Yu gɛt tayp 1 dayabitis bak? (Type 1 Diabetes) Lɛ wi tɔk bɔt ɔl dis

We yu tink bɔt dayabitis, yu go mɔs tink bɔt di kayn 2 dayabitis we kin apin mɔ we yu de ol. Bɔt yu no se ɔda kayn dayabitis de we kin gɛt ɛni ej, mɔ pan pikin dɛn ɛn yɔŋ pipul dɛn? Na dat wi kin kɔl Tayp 1 Dayabitis. I difrɛn smɔl, bɔt if yu manej am fayn, i nɔ go stɔp yu fɔ liv wɛl bɔdi layf. Lɛ wi tɔk bɔt am simpul wan we yu go ɔndastand.

Wetin na Tayp 1 Dayabitis?

Fɔ tɔk am simpul wan, tayp 1 dayabitis na ɔtoimyun kɔndishɔn we wi bɔdi in yon imyun sistɛm de atak ɛn pwɛl di sɛl dɛn we de mek insulin (beta sɛl dɛn) na wi yon pankrias.

Tink bɔt dis, insulin tan lɛk ki we de ɛp di shuga ɔ glukɔs we de na wi blɔd fɔ go insay wi sɛl dɛn fɔ mek ɛnaji. Naw, we dɛn sɛl ya we de mek insulin nɔ de igen, di bɔdi nɔ de mek di insulin we i nid. Dɔn di shuga we de na di blɔd nɔ kin ebul fɔ go insay di sɛl dɛn ɛn i kin gɛda na di blɔd. Na dat kin apin pan dis sik.

Wetin na di difrɛns bitwin Tayp 1 ɛn Tayp 2?

Dis na ples we bɔku pipul dɛn kin stɔp.

• Insay Tayp 1: Di bɔdi kin stɔp fɔ mek insulin kpatakpata , so i nid fɔ tek insulin frɔm ɔdasay fɔ mek i kɔntinyu fɔ liv.
• Insay Tayp 2: Ivin if di bɔdi de mek insulin, di amount nɔ go du, ɔ di bɔdi in sɛl dɛn nɔ de ansa fayn to da insulin de (insulin resistance).

Wetin na di sayn dɛm we de sho se yu gɛt dis sik? Aw yu go no am kwik kwik wan?

Bɔrku tɛm, de sayn dɛm fɔ dis sik kin apia kwik kwik wan , sɔmtɛm insay sɔm wiks. E fayn fɔ no bɔt dɛn sik ya.

Tayp 1 dayabitis we kin bigin let (LADA) .

Sɔmtɛm dɛn kin no dis sik we dɛn big (bitwin di ej fɔ 30-50 ia). Dɛn kɔl dis ‘Latent Autoimmune Diabetes in Adults’ (LADA). Bikɔs di sayn dɛm kin apia sloslo, sɔmtɛm dɛn kin mistek kɔl am tayp 2 dayabitis.

Aw dɛn kin mɛn yɔŋ pikin dɛn?

Apat frɔm di sayn dɛm we dɛn kin si pan big pipul dɛm, yɔŋ pikin ɛn bebi kin sho difrɛn sayn dɛm we dɛn gɛt dis sik. I impɔtant fɔ mek mama ɛn papa dɛn no bɔt dis.

• Fɔ chenj di pikin in dayapɛt bɔku tɛm pas aw i kin chenj.
• Fɔ gɛt daypa rash we nɔ de wɛl pan ɔl we dɛn dɔn trit yu.
• Wan pikin we dɛn tren fɔ go na tɔylɛt kin bigin fɔ wet di bed.
• Di rεt we yu de blo de go ɔp.
• Bɛlɛ de at ɛn vɔmit.
• Chenj na di pikin in bihayvya (bɔku tɛm i kin vɛks, i kin vɛks).
• Wan swit, frut smel frɔm di briz (dis na denja simptom).

Wetin na di rizin dɛn we mek dɛn de du dis? Yu tink se na frɔm dɛn gret gret granpa dɛn?

Wi nɔ stil no di rayt rizin we mek wi bɔdi in imyun sistɛm de atak in yon sɛl dɛn, bɔt dɛn biliv se na bikɔs ɔf di jɛnɛtik prɛdispɔzishɔn ɛn di tin dɛn we de arawnd wi .

Fɔ tɔk am simpul wan, if pɔsin na yu famili (mama, papa, brɔda ɛn sista) gɛt tayp 1 dayabitis, yu de pan smɔl risk fɔ gɛt am, bɔt dat nɔ min se yu go mɔs gɛt am.

Imajin, ivin if yu gɛt jin dɛm we de mek yu gɛt di sik, i kin tek wan tin we de na di envayrɔmɛnt, lɛk vayral infɛkshɔn, fɔ mek dɛn jin dɛm "aktiv".

Aw dɛn kin no di sik? Us tɛst dɛn dɛn kin du?

If yu dɔktɔ tink se yu gɛt tayp 1 dayabitis, di fɔs tin we dɛn go du na fɔ tɛst yu blɔd shuga.

