Lɛ wi lan mɔ bɔt di lipoproteins, we dɛn mek wit fat ɛn protin.

Yu dɔn ɛva wɔnda aw di fat we de na wi bɔdi (we dɛn kin kɔl bak fat we wi de tɔk) kin kɔmɔt na wan ples to ɔda ples? Lipoprotein na spεshal patikyula dεm we de εp wit dis. Fɔ tɔk am simpul wan, dɛn tan lɛk smɔl smɔl motoka dɛn we de kɛr tin dɛn lɛk fat ɛn kɔlɔstrel na wi bɔdi go usay dɛn nid fɔ de tru di blɔd. Dɛn kin travul tru wi blɔd vesel dɛn. Sɔm lipoprotein dɛn kin rili fayn fɔ wi at, bɔt sɔm kin mek wi gɛt smɔl prɔblɛm. So, lɛ wi tɔk bɔt dɛn lipoprotein ya insay wan simpul ɛn stret we, jɔs lɛk ɔl ɔda tin.

Wetin rili na kɔlɔstrel ɛn triglisɛrɛyd?

Bifo wi tɔk bɔt lipoprotein, lɛ wi ɔndastand smɔl bɔt dɛn tu wɔd ya: kɔlɔstrel ɛn triglisɛrɛyd. Dɛn tu na waxy fat we de insay wi blɔd. Bɔt di we aw wi bɔdi de mek ɛn yuz dɛn difrɛn smɔl.

• Kɔlɔstrel: Bɔku tɛm, dɛn kin mek kɔlɔstrel na wi liva. Wi kin gɛt kɔlɔstrel bak frɔm sɔm pan di it dɛn we wi kin it, mɔ di animal fat, mit, fish, ɛn tin dɛn we dɛn mek wit milk. Dis kɔlɔstrel nɔ jɔs bad fɔ wi bɔdi. Dis kɔlɔstrel de ɛp fɔ mek vaytamɛn D, ɛn impɔtant ɔmon dɛn lɛk ɛstrojen, tɛstostɛron, ɛn kɔtisol . Nɔto dat nɔmɔ, i impɔtant bak fɔ mek sɛl dɛn na wi nervɔs sistɛm.

• Triglisεrayd: Na wi liva εn in intestin dεm we de na wi dijestiv sistεm de mek dεn tin ya. We wi it bɔku it dɛn we gɛt fat ɛn we gɛt bɔku kalori, wi bɔdi kin mek mɔ triglisɛrɛyd. di ekstra triglisεrayd dεm we wi de mek de stכr insay wi fεt sεl dεm. Leta, we di bɔdi nid ɛnaji, dɛn triglisɛrɛyd ya we dɛn dɔn kip kin kɔmɔt na di blɔd.

Naw yu go dɔn ɔndastand wetin na kɔlɔstrel ɛn triglisɛrɛyd ɛn aw dɛn impɔtant to wi bɔdi. Bɔt, di prɔblɛm na we dɛn kin ay pasmak.

Us kayn lipoprotein dɛn de?

Fayv men kayn lipoprotein dεm de na wi bכdi. Lɛ wi tek wan luk pan wetin dɛn bi.

• High-density lipoprotein (HDL): Dis na wetin wi kin kɔl "gud kɔlɔstrel." HDL de ɛp fɔ kɛr di kɔlɔstrel we pasmak frɔm di bɔdi go bak na di liva, usay i de kɔmɔt. Di ay levul fɔ HDL de ridyus wi risk fɔ gɛt di sik we de mek wi gɛt di sik we de na di at ɛn di blɔd. I tan lɛk padi to wi.

• lכw dεnsiti lipoprotein (LDL): dis na wetin wi kכl "bad kכlestכl".Dɛn kɔl am. We di LDL lɛvɛl go ɔp, i kin mek pɔsin gɛt sik dɛn lɛk korona at sik, at atak, ɛn strok. LDL kin kɛr kɔlɔstrel ɛn put am na wi blɔd vesel dɛn. We dɛn put am dis kayn we, i kin tan lɛk dɔti na paip, we kin mek di ples we de insay di blɔd vesel dɛn nɔ bɔku ɛn i kin mek di blɔd nɔ flɔ. Wi kin kɔl dis kɔndishɔn atrɔsklɛrosis .

• di lipoprotein dεm we rili lכw dεnsiti (VLDL): Dis na כda kayn "bad kכlestכl." VLDL de mεntal kכri triglisεrayd to wi tisu dεm. i de kכntribyut bak fכ transpכt kכlestכl to sכm mak.

• intamεdiεt-dεnsiti lipoprotein dεm (IDL): dεn kin fכm afta we VLDL dכn pul sכm pan in fεt asid dεm. Dɔn di liva kin pul dɛn IDL ya ɔ dɛn kin chenj dɛn to LDL.

