Iintlungu zomqolo ezihlala zihleli, ukuqina... Ingaba le yi-Ankylosing Spondylitis (AS)?

Ngaba nawe ubusoloko uziva iintlungu ezingapheliyo emqolo nasesinqeni sakho ukususela usemncinci? Ingakumbi xa uvuka kusasa, ngaba umqolo wakho uziva uqinile kwaye uziva kunzima ukugoba okanye ukwenza nawuphi na umsebenzi okwethutyana? Sihlala silibala ngezinto ezinjalo, sicinga ukuba "kusenokuba kukukruneka" okanye "kusenokuba kungenxa yokudinwa ngumsebenzi." Nangona kunjalo, le isenokuba yimeko efuna ingqalelo engakumbi. Namhlanje sithetha ngesifo esifana nesi, kwaye abantu abaninzi abasazi kakuhle, kodwa kubaluleke kakhulu ukuba siqaphele. Yi- Ankylosing Spondylitis leyo.

Yintoni i-Ankylosing Spondylitis (AS) ngokulula?

Ngamafutshane, luhlobo lwesifo samathambo esichaphazela amalungu omqolo. Sichaphazela kakhulu umqolo wakho kunye namalungu e-sacroiliac, apho inxalenye esezantsi yomqolo wakho idibana namathambo akho esinqeni. Esi sifo sibangela ukuba amasosha omzimba wakho ahlasele ngempazamo la malungu, nto leyo ebangela ukudumba okungapheliyo .

Njengoko oku kuqandusela kuqhubeka ixesha elide, kunokubangela iintlungu zomqolo nethanga, kunciphise ukuguquguquka komqolo, kwaye kuthintele intshukumo . Abanye abantu banokuba nentamo eqinileyo ekuhambeni kwexesha.

Kuthiwa malunga nomntu omnye kwabayi-1000 kwihlabathi liphela unokuba nale meko. Okwangoku akukho nyango lwale meko. Kodwa ungakhathazeki , ukuba isifo sichongiwe ngokuchanekileyo kwaye sinyangwa phantsi kwengcebiso kagqirha ofanelekileyo, ungasilawula kakuhle esi sifo kwaye uphile ubomi obuqhelekileyo.

Ngubani osengozini enkulu yokuhlaselwa sesi sifo?

Kutheni abanye abantu befumana esi sifo? Nangona unobangela ochanekileyo ungekafunyanwa, izazinzulu zichonge izinto ezininzi ezonyusa umngcipheko.

Impembelelo yemfuza (iiMfuza)

Kufunyaniswe ukuba iimpembelelo zemfuza zezona zibangela esi sifo.

• I-HLA-B27 gene: Le yeyona gene inxulunyaniswa kakhulu nesi sifo. Le gene inceda inkqubo yomzimba yethu yokuzikhusela ukuba iqaphele ukuba 'ezi ziiseli zethu' kunye 'nentsholongwane okanye iibhaktheriya zasemzini'. Abantu abanohlobo oluthile lwale gene (uhlobo lwe-'HLA-B27`) basengozini enkulu yokuhlaselwa yi-AS.
• Eyona nto ibalulekileyo: Kodwa eyona nto ibalulekileyo apha kukuba ngenxa yokuba une-'HLA-B27' gene emzimbeni wakho, awunakutsho ukuba uza kuba nesi sifo. Uninzi lwabantu abanale gene abasoze babe nesi sifo. Kwakhona, abantu abangenayo le gene nabo banokuba nesi sifo.
• Ezinye ii-genes: Ukongeza, ezinye ii-genes ezininzi ezinxulumene nenkqubo yomzimba yokuzikhusela, ezifana ne-'ERAP1`, `IL1A`, kunye ne-`IL23R`, zifunyenwe zidibene nesi sifo.

Ezinye iimeko zokudumba

Abanye abantu abane- AS banokuba nezinye iimeko zokudumba. Umzekelo:

• I-Psoriasis
• I-Ulcerative Colitis
• Isifo sikaCrohn

Ngoko ke, ukuba umntu kusapho lwakho unezi meko, okanye i-Ankylosing Spondylitis, nawe usenokuba sengozini.

Indlela isini kunye nobudala obuchaphazela ngayo

Iimpawu ze-AS zihlala ziqala ngaphambi kokuba zibe neminyaka engama-40 ubudala. Ngamanye amaxesha, iimpawu zinokubonakala kwangethuba zineminyaka eli-16 ubudala, nto leyo ebizwa ngokuba yi-'juvenile-onset ankylosing spondylitis'.

Indlela esiziphatha ngayo esi sifo yahlukile ngokuxhomekeke kwisini. Ukwazi la mahluko kuya kunceda kakhulu ekuxilongweni kwesi sifo kwangethuba.

Esinye sezizathu zokuba abafazi badla ngokulibaziseka ekufumaneni uxilongo kukungazi kakuhle phakathi koluntu kwaye mhlawumbi noogqirha ukuba esi 'sisifo samadoda'. Kwakhona, ngenxa yokuba iimpawu zabasetyhini zahlukile kwezamadoda (kwanezikrini zeMRI zinokubonisa umonakalo omncinci womqolo), kungathatha ixesha ukufumanisa esi sifo.

Ngaba uhlanga kunye nobuhlanga nazo zidlala indima?

Ewe, uhlanga ludlala indima ngandlela ithile. Izifundo zibonise ukuba esi sifo sixhaphake kakhulu kubantu abamhlophe, ingakumbi kumazwe asemantla Yurophu. Asixhaphakanga kangako kubantu abamnyama baseAfrika.

Kodwa kukho ingongoma ebalulekileyo apha. Nangona umngcipheko wokufumana esi sifo uphantsi, izifundo zibonisa ukuba ukuba umntu omnyama ufumana i-AS, ubukhali besifo bunokwanda .

Kwakhona, phantse ama-90% abantu abamhlophe abane-AS bane-gene ye-HLA-B27. Nangona kunjalo, yi-50%-60% kuphela yabantu abamnyama abane-AS abanale gene. Ke ngoko, kubalulekile ukuqwalasela uhlanga kunye nemvelaphi ye-genetic xa uxilonga.

Ukulibaziseka kokuxilongwa kunye nempembelelo yako

Kwabaninzi, kuthatha malunga neminyaka esi-8 ukuya kweli-10 ukuba esi sifo sichongwe ngokufanelekileyo emva kokuba iimpawu ziqalile. Oku kulibaziseka kunokuba ixesha elide ngakumbi kubafazi, ngakumbi.

Oku kulibaziseka kubangela ukuba unyango lulibaziseke. Oku kwandisa umonakalo onokwenziwa sisifo. Ekuhambeni kwexesha, amathambo emgogodleni anokudibana (ukudibana), kwaye intshukumo yomqolo inokulahleka ngokupheleleyo.

Ngoko ke, ukuba uneentlungu ezingapheliyo emqolo, ukuqina kwasekuseni, okanye ezinye iimpawu, ingakumbi ukuba ungumfazi , nceda ungakutyesheli oko. Bona ugqirha ofanelekileyo kwaye uthethe ngeempawu zakho ngokucacileyo. Ungoyiki ukucela i-X-ray okanye i-MRI scan ukuba kuyimfuneko. Unelungelo lokuthetha ngokwakho.