Dɛn tɛst ya kin no if yu gɛt dayabitis. Bɔt fɔ no fɔ tru if na tayp 1 ɔ tayp 2, yu dɔktɔ go du sɔm ɔda tɛst dɛn.

• Autoantibodi test: Dis na di test we impɔtant pas ɔl. Insay tayp 1 dayabitis, spɛshal prɔtin (ɔtoantibodi) dɛn de na di blɔd we de atak di sɛl dɛn we de mek insulin. Dis tɛst de luk fɔ dɛn tin ya.
• Keton test: We insulin dɔn lɔs, di bɔdi kin bigin fɔ bɔn fat fɔ gɛt ɛnaji. di asid dεm we de kכmכt na di kεtכn dεm dεn kכl dεm kεtכn. Dɛn kin no dɛn tin ya na di urine.

Tritmɛnt ɛn aw fɔ mɛn pipul dɛn layf

Yu layf nɔ dɔn jɔs bikɔs dɛn dɔn no se yu gɛt tayp 1 dayabitis. Yu kin liv lɔng, wɛlbɔdi, ɛn gladi at. Di men tin na fɔ kɔntrol di shuga we yu gɛt na yu blɔd.

Na sɔntin we pɔsin fɔ yuz insulin.

Insay tayp 1 dayabitis, yu bɔdi nɔ de mek insulin, so yu fɔ tek insulin frɔm ɔdasay ɛvride . Yu nɔ kin tek insulin insay pil fɔm, bikɔs i kin brok dɔŋ na yu bɛlɛ. So yu fɔ tek am insay injɛkshɔn ɔ wit insulin pɔmp.

Bɔku kayn insulin de. Dɛn kin difrɛn pan di tɛm we i kin tek fɔ wok ɛn aw lɔng i kin te. Yu dɔktɔ go no di kayn insulin we fayn fɔ yu ɛn di doz fɔ yu bay bɔku tin dɛn lɛk yu ej, aw yu de liv yu layf, ɛn aw yu de it.

Fɔ chɛk di blɔd shuga ɔltɛm

Di tin we impɔtant pas ɔl na fɔ kip yu blɔd shuga lɛvɛl insay wan target rɛnj ɔl di de. Tu we dɛn de fɔ du dis:

1. Glukomita: Di tradishɔnal we fɔ tɛst na fɔ tek smɔl drop blɔd frɔm di tip na di finga.

2. Continuous Glucose Monitor (CGM): Na smɔl sɛns we dɛn put ɔnda di skin kin chɛk yu blɔd shuga lɛvɛl ɛvri sɔm minit ɛn sɛn di data to yu fon ɔ ɔda divays. I de wɔn yu if yu blɔd shuga lɛvɛl ay ɔ smɔl.

Fɔ it ɛn di we aw pɔsin de liv in layf

If yu gɛt tayp 1 dayabitis, dat nɔ min se yu nɔ go ebul fɔ it. Bɔt yu nid fɔ tek tɛm wit di bɔku it dɛn we yu de it, mɔ di kabɔhaydrɛt (stach). Yu nid fɔ ajɔst yu insulin doz fɔ mek i mach di it we yu de it.

• It fayn it . Put mɔ vɛjitebul, frut, ɛn ɔl di tin dɛn we dɛn kin it.
• Limit drink dɛn we gɛt shuga, swit it dɛn, ɛn rifin kabɔhaydrɛt (bred, pasta).
• Ɛksesaiz ɔltɛm. We yu de du ɛksɛsayz, dat kin mek di bɔdi ebul fɔ gɛt insulin mɔ ɛn mɔ.
• Manej strɛs ɛn slip fayn . Dɛn tin ya kin afɛkt di blɔd shuga lɛvɛl bak.

Wetin na de kɔmplikeshɔn wae kin kam bikɔs ɔf dayabitis?

If dɛn nɔ kɔntrol di blɔd shuga fayn fayn wan fɔ lɔng tɛm, difrɛn pat dɛn na di bɔdi kin pwɛl. Bɔt nɔ wɔri. If yu kɔntinyu fɔ kɔntrol gud gud wan, yu go ebul fɔ avɔyd bɔku pan dɛn prɔblɛm dɛn ya.

Di kɔmplikeshɔn we impɔtant pas ɔl ɛn we kin apin kwik kwik wan na dayabitik ketoacidosis (DKA) . dis kin apin we di bכdi nכ gεt insulin, we kin mek di kεtכn εn shuga bכku na di bכdi, we kin mek di bכdi bi asid. Dis na imejensi we kin mek pɔsin in layf de pan denja.

Di prɔblɛm dɛn we kin apin fɔ lɔng tɛm kin bi at sik, kidni sik (nephropathy), yay we kin pwɛl (retinopathy), ɛn di nerve we kin pwɛl. Di bɛst we fɔ mek dɛn nɔ gɛt dɛn tin ya na fɔ fala wetin yu dɔktɔ tɛl yu ɛn kɔntrol di shuga, blɔd prɛshɔn, ɛn kɔlɔstrel we yu gɛt.