• Chylomicrons: Dis na di big big lipoprotein patikyula dεm. Dɛn kin transpɔt triglisɛrɛyd dɛn bak.

Yu si, difrɛn kayn lipoprotein dɛn de, ɛn dɛn kin du difrɛn tin dɛn.

Wetin na lipoprotein (a) ɔ LP(a)?

Dis wan spɛshal smɔl. lipoprotein (a) כ LP(a) na di sem kayn LDL bak, di "bad kכlestכl" we wi bin tכk bכt. Bɔt dis wan spɛshal. כda protin we dεn k כl apolipoprotein (a) כ apo(a) de rap rawnd dis LDL patikyula. dis apo(a) de mek di LDL patikyula "stika". fכ dat, dis LP(a) kin izi fכ deposit insay di bכdi vεsul dεm, we de inkrεs di risk fכ kכdivaskyul prכblεm. fכ gεt hכy lεvεl dεm fכ LP(a) na big risk fכ divεlכp atεrosklεrosis.

pan ɔl we wi it ɛn layf we wi de liv kin afɛkt ɔda HDL ɛn LDL lɛvɛl dɛn, wi LP(a) lɛvɛl dɛn kin bi mɔ bay wi jin dɛn. sכm pipul dεn gεt hכy LP(a) lεvεl bikoz fכ di jεnεtik mכtεshכn. Dɛn kayn pipul ya kin gɛt ay risk fɔ gɛt at sik we dɛn yɔŋ.

Wetin na Lipid Panel blɔd tɛst?

Yu dɔktɔ kin dɔn aks yu fɔ gɛt "lipid profayl." Na dat dɛn kɔl dis lipid panɛl blɔd tɛst. I de mɛzhɔ di ɔl kɔlɔstrel we de na yu blɔd. I de mɛzhɔ bak di lɛvɛl dɛn we yu gɛt HDL, LDL, ɛn triglisɛrɛyd, ɛn yu ɔl di kɔlɔstrel.

Dis na wan rili simpul blɔd tɛst. I nɔ kin tek fayv minit. Yu kin mek dɛn du am na yu dɔktɔ in ɔfis, na lab, ɔ na ɔspitul. Fɔ mek yu gɛt di bɛst rizɔlt, yu fɔ fast fɔ 8 to 12 awa bifo yu du dis blɔd tɛst.Dat min se yu nɔ go it ɔ drink ɛnitin. Dɛn kin aks yu bak fɔ stɔp fɔ tek ɛni mɛrɛsin we yu de tek fɔ sɔm tɛm. So, yu fɔ fala di dɔktɔ in instrɔkshɔn dɛn di rayt we.

Aw ɛn ustɛm dɛn kin tɛst di lipoprotein (a) [LP(a)]?

Bɔku tɛm, nɔto ɔlman kin gɛt dɛn LP(a) lɛvɛl we dɛn kin chɛk. Na if yu dɔktɔ spɛshal wan aks fɔ am, dɛn go chɛk di LP(a) lɛvɛl wit di lipid panɛl.

Bɔrku tɛm na fɔ pipul dɛm wae gɛt sɔm tin dɛm wae kin mek pɔrsin gɛt at sik nɔmɔ dɛn kin advays dis tɛst. Dɛn tin ya na:

• If yu dɔn gɛt at prɔblɛm bifo.
• If ɛnibɔdi na yu famili, mɔ di fɔs digri fambul dɛm (mama ɛn papa, brɔda ɛn sista dɛm), dɔn gɛt at sik we i yɔŋ.
• If yu LDL lɛvɛl nɔ bɔku.
• If yu gɛt tin dɛn we kin mek yu gɛt famili haypa kɔlɔstrelemia , we na wan sik we de mek yu gɛt bɔku LDL.

Wetin na di impɔtant tin fɔ dis lipid blɔd tɛst?

Bɔku tɛm, if yu gɛt bɔku kɔlɔstrel, dat nɔ kin mek yu gɛt ɛni sayn, so fɔ du dis blɔd tɛst na di bɛst we fɔ no di prɔblɛm dɛn we gɛt fɔ du wit kɔlɔstrel we kin afɛkt yu at ɛn yu blɔd vesel dɛn.

