Yu gɛt wik bon dɛn bak? Dis kin bi sayn fɔ Mɛtabolik Bɔn Disease!

Sɔntɛnde yu kin fil lɛk se yu bon dɛn wik smɔl? Sɔntɛm ivin we pɔsin fɔdɔm smɔl, i kin brok in an ɔ in leg. Ɔ yu kin fil lɛk se yu ayt de go dɔŋ as yu de ol, ɛn yu bɔdi de bɛn smɔl smɔl? Wan pɔsibul rizin bihayn dɛn tin ya na wan kɔndishɔn we dɛn kɔl mɛtabolik bon sik, we wi go tɔk bɔt tide. Lɛ wi tek wan luk pan dis insay wan simpul we.

Wetin na dis Mɛtabolik Bɔn Sik?

Fɔ tɔk am simpul wan, mɛtabolik bon sik na grup fɔ sik dɛn we de afɛkt di trɛnk we yu bon dɛn gɛt. I kin apin we di balans we di minral dεm na wi bכdi de bכn, i kin bi we tin dεm lεk kalsiכm, fכsfכr, כ vaytam in D. Dis minral imbalans de ambɔg di bon mas, gro, εn bon tכnכv (di prכsεs we nyu bon tisu de riples wit ol bon tisu).

Tink bɔt am, wi bon dɛn nɔto jɔs tin dɛn. Dɛn nid fɔ de mek dɛn nyu ɛn gɛt trɛnk ɔltɛm. if wi nכ gεt di rayt nyutriεnt dεm, כ if tin dεm lεk jεnεtiks כ vaytamεn dεm we de fכm de afekt di wok we di sεl dεm na wi bon dεm de du, di minral imbalans go apin εn wi bon dεm go bigin fכ wik. Dɛn mɛtabolik bon sik ya kin bi we pɔsin bɔn ɔ we pɔsin kin gɛt.

Wetin na ɛgzampul dɛn bɔt dɛn sik ya?

Bɔku sik dɛn kin fɔdɔm insay dis kategori fɔ mɛtabolik bon sik dɛn. Lɛ wi luk sɔm pan dɛn:

• Ɔstioporosis: Dis na di sik we bɔku pipul dɛn kin gɛt ɛn we pipul dɛn sabi bɔt dis kayn sik. Dɛn se na lɛk 200 milyɔn pipul dɛn ɔlsay na di wɔl gɛt dis sik. Fɔ tɔk am simpul wan, ɔstioporosis na wan sik we di density na wi bon dɛm, dat na di amɔnt ɔf minral sɔl dɛm insay di bon dɛm (wi kin kɔl dis bon mas bak), de go dɔŋ smɔl smɔl. Dɔn di bon dɛn kin wik ɛn dɛn kin brok izi wan. Ivin if yu fɔdɔm smɔl, chans de fɔ mek yu brok yu an ɔ yu leg.
• Ostiopenia: Dis na bak we di bon dεm de dכn, bכt i nכ de siriכs lεk osteoporosis, εn na wan sik we nכ de sכmtεm sכmtεm.

Ɔda kayn dɛn de:

• Cushing’s syndrome: Dis na we yu bon wik bikɔs ɔf prɔblɛm we gɛt fɔ du wit ɔmon.
• Hypophosphatasia: Dis kin bi bikɔs ɔf di ɛnzaym we nɔ de.
• Osteogenesis imperfecta: Dɛn kin kɔl am bak "brittle bone disease," dis na wan sik we kin de bɔku tɛm we dɛn bɔn am. Di bon dɛn kin brok izi wan.
• Osteomalacia ɛn Rickets: Dis na tin we de mek di bon dɛn sof. We dis sik apin to big pipul, dεn kכl am osteomalacia, εn we i apin to pikin dεm, dεn kכl am rikεt.
• Di sik we dɛn kɔl Paget:Dis na we di bon dεm de gro abnכmal εn kwik kwik wan, we de mek dεn wik.

Aw dɛn sik ya kin bɔku?

