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Sik ɛn Kɔndishɔn dɛnDecember 13, 2025🔄 Updated: Jul 14, 2026⏱ 13 min

Na di tɔp nɔmba nɔmɔ fɔ yu blɔd prɛshɔn ay? Na dat na Isolated Systolic Hypertension?

Dr. Priya Sammani, MBBS, PG Dip. FMMedical Verification

Yu dɔn ɛva chɛk yu blɔd prɛshɔn ɛn na di nɔmba we de ɔp nɔmɔ ay, bɔt di nɔmba we de dɔŋ nɔmal? Sɔntɛm wan dɔktɔ dɔn tɛl yu dat. Dis na wetin wi kin kɔl Isolated Systolic Hypertension. Bɔku pipul dɛn nɔ no bɔku tin bɔt dis. So, lɛ wi tɔk bɔt am mɔ ditayli tide.

Wetin na Isolated Systolic Hypertension?

Fɔ tɔk am simpul wan, dis na we di nɔmba we de ɔp pan yu blɔd prɛshɔn, we na di sistolik nɔmba , pas di lɛvɛl we dɛn kin se. di nכmba we de dכn, di dayastolik nכmba, kin bi nכmal כ lכw. fכ bi prεsis, dis na we di sistolik nכmba hכy pas 130 milimita mεkkyuri (mmHg) εn di dayastolik nכmba de lכs pas 90 milimita mεkkyuri. Nɔmal wan, pɔsin we gɛt wɛlbɔdi in blɔd prɛshɔn kin smɔl pas 120/80 mmHg. Yu no wetin sistolik prɛshɔn min? We wi at de bit ɛn pɔmp blɔd ɔlsay na di bɔdi, da blɔd de kin put prɛshɔn pan di wɔl dɛn na wi blɔd vesel dɛn. diastolik prεshכn de tכk bכt di rεsistεns na di bכdi vεsul dεm we di at rilaks sכmtεm, dat na we i nכ de pכmp bכdi.

Aw kɔmɔn tin dis? Yu tink se i kin bɔku we pɔsin de ol?

Dis sik kin rili kɔmɔn, mɔ as wi de ol. Akɔdin to sɔm statystik, lɛk 15% pan pipul dɛm wae dɔn pas 60 ia gɛt Isolated Systolic Hypertension. Infakt, dis na di kayn haypatɛyshɔn we kin kam pan pipul dɛn we dɔn pas 70 ia. So i klia se dis risk kin bɔku we dɛn de ol, nɔto so?

Yu tink se Isolated Systolic Hypertension Denja?

Yɛs, i kin denja if dɛn nɔ trit am fayn. If yu nɔ trit am, lɛk ɔda kayn ay blɔd prɛshɔn, i kin pwɛl difrɛn pat dɛn na yu bɔdi. Jɔs tink bɔt, na risk factor fɔ di sik dɛm na di at ɛn di blɔd (i.e. sik dɛm na di at, di blɔd vesel dɛm na di bren ɛn big blɔd vesel dɛm ɔlsay na di bɔdi), day, ɛn ivin siriɔs tin dɛm lɛk we di kidni nɔ de wok fayn. So dis nɔto sɔntin we wi fɔ tek am layt.

Dis na imejensi?

Isolated Systolic Hypertension nɔ kin bi imejensi. Bɔt if yu sistolik ridin go ɔp wantɛm wantɛm pas 180 mmHg, na imejensi. Wi kin kɔl dis ‘Hypertensive crisis’. If dis apin, yu fɔ go to dɔktɔ wantɛm wantɛm.

Wetin na di sayn dɛm fɔ dis?

Lɛk ɔda kayn ay blɔd prɛshɔn, dis wan nɔ de sho big big sayn dɛn we i bigin.Bɔrku pipul nɔr kin ivin no se dɛn gɛt dis sik. Na dat mek i impɔtant fɔ de chɛk yu blɔd prɛshɔn ɔltɛm. Bɔt we dɛn nɔ trit am fɔ lɔng tɛm ɛn bigin fɔ pwɛl yu bɔdi in ɔgan dɛn, sɔm sayn dɛn kin sho. Dɛn na:

Chɛst de pen.
Di shɔt we aw yu de blo .
Di ed we kin at bɔku tɛm .
Vishɔn we nɔ klia bɛtɛ .

If yu gɛt sayn dɛn lɛk dis, yu fɔ rili go to dɔktɔ.

Wetin mek dis de apin? Wetin na di rizin dɛn?

di men rizin fכ dis na biכs wi bכdi vεsεl dεm (atεri dεm) de chenj as wi de ol.

Di ol we pɔsin de ol ɛn di chenj we de apin na di at

As wi de ol, wi at dɛn kin stif ɛn nɔ kin ebul fɔ chenj. Dɛn nɔ kin strɛch ɛn kɔntrakt izi wan lɛk aw dɛn bin de du we wi bin yɔŋ. we dis kin apin, di prεshכn we dεn kin mek we di at de pכmp bכdi, we dεn kכl di sistolik prεshכn kin bכku. di sem tεm, di at dεm de lכs dεn elastik, so di dayastolik prεshכn de dכp. I tan lɛk wan ol rɔba tiub we kin stif bɔt nɔ kin strɛch izi wan.