Dɔktɔ dɛn kin yuz dis lipid blɔd tɛst fɔ tin dɛn lɛk:

• Skrin: Dɛn kin du dis as pat pan yu mɛdikal chɛk-ap ɛvri ia. Dis kin ɛp yu fɔ no if yu gɛt mɔ prɔblɛm wit yu at ɛn blɔd. Wae yu no se yu de pan de risk pasmak, yu kin tek step fɔ mek yu nɔr gɛt siriɔs sik lɛk at atak ɛn strok.
• Diagnosis: Dis kɔlɔstrel blɔd tɛst de ɛp bak fɔ no di sik dɛn lɛk atherosclerosis .
• Monitoring: Dɛn tɛst rizɔlt ya kin ɛp yu fɔ si if yu kɔlɔstrel lɛvɛl de wɔs, nid tritmɛnt, ɔ yu de ansa fayn to tritmɛnt.

Aw ɔltɛm dɛn fɔ du dɛn lipoprotein tɛst ya?

i nכmal fכ di lipoprotein εn tכtal kכlestכl lεvεl fכ inkrεs wit di ej. As yu de gɛt mɔ prɔblɛm, yu dɔktɔ kin tɛl yu fɔ du tɛst ɔltɛm.

Dɛn kin rɛkɔmɛnd fɔ du lipid blɔd tɛst: dɛn tin ya:

• Fɔ man dɛn we dɔn pas 35 ia ɛn uman dɛn we dɔn pas 45 ia.
• Atɛrosklɛrosis kin kam pan man dɛn we ol bitwin 20 ɛn 35 ia ɛn uman dɛn we ol bitwin 20 ɛn 45 ia.
• Fɔ pikin dɛn, i bɛtɛ fɔ tek dis tɛst wan tɛm bitwin 9 ɛn 11 ia, ɛn afta dat bak bitwin 17 ɛn 21 ia.

Wetin na di hεlty lεvεl dεm fכ di lipoprotein dεm?

di lab dεm de mכsu kכlestכl εn lipoprotein dεm insay miligram pan wan dεsilita (mg/dL). Di we aw ɛni lab de yuz kin difrɛn. Dis min se di rizɔlt frɔm di sem blɔd sɛmpul kin difrɛn smɔl frɔm wan lab to ɔda lab. Ɛnitɛm we i pɔsibul, yu dɔktɔ go want fɔ yuz di sem lab fɔ wach yu lɛvɛl.

di hεlth lεvεl dεm fכ HDL, LDL, triglisεrayd, εn tכtal kכlestכl de difrεn bay di ej εn jεnda. Tɔk to yu dɔktɔ bɔt wetin na wɛl bɔdi lɛvɛl fɔ yu. In jɛnɛral, if yu nɔr gɛt ɔda risk factors, dɛn kin tek dɛn valyu ya as wɛl bɔdi:

• Tכtal kכlestכl: 100-199 mg/dL fכ dεn wan dεm we pas 21 ia; 75-169 mg/dL fɔ di wan dɛn we ol 20 ia ɛn smɔl pas dat.
• HDL (gud kɔlɔstrel): 45 mg/dL ɔ pas dat. (55 mg/dL ɔ pas dat bɛtɛ fɔ uman, 45 mg/dL ɔ pas dat fɔ man dɛn)
• LDL (bad kɔlɔstrel): i nɔ pas 100 mg/dL.
• Triglisɛrɛyd: I nɔ rich 150 mg/dL.

wan lipid panεl nכ kin mכsu di VLDL lεvεl dεm dairekt wan, εn nכ spεsifi k tεst de fכ am. Bifo dat, di lab dεm de εstimat yu VLDL lεvεl bay we dεn de sheb yu triglisεrayd dεm bay fayv. wan hεlty VLDL lεvεl fכ less dan 30 mg/dL. Bɔt dis we fɔ du tin nɔ kin kɔrɛkt ɔltɛm if yu triglisɛrɛyd dɛn rili bɔku.

Wetin na di nכmal valyu fכ lipoprotein (a) [LP(a)]?

LP(a) lεvεl dεm dεn ripot in εither mg/dL כ nanomoles pan wan lita (nmol/L). di valyu we pas 50 mg/dL כ 75 nmol/L dεn kin tek am se i hכy, εn i de mek di risk fכ kכndyushכn lεk atεrosklεrosis, at atak, εn strכk .

Wetin na di tin dɛn we kin mek pɔsin gɛt bad kɔlɔstrel (LDL)?

Uman dɛn we dɔn pas 40 ia we dɔn pas 40 ia kin gɛt di ay risk fɔ gɛt ay LDL lɛvɛl. Bɔt ivin smɔl pikin dɛn kin gɛt prɔblɛm wit di kɔlɔstrel. As yu de ol, yu mɛtabolism de slo, ɛn yu liva nɔ de ebul fɔ pul bad kɔlɔstrel na yu bɔdi.

Di kɔlɔ ɛn trayb bak kin ɛp. Di pipul dɛm we kɔmɔt Eshia na Amɛrika kin gɛt bɔku bɔku LDL lɛvɛl, ɛn di wayt pipul dɛm kin gɛt bɔku bɔku kɔlɔstrel lɛvɛl.