As wi bin dɔn tɔk, sɔm mɛtabolik bon sik dɛn kin bɔku pas ɔda wan dɛn. Ɔstioporosis na di wan we kin bɔku pan dɛn tin ya. Fɔ ɛgzampul, na Amɛrika, lɛk 12.6% pan pipul dɛm wae dɔn pas 50 ia gɛt dis sik.

Bɔt kɔndishɔn dɛn lɛk Osteogenesis imperfecta nɔ kin bɔku. Ivin na Amɛrika, na smɔl pipul dɛn nɔmɔ dis sik kin ambɔg, lɛk 25,000 to 50,000. Kɔndishɔn lɛk Osteomalacia, Hypophosphatasia, ɛn Paget in sik bak nɔ kin bɔku lɛk ɔstioporosis.

Wetin mek dɛn mɛtabolik bon sik ya kin apin?

Di men rizin na bikɔs wi bon dɛn nɔ kin gɛt inof minral. Bikɔs ɔf dis we aw di minral dɛn nɔ de, di bon dɛn kin wik smɔl smɔl ɛn pwɛl, we kin mek pɔsin gɛt dɛn sik ya. dis minral imbalans kin de we dεn bכn am, כ i kin divεlכp leta.

Wi bɔdi gɛt wan sistɛm we de kip tin dɛn lɛk kalsiɔm, vaytamɛn D, ɛn fɔsfɔr insay di rayt amɔnt. Dɛn kɔl dis minral homyostasis . Prɔblɛm kin bigin we dɛn ambɔg dis. Bikɔs wi bon dɛn kin kip dɛn minral dɛn ya, yuz dɛn fɔ gro nyu wan dɛn, ɛn strɔng.

Wetin na di tin dɛm wae kin mek di risk bɔku?

Sɔm tin dɛn de we kin mek yu gɛt dis mɛtabolik bon sik. Lɛ wi tek wan luk pan wetin dɛn bi:

• Fɔ bi 65 ia ɔ pas dat.
• Fɔ smok.
• di fכs mεnopos kin apin bifo i rich 45 ia.
• If yu nɔ gɛt bɛtɛ layt na di san (we yu nɔ gɛt vitamin D). Pan ɔl we wi kɔntri gɛt bɔku san, sɔm pipul dɛn nɔ kin gɛt bɛtɛ say fɔ si di san. Dis kin mek di vaytamɛn D nɔ bɔku.
• Fɔ gɛt smɔl bɔdi (Petite frame).
• Fɔ dɔn gɛt ɔmon tɛrapi fɔ brɔst kansa ɔ prɔstat kansa.
• Fɔ bi pɔsin we de gɛt ɔgan transplant.
• Yuz mɛrɛsin fɔ lɔng tɛm fɔ mek yu nɔ gɛt sik.
• If yu gɛt famili histri bɔt ɔstioporosis, mɔ if yu mama ɔ papa bin gɛt ɔstioporosis ɔ yu hip brok, dat kin mek yu gɛt mɔ risk.
• Nɔ de du ɛksɛsayz inof.

Ɔda wɛl bɔdi prɔblɛm dɛn we kin mek dɛn sik ya kam

Pipul wae gɛt sɔm kayn wɛl bɔdi prɔblɛm kin gɛt dis mɛtabolik bon sik bak. Dɛn kayn kɔndishɔn dɛn ya na:

• Hyperparathyroidism: We yu de mek paratayroyd ɔmon pasmak.
• Hypophosphatemia: Fɔsfɔr we de na di blɔd kin smɔl.
• Vitamin D we nɔ de.
• Di we aw pɔsin nɔ gɛt kalsiɔm.
• Kidni sik.
• Dizayd fɔ yuz rɔm.
• Malabsorpshɔn we de mek pɔsin nɔ ebul fɔ du sɔntin.
• Malnutrishɔn.

Wetin na di sayn dɛm fɔ dɛn sik ya?