Ɔda tin dɛn we kin apin to pɔsin we sik

Wan ɔda tin we kin mek i apin na wan sik we dɛn kɔl atherosclerosis. dis na we tin dεm lεk kalsiכm εn kolagen de dכn insay di at dεm, we de mek dεn at εn sכmtεm. dis lכs fכ elastik na di at dεm kin mek i gεt isol sistolik haypatεnshכn.

Wetin na di tin dɛn we kin mek pɔsin gɛt dis sik?

Bɔrku tin de wae kin ad mɔr risk to dis situeshɔn. Dɛn na:

Di bɔku bɔku kɔlɔstrel we de na di blɔd.
Fɔ gɛt dayabitis .
Fɔ liv layf we yu nɔ de sidɔm, we yu nɔ gɛt ɛni ɛksesaiz. Imajin pɔsin we de wok na wan ples ɔl di de.
Bɔdi mas indeks (BMI) we pas 30 ( ɔbisiti ).
Yuz tin dɛn we dɛn kin yuz fɔ smok (lɛk sigrɛt, beedis).
Fɔ it tin dɛn we dɛn dɔn prosɛs bɔku tɛm ɛn it dɛn we gɛt bɔku sɔl ɛn ɔyl, lɛk rays we dɛn dɔn frɛsh, sosish, ɛn it dɛn we dɛn pak.

Yɔŋ pipul dɛnsɛf kin gɛt dis?

Pan ɔl we dis kin apin mɔ pan pipul dɛn we dɔn ol, i kin apin bak to yɔŋ pipul dɛn we nɔ rich 40 ia yet.Naw dɛn dɔn kam fɔ no se dis sik we dɛn kɔl Isolated Systolic Hypertension, de kam mɔ ɛn mɔ. Yu nɔ tink se i de sɔprayz? Bɔrku pipul dɛm wae kin gɛt dis sik wae dɛn yɔŋ:

Di bɔdi mas indeks (BMI) pas 30 (we min se pɔsin fat pasmak).
Mɔs pan dɛn na man dɛn.
Yuz tin dɛn we dɛn mek wit tabak.
Yu kin si pipul dɛn bak we nɔ gɛt wɛlbɔdi inshɔrans.

Us prɔblɛm dɛn kin apin if dɛn nɔ trit am?

As wi bin dɔn tɔk bifo tɛm, if dɛn nɔ de manej dis fayn, dat na if dɛn nɔ trit am, denja kɔmplikeshɔn kin apin. Dɛn tin ya na:

At we nɔ de wok fayn
At atak
Strok
Kidni sik we nɔ de mɛn
di pεrifεral atεri sik - Dis na di sכmtεm we di at dεm we de gi bכdi to di limb dεm de sכmtεm sכmtεm.
Aneurysm - Dis na we di bכdi de wik εn bulge lεk balכn.

Aw yu no dis?

Bifo yu no se yu gɛt dis sik, yu dɔktɔ go aks yu bɔt yu famili histri, aw yu de it, aw yu de du tin, ɛn ɛni ɔda mɛrɛsin we yu go gɛt. Jɔs lɛk ɛni sik, fɔ no am kwik na di bɛst we fɔ mɛn am.

Us kayn tɛst dɛn kin du?

Bifo dɔktɔ no klia wan se yu gɛt Isolated Systolic Hypertension, dɛn go chɛk yu blɔd prɛshɔn tu ɔ tri tɛm. Dɛn nɔ go jɔs chɛk am wan tɛm ɛn se. Apat frɔm dat, dɛn kin du sɔm ɔda tɛst dɛn fɔ si aw yu gɛt prɔblɛm wit yu at ɛn blɔd. Fɔ ɛgzampul:

Wan ECG (ilɛktrokardiogram - EKG) tɛst. Dis de mekɔp di ilɛktrik wok we di at de du.
Blɔd tɛst dɛn. sכm spεshal wan, wan lipid panεl fכ chεk di kכlestכl lεvεl.

Wetin a fɔ du if a de pan dis kayn tin?

If yu dɔktɔ tɛl yu se yu gɛt Isolated Systolic Hypertension, di tin we impɔtant pas ɔl na fɔ fala dɛn instrɔkshɔn dɛn kɔrɛkt wan. Nɔ panik. Dɛn go mɔs tɛl yu fɔ mek sɔm chenj dɛn na di tin dɛn we yu de du ɛvride, ɛn sɔntɛm dɛn go ivin bigin fɔ tek mɛrɛsin.

Aw dɛn kin trit am?

We yu de trit dis, yu dɔktɔ kin tɛl yu fɔ chenj yu layf ɛn if nid de, yu fɔ bigin fɔ tek mɛrɛsin dɛn we de mek yu gɛt ay blɔd prɛshɔn.