Ɔda tin dɛn we kin mek di LDL bɔku na:

• Wan it we gɛt bɔku kalori, sɛtyuret fat, ɛn kɔlɔstrel. (Wi fɔ tink smɔl bak bɔt di it dɛn we gɛt fat ɛn we dɛn dɔn frɛsh we wi de it.)
• Kɔndishɔn lɛk dayabitis ɛn kidni sik.
• Wan famili histri we gɛt ay kɔlɔstrel.
• Fɔ drink pasmak ɛn fɔ smok pasmak.
• Mɛrɛsin lɛk kɔtikosterɔyd ɛn sɔm mɛrɛsin fɔ ay blɔd prɛshɔn ɛn HIV/AIDS.
• Ovaweit (Fɔ fat pasmak).

Aw fɔ trit di ay LDL lɛvɛl?

Pipul dɛn we gɛt ay LDL lɛvɛl, we kin mek dɛn gɛt ay risk fɔ gɛt at sik ɛn strok, kin nid mɛrɛsin dɛn we de mek dɛn nɔ gɛt bɔku kɔlɔstrel, lɛk statin, ezetimibe, ɛn PCSK9 inhibitors . Yu kin ɛp bak fɔ mek dɛn kayn tin ya bɛtɛ bay we yu chenj di we aw yu de it ɛn aw yu de liv yu layf.

Wetin yu kin du fɔ mek di lipoprotein lɛvɛl bɛtɛ?

If yu mek sɔm chenj dɛn na yu it ɛn layf, dat kin ɛp fɔ mek yu nɔ gɛt bɔku bɔku kɔlɔstrel (LDL) ɛn mek yu gɛt bɔku kɔlɔstrel (HDL). Dɛn step ya kin ɛp:

• Bi fyzikal aktiv fɔ at le 150 minit insay di wik (dat na lɛk 30 minit insay di de). Ivin fɔ waka fɔ shɔt tɛm ɔ fɔ ɛksesaiz kin fayn.
• It tin dɛn we go ɛp yu at. Dat min se yu fɔ it tin dɛn we nɔ gɛt bɔku fat we nɔ fayn fɔ yu, we gɛt simpul shuga (kabɔhaydrɛt), ɛn we gɛt bɔku fayv.
• Kɔntrol ay blɔd prɛshɔn ɛn dayabitis.
• Slip fayn fayn wan.
• Mek yu gɛt wɛlbɔdi wet. If yu ova wet, if yu lɔs 5% to 10% pan yu bɔdi wet kin mek yu kɔlɔstrel lɛvɛl bɛtɛ.
• Fɛn wɛlbɔdi we fɔ kɔntrol strɛs.
• Ridyus di we aw yu de drink rɔm.
• Aks fɔ ɛp fɔ lɛf fɔ smok (nikotin de mek di LDL lɛvɛl go ɔp ɛn i de mek di HDL lɛvɛl go dɔŋ).

Mɛsej we dɛn kin kɛr go na os:

Okay, so wi don tok plenti boht lipoproteins tide, no so? Fɔ tɔk am simpul wan, na di men tin dɛn we wi fɔ mɛmba:

• Lipoprotein dεm lεk motoka dεm we de transpכt fεt εn kכlestכl na wi bכdi.
• HDL na "gud kכlestכl," we de εp fכ pul di kכlestכl we pasmak na di bכdi.
• LDL na "bad kɔlɔstrel." If i bɔku, i kin gɛda na di blɔd vesel dɛn ɛn mek pɔsin gɛt at sik.
• LP(a) na ɔda kayn "stik" bad kɔlɔstrel, we de mek bak di risk fɔ gɛt at sik.
• Tin dɛm lɛk aw yu de liv yu layf, aw yu de it, ɛn ɛksesaiz kin gɛt big impak pan yu lipoprotein lɛvɛl.
• Sɔntɛnde dɛn kin nid mɛrɛsin (lɛk ``Statins'') fɔ mek di LDL ɛn LP(a) lɛvɛl go dɔŋ ɛn fɔ mek di HDL lɛvɛl go ɔp.
• Di tin we impɔtant pas ɔl na fɔ du blɔd tɛst ɔltɛm fɔ no bɔt yu lɛvɛl ɛn fala yu dɔktɔ in instrɔkshɔn.

A op se dis infɔmeshɔn go ɛp yu fɔ mek yu at gɛt wɛlbɔdi!

` lipoprotein, kכlestכl, triglisεrayd, HDL, LDL, VLDL, at sik, lipid panεl, atεrosklεrosis, LP(a), kכlestכl