kɔntrol am, tɔk to yu dɔktɔ bɔt LADA. Nɔ ɛva stɔp ɔ chenj yu tritmɛnt fɔ yusɛf. Ɔltɛm fala wetin yu dɔktɔ tɛl yu fɔ du.

` LADA, dayabitis, dayabitis we kin bigin fɔ big pipul, tayp 1.5 dayabitis, ɔtoimyun dayabitis, insulin, blɔd shuga

Frequently Asked Questions (FAQ)

Ɛni spɛshal trig de fɔ LADA?

No patikyula ‘trig’ nɔ de fɔ LADA. Dis na bikɔs na sɔntin we yu nɔ ebul fɔ kɔntrol, we yu imyun sistɛm de mek. Sɔntɛnde yu nɔ kin ivin no se yu gɛt LADA. Dis na bikɔs yu pankrias stil de mek inof insulin fɔ mek i nɔ mek yu gɛt di sik.

⚠️ Important: The medical articles and information on Nirogi Lanka are for general awareness only, and are by no means a substitute for professional medical advice, diagnosis, or treatment. For any medical problem you have, consult a qualified physician immediately.

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