Di we aw pipul dɛn de liv dɛn layf kin chenj

Dis na sɔm pan di impɔtant tin dɛn we yu go du:

Fɔ ridyus di sɔl we dɛn kin ad to it. I fayn fɔ mek yu nɔ it pas 1.5 gram sɔl fɔ wan de.
Fɔ fala di DASH it.Dis na spɛshal it plan we de ɛp fɔ kɔntrol ay blɔd prɛshɔn.
Fɔ it mɔ fresh frut ɛn vɛjitebul dɛn.
Fɔ ridyus di it dɛn we gɛt bɔku fat we gɛt dairy (e.g. chiz, bɔta).
fכ ridyus di it dεm we gεt satεrayt fεt (e.g. rεd mit, fεt it dεm).
Fɔ lɔs yu wet (if yu fat).
Fɔ gɛt mɔ ɛksesaiz. Sɔntin lɛk fɔ waka fɔ at le 30 minit ɛvride.
If yu de drink rɔm, nɔ fɔ drink am.
Fɔ kɔntrol di strɛs. Sɔntɛnde, fɔ tink gud wan ɔ fɔ du yoga kin ɛp.
Tray fɔ gɛt mɔ potashɔm frɔm it (e.g. banana, ɔrɛnj, spinach) .
Fɔ stɔp di tin dɛn we dɛn kin mek wit tabak kpatakpata.

Imajin aw dɛn smɔl chenj dɛn ya kin afɛkt yu wan ol layf!

Mɛrɛsin dɛn we dɛn kin yuz

Apat frɔm di chenj dɛn we pɔsin kin chenj in layf, dɔktɔ dɛn kin gi mɛrɛsin dɛn we dɛn kɔl antihypertensives fɔ mɛn Isolated Systolic Hypertension. Sɔm pan di mɛrɛsin dɛn we dɛn kin yuz mɔ na:

Diuretics: Dɛn tin ya kin mek di bɔdi pul di wata ɛn sɔl we pasmak tru di urine, we kin mek di blɔd prɛshɔn go dɔŋ.
Calcium channel blockers: Dɛn tin ya kin mek di wɔl dɛn na di blɔd vesel dɛn rilaks, ɛn dis kin mek i izi fɔ mek di blɔd flɔ.
Angiotensin-converting enzyme inhibitors (ACE inhibitors): Dɛn wan ya kin kɔntrol blɔd prɛshɔn bak bay we dɛn de ridyus di blɔd vesel dɛn we de kɔnstrikt.
Angiotensin II Receptor Blockers (ARBs): Dɛn mɛrɛsin ya de wok lɛk aw di ACE inhibitor dɛn de wok.

Yu dɔktɔ kin bigin yu fɔ tek wan pan di fɔs tu mɛrɛsin dɛn we de na dis list. Dɔn, i go dipen pan aw yu bɔdi de du tin, dɛn kin ad sɛkɔn ɔ tɔd mɛrɛsin if nid de. Blɔd vesel dɛn kin stif as yu de ol, ɛn dis kin mek i nɔ izi fɔ kɔntrol di ay blɔd prɛshɔn. Bɔt dɛn antihaypatensives ya kin ɛp fɔ ridyus di stiffness of yu arteries to some extent.

Ɛni bad tin de we di tritmɛnt kin du?

Jɔs lɛk ɛni ɔda mɛrɛsin, sɔm pipul dɛn kin gɛt smɔl smɔl sayd ɛfɛkt dɛn frɔm dɛn tritmɛnt ya. Nɔto ɔlman go ɛkspiriɛns dɛn, bɔt i fayn fɔ no bɔt dɛn.

Ed de at
Diziz we pɔsin kin gɛt
Fil fɔ it
Dray kɔf (especially frɔm ACE inhibitors) .
Di ia we de lɔs (rarely) .

If yu gɛt ɛnitin lɛk dis, nɔ fred fɔ tɛl yu dɔktɔ. I kin ebul fɔ chenj di mɛrɛsin ɔ ridyus di doz.

Wetin dɛn kin du fɔ ridyus dis prɔblɛm?

Wi ɔl kin ol. Sɔm pan di tin dɛm wae kin kam wit am na tin dɛm wae kin mek yu gɛt Isolated Systolic Hypertension. Bɔt bɔku tin dɛn de we yu kin du fɔ mek yu nɔ gɛt dis sik. Dɛn tin ya na:

If yu gɛt dayabitis, kɔntrol am fayn fayn wan.
If yu gɛt ay kɔlɔstrel, kɔntrol dat bak.
Ɛksesaiz ɔltɛm. At least 5 dez fo wik, 30 minit fo de.
Kip yu bɔdi mas indeks (BMI) dɔŋ 30.
Nɔ yuz tin dɛn we dɛn mek wit tabak.
Ridyus di it dɛn we dɛn dɔn prosɛs ɛn it dɛn we gɛt bɔku fat ɛn sɔl as yu ebul. Na di bɛst it dɛn we dɛn kin kuk na os.

Wetin yu fɔ ɛkspɛkt we yu de liv wit dis sik?

Di bɛst tritmɛnt na fɔ bigin jɔs lɛk aw dɛn no se yu gɛt dis sik, bikɔs ɛni kayn ay blɔd prɛshɔn kin mek yu gɛt ɔda wɛlbɔdi prɔblɛm dɛn lɛk at atak ɛn strok.

