Ngaba ngamanye amaxesha uvuka kusasa unomqolo oqinileyo kunye nentlungu eyenza kube nzima ukuyolula umqolo wakho? Olu hlobo lwentlungu, ingakumbi kumqolo ongezantsi nasemaqakaleni, lunokuba sisiqalo sento enzulu kakhulu kunentlungu nje yomqolo, ingakumbi xa usemncinci. Oku akusiyo intlungu yomqolo nje. Namhlanje siza kuthetha ngenye yeemeko ezinjalo, i-ankylosing spondylitis (AS), uhlobo olungaqhelekanga lwe-arthritis. Makhe sithethe ngento ebangela le ntlungu kwaye ngubani osengozini enkulu yokuyibangela.
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Ngamafutshane, yintoni i-Ankylosing Spondylitis (AS)?
I-Ankylosing Spondylitis, okanye i-AS ngamafutshane, luhlobo lwe-arthritis. Ichaphazela kakhulu umqolo. Ibangela ukudumba, oko kuthetha ukuba ibangela ukudumba nokuqina kwamalungu omqolo kwaye ngamanye amaxesha amanye amalungu, njengeempundu. Oku kunokubangela ukuqina kunye nentlungu kusasa.
Ukuba oku kuvutha kuqhubeka ixesha elide, njengoko umzimba wethu uzama ukuphola, kwakheka amaqhekeza amatsha amathambo. Ngenxa yesiphumo esibi soku, amathambo anokucinezelwa kwaye umqolo unokulahlekelwa bubugophe bawo. Oku kwenza kube nzima ukugcina umqolo uthe tye kwaye ugobe.
Ngaba oogqirha bayasazi isizathu esichanekileyo soku?
Inyaniso kukuba, oogqirha abanakutsho ngokuqinisekileyo ngokupheleleyo ukuba yintoni kanye kanye ebangela i-AS. Nangona kunjalo, okwangoku kuyavunywa ukuba ibangelwa kukudibana kwezinto ezimbini: ii-genes zethu kunye nezinto ezisingqongileyo.
Cinga ngale ndlela: iijini zethu zifana nenqwaba yempuphu. Kodwa ukuze ivuthe, ifuna intlantsi yangaphandle. Kwenzeka into efanayo apha. Nangona kukho utyekelo lwemfuza lokuphuhlisa i-AS, kufuneka uhlobo oluthile 'lwesiqhumiso' okanye 'intlantsi' oluvela kwindawo esingqongileyo ukuze isifo senzeke.
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Check Availability →Impembelelo ye-genetic kunye ne-HLA-B27
Xa sithetha nge-AS, into enye ekufuneka ixoxwe ngayo yi-gene ebizwa ngokuba yi-HLA-B27 . Le yi-gene evelisa iproteni ekhethekileyo, okanye 'uphawu lwe-genetic', emizimbeni yethu. Umsebenzi ophambili wale gene kukunceda amasosha omzimba ethu. Oko kukuthi, ukunceda ekuchongeni nasekulweni nezinto ezinobungozi ezifana neebhaktheriya kunye neentsholongwane ezingena emzimbeni.
Uninzi lwabantu abane-AS lune-HLA-B27 gene emizimbeni yabo. Nangona kunjalo, eyona nto ibalulekileyo apha yile:
Asinguye wonke umntu ophuhlisa i-AS kuba nje ene-HLA-B27 gene.Enyanisweni, uninzi lwabantu abanale jini abasayi kuze bakhulele kwi-AS ebomini babo. Kwakhona, kunokwenzeka ukuba umntu ongenale jini akhulele kwi-AS.
Oko kuthetha ukuba i-HLA-B27 yinxalenye nje yebali. Izazinzulu zichonge ezinye iijini ezininzi ezinxulumene ne-AS. Phantse zonke ezi jini zibandakanyeka ngandlela ithile ekusebenzeni kwenkqubo yethu yokuzikhusela komzimba.
I-Trigger
Kulungile, masithi unesifo sofuzo, okanye 'isibhamu', ukuze uvelise i-AS. Ngoku udinga 'intlantsi' ukuze uyivuthele. Izazinzulu zisaphanda ukuba yintoni le ntlantsi.