De sayn dɛm fɔ dɛn sik ya kin difrɛn frɔm wan to ɔda. Dɛn kin difrɛn bak fɔ di kayn sik. Na sɔm kɔmɔn sayn dɛn ya:

• Ayt we yu de lɔs (we yu lɔs at le wan inch).
• Bɔn kin brok bɔku tɛm. Ivin smɔl fɔdɔm ɔ aksidɛnt kin mek in bon brok.
• di we aw yu de tinap de chenj (we yu de ledɔm bifo, yu de butu).
• I nɔ izi fɔ waka.
• Di pen we de na di lɔwa bak.
• Di mɔsul dɛn we wik.
• Pen na di bon ɔ di hip eria.
• I nɔ kin izi fɔ yu fɔ blo. dis kin apin we di lכng kapasiti dכn dכn bikoz fכ kכmpreshכn di spεnal kכd.

Bɔt wan tin de we yu fɔ rili mɛmba. Bɔrku tɛm, ɔstioporosis na "saylent sik." Dis min se we di bon dεm bigin, di bon dεm kin wik sכmtεm frכm insay we dεn nכ de mek dεn fil pen כ big big sayn dεm.

Aw dɛn kin no bɔt dɛn sik ya?

If yu gɛt ɛni wan pan dɛn sik ya, ɔ if yu gɛt tin dɛn we kin mek yu gɛt dis sik, i go fayn fɔ mek yu go to dɔktɔ fɔ mek dɛn chɛk yu. Di dɔktɔ go chɛk yu ɛn if nid de, ɔda wan ɔ mɔ pan dɛn tɛst ya:

Blɔd tɛst dɛn

Dɛn tɛst ya kin chɛk di lɛvɛl we di kalsiɔm, fɔsfɔr, ɛn vaytamɛn D de na di blɔd. Fɔ ɛgzampul, if yu nɔ gɛt bɔku kalsiɔm, vaytamɛn D, ɔ fosfɔr, dat kin bi sayn fɔ wan sik we dɛn kɔl ɔstiomalasia. semweso, we di alkaline phosphatase dכn lכw εn di vaytam in B6 we de na di bכdi kin bi sayn fכ haypofosfatasia.

X-ray we dɛn kin du

Dɛn kin yuz ɛkstrem rayt fɔ chɛk fɔ si if di bon dɛn krak ɛn brok, we kin bi sayn fɔ tin dɛn lɛk ɔstioporosis.

Bɔn Minral Dɛnsiti (BMD) tɛst

Dɛn kin kɔl dis bak DEXA skan ɔ DXA skan . I de mɛzhɔ di trɛnk we yu bon dɛn gɛt na say dɛn lɛk yu an, yu hip, ɛn yu spayna. I de yuz smɔl raytin, so natin nɔ de fɔ wɔri bɔt. In jɛnɛral, as prɛvɛntiv mɛsej, i fayn fɔ mek dɛn du dis BMD tɛst we dɛn ol 65 ia fɔ uman dɛn ɛn 70 ia fɔ man dɛn. Bɔt if yu gɛt bɔku risk fɔ lɛ yu bon lɔs ɛn brok, yu dɔktɔ go se yu fɔ du dis skan bifo tɛm.

Yu tink se tritmɛnt de fɔ dis?

Yes, tritmεnt dεm de fכ tru fכ dεn mεtabolik bon sik dεm ya.De tritmɛnt wae go fayn fɔ yu go dipen pan de ɔndalayn kɔz fɔ de sik. Bɔku tritmɛnt dɛn de we yu kin pik:

• Vitamin ɛn nyutrishɔn sɔpɔtmɛnt: Dɛn kin gi dɛn tin ya, mɔ if di kalsiɔm ɛn vaytamɛn D nɔ de.
• Anabolic agents: Dis na mεdikeshכn dεm we de bil nyu bon dεm εn mek dεn trεnk.
• Bisphosphonates: Dis na mεdikal dεm we de ridyus di bon dεm we de lכs.
• Denosumab injεkshכn dεm.
• Ɔmon tritmɛnt: Sɔntɛnde kin de we dɛn kin yuz ɔmon lɛk tɛstostɛron, kalsitɔnin, ɛn ɛstrojen.
• Fɔ trit yu bɔdi: Dis kin min fɔ du ɛksɛsayz ɛn ɔda tritmɛnt dɛn fɔ mek yu mɔsul dɛn strɔng, fɔ mek yu balans fayn, ɛn fɔ mek i izi fɔ waka.
• Ɛksesaiz fɔ bia wet: Tin dɛn lɛk fɔ waka, fɔ jok, ɛn ɔda tin dɛn.
• Ɔpreshɔn: Bɔku tɛm dɛn nɔ kin du dis ɔltɛm, mɔ if sɔntin we kɔmplikt, lɛk we pɔsin brok, apin.