Yu tink se dɛn go ebul fɔ mɛn dis?

Nɔ, i nɔ go ebul fɔ mɛn am kpatakpata. Bɔt nɔ wɔri! Yu kin ebul fɔ kɔntrol am ɛn liv nɔmal layf bay we yu chenj yu layf ɛn/ɔ tek di mɛrɛsin we yu dɔktɔ tɛl yu fɔ du.

Aw lɔng dis tin go las?

Isolated Systolic Hypertension na wan sik wae de mɛk pɔrsin kin fil fɔ lɔng tɛm. Dis min se yu go nid fɔ kia fɔ am ɛn trit am fɔ di res ɔf yu layf. Risach dɔn sho se fɔ trit Isolated Systolic Hypertension fayn fayn wan kin mek big difrɛns. I kin ridyus di risk fɔ gɛt at sik, strok, ɛn day bay lɛk 30%! Yu nɔ tink se dat na big tin?

Aw a kin kia fɔ misɛf?

Fɔ mɛn dis sik, i impɔtant fɔ tek yu mɛrɛsin jɔs lɛk aw yu dɔktɔ tɛl yu fɔ tek, di rayt tɛm. Mek am bi yu abit. Sɔntɛm if yu sɛt sɔntin fɔ mɛmba yu na yu fon, dat go ɛp yu. Dɔn bak, fɔ du ɛksɛsayz ɛn it tin dɛn we nɔ gɛt bɔku sɔl ɛn fat kin rili fayn fɔ yu blɔd prɛshɔn ɛn yu at wɛlbɔdi.

Ustɛm a fɔ go to di dɔktɔ?

If yu dɔktɔ bigin fɔ tek mɛrɛsin fɔ dis sik, i go mɔs aks yu fɔ kam bak afta lɛk wan mɔnt so. Na da tɛm de i kin si if di mɛrɛsin de wok ɔ if i de mek ɛni sayd ɛfɛkt we de mɔna am. Insay da tɛm de, dɛn kin aks yu fɔ chɛk yu blɔd prɛshɔn na os wan tɛm insay di wik, ɔ mɔ.If yu gɛda dɛn ridin dɛn de fɔ lɛk wan mɔnt so ɛn sho dɛn to yu dɔktɔ, i go izi fɔ am fɔ ɔndastand if di mɛrɛsin de wok fayn. Dɔn bak, di dɔktɔ go luk if yu sistolik valyu ɛn yu dayastolik valyu de drɔp tumɔs. Bikɔs if yu dayastolik valyu go dɔŋ tumɔs, i kin min se yu ɔgan dɛn nɔ de gɛt inof blɔd.

Ustɛm yu fɔ go to tritmɛnt kwik kwik wan?

If yu gɛt ‘haypertensive crisis’ , we min se yu blɔd prɛshɔn na 180/120 mmHg ɔ pas dat, yu fɔ go to imejensi mɛdikal kia wantɛm wantɛm. Yu kin gɛt dɛn sayn ya:

Chɛst de pen
I nɔ kin izi fɔ yu fɔ blo
Diziz we pɔsin kin gɛt
Di ed we de at bad bad wan

Us kwɛstyɔn dɛn yu fɔ aks di dɔktɔ?

We yu go si dɔktɔ, i go fayn fɔ aks kwɛstyɔn dɛn lɛk dɛn wan ya:

Aw ɔltɛm a nid fɔ kam si yu?
Aw a go bigin fɔ du ɛksɛsayz program? Us ɛgzampul dɛn we rayt fɔ mi?
A nid fɔ tek mɛrɛsin, ɔ fɔ chenj di we aw a de liv mi layf go du fɔ mi sik?
Us sayd ɛfɛkt a kin ɛkspɛkt frɔm dis mɛrɛsin?
Us valyu dɛn a fɔ wɔri bɔt we a de chɛk mi blɔd prɛshɔn na os?

Fɔ dɔn, tin dɛn fɔ mɛmba (Take-Home Message) .

If yu gɛt Isolated Systolic Hypertension, nɔto yu wan de. Bɔku pipul dɛn kin gɛt dis sik we dɛn kɔl ay blɔd prɛshɔn. Nɔ wɔri. Bɔku chenj dɛn de we go ɛp yu fɔ chenj di we aw yu de liv yu layf. Yusɛf kin mek dɛn chenj dɛn de. If nid de, mɛrɛsin dɛn de bak.

Fɔ mɛn dis sik na big invɛstismɛnt fɔ yu wɛlbɔdi tumara bambay. Yu fiuja self go tank yu fo am!

So, tek kia ɔf yu blɔd prɛshɔn, mek dɛn chɛk am ɔltɛm, ɛn fala wetin yu dɔktɔ tɛl yu fɔ du. Dɔn yu go ebul fɔ liv fayn layf ɛn gladi at.

` Isolεt Sistolik Haypatεnshכn, Blכd Prεshכn, Sistolik, Dayastolik, Haypatεnshכn, At Wεlth, Sri Lanka

⚠️ Important: The medical articles and information on Nirogi Lanka are for general awareness only, and are by no means a substitute for professional medical advice, diagnosis, or treatment. For any medical problem you have, consult a qualified physician immediately.