Enye ingcamango ephambili kukuba oku kuqhambuka kubangelwa lusulelo lwebhaktiriya . Ngokukodwa, kucingelwa ukuba oku kusenokubangelwa kukuphazamiseka kwendlela yokuzikhusela yendalo yenkqubo yethu yokugaya ukutya, oko kukuthi amathumbu . Xa oku kusenzeka, iintlobo ezithile zeebhaktiriya ezivela emathunjini zinokungena egazini.
Xa iintsholongwane zingena egazini ngale ndlela, inkqubo yomzimba yokuzikhusela yomntu onesifo semfuza isabela ngokugqithisileyo, iphambane. Ngenxa yoko, inkqubo yomzimba yokuzikhusela iqala ukuhlasela amalungu omzimba asempilweni, ingakumbi amalungu omqolo. Le yindlela i-AS eqala ngayo.
Ngubani osengozini enkulu yoku?
Nangona nabani na enokuba ne-AS, abanye abantu basengozini enkulu kancinci. Makhe sijonge ukuba bangoobani.
| Into Engumngcipheko | Inkcazo |
|---|---|
| Imbali yosapho | Ukuba umntu kusapho lwakho, ingakumbi umama, utata, okanye umntakwenu, une-AS, usemngciphekweni omkhulu wokuba nayo kunomntu oqhelekileyo. Oku kubonisa impembelelo yezakhi zofuzo. |
| Ubudala | I-AS idla ngokuqala kubantu abancinci abaphakathi kweminyaka eli-17 nengama-45 ubudala. Idla ngokuqala kwangethuba kakhulu kunezinye iintlobo ze-arthritis ezikhula xa abantu bekhula. |
| Isini | Amadoda anamathuba aphindwe kabini okukhula njengokwabasetyhini. |
| Usulelo lwamathumbu rhoqo | Le ngozi inokwanda kancinci kumntu ohlala enengxaki yesisu kunye nosulelo lokugaya ukutya. Oku kungenxa 'yentlantsi yomlilo' esithethe ngayo ngaphambili. |
| Ezinye izifo zokuzikhusela komzimba | Ukuba sele unesinye isifo sokuzikhusela komzimba, esifana ne -psoriasis , isifo sikaCrohn , okanye i-ulcerative colitis , usemngciphekweni omkhulu wokufumana i-AS. Into efanayo kuzo zonke ezi zifo kukungasebenzi kakuhle kwenkqubo yomzimba yokuzikhusela. |
Ukuba uneentlungu zomqolo okanye ukuqina okunje, ingakumbi ukuba zimbi kakhulu kusasa kwaye zihlala ixesha elingaphezu kwesiqingatha seyure, musa ukuzibetha ngoyaba. Musa ukucinga nje ukuba, 'Oku kwenzeka xa ukhula.' Bona ugqirha wakho ngoko nangoko uze uthethe ngayo. Okukhona uyifumanisa ngokukhawuleza le meko, kokukhona amathuba akho okuyilawula, ukunciphisa umonakalo kumalungu akho, nokuphila ubomi obusempilweni.
Umyalezo Wokuya Ekhaya
- I-Ankylosing Spondylitis (AS) luhlobo lwe-arthritis oluchaphazela umqolo, kungekuphela nje iintlungu eziqhelekileyo zomqolo.
- Nangona kungekho sizathu sicacileyo soku, kucingelwa ukuba kubangelwa kukudibana kwezakhi zofuzo ezifana ne-HLA-B27 kunye nezinto ezibangela okungqongileyo ezifana nosulelo lweebhaktheriya.
- Ukuba ne-HLA-B27 gene okanye ukuba nezinye izinto ezinobungozi akuthethi ukuba ngokuqinisekileyo uza kuba ne-AS.
- Esi sifo sidla ngokuqala ebantwaneni, phakathi kweminyaka eli-17-45 ubudala.
- Ukuba uneentlungu ezingapheliyo zomqolo kunye nokuqina kwawo, ingakumbi kusasa, musa ukuchitha ixesha uye kugqirha.
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