Ɛni bad tin de we di tritmɛnt kin du?

Sɔm mɛrɛsin dɛn kin mek pɔsin gɛt sayd ɛfɛkt. Bɔt i dipen pan di kayn mɛrɛsin we dɛn de yuz. Sɔm pan di kɔmɔn sayd ɛfɛkt dɛm na:

• Simptom dɛm we tan lɛk flu.
• Ed de at.
• At bɔn.
• Efεkt dεm pan di kidni fכnshכn.
• Mɔsul dɛn kin pen.
• Skin de rɛd, dray, it.
• di bon pen we de go כp (insay di fכs stej dεm).

Dis list nɔ kɔmplit. If yu notis ɛnitin we nɔ kɔmɔn ɔ yu gɛt ɛni nyu sayn we yu de gɛt tritmɛnt, yu fɔ rili tɛl yu dɔktɔ.

Aw kwik a go ebul fɔ ɛkspɛkt fɔ wɛl afta dɛn dɔn trit mi?

Bɔrku pipul kin bigin fɔ fil sɔm kayn impɔtant tin na dɛn trɛnk jɔs afta dɛn dɔn bigin fɔ trit dɛn. Bɔt i kin tek tɛm fɔ mek di bon dɛn bɔku ɛn fɔ mek nyu bon dɛn we gɛt wɛlbɔdi. Yu dɔktɔ go ɛksplen aw lɔng i go tek ɛn wetin fɔ ɛkspɛkt, i go dipen pan yu kɔndishɔn.

Wetin kin apin if a gɛt dis kayn bon sik?

If dɛn no se yu gɛt mɛtabolik bon sik lɛk ɔstioporosis ɔ ɔstiɔmalasia, yu dɔktɔ go gi yu tritmɛnt plan we go fayn fɔ yu. Dis kin inklud mɛrɛsin, tin dɛn we de ɛp yu fɔ it, tritmɛnt fɔ yu bɔdi, ɛn, nɔto ɔltɛm, ɔpreshɔn. I kin tek sɔm tɛm fɔ mek yu fil fayn afta di tritmɛnt bigin. Bɔt if yu trit yu di rayt tritmɛnt, yu bon dɛn go bigin fɔ strɔng as tɛm de go.

Wetin na di lukin-grɔn fɔ dɛn kayn tin ya?

If yu gɛt di rayt tritmɛnt, di luk fɔ dɛn mɛtabolik bon sik ya kin jɔs fayn.If yu trit yu di rayt tɛm, i kin ɛp fɔ bil yu bon dɛn bak ɛn kɔntrol di bon dɛn we de lɔs. Dis kin mek yu nɔ gɛt bɔku prɔblɛm wit yu bɔdi ɛn i kin mek yu layf bɛtɛ.

Aw yu go ridyus di risk fɔ gɛt dɛn sik ya?

Bikɔs sɔm mεtabolik bon sik dεm gεt jεnεtik inflכεns, i nɔr kin izi fɔ avɔyd dɛn ɔltɛm. Bɔt tin dɛn de we wi kin du fɔ ridyus di prɔblɛm we wi gɛt:

• Gɛt prɛvɛntiv skrinin fɔ sik dɛn lɛk ɔstioporosis ɔltɛm.
• If yu de smok, stɔp.
• If yu de drink rɔm, nɔ fɔ drink am.
• Ɛksesaiz ɔltɛm.
• It it we gɛt bɔku kalsiɔm ɛn vaytamɛn D (milk, chiz, yogɔt, grɛn vɛjitebul, smɔl fish).
• Aks yu dɔktɔ bɔt tin dɛn we go ɛp yu fɔ mek yu bon dɛn gɛt wɛlbɔdi.