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Sik ɛn Kɔndishɔn dɛnDecember 13, 2025

Na di tɔp nɔmba nɔmɔ fɔ yu blɔd prɛshɔn ay? Na dat na Isolated Systolic Hypertension?

Dr. Priya SammaniMedical Verification

⏱ 13 min read

Yu dɔn ɛva chɛk yu blɔd prɛshɔn ɛn na di nɔmba we de ɔp nɔmɔ ay, bɔt di nɔmba we de dɔŋ nɔmal? Sɔntɛm wan dɔktɔ dɔn tɛl yu dat. Dis na wetin wi kin kɔl Isolated Systolic Hypertension. Bɔku pipul dɛn nɔ no bɔku tin bɔt dis. So, lɛ wi tɔk bɔt am mɔ ditayli tide.

Wetin na Isolated Systolic Hypertension?

Fɔ tɔk am simpul wan, dis na we di nɔmba we de ɔp pan yu blɔd prɛshɔn, we na di sistolik nɔmba , pas di lɛvɛl we dɛn kin se. di nכmba we de dכn, di dayastolik nכmba, kin bi nכmal כ lכw. fכ bi prεsis, dis na we di sistolik nכmba hכy pas 130 milimita mεkkyuri (mmHg) εn di dayastolik nכmba de lכs pas 90 milimita mεkkyuri. Nɔmal wan, pɔsin we gɛt wɛlbɔdi in blɔd prɛshɔn kin smɔl pas 120/80 mmHg. Yu no wetin sistolik prɛshɔn min? We wi at de bit ɛn pɔmp blɔd ɔlsay na di bɔdi, da blɔd de kin put prɛshɔn pan di wɔl dɛn na wi blɔd vesel dɛn. diastolik prεshכn de tכk bכt di rεsistεns na di bכdi vεsul dεm we di at rilaks sכmtεm, dat na we i nכ de pכmp bכdi.

Aw kɔmɔn tin dis? Yu tink se i kin bɔku we pɔsin de ol?

Dis sik kin rili kɔmɔn, mɔ as wi de ol. Akɔdin to sɔm statystik, lɛk 15% pan pipul dɛm wae dɔn pas 60 ia gɛt Isolated Systolic Hypertension. Infakt, dis na di kayn haypatɛyshɔn we kin kam pan pipul dɛn we dɔn pas 70 ia. So i klia se dis risk kin bɔku we dɛn de ol, nɔto so?

Yu tink se Isolated Systolic Hypertension Denja?

Yɛs, i kin denja if dɛn nɔ trit am fayn. If yu nɔ trit am, lɛk ɔda kayn ay blɔd prɛshɔn, i kin pwɛl difrɛn pat dɛn na yu bɔdi. Jɔs tink bɔt, na risk factor fɔ di sik dɛm na di at ɛn di blɔd (i.e. sik dɛm na di at, di blɔd vesel dɛm na di bren ɛn big blɔd vesel dɛm ɔlsay na di bɔdi), day, ɛn ivin siriɔs tin dɛm lɛk we di kidni nɔ de wok fayn. So dis nɔto sɔntin we wi fɔ tek am layt.

Dis na imejensi?

Isolated Systolic Hypertension nɔ kin bi imejensi. Bɔt if yu sistolik ridin go ɔp wantɛm wantɛm pas 180 mmHg, na imejensi. Wi kin kɔl dis ‘Hypertensive crisis’. If dis apin, yu fɔ go to dɔktɔ wantɛm wantɛm.

Wetin na di sayn dɛm fɔ dis?

Lɛk ɔda kayn ay blɔd prɛshɔn, dis wan nɔ de sho big big sayn dɛn we i bigin.Bɔrku pipul nɔr kin ivin no se dɛn gɛt dis sik. Na dat mek i impɔtant fɔ de chɛk yu blɔd prɛshɔn ɔltɛm. Bɔt we dɛn nɔ trit am fɔ lɔng tɛm ɛn bigin fɔ pwɛl yu bɔdi in ɔgan dɛn, sɔm sayn dɛn kin sho. Dɛn na:

Chɛst de pen.
Di shɔt we aw yu de blo .
Di ed we kin at bɔku tɛm .
Vishɔn we nɔ klia bɛtɛ .

If yu gɛt sayn dɛn lɛk dis, yu fɔ rili go to dɔktɔ.

Wetin mek dis de apin? Wetin na di rizin dɛn?

di men rizin fכ dis na biכs wi bכdi vεsεl dεm (atεri dεm) de chenj as wi de ol.

Di ol we pɔsin de ol ɛn di chenj we de apin na di at

As wi de ol, wi at dɛn kin stif ɛn nɔ kin ebul fɔ chenj. Dɛn nɔ kin strɛch ɛn kɔntrakt izi wan lɛk aw dɛn bin de du we wi bin yɔŋ. we dis kin apin, di prεshכn we dεn kin mek we di at de pכmp bכdi, we dεn kכl di sistolik prεshכn kin bכku. di sem tεm, di at dεm de lכs dεn elastik, so di dayastolik prεshכn de dכp. I tan lɛk wan ol rɔba tiub we kin stif bɔt nɔ kin strɛch izi wan.

Ɔda tin dɛn we kin apin to pɔsin we sik

Wan ɔda tin we kin mek i apin na wan sik we dɛn kɔl atherosclerosis. dis na we tin dεm lεk kalsiכm εn kolagen de dכn insay di at dεm, we de mek dεn at εn sכmtεm. dis lכs fכ elastik na di at dεm kin mek i gεt isol sistolik haypatεnshכn.

Wetin na di tin dɛn we kin mek pɔsin gɛt dis sik?

Bɔrku tin de wae kin ad mɔr risk to dis situeshɔn. Dɛn na:

Di bɔku bɔku kɔlɔstrel we de na di blɔd.
Fɔ gɛt dayabitis .
Fɔ liv layf we yu nɔ de sidɔm, we yu nɔ gɛt ɛni ɛksesaiz. Imajin pɔsin we de wok na wan ples ɔl di de.
Bɔdi mas indeks (BMI) we pas 30 ( ɔbisiti ).
Yuz tin dɛn we dɛn kin yuz fɔ smok (lɛk sigrɛt, beedis).
Fɔ it tin dɛn we dɛn dɔn prosɛs bɔku tɛm ɛn it dɛn we gɛt bɔku sɔl ɛn ɔyl, lɛk rays we dɛn dɔn frɛsh, sosish, ɛn it dɛn we dɛn pak.

Yɔŋ pipul dɛnsɛf kin gɛt dis?

Pan ɔl we dis kin apin mɔ pan pipul dɛn we dɔn ol, i kin apin bak to yɔŋ pipul dɛn we nɔ rich 40 ia yet.Naw dɛn dɔn kam fɔ no se dis sik we dɛn kɔl Isolated Systolic Hypertension, de kam mɔ ɛn mɔ. Yu nɔ tink se i de sɔprayz? Bɔrku pipul dɛm wae kin gɛt dis sik wae dɛn yɔŋ:

Di bɔdi mas indeks (BMI) pas 30 (we min se pɔsin fat pasmak).
Mɔs pan dɛn na man dɛn.
Yuz tin dɛn we dɛn mek wit tabak.
Yu kin si pipul dɛn bak we nɔ gɛt wɛlbɔdi inshɔrans.

Us prɔblɛm dɛn kin apin if dɛn nɔ trit am?

As wi bin dɔn tɔk bifo tɛm, if dɛn nɔ de manej dis fayn, dat na if dɛn nɔ trit am, denja kɔmplikeshɔn kin apin. Dɛn tin ya na:

At we nɔ de wok fayn
At atak
Strok
Kidni sik we nɔ de mɛn
di pεrifεral atεri sik - Dis na di sכmtεm we di at dεm we de gi bכdi to di limb dεm de sכmtεm sכmtεm.
Aneurysm - Dis na we di bכdi de wik εn bulge lεk balכn.

Aw yu no dis?

Bifo yu no se yu gɛt dis sik, yu dɔktɔ go aks yu bɔt yu famili histri, aw yu de it, aw yu de du tin, ɛn ɛni ɔda mɛrɛsin we yu go gɛt. Jɔs lɛk ɛni sik, fɔ no am kwik na di bɛst we fɔ mɛn am.

Us kayn tɛst dɛn kin du?

Bifo dɔktɔ no klia wan se yu gɛt Isolated Systolic Hypertension, dɛn go chɛk yu blɔd prɛshɔn tu ɔ tri tɛm. Dɛn nɔ go jɔs chɛk am wan tɛm ɛn se. Apat frɔm dat, dɛn kin du sɔm ɔda tɛst dɛn fɔ si aw yu gɛt prɔblɛm wit yu at ɛn blɔd. Fɔ ɛgzampul:

Wan ECG (ilɛktrokardiogram - EKG) tɛst. Dis de mekɔp di ilɛktrik wok we di at de du.
Blɔd tɛst dɛn. sכm spεshal wan, wan lipid panεl fכ chεk di kכlestכl lεvεl.

Wetin a fɔ du if a de pan dis kayn tin?

If yu dɔktɔ tɛl yu se yu gɛt Isolated Systolic Hypertension, di tin we impɔtant pas ɔl na fɔ fala dɛn instrɔkshɔn dɛn kɔrɛkt wan. Nɔ panik. Dɛn go mɔs tɛl yu fɔ mek sɔm chenj dɛn na di tin dɛn we yu de du ɛvride, ɛn sɔntɛm dɛn go ivin bigin fɔ tek mɛrɛsin.

Aw dɛn kin trit am?

We yu de trit dis, yu dɔktɔ kin tɛl yu fɔ chenj yu layf ɛn if nid de, yu fɔ bigin fɔ tek mɛrɛsin dɛn we de mek yu gɛt ay blɔd prɛshɔn.