Aw a go tek kia ɔf misɛf wit dis kayn sik?

Apat frɔm di skrinin we dɛn kin du fɔ mek dɛn nɔ gɛt sik ɛn chenj dɛn layf, i impɔtant fɔ tek tɛm we pɔsin fɔdɔm, bikɔs i kin mek di bon dɛn brok mɔ ɛn mɔ. Yu kin du tin dɛn lɛk:

• Lan bɔt aw fɔ mek pɔsin nɔ fɔdɔm.
• No di bad tin dɛn we kin mek yu fɔdɔm na yu os ɛn mek chenj dɛn we yu nid (fɔ ɛgzampul, fɔ put rug dɛn na say dɛn we slipul, fɔ put grab bar dɛn na di bafa).
• Kɔnsul yu dɔktɔ fɔ no if yu fɔdɔm ɛn if nid de, gɛt tin dɛn we go ɛp yu fɔ waka.

Dɛn sik ya we de mek pɔsin gɛt bɔdi we de wok na di bɔdi kin denja?

If dɛn nɔ no ɛn trit am kwik, bɔku mɛtabolik bon sik dɛn kin siriɔs. Sɔm kin ivin mek dɛn layf de pan denja. Fɔ ɛgzampul, ɔstioporosis kin mek di bon dɛn brok mɔ ɛn mɔ. Kɔndishɔn lɛk Cushing’s syndrome kin kil pɔsin if dɛn nɔ trit am.

So, mεtabolik bon sik na grup כf kכndishכn dεm we de afekt bon mas, bon tכnכv, εn bon gכd. Sɔm kin bɔku pas ɔda wan dɛn, bɔt dɛn ɔl kin afɛkt di wɛlbɔdi na yu bon dɛn. Bɔt mɛmba se if yu no di sik ɛn trit yu fayn fayn wan, yu go ebul fɔ liv layf we go mek yu gladi. If yu gɛt ɛni kwɛstyɔn ɔr wɔri bɔt dɛn sik ya, yu tritmɛnt plan, ɔr aw yu de si tin, nɔr shem fɔ tɔk to yu dɔktɔ. I kin ɛp yu.

So, wetin na di tin dɛn we wi nid fɔ mɛmba frɔm ɔl dɛn tin ya?

Okay, so a op se yu dɔn ɔndastand mɔ bɔt di mɛtabolik bon sik dɛn we wi bin tɔk bɔt tide. Fɔ tɔk smɔl:

• Tek kia ɔf yu bon wɛlbɔdi. Strɔng bon dɛn impɔtant fɔ mek yu gɛt wɛlbɔdi.
• Mεtabolik bon sik dεm na kכndyushכn dεm we de wikεd bon dεm εn rεdכks dεn trεnk.
• di minral imbalans (especially calcium, vitamin D, phosphorus) na di men tin we kin mek dis apin.
• Tin dɛm lɛk di ej, di we aw pɔsin de liv in layf, ɛn di famili istri kin mek di prɔblɛm bɔku.
• Sɔm sik, lɛk ɔstioporosis, nɔ kin sho ɛni sayn fɔs, so i bɛtɛ fɔ mek dɛn tɛst yu if yu gɛt tin dɛn we kin mek yu gɛt dis sik.
• We dɛn de fɔ no ɛn trit dɛn sik ya. So, if yu gɛt sɔm kayn sik, nɔr delay fɔ go to dɔktɔ.
• If yu gɛt di rayt tritmɛnt ɛn chenj dɛn layf, yu kin ebul fɔ kɔntrol dɛn sik ya fayn fayn wan ɛn liv gud layf.

Tek kia ɔf yu bon dɛn! Bikɔs bon dɛn we gɛt wɛlbɔdi na di trɛnk we de gi yu fridɔm ɛn gladi at fɔ liv fri ɛn gladi layf.

` Mεtabolik Bon Disiz, Ostioporosis, Bon Wik, Kalsiכm, Vitamin D, Bon Density, Frakshכn