Di we aw pipul dɛn de liv dɛn layf kin chenj

Dis na sɔm pan di impɔtant tin dɛn we yu go du:

Fɔ ridyus di sɔl we dɛn kin ad to it. I fayn fɔ mek yu nɔ it pas 1.5 gram sɔl fɔ wan de.
Fɔ fala di DASH it.Dis na spɛshal it plan we de ɛp fɔ kɔntrol ay blɔd prɛshɔn.
Fɔ it mɔ fresh frut ɛn vɛjitebul dɛn.
Fɔ ridyus di it dɛn we gɛt bɔku fat we gɛt dairy (e.g. chiz, bɔta).
fכ ridyus di it dεm we gεt satεrayt fεt (e.g. rεd mit, fεt it dεm).
Fɔ lɔs yu wet (if yu fat).
Fɔ gɛt mɔ ɛksesaiz. Sɔntin lɛk fɔ waka fɔ at le 30 minit ɛvride.
If yu de drink rɔm, nɔ fɔ drink am.
Fɔ kɔntrol di strɛs. Sɔntɛnde, fɔ tink gud wan ɔ fɔ du yoga kin ɛp.
Tray fɔ gɛt mɔ potashɔm frɔm it (e.g. banana, ɔrɛnj, spinach) .
Fɔ stɔp di tin dɛn we dɛn kin mek wit tabak kpatakpata.

Imajin aw dɛn smɔl chenj dɛn ya kin afɛkt yu wan ol layf!

Mɛrɛsin dɛn we dɛn kin yuz

Apat frɔm di chenj dɛn we pɔsin kin chenj in layf, dɔktɔ dɛn kin gi mɛrɛsin dɛn we dɛn kɔl antihypertensives fɔ mɛn Isolated Systolic Hypertension. Sɔm pan di mɛrɛsin dɛn we dɛn kin yuz mɔ na:

Diuretics: Dɛn tin ya kin mek di bɔdi pul di wata ɛn sɔl we pasmak tru di urine, we kin mek di blɔd prɛshɔn go dɔŋ.
Calcium channel blockers: Dɛn tin ya kin mek di wɔl dɛn na di blɔd vesel dɛn rilaks, ɛn dis kin mek i izi fɔ mek di blɔd flɔ.
Angiotensin-converting enzyme inhibitors (ACE inhibitors): Dɛn wan ya kin kɔntrol blɔd prɛshɔn bak bay we dɛn de ridyus di blɔd vesel dɛn we de kɔnstrikt.
Angiotensin II Receptor Blockers (ARBs): Dɛn mɛrɛsin ya de wok lɛk aw di ACE inhibitor dɛn de wok.

Yu dɔktɔ kin bigin yu fɔ tek wan pan di fɔs tu mɛrɛsin dɛn we de na dis list. Dɔn, i go dipen pan aw yu bɔdi de du tin, dɛn kin ad sɛkɔn ɔ tɔd mɛrɛsin if nid de. Blɔd vesel dɛn kin stif as yu de ol, ɛn dis kin mek i nɔ izi fɔ kɔntrol di ay blɔd prɛshɔn. Bɔt dɛn antihaypatensives ya kin ɛp fɔ ridyus di stiffness of yu arteries to some extent.

Ɛni bad tin de we di tritmɛnt kin du?

Jɔs lɛk ɛni ɔda mɛrɛsin, sɔm pipul dɛn kin gɛt smɔl smɔl sayd ɛfɛkt dɛn frɔm dɛn tritmɛnt ya. Nɔto ɔlman go ɛkspiriɛns dɛn, bɔt i fayn fɔ no bɔt dɛn.

Ed de at
Diziz we pɔsin kin gɛt
Fil fɔ it
Dray kɔf (especially frɔm ACE inhibitors) .
Di ia we de lɔs (rarely) .

If yu gɛt ɛnitin lɛk dis, nɔ fred fɔ tɛl yu dɔktɔ. I kin ebul fɔ chenj di mɛrɛsin ɔ ridyus di doz.

Wetin dɛn kin du fɔ ridyus dis prɔblɛm?

Wi ɔl kin ol. Sɔm pan di tin dɛm wae kin kam wit am na tin dɛm wae kin mek yu gɛt Isolated Systolic Hypertension. Bɔt bɔku tin dɛn de we yu kin du fɔ mek yu nɔ gɛt dis sik. Dɛn tin ya na:

If yu gɛt dayabitis, kɔntrol am fayn fayn wan.
If yu gɛt ay kɔlɔstrel, kɔntrol dat bak.
Ɛksesaiz ɔltɛm. At least 5 dez fo wik, 30 minit fo de.
Kip yu bɔdi mas indeks (BMI) dɔŋ 30.
Nɔ yuz tin dɛn we dɛn mek wit tabak.
Ridyus di it dɛn we dɛn dɔn prosɛs ɛn it dɛn we gɛt bɔku fat ɛn sɔl as yu ebul. Na di bɛst it dɛn we dɛn kin kuk na os.

Wetin yu fɔ ɛkspɛkt we yu de liv wit dis sik?

Di bɛst tritmɛnt na fɔ bigin jɔs lɛk aw dɛn no se yu gɛt dis sik, bikɔs ɛni kayn ay blɔd prɛshɔn kin mek yu gɛt ɔda wɛlbɔdi prɔblɛm dɛn lɛk at atak ɛn strok.

Yu tink se dɛn go ebul fɔ mɛn dis?

Nɔ, i nɔ go ebul fɔ mɛn am kpatakpata. Bɔt nɔ wɔri! Yu kin ebul fɔ kɔntrol am ɛn liv nɔmal layf bay we yu chenj yu layf ɛn/ɔ tek di mɛrɛsin we yu dɔktɔ tɛl yu fɔ du.

Aw lɔng dis tin go las?

Isolated Systolic Hypertension na wan sik wae de mɛk pɔrsin kin fil fɔ lɔng tɛm. Dis min se yu go nid fɔ kia fɔ am ɛn trit am fɔ di res ɔf yu layf. Risach dɔn sho se fɔ trit Isolated Systolic Hypertension fayn fayn wan kin mek big difrɛns. I kin ridyus di risk fɔ gɛt at sik, strok, ɛn day bay lɛk 30%! Yu nɔ tink se dat na big tin?

Aw a kin kia fɔ misɛf?

Fɔ mɛn dis sik, i impɔtant fɔ tek yu mɛrɛsin jɔs lɛk aw yu dɔktɔ tɛl yu fɔ tek, di rayt tɛm. Mek am bi yu abit. Sɔntɛm if yu sɛt sɔntin fɔ mɛmba yu na yu fon, dat go ɛp yu. Dɔn bak, fɔ du ɛksɛsayz ɛn it tin dɛn we nɔ gɛt bɔku sɔl ɛn fat kin rili fayn fɔ yu blɔd prɛshɔn ɛn yu at wɛlbɔdi.

Ustɛm a fɔ go to di dɔktɔ?

If yu dɔktɔ bigin fɔ tek mɛrɛsin fɔ dis sik, i go mɔs aks yu fɔ kam bak afta lɛk wan mɔnt so. Na da tɛm de i kin si if di mɛrɛsin de wok ɔ if i de mek ɛni sayd ɛfɛkt we de mɔna am. Insay da tɛm de, dɛn kin aks yu fɔ chɛk yu blɔd prɛshɔn na os wan tɛm insay di wik, ɔ mɔ.If yu gɛda dɛn ridin dɛn de fɔ lɛk wan mɔnt so ɛn sho dɛn to yu dɔktɔ, i go izi fɔ am fɔ ɔndastand if di mɛrɛsin de wok fayn. Dɔn bak, di dɔktɔ go luk if yu sistolik valyu ɛn yu dayastolik valyu de drɔp tumɔs. Bikɔs if yu dayastolik valyu go dɔŋ tumɔs, i kin min se yu ɔgan dɛn nɔ de gɛt inof blɔd.

Ustɛm yu fɔ go to tritmɛnt kwik kwik wan?

If yu gɛt ‘haypertensive crisis’ , we min se yu blɔd prɛshɔn na 180/120 mmHg ɔ pas dat, yu fɔ go to imejensi mɛdikal kia wantɛm wantɛm. Yu kin gɛt dɛn sayn ya:

Chɛst de pen
I nɔ kin izi fɔ yu fɔ blo
Diziz we pɔsin kin gɛt
Di ed we de at bad bad wan

Us kwɛstyɔn dɛn yu fɔ aks di dɔktɔ?

We yu go si dɔktɔ, i go fayn fɔ aks kwɛstyɔn dɛn lɛk dɛn wan ya:

Aw ɔltɛm a nid fɔ kam si yu?
Aw a go bigin fɔ du ɛksɛsayz program? Us ɛgzampul dɛn we rayt fɔ mi?
A nid fɔ tek mɛrɛsin, ɔ fɔ chenj di we aw a de liv mi layf go du fɔ mi sik?
Us sayd ɛfɛkt a kin ɛkspɛkt frɔm dis mɛrɛsin?
Us valyu dɛn a fɔ wɔri bɔt we a de chɛk mi blɔd prɛshɔn na os?

Fɔ dɔn, tin dɛn fɔ mɛmba (Take-Home Message) .

If yu gɛt Isolated Systolic Hypertension, nɔto yu wan de. Bɔku pipul dɛn kin gɛt dis sik we dɛn kɔl ay blɔd prɛshɔn. Nɔ wɔri. Bɔku chenj dɛn de we go ɛp yu fɔ chenj di we aw yu de liv yu layf. Yusɛf kin mek dɛn chenj dɛn de. If nid de, mɛrɛsin dɛn de bak.

Fɔ mɛn dis sik na big invɛstismɛnt fɔ yu wɛlbɔdi tumara bambay. Yu fiuja self go tank yu fo am!

So, tek kia ɔf yu blɔd prɛshɔn, mek dɛn chɛk am ɔltɛm, ɛn fala wetin yu dɔktɔ tɛl yu fɔ du. Dɔn yu go ebul fɔ liv fayn layf ɛn gladi at.

` Isolεt Sistolik Haypatεnshכn, Blכd Prεshכn, Sistolik, Dayastolik, Haypatεnshכn, At Wεlth, Sri Lanka

⚠️ Important: The medical articles and information on Nirogi Lanka are for general awareness only, and are by no means a substitute for professional medical advice, diagnosis, or treatment. For any medical problem you have, consult a qualified physician immediately.

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